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1 nucleinopathies, multiple system atrophy and pure autonomic failure.
2                It was indistinguishable from pure autonomic failure.
3 glycol higher in Parkinson's disease than in pure autonomic failure.
4 ts (13.2 [7.0-18.6] mU/L), and patients with pure autonomic failure (12.5 [5.6-18.2] mU/L).
5                                Patients with pure autonomic failure also had very low levels of plasm
6 rance in 19 patients with severe NOH (8 with pure autonomic failure and 11 with multiple-system atrop
7 on's disease, 54 multiple system atrophy, 20 pure autonomic failure) and 38 controls.
8  chronically deficient sympathetic activity (pure autonomic failure), and 15 normal age-matched contr
9 nian, cerebellar, and mixed), 19 people with pure autonomic failure, and 27 healthy participants.
10 onomic dysfunction (multiple system atrophy, pure autonomic failure, and baroreflex failure) were com
11 nic cases, some being indistinguishable from pure autonomic failure, and support the concept that gan
12     INTERPRETATION: Patients presenting with pure autonomic failure are at high risk of phenoconverti
13 inson's disease, multiple system atrophy and pure autonomic failure groups all had lower cerebrospina
14 redominant cerebellar involvement) in eight, pure autonomic failure in two and Parkinson's disease in
15 n tomography and a human neurological model (pure autonomic failure), in which peripheral autonomic d
16                      Parkinson's disease and pure autonomic failure involve differential dopaminergic
17                In chronic autonomic failure (pure autonomic failure, multiple system atrophy, or auto
18  without SH (PD - SH, n = 19), patients with pure autonomic failure (n = 8), and controls (n = 16).
19 n both the multiple system atrophy (n=5) and pure autonomic failure (n=4) groups, all initial pressur
20                                Patients with pure autonomic failure or parkinsonism and sympathetic n
21 orthostatic hypotension (OH) (PD+OH) or with pure autonomic failure (PAF) have markedly decreased myo
22 tients with multiple system atrophy (MSA) or pure autonomic failure (PAF), we studied the effect of o
23 le system atrophy (MSA), and 8 patients with pure autonomic failure (PAF).
24 otension (PD+OH, PD-No-OH); in patients with pure autonomic failure (PAF, a Lewy body disease without
25    The small group of patients retaining the pure autonomic failure phenotype had very low plasma nor
26  with no signs of central neurodegeneration (pure autonomic failure), two with parkinsonism responsiv
27      One hundred patients who presented with pure autonomic failure were recruited at 5 medical cente
28  biomarkers that predict which patients with pure autonomic failure will develop Parkinson disease, d
29 GH in serum in healthy people and those with pure autonomic failure (with peripheral lesions), but no

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