ライフサイエンスコーパス: pulse wave

1 gestures, acoustic vibrations, and real-time pulse wave.

2 the push and pull regions associated with a pulse wave.

3 ant to the formation of solitons in arterial pulse waves.

4 Hence, we compared finger pulse-wave amplitude (PWA) responses to exercise among 5

5 vascular responses to inhaled salbutamol by pulse wave analysis (PWA) or pulse contour analysis (PCA

6 s), arterial stiffness [pulse wave velocity, pulse wave analysis (PWA)], 24-h ambulatory blood pressu

7 sing radial artery applanation tonometry for pulse wave analysis and modeled in a mixed effects regre

8 automated oscillometric sphygmomanometer and pulse wave analysis every 2 weeks on up to five occasion

9 re derived from brachial pressure and radial pulse wave analysis in 2,073 patients, and 7,146 measure

10 Pulse wave analysis was used to determine augmentation i

11 Pulse wave analysis was used to determine central arteri

12 Radial artery applanation tonometry and pulse wave analysis were used to derive central aortic p

13 Radial artery applanation tonometry and pulse wave analysis were used to derive central aortic p

14 lacement (PAVR) and developments in coronary pulse wave analysis, it is now possible to instantaneous

15 Central hemodynamics was measured by Pulse Wave Analysis, left ventricular mass was assessed

16 sive individuals (age 21-78 yr; 43 male) and pulse wave analysis, wave intensity analysis and wave se

17 Using coronary pulse wave analysis, we calculated the intracoronary dia

18 plethysmography, flow-mediated dilation, and pulse wave analysis.

19 Radial artery applanation tonometry and pulse-wave analysis were used to derive central aortic p

20 latory blood pressure monitoring, peripheral pulse-wave analysis, and carotid intima-media thickness.

21 and evaluated central arterial stiffness by pulse-wave analysis.

22 tive self-oscillating gel, in which chemical pulse waves and a stimulus-responsive medium play roles

23 ing) with Doppler assessment (continuous and pulse wave as well as color-flow mapping).

24 Pulsed-wave assessment of PVD flow included S-, D-, and

25 a novel form of mechanical stimulation, or a pulsed wave at the frequency of 1.5 MHz and the duty cyc

26 l artery pulse wave velocity, carotid artery pulse waves (by applanation tonometry) and the arrival t

27 We studied how the pulsed wave can further increase algal lipid production

28 lastic blood vessels provide capacitance and pulse-wave dampening, which are critically important in

29 Thirteen pulse wave Doppler and 14 tissue Doppler imaging measure

30 We present Z scores for normalized pulse wave Doppler and tissue Doppler imaging in pediatr

31 Z score equations for most left ventricular pulse wave Doppler and tissue Doppler imaging measuremen

32 ETHODS: Ultrasound B-mode, color Doppler and pulse wave Doppler imaging of foot arteries was conducte

33 itant jet-derived pulmonary artery pressure, pulse wave Doppler pulmonary venous flow pattern and two

34 In pediatric echocardiography, pulse wave Doppler, and tissue Doppler imaging velocitie

35 tying velocity and calculated shear rates by pulsed wave Doppler and two-dimensional echocardiography

36 rium and in the appendage by transesophageal pulsed wave Doppler echocardiography in 89 patients with

37 using native tissue harmonics or transmitral pulsed wave Doppler have quantitated PFO functional size

38 low velocities obtained with transesophageal pulsed wave Doppler imaging were recorded together with

39 The pulsed wave Doppler ratio of peak early transmitral flow

40 ry venous flow velocity (PVFV) recorded with pulsed wave Doppler technique is currently used in the n

41 Using transesophageal pulsed wave Doppler technique, four PVFV components are

42 Pulsed wave Doppler transmitral and pulmonary venous flo

43 ated with hypoxic-ischemic injury, power and pulsed wave Doppler US may enable identification of pret

44 rhythm using epicardial and transesophageal pulsed wave Doppler.

45 pared with thermodilution (TD), aortic valve pulsed-wave Doppler (PWAO), and left ventricular echocar

46 luded LV volumes and ejection fraction (EF), pulsed-wave Doppler (PWD)-derived transmitral filling in

47 Pulsed-wave Doppler determination of the pulmonary arter

48 ntricular systolic function using M mode and pulsed-wave Doppler echocardiography revealed decreases

49 tored by (1) TEE of the ascending aorta, (2) pulsed-wave Doppler of the right carotid artery, (3) bal

50 Pulsed-wave Doppler recording of mitral and superior ven

51 Pulsed-wave Doppler transesophageal echocardiography (TE

52 Standard pulsed-wave Doppler transmitral and pulmonary vein flow

53 ects on diastolic function by load-dependent pulsed-wave Doppler transmitral indices has been variabl

54 myocardial performance indexes quantified by pulsed-wave Doppler ultrasound at day 30, followed by no

55 Pulse wave imaging (PWI) was performed just before infus

56 of spectral analysis of intraocular pressure pulse wave in healthy eyes of a control group (CG), pati

57 h resin casting at electronic microscopy and pulse-wave measurements, respectively.

58 Resin casting and pulse-wave measurements, showed that hrECMs preserves th

59 en to output either continuous wave (CW), or pulsed wave modes (PW).

60 ive, high fidelity, continuous radial artery pulse wave monitoring, which may lead to the use of flex

61 The pulsed waves of ultrasound, generated by a carbon black/

62 Results In normal aortas, the pulse waves propagated at relatively constant velocities

63 was used to measure the local homogeneity of pulse wave propagation within the saccular wall, which i

64 urysms is associated with the homogeneity of pulse wave propagation within the saccular wall.

65 ogram tracings with continuous wave (CW) and pulsed wave (PW) Doppler tracings recorded on the same s

66 ls were degraded by continuous wave (CW) and pulsed wave (PW) ultrasound at 205 kHz using deionized w

67 Here we report the utility of pulsed-wave (PW) Doppler-measured instantaneous flow and

68 lectron microscope results also suggest that pulsed wave stimulation induces shear stress and thus in

69 tty acid composition remains unchanged after pulsed-wave stimulation.

70 Long-axis M-mode, pulsed-wave tissue Doppler echograms (lateral, septal, a

71 ipients have biventricular dysfunction using pulsed-wave tissue Doppler imaging early after HT with m

72 ate-diastolic (A') velocities obtained using pulsed-wave tissue Doppler imaging in 380 healthy childr

73 Pulsed-wave tissue Doppler imaging studies </=10 days po

74 after HT in children and young adults using pulsed-wave tissue Doppler imaging.

75 Aortic pulse wave transit time from the root of the subclavian

76 Arterial elastic modulus and pulse wave transit time were assessed using ultrahigh fr

77 crease in Ep (+155 +/- 193% vs. -5 +/- 28%), pulse wave velocity (+20 +/- 30% vs. -7 +/- 24%), and Ea

78 Carotid-femoral pulse wave velocity (-0.095 +/- 0.043 SD/SD, P = 0.028)

79 ss (-14 +/- 13 g vs. +3 +/- 11 g, p < 0.01), pulse wave velocity (-0.8 +/- 1.0 m/s vs. -0.1 +/- 0.9 m

80 Measurements of aortic-femoral pulse wave velocity (afPWV; n = 446) and large- and smal

81 Aortic pulse wave velocity (Ao-PWV) and albumin creatinine rati

82 Increased aortic pulse wave velocity (aPWV) has been associated with mort

83 f this study was to determine whether aortic pulse wave velocity (aPWV) improves prediction of cardio

84 he basis of having either low or high aortic pulse wave velocity (aPWV), a robust measure of aortic s

85 ns, central augmentation index (AIx), aortic pulse wave velocity (aPWV), blood pressure and heart rat

86 Brachial-ankle pulse wave velocity (baPWV) was measured to determine ar

87 ve hyperemia index (beta = 0.23, p < 0.001), pulse wave velocity (beta = -0.09, p = 0.04), augmentati

88 g flow-mediated vasodilation (FMD), brachial pulse wave velocity (bPWV), circulating angiogenic cells

89 io measure, and a measure of carotid-femoral pulse wave velocity (cf-PWV) and augmentation index (AI)

90 Carotid-femoral pulse wave velocity (CF-PWV; the gold standard index of

91 mean arterial pressure, and carotid-femoral pulse wave velocity (CFPWV) in 1480 participants represe

92 Carotid-femoral pulse wave velocity (CFPWV) is a heritable measure of ao

93 teries (by ultrasonography), carotid-femoral pulse wave velocity (cfPWV), aortic augmentation index,

94 ss: brachial pulse pressure; carotid-femoral pulse wave velocity (CFPWV), which is related directly t

95 ently measured compared with carotid-femoral pulse wave velocity (cfPWV).

96 ic stiffness as estimated by carotid-femoral pulse wave velocity (cfPWV).

97 ar risk factors, both higher carotid-femoral pulse wave velocity (hazard ratio [HR], 1.32; 95% confid

98 A small improvement in the aortic pulse wave velocity (i.e., a decrease of 0.22 m/s; 95% C

99 Pulse wave velocity (index of arterial stiffness) was al

100 peptide were associated with carotid-femoral pulse wave velocity (men: partial correlation, 0.069, P

101 d r = -0.062, P = 0.040), and carotid-radial pulse wave velocity (men: r = -0.090, P = 0.009 and r =

102 ng aortic distensibility and positively with pulse wave velocity (P<0.05).

103 ratio of MPA to aortic size correlated with pulse wave velocity (P=0.0098), strain (P=0.0099), and d

104 Carotid-femoral pulse wave velocity (P=0.02), central pulse pressure (P<

105 Indexed MPA diameter correlated with pulse wave velocity (P=0.04) and with aortic strain (P=0

106 ior diameter (increase of 54.9% +/- 2.5) and pulse wave velocity (PWV) (decrease of 1.3 m/sec +/- 0.8

107 ardiovascular magnetic resonance measures of pulse wave velocity (PWV) and aortic distensibility (AoD

108 of arterial stiffness indices [i.e., aortic pulse wave velocity (PWV) and augmentation (AGI) of caro

109 Aortic blood pressure (BP), pulse wave velocity (PWV) and augmentation index (AIx) w

110 rced vital capacity [FVC]) and a decrease in pulse wave velocity (PWV) and augmentation index up to 2

111 We measured aortic pulse wave velocity (PWV) and brachial PWV to evaluate t

112 Aortic pulse wave velocity (PWV) and carotid augmentation index

113 ein, and arterial stiffness [carotid-femoral pulse wave velocity (PWV) and carotid augmentation index

114 and arterial compliance as assessed by using pulse wave velocity (PWV) and central augmentation index

115 outcomes were changes in carotid to femoral pulse wave velocity (PWV) and plasma 8-isoprostane F2alp

116 Pulse wave velocity (PWV) and the augmentation index (AI

117 ar stiffness was measured by carotid-femoral pulse wave velocity (PWV) and total arterial compliance.

118 We tested this by examining pulse wave velocity (PWV) in brachial arteries of twin s

119 Previous studies have suggested that AIx and pulse wave velocity (PWV) increase linearly with age, ye

120 assessed by magnetic resonance imaging (MRI) pulse wave velocity (PWV) measurements.

121 Higher pulse wave velocity (PWV) reflects increased arterial st

122 Pulse wave velocity (PWV) was calculated by the foot-to-

123 Pulse wave velocity (PWV) was calculated using the foot-

124 Pulmonary pulse wave velocity (PWV) was determined by the interval

125 Pulse wave velocity (PWV) was measured in the central (c

126 Pulse wave velocity (PWV) was measured invasively (aorti

127 ain, incremental elastic modulus (Einc), and pulse wave velocity (PWV) were measured over a TP range

128 at there is a progressive increase in aortic pulse wave velocity (PWV) with age.

129 ere 1) arterial stiffness measured by aortic pulse wave velocity (PWV), 2) oxidative stress assessed

130 This study sought to evaluate whether pulse wave velocity (PWV), a noninvasive index of arteri

131 mediated vasodilation (FMD), carotid-femoral pulse wave velocity (PWV), and aortic augmentation index

132 arterial pressure (MAP), augmentation index, pulse wave velocity (PWV), and intima-media thickness.

133 assess hepatic triglyceride content, aortic pulse wave velocity (PWV), and visceral fat.

134 ness of the common carotid artery (CCA-IMT), pulse wave velocity (PWV), augmentation index, blood pre

135 brachial artery blood pressure (BP), aortic pulse wave velocity (PWV), B-mode ultrasonography and wa

136 Disease activity, blood pressure, aortic pulse wave velocity (PWV), brachial artery flow-mediated

137 ently, vascular stiffness was assessed using pulse wave velocity (PWV).

138 ng 2007 to 2012, we measured carotid-femoral pulse wave velocity (PWV; SphygmoCor apparatus) 8 weeks

139 Carotid-to-femoral pulse wave velocity (PWVc-f) was assessed at baseline, a

140 cysteine was associated with carotid-femoral pulse wave velocity (r = 0.072, P = 0.036), forward pres

141 ectively measured arterial stiffness (aortic pulse wave velocity [aPWV]) and cardiac biomarkers in 98

142 ved from arterial tonometry (carotid-femoral pulse wave velocity [CFPWV], forward wave amplitude [FWA

143 Measures of arterial stiffness (pulse wave velocity [PWV] and augmentation index correct

144 ple (n = 42), cPP, arterial stiffness (using pulse wave velocity [PWV]) and arterial diameters (using

145 heir relation to central arterial stiffness (pulse wave velocity [PWV]) and arterial diameters, and t

146 Arterial stiffness (carotid to femoral pulse wave velocity [PWV]) was measured and peripheral b

147 nction (local aortic distensibility and arch pulse wave velocity [PWV]), and LV volumes and mass.

148 r stroke) in relation to arterial stiffness (pulse wave velocity [PWV]), wave reflection (augmentatio

149 ing with iontophoresis), arterial stiffness (pulse wave velocity and analysis), blood pressure, and p

150 male rats characterized for abdominal aortic pulse wave velocity and aortic strain by high-resolution

151 Pulse wave velocity and augmentation index were improved

152 Measures of arterial stiffness (pulse wave velocity and augmentation index) and blood pr

153 aldosterone levels, and arterial stiffness (pulse wave velocity and augmentation index) in 20 adult

154 cardiography, (2) coronary flow reserve, (3) pulse wave velocity and augmentation index, (4) circulat

155 dary outcomes included decreases in arterial pulse wave velocity and carotid artery echodensity and i

156 glyceride content was associated with aortic pulse wave velocity and carotid IMT.

157 condary outcome measures included changes in pulse wave velocity and circulating biomarkers.

158 also led to significant favorable changes in pulse wave velocity and circulating IL-6 levels.

159 In contrast to controls pulse wave velocity and distensibility correlated with a

160 orta and the left ventricle (eg, aortic arch pulse wave velocity and distensibility) as well as the v

161 thelial dysfunction as determined in vivo by pulse wave velocity and ex vivo by atomic force microsco

162 2 aortic stiffness measures, carotid-femoral pulse wave velocity and forward pressure wave amplitude,

163 In vivo MRI revealed that baseline pulse wave velocity and morphology were similar in 6-wee

164 Carotid-femoral pulse wave velocity and radial tonometry-derived central

165 sex-specific genetic determinants for aortic pulse wave velocity and suggest distinct polygenic susce

166 ential relationships observed between aortic pulse wave velocity and telomere length in younger and o

167 tly modifies the relationship between aortic pulse wave velocity and telomere length.

168 r elasticity locally, specifically the local pulse wave velocity and the arterial wall thickness.

169 Local pulse wave velocity and the mean arterial wall thickness

170 t) rats exhibited significantly lower aortic pulse wave velocity and vascular media thickness compare

171 Carotid femoral pulse wave velocity associated with both urinary albumin

172 ApoE(-/-) and WT mice showed that increased pulse wave velocity coincided with the fragmentation of

173 Pulse wave velocity declined 8% with ALT-711 (P<0.05 at

174 artery wall echodensity and carotid-femoral pulse wave velocity demonstrated no significant changes.

175 onometry, blood pressure, and carotid-radial pulse wave velocity did not change.

176 = 0.03), and reduced (i.e. improved) aortic pulse wave velocity from 7.1 +/- 0.3 to 6.1 +/- 0.3 m s(

177 hildren with PAH had significantly increased pulse wave velocity in the ascending aorta (3.4 versus 2

178 aseline independently associated with aortic pulse wave velocity in the complete cohort and progressi

179 Pulse wave velocity is an independent predictor of the l

180 ders of magnitude higher), as illustrated by pulse wave velocity measurements, toward hypertension de

181 wave reflection, reflected wave timing, and pulse wave velocity noninvasively in 6417 (age range, 19

182 Mean pulse wave velocity remained stable with both everolimus

183 Carotid to femoral pulse wave velocity showed a significant reduction from

184 Treatment also reduced aortic pulse wave velocity significantly (from 9.09+/-1.77 to 8

185 he weight-loss group, but carotid-to-femoral pulse wave velocity tended to decrease by 0.5 m/s (P = 0

186 systolic blood pressure and carotid-femoral pulse wave velocity to the model, forward pressure wave

187 We newly report that the assessment of local pulse wave velocity via MRI provides early information a

188 was 0.13 (95% CI: 0, 0.26; P = 0.044) lower, pulse wave velocity was 0.29 m/s (95% CI: 0.07, 0.52 m/s

189 Carotid-femoral pulse wave velocity was associated with higher white mat

190 Aortic pulse wave velocity was high-normal (9.2 +/- 2.2 m/s), i

191 Pulse wave velocity was higher in adults after ASO (5.0+

192 Pulse wave velocity was measured at baseline in 449 norm

193 mice, whereas at the age of 18 weeks, local pulse wave velocity was significantly elevated in ApoE(-

194 oral pulse wave velocity, and carotid-radial pulse wave velocity were assessed by tonometry in 1962 p

195 arget-to-background ratios (TBRs) and aortic pulse wave velocity were assessed.

196 ssure, pulsatility index and carotid-femoral pulse wave velocity were each associated with increased

197 n the brachial artery, and carotid to radial pulse wave velocity were measured in all children.

198 interval, 2.4-20.7), augmentation index, and pulse wave velocity without changing peripheral blood pr

199 active hyperemia index, aortic hemodynamics, pulse wave velocity) were not differentially altered by

200 measures (distensibility, aortic strain, and pulse wave velocity) were similar across all groups.

201 thickness, peripheral arterial disease, and pulse wave velocity).

202 Mean (+/-SD) carotid-femoral pulse wave velocity, a measure of central aortic stiffne

203 Aortic calcium was reduced by 31%, and pulse wave velocity, an index of stiffness, was decrease

204 elastance (Ea), arterial compliance, aortic pulse wave velocity, and carotid Peterson modulus (Ep).

205 ve, reflected pressure wave, carotid-femoral pulse wave velocity, and carotid-radial pulse wave veloc

206 carotid ultrasound (intima-media thickness), pulse wave velocity, and Doppler examination of kidney g

207 , total arterial compliance, carotid-femoral pulse wave velocity, and drug tolerability were assessed

208 -femoral pulse wave velocity, carotid-radial pulse wave velocity, and venous occlusion plethysmograph

209 Aortic dimensions, distensibility, pulse wave velocity, aortic arch angle, left ventricular

210 aortic stiffness was evaluated by measuring pulse wave velocity, aortic strain, and distensibility.

211 e contour analysis, partial rebreathing, and pulse wave velocity, are far less in number and are prim

212 ce or stiffness, elastic modulus, impedance, pulse wave velocity, augmentation index, and pulse press

213 OH)D(3) was not associated with adult aortic pulse wave velocity, blood pressure, fasting glucose, HD

214 d carotid pressure and flow, carotid-femoral pulse wave velocity, brain magnetic resonance images and

215 ) present repeated measures of aorto-femoral pulse wave velocity, capacitive compliance (C1), and osc

216 diac cycle length, carotid to femoral artery pulse wave velocity, carotid artery pulse waves (by appl

217 elial cells associated with increased aortic pulse wave velocity, carotid intima-media thickness, and

218 rin-mediated dilation (NMD), carotid-femoral pulse wave velocity, carotid-radial pulse wave velocity,

219 RI with gadolinium injection, measurement of pulse wave velocity, extracellular water, 24-hour ambula

220 arterial stiffness were the carotid femoral pulse wave velocity, forward pressure wave amplitude, ce

221 iffness increased markedly with age, eg, for pulse wave velocity, from a few percent in both sexes ag

222 rial stiffness, measured via carotid-femoral pulse wave velocity, has a better predictive value than

223 thickness, echocardiography, measurement of pulse wave velocity, hepatic ultrasonography, retinal fu

224 nces between treatment in carotid-to-femoral pulse wave velocity, high-sensitivity C-reactive protein

225 rtic stiffening, assessed by carotid-femoral pulse wave velocity, is associated with CKD.

226 The values of age, serum phosphate, pulse wave velocity, left ventricular mass (LVM), and LV

227 Patients also underwent assessment of pulse wave velocity, measurement of circulating superoxi

228 ing with iontophoresis), arterial stiffness [pulse wave velocity, pulse wave analysis (PWA)], 24-h am

229 Pulse wave velocity, superoxide, and C-reactive protein

230 Aortic stiffness, measured by pulse wave velocity, was approximately 35% greater in El

231 Pulse wave velocity, wave travel times, and lumped press

232 The mean +/- SD carotid-femoral pulse wave velocity, which reflects central aortic stiff

233 ry juice consumption reduced carotid femoral pulse wave velocity-a clinically relevant measure of art

234 nce, pulse contour, partial rebreathing, and pulse wave velocity-based devices have not been studied

235 sessed arterial stiffness by carotid-femoral pulse wave velocity.

236 LV mass, systolic and diastolic function, or pulse wave velocity.

237 ter region of DDB2 gene with carotid-femoral pulse wave velocity.

238 e groups despite considerable differences in pulse wave velocity.

239 patients, arterial stiffness as measured by pulse wave velocity.

240 chromosome11, LOD 8.9) in females affecting pulse wave velocity.

241 e severity of arterial stiffness assessed by pulse wave velocity.

242 magnetic resonance) and arterial stiffness (pulse wave velocity/analysis, aortic distensibility) wer

243 8.1 +/- 3.3%), and lower arterial stiffness (pulse wave velocity: mean 6.99 +/- 1.0 m/s vs. 7.05 +/-

244 s, compliance, and distensibility; 2) aortic pulse wave velocity; 3) coronary calcification; and 4) b

245 erformance index (MPI) and aortic stiffness (pulse wave velocity; PWV) were evaluated before and afte

246 ry flow-mediated dilation (FMDBA) and aortic pulse-wave velocity (aPWV) after 4, 8, and 12 weeks.

247 line vascular stiffness, indexed by arterial pulse-wave velocity (Doppler) and augmentation index (ca

248 Arterial stiffness was determined by pulse-wave velocity (PWV) of the brachioradial and femor

249 rial distensibility measures, generally from pulse-wave velocity (PWV), are widely used with little k

250 ents (62%) were found to present supranormal pulse-wave velocity and 14 patients (38%) presented left

251 elasticity was evaluated by Doppler-derived pulse-wave velocity and left ventricular function by ech

252 surement of AS by applanation tonometry with pulse-wave velocity has been the gold-standard method an

253 terial distensibility, assessed by measuring pulse-wave velocity in vivo.

254 Aortic pulse-wave velocity measured vascular stiffness.

255 there were significant associations between pulse-wave velocity values and left ventricular ejection

256 IMT was 0.71 +/- 0.1 mm, and the mean +/- SD pulse-wave velocity was 5.96 +/- 1.6 meters/second.

257 Pulse-wave velocity was assessed from tonometry and body

258 Pulse-wave velocity was higher in hypertensives (P=0.001

259 Carotid-femoral pulse-wave velocity was significantly (P<0.001) faster a

260 Doppler probes were used to collect pulse-wave velocity waveforms from the right carotid and

261 Left ventricular ejection fraction and pulse-wave velocity were both associated with Hunt and H

262 Left ventricular ejection fraction and pulse-wave velocity were improved between acute aneurysm

263 urine ET-1/creatinine, whereas reduction in pulse-wave velocity, a measure of arterial stiffness, wa

264 ow-mediated dilation of the brachial artery, pulse-wave velocity, and carotid intima-media thickness)

265 ection fraction, B-type natriuretic peptide, pulse-wave velocity, and pulse-wave velocity/left ventri

266 ice a Western diet markedly increased aortic pulse-wave velocity, intima-media thickening, oxidized l

267 res (central pulse pressure, carotid-femoral pulse-wave velocity, mean arterial pressure, forward pre

268 increased carotid intima-media thickness and pulse-wave velocity.

269 Pulse-wave velocity/left ventricular ejection fraction r

270 atriuretic peptide, pulse-wave velocity, and pulse-wave velocity/left ventricular ejection fraction s

271 WCH, MH, sustained hypertension, and aortic pulsed wave velocity by magnetic resonance imaging; urin

272 dependently associated with increased aortic pulsed wave velocity, cystatin C, and urinary albumin-to

273 ntent up to the 6th harmonic of the pressure pulse wave was considered.

274 t of central aortic waveforms analyzed using pulse wave, wave separation, and arterial reservoir mode