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1 nucleinopathies, multiple system atrophy and pure autonomic failure.
2 It was indistinguishable from pure autonomic failure.
3 OVID, control subjects, and individuals with pure autonomic failure.
4 dysfunction that is similar to patients with pure autonomic failure.
5 glycol higher in Parkinson's disease than in pure autonomic failure.
8 rance in 19 patients with severe NOH (8 with pure autonomic failure and 11 with multiple-system atrop
9 h Parkinson disease or the Lewy body form of pure autonomic failure and 15 controls underwent (18)F-D
11 chronically deficient sympathetic activity (pure autonomic failure), and 15 normal age-matched contr
12 nian, cerebellar, and mixed), 19 people with pure autonomic failure, and 27 healthy participants.
13 onomic dysfunction (multiple system atrophy, pure autonomic failure, and baroreflex failure) were com
14 nic cases, some being indistinguishable from pure autonomic failure, and support the concept that gan
15 INTERPRETATION: Patients presenting with pure autonomic failure are at high risk of phenoconverti
16 ID-19 negative, normal autonomic tests), and pure autonomic failure (COVID-19 negative, abnormal auto
18 inson's disease, multiple system atrophy and pure autonomic failure groups all had lower cerebrospina
19 redominant cerebellar involvement) in eight, pure autonomic failure in two and Parkinson's disease in
20 n tomography and a human neurological model (pure autonomic failure), in which peripheral autonomic d
23 without SH (PD - SH, n = 19), patients with pure autonomic failure (n = 8), and controls (n = 16).
24 n both the multiple system atrophy (n=5) and pure autonomic failure (n=4) groups, all initial pressur
26 orthostatic hypotension (OH) (PD+OH) or with pure autonomic failure (PAF) have markedly decreased myo
28 cribe the clinical features of patients with pure autonomic failure (PAF) preceding phenoconversion t
31 tients with multiple system atrophy (MSA) or pure autonomic failure (PAF), we studied the effect of o
34 otension (PD+OH, PD-No-OH); in patients with pure autonomic failure (PAF, a Lewy body disease without
35 es [DLB], multiple system atrophy [MSA], and pure autonomic failure [PAF]) is an urgent unmet need.
37 The small group of patients retaining the pure autonomic failure phenotype had very low plasma nor
38 with no signs of central neurodegeneration (pure autonomic failure), two with parkinsonism responsiv
40 in retrospective samples from patients with pure autonomic failure who later developed PD or MSA.
41 biomarkers that predict which patients with pure autonomic failure will develop Parkinson disease, d
42 GH in serum in healthy people and those with pure autonomic failure (with peripheral lesions), but no