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1 yasthenia gravis, hypogammaglobulinemia, and pure red cell aplasia.
2 and, unlike EPO products, reduce the risk of pure red cell aplasia.
3 atible transplant and may be associated with pure red cell aplasia.
4 set disorders), the most common of which was pure red-cell aplasia.
5 s the disorder most commonly associated with pure red cell aplasia, (2) the presence of clonal cytoge
7 he association between rituximab therapy and pure red cell aplasia, but the diagnostic and therapeuti
8 receptor can correct anemia in patients with pure red-cell aplasia caused by antierythropoietin antib
9 nus, graft-vs-host disease-like colitis, and pure red cell aplasia, different from the pattern observ
11 We obtained reports of epoetin-associated pure red-cell aplasia from the Food and Drug Administrat
12 sease (erythema infectiosum) in children and pure red cell aplasia in immunocompromised patients.
13 e identified 47 adult patients with acquired pure red cell aplasia (median age, 64 years; range, 22 t
14 patients with chronic kidney disease who had pure red-cell aplasia or hypoplasia due to antierythropo
17 d 47 months, respectively, before developing pure red cell aplasia (PRCA) confirmed by bone marrow bi
20 munoglobulin (IVIG) therapy in patients with pure red cell aplasia (PRCA) related to human parvovirus
23 equence of this effect of rituximab therapy: pure red cell aplasia resulting from chronic parvovirus
25 oute of administration, the country in which pure red-cell aplasia was identified, and the date on wh
26 After the peak incidence of Eprex-associated pure red-cell aplasia was reached in 2001, interventions
27 Between 1988 and 1998, antibody-associated pure red-cell aplasia was reported in three patients who
30 April 2004, 175 cases of epoetin-associated pure red-cell aplasia were reported for Eprex, 11 cases
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