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1 rse than those for patients with asystole or pulseless electrical activity.
2 m survival rates in patients presenting with pulseless electrical activity.
3 rolonged VF typically results in asystole or pulseless electrical activity.
4 ut was offset by more cases with asystole or pulseless electrical activity.
5 developed hypotension, dyspnea, hypoxia, and pulseless electrical activity 10 days after resection of
6 (49.84%), ventricular fibrillation (32.0%), pulseless electrical activity (14.6%), and asystole (9.1
7 en groups, as was the incidence of postshock pulseless electrical activity (15/18 MTE, 18/20 BTE) and
8 ar fibrillation/ventricular tachycardia 24%, pulseless electrical activity 16%, asystole 48%, other n
9 ibrillation), ventricular fibrillation (40), pulseless electrical activity (20), and asystole (20), i
10 atients whose initial rhythm was asystole or pulseless electrical activity, AEDs were associated with
11 suscitation followed by countershock-induced pulseless electrical activity, after which animals were
12 acing) and, after successful defibrillation, pulseless electrical activity and asystole (by high-dose
13 2.2%) had nonshockable rhythms (asystole and pulseless electrical activity) and 2079 (17.8%) had shoc
14 nd separately for nonshockable (asystole and pulseless electrical activity) and shockable (ventricula
15 , 79.3% had an initial rhythm of asystole or pulseless electrical activity, and 20.7% had ventricular
16 tubation, and time to asystole, hypotension, pulseless electrical activity, and declaration of death
19 ardia/ventricular fibrillation as opposed to pulseless electrical activity/asystole (epilepsy, 26%; n
20 al oxygen extraction in adult patients after pulseless electrical activity/asystole or resistant vent
23 day after he first became ill, he suffered a pulseless electrical activity cardiorespiratory arrest f
24 nimals developed refractory postcountershock pulseless electrical activity compared with 0 of 16 trea
25 Patients in the subgroup presenting with pulseless electrical activity had intensive care unit ad
27 itial cardiac arrest rhythm was asystole and pulseless electrical activity in 874 children (84.8%) an
28 ortion of cardiac arrests due to asystole or pulseless electrical activity increased over time (P<0.0
29 15 defibrillation 1st animals (p <.001), and pulseless electrical activity occurred in only one of 15
31 of >4.0 mmol/L (normal range 0.0 to 2.2); c) pulseless electrical activity or ventricular fibrillatio
32 , with an increase in cardiac arrests due to pulseless electrical activity over time (P for trend <0.
33 initial rhythm was perfusing (p < .0001) or pulseless electrical activity (p = .0002), and not relat
34 en the initial cardiac rhythm is asystole or pulseless electrical activity, particularly in patients
35 scitation for the diagnosis and treatment of pulseless electrical activity (PEA) correctly stress the
40 ith an initial cardiac rhythm of asystole or pulseless electrical activity, pre-hospital cooling usin
42 more asystole (8.8% vs. 7%) and (organized) pulseless electrical activity than men (24% vs. 18%; p <
43 perienced nonshockable OHCA (ie, asystole or pulseless electrical activity) were assessed for eligibi
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