1 pin-sorting determinant in vivo that directs pulsatile secretion.

2 ypoglycemia and that paracrine factors shape pulsatile secretion in hyperglycemia.

3 s inhibitory effect on the GnRH/LH surge and pulsatile secretion is mediated by its receptor in the k

4 e for kisspeptin in GnRH neuron firing, GnRH pulsatile secretion, negative feedback by gonadal steroi

5 itive burst discharges that may underlie the pulsatile secretion of GnRH, and glutamatergic inputs ma

6 pothalamus, followed by proper synthesis and pulsatile secretion of GnRH, functions absent in patient

7 us followed by the appropriate synthesis and pulsatile secretion of GnRH.

8                                          The pulsatile secretion of gonadotropin-releasing hormone (G

9  puberty is first detected as an increase in pulsatile secretion of gonadotropin-releasing hormone (G

10                                              Pulsatile secretion of gonadotropin-releasing hormone-1

11 rons in the Arc to synchronize and shape the pulsatile secretion of kisspeptin and drive the release

12 tion, which is represented by the downstream pulsatile secretion of luteinizing hormone.

13 ons in vivo, burst firing is associated with pulsatile secretion of OT in the milk ejection reflex, a

14 ency per day, total basal (constitutive) and pulsatile secretion per day, and half-lives of eliminati

15 esis that progesterone inhibits LH surge and pulsatile secretion via its receptor in the ARC and/or A