ライフサイエンスコーパス: pulseless

1 ital discharge than those who were initially pulseless.

2 rse event outcomes than those who were never pulseless.

3 42/47; p < 0.001) than those who were never pulseless.

4 e CPR compared with those who were initially pulseless.

5 rdia, 869 (31.0%, or 15.5% of cohort) became pulseless after a median of 3 minutes of CPR (interquart

6 ferences in survival compared with initially pulseless arrest are unknown.

7 her demonstrates that early in the asphyxial pulseless arrest process doing something (mouth-to-mouth

8 lmonary resuscitation, only first documented pulseless arrest rhythm remained significantly associate

9 ospital cardiac arrest, the first documented pulseless arrest rhythm was typically asystole or PEA in

10 rable neurologic outcomes than patients with pulseless arrests, although there were no differences in

11 oves outcome in the early stages of apparent pulseless asphyxial cardiac arrest.

12 tic external defibrillator shock or who were pulseless but received no resuscitation by emergency med

13 ssness, and 37.5% (1046 of 2793) for initial pulseless cardiac arrest (P for difference across groups

14 A total of 7433 patients with a pulseless cardiac arrest and 5751 patients with a nonpul

15 analyses were performed for patients with a pulseless cardiac arrest and patients with a nonpulseles

16 l for pediatric patients with an in-hospital pulseless cardiac arrest and pediatric patients with a n

17 However, their rate of progression to pulseless cardiac arrest despite CPR and the differences

18 of survival to hospital discharge following pulseless cardiac arrest was higher in children than adu

19 a with subsequent pulselessness, and initial pulseless cardiac arrest.

20 poor perfusion and 2793 (49.9%) for initial pulseless cardiac arrest.

21 prediction interval, 4900-11 200) cases were pulseless cardiac arrests and 11 600 (95% prediction int

22 % prediction interval, 4400-9900) cases were pulseless cardiac arrests and 8100 (95% prediction inter

23 bsolute increase in survival for in-hospital pulseless cardiac arrests and a 9% absolute increase in

24 However, survival from pulseless cardiac arrests appeared to have reached a pla

25 =18 years) and 880 children (<18 years) with pulseless cardiac arrests requiring chest compressions,

26 For pulseless cardiac arrests, survival was 19% (95% CI, 11%

27 radycardia and poor perfusion and those with pulseless cardiac arrests.

28 SCD was defined as a sudden pulseless condition from a cardiac cause in a previously

29 SCD was defined as a sudden pulseless condition of cardiac origin in a previously st

30 Children who became pulseless despite CPR for bradycardia had a 19% lower li

31 G alerts, specifically asystole (n = 5), and pulseless electric activity (n = 8).

32 r sudden cardiac arrest (SCA) manifesting as pulseless electric activity (PEA) and survival rates are

33 as prompt epinephrine treatment for asystole/pulseless electric activity was associated with greater

34 eas patients with IHCA caused by asystole or pulseless electric activity were stratified by prompt (<

35 ents with nonshockable rhythms (asystole and pulseless electric activity).

36 d with the initiating OHCA rhythm (asystole, pulseless electric activity, or VF/VT).

37 842 patients with an IHCA caused by asystole/pulseless electric activity, the rate of 1-year survival

38 jury, new strategies specifically focused on pulseless electric activity, which is the presenting rhy

39 were male, and 60% had an initial rhythm of pulseless electric activity.

40 Although our understanding of SCD because of pulseless electric activity/asystole is growing, the ove

41 d by ventricular tachycardia/fibrillation or pulseless electric activity/asystole.

42 (49.84%), ventricular fibrillation (32.0%), pulseless electrical activity (14.6%), and asystole (9.1

43 en groups, as was the incidence of postshock pulseless electrical activity (15/18 MTE, 18/20 BTE) and

44 ibrillation), ventricular fibrillation (40), pulseless electrical activity (20), and asystole (20), i

45 The most common initial rhythms were pulseless electrical activity (58.7%) and asystole (33.3

46 .37 [95% CI, 5.70-11.03] minutes; P < .001), pulseless electrical activity (8.22 [95% CI, 5.44-11.00]

47 initial rhythm was perfusing (p < .0001) or pulseless electrical activity (p = .0002), and not relat

48 scitation for the diagnosis and treatment of pulseless electrical activity (PEA) correctly stress the

49 ciated with better outcomes than asystole or pulseless electrical activity (PEA).

50 ock is most commonly followed by asystole or pulseless electrical activity (PEA).

51 been a significant rise in the prevalence of pulseless electrical activity (PEA).

52 developed hypotension, dyspnea, hypoxia, and pulseless electrical activity 10 days after resection of

53 ar fibrillation/ventricular tachycardia 24%, pulseless electrical activity 16%, asystole 48%, other n

54 e nonshockable (83.9%, [asystole = 36.4% and pulseless electrical activity = 47.6%]).

55 to resultant postdefibrillation rhythms (ie, pulseless electrical activity [PEA] or asystole).

56 Asystole and pulseless electrical activity account for a high proport

57 acing) and, after successful defibrillation, pulseless electrical activity and asystole (by high-dose

58 ng, anaphylaxis with a difficult airway, and pulseless electrical activity arrest.

59 eased survival after in-hospital asystole or pulseless electrical activity arrest.

60 A post hoc analysis of patients with pulseless electrical activity at any time during the car

61 after prolonged ventricular fibrillation and pulseless electrical activity cardiac arrest.

62 day after he first became ill, he suffered a pulseless electrical activity cardiorespiratory arrest f

63 nimals developed refractory postcountershock pulseless electrical activity compared with 0 of 16 trea

64 Patients in the subgroup presenting with pulseless electrical activity had intensive care unit ad

65 Asystole occurred in 23 (17 died), pulseless electrical activity in 2, and respiratory arre

66 itial cardiac arrest rhythm was asystole and pulseless electrical activity in 874 children (84.8%) an

67 scitation was used after cardiac arrest with pulseless electrical activity in a patient with recurren

68 ortion of cardiac arrests due to asystole or pulseless electrical activity increased over time (P<0.0

69 15 defibrillation 1st animals (p <.001), and pulseless electrical activity occurred in only one of 15

70 bserved in patients whose initial rhythm was pulseless electrical activity or asystole.

71 Women are also more likely to present in pulseless electrical activity or systole rather than ven

72 of >4.0 mmol/L (normal range 0.0 to 2.2); c) pulseless electrical activity or ventricular fibrillatio

73 , with an increase in cardiac arrests due to pulseless electrical activity over time (P for trend <0.

74 more asystole (8.8% vs. 7%) and (organized) pulseless electrical activity than men (24% vs. 18%; p <

75 2.2%) had nonshockable rhythms (asystole and pulseless electrical activity) and 2079 (17.8%) had shoc

76 nd separately for nonshockable (asystole and pulseless electrical activity) and shockable (ventricula

77 ients with nonshockable rhythms (asystole or pulseless electrical activity) is debated.

78 perienced nonshockable OHCA (ie, asystole or pulseless electrical activity) were assessed for eligibi

79 electrocardiographic presentations (asystole/pulseless electrical activity).

80 st often (81%) nonshockable (ie, asystole or pulseless electrical activity).

81 atients whose initial rhythm was asystole or pulseless electrical activity, AEDs were associated with

82 suscitation followed by countershock-induced pulseless electrical activity, after which animals were

83 , 79.3% had an initial rhythm of asystole or pulseless electrical activity, and 20.7% had ventricular

84 manifested ventricular fibrillation (VF), 45 pulseless electrical activity, and 55 asystole as the in

85 tubation, and time to asystole, hypotension, pulseless electrical activity, and declaration of death

86 eless ventricular tachycardia, asystole, and pulseless electrical activity, as well as peri-arrest co

87 en the initial cardiac rhythm is asystole or pulseless electrical activity, particularly in patients

88 ith an initial cardiac rhythm of asystole or pulseless electrical activity, pre-hospital cooling usin

89 In animals with pulseless electrical activity, sodium nitroprusside-enha

90 of ventricular tachycardia/fibrillation and pulseless electrical activity, while the comprehensive (

91 rse than those for patients with asystole or pulseless electrical activity.

92 m survival rates in patients presenting with pulseless electrical activity.

93 rolonged VF typically results in asystole or pulseless electrical activity.

94 ut was offset by more cases with asystole or pulseless electrical activity.

95 entricular fibrillation; and (4) infant with pulseless electrical activity.

96 ardia/ventricular fibrillation as opposed to pulseless electrical activity/asystole (epilepsy, 26%; n

97 al oxygen extraction in adult patients after pulseless electrical activity/asystole or resistant vent

98 tricular fibrillation; (2) adult female with pulseless electrical activity; (3) school-aged child wit

99 kable versus nonshockable initial rhythms in pulseless events.

100 ion model to estimate the incidence of index pulseless in-hospital cardiac arrest based on hospital-l

101 s, and nearly one-third of these progress to pulseless in-hospital cardiac arrest despite CPR.

102 of children who progress from bradycardia to pulseless in-hospital cardiac arrest despite CPR.

103 All patients were pulseless on EMS arrival.

104 f VF or pulseless VT as the first documented pulseless rhythm was 14% (120/880) in children and 23% (

105 have ventricular arrhythmias as their first pulseless rhythm, to be monitored and hospitalized in th

106 fibrillation or tachycardia was the initial pulseless rhythm; in 149 patients (15 percent), it devel

107 Compared to those with initial pulseless rhythms, these children were younger (0.4 vs 1

108 as a major contributor to the development of pulseless rhythms.

109 Unchecked disease progression leads to the "pulseless" stage, manifest clinically by missing pulses,

110 nitial rhythms of ventricular tachycardia or pulseless ventricular fibrillation (VT/VF).

111 [95% CI, 5.44-11.00] minutes; P < .001), and pulseless ventricular tachycardia (6.17 [95% CI, 0.09-12

112 Ventricular fibrillation (VF) or pulseless ventricular tachycardia (pVT) are the most tre

113 hockable rhythms of ventricular fibrillation/pulseless ventricular tachycardia (VF/pVT).

114 care unit, younger age, an initial rhythm of pulseless ventricular tachycardia (VT) or ventricular fi

115 ften due to ventricular fibrillation (VF) or pulseless ventricular tachycardia (VT), which are associ

116 for victims with ventricular fibrillation or pulseless ventricular tachycardia (VT/VF), compared with

117 shockable rhythms (ventricular fibrillation/pulseless ventricular tachycardia [VF/VT]) during resusc

118 ory OHCA (incessant ventricular fibrillation/pulseless ventricular tachycardia after >=3 direct-curre

119 shock-refractory ventricular fibrillation or pulseless ventricular tachycardia after at least one sho

120 e frequencies of ventricular fibrillation or pulseless ventricular tachycardia and of survival to hos

121 graphy rhythm of ventricular fibrillation or pulseless ventricular tachycardia and still unconscious

122 hockable rhythms of ventricular fibrillation/pulseless ventricular tachycardia are not well-character

123 The incidence of ventricular fibrillation or pulseless ventricular tachycardia as the first recorded

124 ac arrest due to ventricular fibrillation or pulseless ventricular tachycardia at 369 hospitals parti

125 l adult primary ventricular fibrillation and pulseless ventricular tachycardia cardiac arrests 36 mon

126 c arrests due to ventricular fibrillation or pulseless ventricular tachycardia enrolled between Janua

127 ren (84.8%) and ventricular fibrillation and pulseless ventricular tachycardia in 157 children (15.2%

128 atio for initial ventricular fibrillation or pulseless ventricular tachycardia in public versus at ho

129 sts with initial ventricular fibrillation or pulseless ventricular tachycardia is much greater in pub

130 f 204 patients with ventricular fibrillation/pulseless ventricular tachycardia OHCA (median age 62; m

131 Refractory ventricular fibrillation/pulseless ventricular tachycardia OHCA was associated wi

132 Patients with IHCA caused by pulseless ventricular tachycardia or ventricular fibrill

133 th shock-refractory ventricular fibrillation/pulseless ventricular tachycardia out-of-hospital cardia

134 nitial rhythm of ventricular fibrillation or pulseless ventricular tachycardia refractory to at least

135 des of recurrent ventricular fibrillation or pulseless ventricular tachycardia requiring additional e

136 the incidence of ventricular fibrillation or pulseless ventricular tachycardia was 25% when the arres

137 arrest rhythm of ventricular fibrillation or pulseless ventricular tachycardia was refractory to shoc

138 iac arrest with ventricular fibrillation (or pulseless ventricular tachycardia) and who had not been

139 and shockable (ventricular fibrillation and pulseless ventricular tachycardia) cardiac arrests.

140 ckable rhythms (ventricular fibrillation and pulseless ventricular tachycardia).

141 arrest, including ventricular fibrillation, pulseless ventricular tachycardia, asystole, and pulsele

142 shock-refractory ventricular fibrillation or pulseless ventricular tachycardia, but without proven su

143 arrest because of ventricular fibrillation, pulseless ventricular tachycardia, or unknown shockable

144 om shock-refractory ventricular fibrillation/pulseless ventricular tachycardia.

145 th shock-resistant ventricular fibrillation /pulseless ventricular tachycardia.

146 shock-refractory ventricular fibrillation or pulseless ventricular tachycardia.

147 y, and 20.7% had ventricular fibrillation or pulseless ventricular tachycardia.

148 ased likelihood of survival in patients with pulseless ventricular tachycardia/ventricular fibrillati

149 e who presented with bradycardia followed by pulseless ventricular tachycardia/ventricular fibrillati

150 in shock-resistant ventricular fibrillation/pulseless ventricular tachycardia; however, the efficacy

151 The prevalence of VF or pulseless VT as the first documented pulseless rhythm wa

152 ored status, time to defibrillation of VF or pulseless VT, intensive care unit location of arrest, an

153 t-of-hospital cardiac arrest caused by VF or pulseless VT, we found early coronary angiography was as

154 s despite fewer cardiac arrests due to VF or pulseless VT.