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1 sceptibility to glomerular damage induced by puromycin aminonucleoside.
2 d can be induced in rats by the injection of puromycin aminonucleoside.
3 romatic substitution with dimethylamine gave puromycin aminonucleoside [9-(3-amino-3-deoxy-beta-D-rib
4 ycin, cycloheximide, emetine, puromycin, and puromycin aminonucleoside, a negative analog, were evalu
5   Treatment of differentiated podocytes with puromycin aminonucleoside, an agent that causes foot pro
6  with proteinuria and hypoalbuminemia in the puromycin aminonucleoside and adriamycin rat models of n
7 cyte injury was induced in C57BL/6 mice with puromycin aminonucleoside, and the selective A(2A)R agon
8 to glomerular injury, because treatment with puromycin aminonucleoside enhanced proteinuria in TG mic
9  Sprague-Dawley rats by an i.v. injection of puromycin aminonucleoside in saline; seven control rats
10 us injection of OX-7 monoclonal antibody and puromycin aminonucleoside in which 10(7) SPIO- and DiI-l
11 docytes, before or after sublethal injury by puromycin aminonucleoside, in the presence or absence of
12  lacking cathepsin L are protected from cell puromycin aminonucleoside-induced cell detachment.
13 sion was underscored by the observation that puromycin aminonucleoside-induced cell migration was slo
14 ne significantly protected podocytes against puromycin aminonucleoside-induced injury (designed to mi
15 n of the actin cytoskeleton that accompanied puromycin aminonucleoside-induced injury in vitro.
16 rintegrin reduced peak proteinuria caused by puromycin aminonucleoside-induced nephropathy.
17 ing the peritoneum permeability of rats with puromycin aminonucleoside-induced nephrotic syndrome.
18 ation of alpha(3) integrin protected against puromycin aminonucleoside-induced podocyte detachment.
19                                We found that puromycin aminonucleoside-induced proteinuria in rats wa
20                                    Rats with puromycin-aminonucleoside-induced nephrotic proteinuria
21 gnificantly increased GEN sprouting, whereas puromycin aminonucleoside-injured podocyte supernatant d
22                              Notably, in the puromycin aminonucleoside model, hyperfibrinogenemia and
23                                           In puromycin aminonucleoside nephrosis (PAN), GIV expressio
24          Here, we show that the induction of puromycin aminonucleoside nephrosis involves podocyte mi
25                         Furthermore, the rat puromycin aminonucleoside nephrosis model, previously su
26  syndrome in humans, in animals treated with puromycin aminonucleoside, or in humans or animals with
27 docytes in the passive Heymann nephritis and puromycin aminonucleoside (PA) nephrosis rat models of p
28 d mouse podocytes in culture were exposed to puromycin aminonucleoside (PA) to induce apoptosis.
29 accumulation and phosphorylation of hsp27 in puromycin aminonucleoside (PAN) -induced experimental NS
30 elevated in podocytes from rats treated with puromycin aminonucleoside (PAN) and from patients with f
31 proteinuria in a rat model of MCD induced by puromycin aminonucleoside (PAN) and in vitro cultured mo
32  the effects of this mutation in response to puromycin aminonucleoside (PAN) nephrosis.
33 , 10, 14, and 21 days after the induction of puromycin aminonucleoside (PAN) nephrosis.
34 ague-Dawley rats by intravenous injection of puromycin aminonucleoside (PAN), whereas control rats re
35 ) on renal function and structure in chronic puromycin aminonucleoside (PAN)-induced nephropathy in r
36                                              Puromycin aminonucleoside (PAN)-induced nephrosis is a w
37 mage from a glomerular injury-inducing agent puromycin aminonucleoside (PAN).
38                          The inactive agent, puromycin aminonucleoside, produced marked repression of
39 d this interaction is disrupted in GECs from puromycin aminonucleoside-, protamine sulfate-, or siali
40 me and GEC foot process effacement using the puromycin aminonucleoside rat model resulted in signific
41                            The peritoneum of puromycin aminonucleoside rats displayed an increase in
42 Stimulation of receptor-operated channels in puromycin aminonucleoside-treated podocytes leads to inc
43 e damage (WT1 heterozygous knockout mice and puromycin aminonucleoside-treated rats).
44                                              Puromycin aminonucleoside treatment up-regulates catheps
45                                              Puromycin aminonucleoside was without effect on these pa

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