ライフサイエンスコーパス: pulseless

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1 her demonstrates that early in the asphyxial pulseless arrest process doing something (mouth-to-mouth

2 lmonary resuscitation, only first documented pulseless arrest rhythm remained significantly associate

3 ospital cardiac arrest, the first documented pulseless arrest rhythm was typically asystole or PEA in

4 oves outcome in the early stages of apparent pulseless asphyxial cardiac arrest.

5 tic external defibrillator shock or who were pulseless but received no resuscitation by emergency med

6 of survival to hospital discharge following pulseless cardiac arrest was higher in children than adu

7 =18 years) and 880 children (<18 years) with pulseless cardiac arrests requiring chest compressions,

8 SCD was defined as a sudden pulseless condition of cardiac origin in a previously st

9 as prompt epinephrine treatment for asystole/pulseless electric activity was associated with greater

10 eas patients with IHCA caused by asystole or pulseless electric activity were stratified by prompt (<

11 ents with nonshockable rhythms (asystole and pulseless electric activity).

12 d with the initiating OHCA rhythm (asystole, pulseless electric activity, or VF/VT).

13 842 patients with an IHCA caused by asystole/pulseless electric activity, the rate of 1-year survival

14 jury, new strategies specifically focused on pulseless electric activity, which is the presenting rhy

15 were male, and 60% had an initial rhythm of pulseless electric activity.

16 Although our understanding of SCD because of pulseless electric activity/asystole is growing, the ove

17 d by ventricular tachycardia/fibrillation or pulseless electric activity/asystole.

18 (49.84%), ventricular fibrillation (32.0%), pulseless electrical activity (14.6%), and asystole (9.1

19 en groups, as was the incidence of postshock pulseless electrical activity (15/18 MTE, 18/20 BTE) and

20 ibrillation), ventricular fibrillation (40), pulseless electrical activity (20), and asystole (20), i

21 initial rhythm was perfusing (p < .0001) or pulseless electrical activity (p = .0002), and not relat

22 scitation for the diagnosis and treatment of pulseless electrical activity (PEA) correctly stress the

23 been a significant rise in the prevalence of pulseless electrical activity (PEA).

24 ciated with better outcomes than asystole or pulseless electrical activity (PEA).

25 ock is most commonly followed by asystole or pulseless electrical activity (PEA).

26 developed hypotension, dyspnea, hypoxia, and pulseless electrical activity 10 days after resection of

27 ar fibrillation/ventricular tachycardia 24%, pulseless electrical activity 16%, asystole 48%, other n

28 to resultant postdefibrillation rhythms (ie, pulseless electrical activity [PEA] or asystole).

29 acing) and, after successful defibrillation, pulseless electrical activity and asystole (by high-dose

30 ng, anaphylaxis with a difficult airway, and pulseless electrical activity arrest.

31 eased survival after in-hospital asystole or pulseless electrical activity arrest.

32 A post hoc analysis of patients with pulseless electrical activity at any time during the car

33 after prolonged ventricular fibrillation and pulseless electrical activity cardiac arrest.

34 day after he first became ill, he suffered a pulseless electrical activity cardiorespiratory arrest f

35 nimals developed refractory postcountershock pulseless electrical activity compared with 0 of 16 trea

36 Patients in the subgroup presenting with pulseless electrical activity had intensive care unit ad

37 Asystole occurred in 23 (17 died), pulseless electrical activity in 2, and respiratory arre

38 itial cardiac arrest rhythm was asystole and pulseless electrical activity in 874 children (84.8%) an

39 ortion of cardiac arrests due to asystole or pulseless electrical activity increased over time (P<0.0

40 15 defibrillation 1st animals (p <.001), and pulseless electrical activity occurred in only one of 15

41 bserved in patients whose initial rhythm was pulseless electrical activity or asystole.

42 of >4.0 mmol/L (normal range 0.0 to 2.2); c) pulseless electrical activity or ventricular fibrillatio

43 , with an increase in cardiac arrests due to pulseless electrical activity over time (P for trend <0.

44 more asystole (8.8% vs. 7%) and (organized) pulseless electrical activity than men (24% vs. 18%; p <

45 2.2%) had nonshockable rhythms (asystole and pulseless electrical activity) and 2079 (17.8%) had shoc

46 nd separately for nonshockable (asystole and pulseless electrical activity) and shockable (ventricula

47 perienced nonshockable OHCA (ie, asystole or pulseless electrical activity) were assessed for eligibi

48 atients whose initial rhythm was asystole or pulseless electrical activity, AEDs were associated with

49 suscitation followed by countershock-induced pulseless electrical activity, after which animals were

50 , 79.3% had an initial rhythm of asystole or pulseless electrical activity, and 20.7% had ventricular

51 tubation, and time to asystole, hypotension, pulseless electrical activity, and declaration of death

52 en the initial cardiac rhythm is asystole or pulseless electrical activity, particularly in patients

53 ith an initial cardiac rhythm of asystole or pulseless electrical activity, pre-hospital cooling usin

54 In animals with pulseless electrical activity, sodium nitroprusside-enha

55 rse than those for patients with asystole or pulseless electrical activity.

56 m survival rates in patients presenting with pulseless electrical activity.

57 rolonged VF typically results in asystole or pulseless electrical activity.

58 ut was offset by more cases with asystole or pulseless electrical activity.

59 ardia/ventricular fibrillation as opposed to pulseless electrical activity/asystole (epilepsy, 26%; n

60 al oxygen extraction in adult patients after pulseless electrical activity/asystole or resistant vent

61 f VF or pulseless VT as the first documented pulseless rhythm was 14% (120/880) in children and 23% (

62 have ventricular arrhythmias as their first pulseless rhythm, to be monitored and hospitalized in th

63 fibrillation or tachycardia was the initial pulseless rhythm; in 149 patients (15 percent), it devel

64 as a major contributor to the development of pulseless rhythms.

65 nitial rhythms of ventricular tachycardia or pulseless ventricular fibrillation (VT/VF).

66 care unit, younger age, an initial rhythm of pulseless ventricular tachycardia (VT) or ventricular fi

67 ften due to ventricular fibrillation (VF) or pulseless ventricular tachycardia (VT), which are associ

68 for victims with ventricular fibrillation or pulseless ventricular tachycardia (VT/VF), compared with

69 shockable rhythms (ventricular fibrillation/pulseless ventricular tachycardia [VF/VT]) during resusc

70 shock-refractory ventricular fibrillation or pulseless ventricular tachycardia after at least one sho

71 e frequencies of ventricular fibrillation or pulseless ventricular tachycardia and of survival to hos

72 graphy rhythm of ventricular fibrillation or pulseless ventricular tachycardia and still unconscious

73 The incidence of ventricular fibrillation or pulseless ventricular tachycardia as the first recorded

74 ac arrest due to ventricular fibrillation or pulseless ventricular tachycardia at 369 hospitals parti

75 l adult primary ventricular fibrillation and pulseless ventricular tachycardia cardiac arrests 36 mon

76 c arrests due to ventricular fibrillation or pulseless ventricular tachycardia enrolled between Janua

77 ren (84.8%) and ventricular fibrillation and pulseless ventricular tachycardia in 157 children (15.2%

78 atio for initial ventricular fibrillation or pulseless ventricular tachycardia in public versus at ho

79 sts with initial ventricular fibrillation or pulseless ventricular tachycardia is much greater in pub

80 Patients with IHCA caused by pulseless ventricular tachycardia or ventricular fibrill

81 des of recurrent ventricular fibrillation or pulseless ventricular tachycardia requiring additional e

82 the incidence of ventricular fibrillation or pulseless ventricular tachycardia was 25% when the arres

83 arrest rhythm of ventricular fibrillation or pulseless ventricular tachycardia was refractory to shoc

84 iac arrest with ventricular fibrillation (or pulseless ventricular tachycardia) and who had not been

85 and shockable (ventricular fibrillation and pulseless ventricular tachycardia) cardiac arrests.

86 ckable rhythms (ventricular fibrillation and pulseless ventricular tachycardia).

87 shock-refractory ventricular fibrillation or pulseless ventricular tachycardia, but without proven su

88 arrest because of ventricular fibrillation, pulseless ventricular tachycardia, or unknown shockable

89 shock-refractory ventricular fibrillation or pulseless ventricular tachycardia.

90 th shock-resistant ventricular fibrillation /pulseless ventricular tachycardia.

91 y, and 20.7% had ventricular fibrillation or pulseless ventricular tachycardia.

92 ased likelihood of survival in patients with pulseless ventricular tachycardia/ventricular fibrillati

93 e who presented with bradycardia followed by pulseless ventricular tachycardia/ventricular fibrillati

94 in shock-resistant ventricular fibrillation/pulseless ventricular tachycardia; however, the efficacy

95 The prevalence of VF or pulseless VT as the first documented pulseless rhythm wa

96 ored status, time to defibrillation of VF or pulseless VT, intensive care unit location of arrest, an

97 t-of-hospital cardiac arrest caused by VF or pulseless VT, we found early coronary angiography was as

98 s despite fewer cardiac arrests due to VF or pulseless VT.

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