ライフサイエンスコーパス: pulse wave velocity

1 thickness, peripheral arterial disease, and pulse wave velocity).

2 ter region of DDB2 gene with carotid-femoral pulse wave velocity.

3 e groups despite considerable differences in pulse wave velocity.

4 patients, arterial stiffness as measured by pulse wave velocity.

5 chromosome11, LOD 8.9) in females affecting pulse wave velocity.

6 e severity of arterial stiffness assessed by pulse wave velocity.

7 sessed arterial stiffness by carotid-femoral pulse wave velocity.

8 LV mass, systolic and diastolic function, or pulse wave velocity.

9 increased carotid intima-media thickness and pulse-wave velocity.

10 Carotid-femoral pulse wave velocity (-0.095 +/- 0.043 SD/SD, P = 0.028)

11 ss (-14 +/- 13 g vs. +3 +/- 11 g, p < 0.01), pulse wave velocity (-0.8 +/- 1.0 m/s vs. -0.1 +/- 0.9 m

12 crease in Ep (+155 +/- 193% vs. -5 +/- 28%), pulse wave velocity (+20 +/- 30% vs. -7 +/- 24%), and Ea

13 s, compliance, and distensibility; 2) aortic pulse wave velocity; 3) coronary calcification; and 4) b

14 Mean (+/-SD) carotid-femoral pulse wave velocity, a measure of central aortic stiffne

15 urine ET-1/creatinine, whereas reduction in pulse-wave velocity, a measure of arterial stiffness, wa

16 ry juice consumption reduced carotid femoral pulse wave velocity-a clinically relevant measure of art

17 Measurements of aortic-femoral pulse wave velocity (afPWV; n = 446) and large- and smal

18 Aortic calcium was reduced by 31%, and pulse wave velocity, an index of stiffness, was decrease

19 magnetic resonance) and arterial stiffness (pulse wave velocity/analysis, aortic distensibility) wer

20 ing with iontophoresis), arterial stiffness (pulse wave velocity and analysis), blood pressure, and p

21 male rats characterized for abdominal aortic pulse wave velocity and aortic strain by high-resolution

22 Pulse wave velocity and augmentation index were improved

23 Measures of arterial stiffness (pulse wave velocity and augmentation index) and blood pr

24 aldosterone levels, and arterial stiffness (pulse wave velocity and augmentation index) in 20 adult

25 cardiography, (2) coronary flow reserve, (3) pulse wave velocity and augmentation index, (4) circulat

26 dary outcomes included decreases in arterial pulse wave velocity and carotid artery echodensity and i

27 glyceride content was associated with aortic pulse wave velocity and carotid IMT.

28 condary outcome measures included changes in pulse wave velocity and circulating biomarkers.

29 also led to significant favorable changes in pulse wave velocity and circulating IL-6 levels.

30 In contrast to controls pulse wave velocity and distensibility correlated with a

31 orta and the left ventricle (eg, aortic arch pulse wave velocity and distensibility) as well as the v

32 thelial dysfunction as determined in vivo by pulse wave velocity and ex vivo by atomic force microsco

33 2 aortic stiffness measures, carotid-femoral pulse wave velocity and forward pressure wave amplitude,

34 In vivo MRI revealed that baseline pulse wave velocity and morphology were similar in 6-wee

35 Carotid-femoral pulse wave velocity and radial tonometry-derived central

36 sex-specific genetic determinants for aortic pulse wave velocity and suggest distinct polygenic susce

37 ential relationships observed between aortic pulse wave velocity and telomere length in younger and o

38 tly modifies the relationship between aortic pulse wave velocity and telomere length.

39 r elasticity locally, specifically the local pulse wave velocity and the arterial wall thickness.

40 Local pulse wave velocity and the mean arterial wall thickness

41 t) rats exhibited significantly lower aortic pulse wave velocity and vascular media thickness compare

42 ents (62%) were found to present supranormal pulse-wave velocity and 14 patients (38%) presented left

43 elasticity was evaluated by Doppler-derived pulse-wave velocity and left ventricular function by ech

44 elastance (Ea), arterial compliance, aortic pulse wave velocity, and carotid Peterson modulus (Ep).

45 ve, reflected pressure wave, carotid-femoral pulse wave velocity, and carotid-radial pulse wave veloc

46 carotid ultrasound (intima-media thickness), pulse wave velocity, and Doppler examination of kidney g

47 , total arterial compliance, carotid-femoral pulse wave velocity, and drug tolerability were assessed

48 -femoral pulse wave velocity, carotid-radial pulse wave velocity, and venous occlusion plethysmograph

49 ow-mediated dilation of the brachial artery, pulse-wave velocity, and carotid intima-media thickness)

50 ection fraction, B-type natriuretic peptide, pulse-wave velocity, and pulse-wave velocity/left ventri

51 Aortic pulse wave velocity (Ao-PWV) and albumin creatinine rati

52 Aortic dimensions, distensibility, pulse wave velocity, aortic arch angle, left ventricular

53 aortic stiffness was evaluated by measuring pulse wave velocity, aortic strain, and distensibility.

54 Increased aortic pulse wave velocity (aPWV) has been associated with mort

55 f this study was to determine whether aortic pulse wave velocity (aPWV) improves prediction of cardio

56 he basis of having either low or high aortic pulse wave velocity (aPWV), a robust measure of aortic s

57 ns, central augmentation index (AIx), aortic pulse wave velocity (aPWV), blood pressure and heart rat

58 ry flow-mediated dilation (FMDBA) and aortic pulse-wave velocity (aPWV) after 4, 8, and 12 weeks.

59 ectively measured arterial stiffness (aortic pulse wave velocity [aPWV]) and cardiac biomarkers in 98

60 e contour analysis, partial rebreathing, and pulse wave velocity, are far less in number and are prim

61 Carotid femoral pulse wave velocity associated with both urinary albumin

62 ce or stiffness, elastic modulus, impedance, pulse wave velocity, augmentation index, and pulse press

63 Brachial-ankle pulse wave velocity (baPWV) was measured to determine ar

64 nce, pulse contour, partial rebreathing, and pulse wave velocity-based devices have not been studied

65 ve hyperemia index (beta = 0.23, p < 0.001), pulse wave velocity (beta = -0.09, p = 0.04), augmentati

66 OH)D(3) was not associated with adult aortic pulse wave velocity, blood pressure, fasting glucose, HD

67 g flow-mediated vasodilation (FMD), brachial pulse wave velocity (bPWV), circulating angiogenic cells

68 d carotid pressure and flow, carotid-femoral pulse wave velocity, brain magnetic resonance images and

69 WCH, MH, sustained hypertension, and aortic pulsed wave velocity by magnetic resonance imaging; urin

70 ) present repeated measures of aorto-femoral pulse wave velocity, capacitive compliance (C1), and osc

71 diac cycle length, carotid to femoral artery pulse wave velocity, carotid artery pulse waves (by appl

72 elial cells associated with increased aortic pulse wave velocity, carotid intima-media thickness, and

73 rin-mediated dilation (NMD), carotid-femoral pulse wave velocity, carotid-radial pulse wave velocity,

74 io measure, and a measure of carotid-femoral pulse wave velocity (cf-PWV) and augmentation index (AI)

75 Carotid-femoral pulse wave velocity (CF-PWV; the gold standard index of

76 mean arterial pressure, and carotid-femoral pulse wave velocity (CFPWV) in 1480 participants represe

77 Carotid-femoral pulse wave velocity (CFPWV) is a heritable measure of ao

78 teries (by ultrasonography), carotid-femoral pulse wave velocity (cfPWV), aortic augmentation index,

79 ss: brachial pulse pressure; carotid-femoral pulse wave velocity (CFPWV), which is related directly t

80 ently measured compared with carotid-femoral pulse wave velocity (cfPWV).

81 ic stiffness as estimated by carotid-femoral pulse wave velocity (cfPWV).

82 ved from arterial tonometry (carotid-femoral pulse wave velocity [CFPWV], forward wave amplitude [FWA

83 ApoE(-/-) and WT mice showed that increased pulse wave velocity coincided with the fragmentation of

84 dependently associated with increased aortic pulsed wave velocity, cystatin C, and urinary albumin-to

85 Pulse wave velocity declined 8% with ALT-711 (P<0.05 at

86 artery wall echodensity and carotid-femoral pulse wave velocity demonstrated no significant changes.

87 onometry, blood pressure, and carotid-radial pulse wave velocity did not change.

88 line vascular stiffness, indexed by arterial pulse-wave velocity (Doppler) and augmentation index (ca

89 RI with gadolinium injection, measurement of pulse wave velocity, extracellular water, 24-hour ambula

90 arterial stiffness were the carotid femoral pulse wave velocity, forward pressure wave amplitude, ce

91 = 0.03), and reduced (i.e. improved) aortic pulse wave velocity from 7.1 +/- 0.3 to 6.1 +/- 0.3 m s(

92 iffness increased markedly with age, eg, for pulse wave velocity, from a few percent in both sexes ag

93 surement of AS by applanation tonometry with pulse-wave velocity has been the gold-standard method an

94 rial stiffness, measured via carotid-femoral pulse wave velocity, has a better predictive value than

95 ar risk factors, both higher carotid-femoral pulse wave velocity (hazard ratio [HR], 1.32; 95% confid

96 thickness, echocardiography, measurement of pulse wave velocity, hepatic ultrasonography, retinal fu

97 nces between treatment in carotid-to-femoral pulse wave velocity, high-sensitivity C-reactive protein

98 A small improvement in the aortic pulse wave velocity (i.e., a decrease of 0.22 m/s; 95% C

99 hildren with PAH had significantly increased pulse wave velocity in the ascending aorta (3.4 versus 2

100 aseline independently associated with aortic pulse wave velocity in the complete cohort and progressi

101 terial distensibility, assessed by measuring pulse-wave velocity in vivo.

102 Pulse wave velocity (index of arterial stiffness) was al

103 ice a Western diet markedly increased aortic pulse-wave velocity, intima-media thickening, oxidized l

104 Pulse wave velocity is an independent predictor of the l

105 rtic stiffening, assessed by carotid-femoral pulse wave velocity, is associated with CKD.

106 The values of age, serum phosphate, pulse wave velocity, left ventricular mass (LVM), and LV

107 Pulse-wave velocity/left ventricular ejection fraction r

108 atriuretic peptide, pulse-wave velocity, and pulse-wave velocity/left ventricular ejection fraction s

109 res (central pulse pressure, carotid-femoral pulse-wave velocity, mean arterial pressure, forward pre

110 8.1 +/- 3.3%), and lower arterial stiffness (pulse wave velocity: mean 6.99 +/- 1.0 m/s vs. 7.05 +/-

111 Aortic pulse-wave velocity measured vascular stiffness.

112 Patients also underwent assessment of pulse wave velocity, measurement of circulating superoxi

113 ders of magnitude higher), as illustrated by pulse wave velocity measurements, toward hypertension de

114 peptide were associated with carotid-femoral pulse wave velocity (men: partial correlation, 0.069, P

115 d r = -0.062, P = 0.040), and carotid-radial pulse wave velocity (men: r = -0.090, P = 0.009 and r =

116 wave reflection, reflected wave timing, and pulse wave velocity noninvasively in 6417 (age range, 19

117 ng aortic distensibility and positively with pulse wave velocity (P<0.05).

118 ratio of MPA to aortic size correlated with pulse wave velocity (P=0.0098), strain (P=0.0099), and d

119 Carotid-femoral pulse wave velocity (P=0.02), central pulse pressure (P<

120 Indexed MPA diameter correlated with pulse wave velocity (P=0.04) and with aortic strain (P=0

121 ing with iontophoresis), arterial stiffness [pulse wave velocity, pulse wave analysis (PWA)], 24-h am

122 ior diameter (increase of 54.9% +/- 2.5) and pulse wave velocity (PWV) (decrease of 1.3 m/sec +/- 0.8

123 ardiovascular magnetic resonance measures of pulse wave velocity (PWV) and aortic distensibility (AoD

124 of arterial stiffness indices [i.e., aortic pulse wave velocity (PWV) and augmentation (AGI) of caro

125 Aortic blood pressure (BP), pulse wave velocity (PWV) and augmentation index (AIx) w

126 rced vital capacity [FVC]) and a decrease in pulse wave velocity (PWV) and augmentation index up to 2

127 We measured aortic pulse wave velocity (PWV) and brachial PWV to evaluate t

128 Aortic pulse wave velocity (PWV) and carotid augmentation index

129 ein, and arterial stiffness [carotid-femoral pulse wave velocity (PWV) and carotid augmentation index

130 and arterial compliance as assessed by using pulse wave velocity (PWV) and central augmentation index

131 outcomes were changes in carotid to femoral pulse wave velocity (PWV) and plasma 8-isoprostane F2alp

132 Pulse wave velocity (PWV) and the augmentation index (AI

133 ar stiffness was measured by carotid-femoral pulse wave velocity (PWV) and total arterial compliance.

134 We tested this by examining pulse wave velocity (PWV) in brachial arteries of twin s

135 Previous studies have suggested that AIx and pulse wave velocity (PWV) increase linearly with age, ye

136 assessed by magnetic resonance imaging (MRI) pulse wave velocity (PWV) measurements.

137 Higher pulse wave velocity (PWV) reflects increased arterial st

138 Pulse wave velocity (PWV) was calculated by the foot-to-

139 Pulse wave velocity (PWV) was calculated using the foot-

140 Pulmonary pulse wave velocity (PWV) was determined by the interval

141 Pulse wave velocity (PWV) was measured in the central (c

142 Pulse wave velocity (PWV) was measured invasively (aorti

143 ain, incremental elastic modulus (Einc), and pulse wave velocity (PWV) were measured over a TP range

144 at there is a progressive increase in aortic pulse wave velocity (PWV) with age.

145 ere 1) arterial stiffness measured by aortic pulse wave velocity (PWV), 2) oxidative stress assessed

146 This study sought to evaluate whether pulse wave velocity (PWV), a noninvasive index of arteri

147 mediated vasodilation (FMD), carotid-femoral pulse wave velocity (PWV), and aortic augmentation index

148 arterial pressure (MAP), augmentation index, pulse wave velocity (PWV), and intima-media thickness.

149 assess hepatic triglyceride content, aortic pulse wave velocity (PWV), and visceral fat.

150 ness of the common carotid artery (CCA-IMT), pulse wave velocity (PWV), augmentation index, blood pre

151 brachial artery blood pressure (BP), aortic pulse wave velocity (PWV), B-mode ultrasonography and wa

152 Disease activity, blood pressure, aortic pulse wave velocity (PWV), brachial artery flow-mediated

153 ently, vascular stiffness was assessed using pulse wave velocity (PWV).

154 ng 2007 to 2012, we measured carotid-femoral pulse wave velocity (PWV; SphygmoCor apparatus) 8 weeks

155 Arterial stiffness was determined by pulse-wave velocity (PWV) of the brachioradial and femor

156 rial distensibility measures, generally from pulse-wave velocity (PWV), are widely used with little k

157 Measures of arterial stiffness (pulse wave velocity [PWV] and augmentation index correct

158 ple (n = 42), cPP, arterial stiffness (using pulse wave velocity [PWV]) and arterial diameters (using

159 heir relation to central arterial stiffness (pulse wave velocity [PWV]) and arterial diameters, and t

160 Arterial stiffness (carotid to femoral pulse wave velocity [PWV]) was measured and peripheral b

161 nction (local aortic distensibility and arch pulse wave velocity [PWV]), and LV volumes and mass.

162 r stroke) in relation to arterial stiffness (pulse wave velocity [PWV]), wave reflection (augmentatio

163 erformance index (MPI) and aortic stiffness (pulse wave velocity; PWV) were evaluated before and afte

164 Carotid-to-femoral pulse wave velocity (PWVc-f) was assessed at baseline, a

165 cysteine was associated with carotid-femoral pulse wave velocity (r = 0.072, P = 0.036), forward pres

166 Mean pulse wave velocity remained stable with both everolimus

167 Carotid to femoral pulse wave velocity showed a significant reduction from

168 Treatment also reduced aortic pulse wave velocity significantly (from 9.09+/-1.77 to 8

169 Pulse wave velocity, superoxide, and C-reactive protein

170 he weight-loss group, but carotid-to-femoral pulse wave velocity tended to decrease by 0.5 m/s (P = 0

171 systolic blood pressure and carotid-femoral pulse wave velocity to the model, forward pressure wave

172 there were significant associations between pulse-wave velocity values and left ventricular ejection

173 We newly report that the assessment of local pulse wave velocity via MRI provides early information a

174 was 0.13 (95% CI: 0, 0.26; P = 0.044) lower, pulse wave velocity was 0.29 m/s (95% CI: 0.07, 0.52 m/s

175 Carotid-femoral pulse wave velocity was associated with higher white mat

176 Aortic pulse wave velocity was high-normal (9.2 +/- 2.2 m/s), i

177 Pulse wave velocity was higher in adults after ASO (5.0+

178 Pulse wave velocity was measured at baseline in 449 norm

179 mice, whereas at the age of 18 weeks, local pulse wave velocity was significantly elevated in ApoE(-

180 IMT was 0.71 +/- 0.1 mm, and the mean +/- SD pulse-wave velocity was 5.96 +/- 1.6 meters/second.

181 Pulse-wave velocity was assessed from tonometry and body

182 Pulse-wave velocity was higher in hypertensives (P=0.001

183 Carotid-femoral pulse-wave velocity was significantly (P<0.001) faster a

184 Aortic stiffness, measured by pulse wave velocity, was approximately 35% greater in El

185 Pulse wave velocity, wave travel times, and lumped press

186 Doppler probes were used to collect pulse-wave velocity waveforms from the right carotid and

187 oral pulse wave velocity, and carotid-radial pulse wave velocity were assessed by tonometry in 1962 p

188 arget-to-background ratios (TBRs) and aortic pulse wave velocity were assessed.

189 ssure, pulsatility index and carotid-femoral pulse wave velocity were each associated with increased

190 n the brachial artery, and carotid to radial pulse wave velocity were measured in all children.

191 Left ventricular ejection fraction and pulse-wave velocity were both associated with Hunt and H

192 Left ventricular ejection fraction and pulse-wave velocity were improved between acute aneurysm

193 active hyperemia index, aortic hemodynamics, pulse wave velocity) were not differentially altered by

194 measures (distensibility, aortic strain, and pulse wave velocity) were similar across all groups.

195 The mean +/- SD carotid-femoral pulse wave velocity, which reflects central aortic stiff

196 interval, 2.4-20.7), augmentation index, and pulse wave velocity without changing peripheral blood pr