ライフサイエンスコーパス: pulsatility

1 osine-coronary flow response curve at higher pulsatility.

2 ith archaerhodopsin for 30 min suppressed LH pulsatility.

3 rmful effects of excessive pressure and flow pulsatility.

4 halorhodopsin were found to reset ongoing LH pulsatility.

5 hite matter integrity or brain microvascular pulsatility.

6 reproduces femoral artery flow waveforms and pulsatility.

7 increased +18+/-2% (P:<0.0001) with enhanced pulsatility.

8 9 versus 0.48 mm Hg/mL; P<0.001), greater LA pulsatility (19 versus 13 mm Hg; P<0.001), and higher wa

9 n 3% and 7%), leukocyte speed 14% lower, and pulsatility 25% higher, but none of these differences wa

10 locities, velocity time integral, resistive, pulsatility, acceleration indices (RI, PI, AI), and flow

11 ers as activated by static pressure, whereas pulsatility activated extracellular Na(+)-dependent Cl(-

12 e performance of the widely employed hormone pulsatility analysis program Cluster.

13 ugh residual myocardial function to maintain pulsatility and aortic root ejection and to maintain, wi

14 Restoring pulsatility and decreasing left ventricular afterload wi

15 Increased aortic pressure wave pulsatility and greater decrease in pulsatility on treat

16 ith ghrelin's property as an amplifier of GH pulsatility and its speculated role in establishing an i

17 ic and mean blood flow velocities as well as pulsatility and resistance (RI) indices were measured.

18 were impaired in CHD women (umbilical artery pulsatility and resistance index at 32 weeks in CHD vers

19 Pulsatility and resistance indexes and cerebral oxygen e

20 were found in cerebral blood velocities and pulsatility and resistance indexes in the overall popula

21 ues that are caused by cardiac motion, blood pulsatility, and any muscle activity.

22 The magnitude, pulsatility, and directional characteristics of FSS are

23 rmal aortic stiffness and increased pressure pulsatility are associated with blunted microvascular re

24 ile (ultradian), the genomic effects of this pulsatility are poorly understood.

25 Vascular stiffness and blood pressure pulsatility are related; however, temporal relationships

26 sed to regulate real-time physiological flow pulsatility at constant mean pressure.

27 Luteinizing hormone (LH) pulsatility, body weight, ovarian variables, and hormone

28 t thrombus growth rate is enhanced by modest pulsatility but less so when pulsations are amplified in

29 herapy displayed higher aortic pressure wave pulsatility (central pulse pressure [PP], reflected pres

30 is a highly accurate method to detect brain pulsatility changes related to cerebrovascular functioni

31 ease in lean and obese subjects with similar pulsatility characteristics.

32 s and mechanisms of pH(i) modulation by flow pulsatility, comparing pressurized steady versus pulse-f

33 Because high pulsatility could represent prodromal cerebrovascular ch

34 ncreased aortic stiffness and excessive flow pulsatility damage the microcirculation, leading to quan

35 measures of vascular stiffness and pressure pulsatility derived from arterial tonometry (carotid-fem

36 ication of mean flow by adenosine and higher pulsatility, despite being administered at doses matchin

37 Increasing pulsatility elevated mean coronary flow +11.5 +/- 1.7 %

38 Pulsatility emerges at intermediate dissociation rates,

39 trategies to provide some degree of arterial pulsatility, even in continuous flow LVADs may be necess

40 f MyoII dynamics that underlie stability and pulsatility: exchange kinetics governed by phosphorylati

41 Reduced pulsatility has been implicated as a contributing cause.

42 We used tissue pulsatility imaging (TPI), a novel ultrasound (US) neuro

43 In a separate pilot study we compared leptin pulsatility in 414 plasma samples collected every 7 minu

44 Reduced pulsatility in patients supported with the continuous-fl

45 dysfunction, related in some part to lack of pulsatility in the pulmonary circulation because of alte

46 investigate the mechanistic role of vascular pulsatility in this context.

47 y serve as a regulator of endogenous insulin pulsatility in vivo.

48 Pulmonary artery pulsatility increased in two of three locations (main +2

49 heart rate STV (CTG STV), early DV changes (pulsatility index >95th percentile; DV p95), or late DV

50 een aortic stiffness and GFR was mediated by pulsatility index (95% confidence interval of indirect e

51 offered as mediators downstream from higher pulsatility index (95% confidence interval of indirect e

52 was evaluated by means of the LVAD parameter pulsatility index (PI) and by the echocardiographic asse

53 artery resistive index (RI), hepatic artery pulsatility index (PI) and hepatic artery flow volume.

54 ad DVT and compared them with respect to the pulsatility index (PI) value of the popliteal artery.

55 Pulsatility index (PI) values of DA, RPA, LPA, and DPA w

56 Use of TTF probes to assess graft flow and pulsatility index (PI) values.

57 CD) on the middle cerebral artery (MCA): MCA pulsatility index (PIa) and an estimator based on diasto

58 lsatility was quantified by using the venous pulsatility index (VPI), the venous equivalent of resist

59 centile] and a high umbilical artery Doppler pulsatility index [>95th percentile]).

60 Carotid pulse pressure, pulsatility index and carotid-femoral pulse wave velocit

61 s and/or kidneys in 12 healthy subjects, the pulsatility index and resistive index were estimated fro

62 Pulsatility index and resistive index were mapped with c

63 Umbilical artery pulsatility index and uterine artery resistance index an

64 At 23 to 25 weeks, uterine artery pulsatility index correlated with serum soluble endoglin

65 k velocity in the LCCA (p = 0.04) and higher pulsatility index of both the RCCA and LCCA (p = 0.006 a

66 In contrast, the pulsatility index of the middle cerebral artery was a po

67 We used a higher umbilical/cerebral artery pulsatility index ratio as an indicator of preferential

68 /s) versus 29 cm/s (23-35 cm/s) (p = 0.004), pulsatility index was 1.10 (0.97-1.18) versus 0.94 (0.89

69 Pulsatility index was associated with lower memory score

70 Pulsatility index was associated with lower whole brain

71 Carotid flow pulsatility index was negatively related to the aorta-ca

72 The pulsatility index within the dorsalis pedis artery decre

73 flow velocity in the middle cerebral artery, pulsatility index, and jugular bulb oxygenation between

74 or left ventricular end-diastolic dimension, pulsatility index, and power were calculated.

75 ernal mean arterial pressure, uterine artery pulsatility index, brachial artery flow-mediated dilatat

76 potential mediators, including renal artery pulsatility index, renal vascular resistance, and arteri

77 feature, including intranodular vascularity, pulsatility index, resistive index, or peak-systolic vel

78 artery resistance index and umbilical artery pulsatility index, respectively.

79 After accounting for pulsatility index, the relation between aortic stiffness

80 he intensity of trophic skin changes and the pulsatility index.

81 es (mean, total, mean/RR interval; Gosling's pulsatility index; and cerebrovascular resistance [mean

82 Resistive and pulsatility indexes and peak systolic velocity were docu

83 id and aorta, transmission of excessive flow pulsatility into the brain, microvascular structural bra

84 Transmission of excessive flow pulsatility into the low-impedance renal microvasculatur

85 to lower GFR by transferring excessive flow pulsatility into the susceptible renal microvasculature,

86 dinated adrenocorticotropic hormone-cortisol pulsatility is maintained.

87 Notably, pulsatility is not an outcome of an upstream Rho1 pacema

88 l for proper liver utilization and perturbed pulsatility is observed in type 2 diabetes.

89 onary flow elevation from enhanced perfusion pulsatility is synergistically amplified by adenosine.

90 e compared the maximum and mean brain tissue pulsatility (MaxBTP and MeanBTP), as identified by TPI,

91 creased BTP related to the mean global brain pulsatility (MeanBTP) and no change related to large ves

92 Ultradian pulsatility of both adrenocorticotropic hormone and cort

93 Coordinated pulsatility of individual islets enhances the first-phas

94 A 27% reduction in the vessel wall pulsatility of intracortical arterioles and widespread l

95 ed good sensitivity to detect changes in the pulsatility of small brain volumes, to identify early an

96 effects of continuous blood flow and reduced pulsatility on major organ function have not been studie

97 rate, pH, hypoxia, venous pressure, and flow pulsatility on NOe were determined.

98 o assess the effects of different degrees of pulsatility on the incidence of nonsurgical bleeding.

99 ure wave pulsatility and greater decrease in pulsatility on treatment are associated with functional

100 evidence of detrimental effects from reduced pulsatility over a 6-month time period.

101 Pulsatility over the cardiac cycle was evaluated by send

102 Further analysis demonstrated that the pulsatility parameters recovered by AutoDecon were indis

103 pulse height; all concentration-independent pulsatility parameters were almost identical in the two

104 ht, as leptin levels rose to their peak, the pulsatility profiles of LH changed significantly and bec

105 Aortic pressure pulsatility significantly decreased in subjects with fun

106 Thus, adenosine and perfusion pulsatility specifically interact to enhance coronary fl

107 (+)(Ca) activation and NO comodulate in vivo pulsatility-stimulated coronary flow, supporting an impo

108 Leptin pulsatility therefore can be preserved in the obese.

109 duced changes in thyroid-stimulating hormone pulsatility, thyroid and IGF-1 hormone levels, hypothala

110 , modulating the amount of pressure and flow pulsatility transmitted into the cerebral microcirculati

111 e increased flow demand and higher perfusion pulsatility typically co-exist.

112 Average blood flow pulsatility was 6% higher in subjects with AMD (0.71 +/-

113 Pulsatility was evaluated by means of the LVAD parameter

114 of injection was unaffected, and subsequent pulsatility was inhibited.

115 Pulsatility was introduced using a programmable pneumati

116 Flow augmentation with pulsatility was not linked to higher regional oxygen con

117 Cardiac pulsatility was quantified by using the venous pulsatili

118 The cardiac pulsatility was recorded with a cardiac signal monitor.

119 nstant mean pressure, and physiological flow pulsatility was set at 40 or 100 mm Hg by computer servo

120 Pulsatility was varied between 40 and 100 mmHg.

121 ffects of 5 microM muscimol to increase GnRH pulsatility were blocked by co-exposure to the GABAA rec

122 nsibilitvy, women develop a higher degree of pulsatility with aging, as compared with men.

123 t can potentially benefit from lowering GnRH pulsatility with consequent diminished levels of plasma

124 oduced rapid inhibition of ACTH and cortisol pulsatility within 30 min in the morning and afternoon.