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3 Xanthomonas oryzae pathovar (pv.) oryzae and pv. oryzicola produce numerous transcription activator-l
4 eudomonas syringae pathovar (pv.) tomato and pv. maculicola were made by insertion of the luxCDABE op
5 gens Xanthomonas oryzae pv. oryzae (Xoo) and pv. oryzicola (Xoc) encode numerous secreted transcripti
7 odis pv. citrumelo to those of X. axonopodis pv. citri and X. campestris pv. vesicatoria provides val
16 identified unique effectors in X. axonopodis pv. citrumelo that may be related to the different host
18 he complete genome sequence of X. axonopodis pv. citrumelo to those of X. axonopodis pv. citri and X.
19 pAI, and hrpW were absent from X. axonopodis pv. citrumelo while present in X. axonopodis pv. citri.
21 anker (CBC) caused by Xanthomonas axonopodis pv. citri (Xac) was first documented in India and Java i
22 The protein Clp from Xanthomonas axonopodis pv. citri regulates pathogenesis and is a member of the
25 ght (CBB), incited by Xanthomonas axonopodis pv. manihotis (Xam), is the most important bacterial dis
27 Mb genome sequence of Xanthomonas axonopodis pv. punicae strain LMG 859, the causal agent of bacteria
28 terial plant pathogen Xanthomonas axonopodis pv. vesicatoria (Xav) and examined the effect of the los
29 v library were conjugated into X. campestris pv. campestris (Xcc) and exconjugants were scored for an
31 tomato bacterial spot pathogen X. campestris pv. vesicatoria 85-10, with a completely different host
32 of X. axonopodis pv. citri and X. campestris pv. vesicatoria provides valuable insights into the mech
33 to B. cinerea infection and to X. campestris pv. vesicatoria, correlated with cuticle permeability an
37 family encoded by the Xanthomonas campestris pv. campestris str. ATCC 33913 genome (GI:21233491).
39 e-genome sequences of Xanthomonas campestris pv. raphani strain 756C and X. oryzae pv. oryzicola stra
40 confers resistance to Xanthomonas campestris pv. vesicatoria (Xcv) pathogenic strains which contain t
41 he bacterial pathogen Xanthomonas campestris pv. vesicatoria (Xcv) uses a type III secretion system (
45 ecreted effector from Xanthomonas campestris pv. vesicatoria, is a desumoylating enzyme with strict s
49 pic mechanism of thermal transport in MgSiO3 pv, and provide reference data for understanding heat co
51 esults support the hypothesis that X. oryzae pv. oryzae commandeers the regulation of otherwise devel
52 ive resistance to PthXo2-dependent X. oryzae pv. oryzae due to promoter variations of OsSWEET13 in ja
53 esults corroborate a model whereby X. oryzae pv. oryzae enhances the release of sucrose from host cel
54 These results suggest that the X. oryzae pv. oryzae PhoPQ TCS functions in virulence and in the p
57 ain and found it to be impaired in X. oryzae pv. oryzae virulence and no longer able to activate the
61 estris pv. raphani strain 756C and X. oryzae pv. oryzicola strain BLS256, pathogens that infect the m
62 he strains in the African clade of X. oryzae pv. oryzicola, representing the first dominant resistanc
63 m and mesophyll pathogens Xanthomonas oryzae pv. oryzae (Xoo) and pv. oryzicola (Xoc) encode numerous
64 hway of the rice pathogen Xanthomonas oryzae pv. oryzae (Xoo) in a semi-isolated environment represen
65 The biotrophic pathogen Xanthomonas oryzae pv. oryzae (Xoo) produces a sulfated peptide named RaxX,
66 ce to a broad spectrum of Xanthomonas oryzae pv. oryzae (Xoo) races that cause bacterial blight disea
67 bacterial blight pathogen Xanthomonas oryzae pv. oryzae (Xoo) that deliver the TAL effector AvrXa27.
68 livered by the pathogenic Xanthomonas oryzae pv. oryzae (Xoo), in revealing the new function of two p
69 ceptor confer immunity to Xanthomonas oryzae pv. oryzae (Xoo), the causal agent of bacterial leaf bli
70 n by pathogenic bacterium Xanthomonas oryzae pv. oryzae (Xoo), which causes a vascular disease in ric
80 xtraordinary diversity of Xanthomonas oryzae pv. oryzae genotypes and races that have been isolated f
81 ing disease by strains of Xanthomonas oryzae pv. oryzae is dependent on major transcription activatio
83 levated upon infection by Xanthomonas oryzae pv. oryzae strain PXO99(A) and depends on the type III e
86 21, confers resistance to Xanthomonas oryzae pv. oryzae strains producing the type one system-secrete
87 ative bacterial pathogen, Xanthomonas oryzae pv. oryzae upon recognition of a small protein, Ax21, th
90 the gamma-proteobacterium Xanthomonas oryzae pv. oryzae, which utilizes a group of type III TAL (tran
93 Gram-negative bacterium, Xanthomonas oryzae pv. oryzae; NH1, the rice ortholog of NPR1, a key regula
95 e rice bacterial pathogen Xanthomonas oryzae pv. oryzicola (Xoc) has been demonstrated to contain a b
98 native TAL effector from Xanthomonas oryzae pv. oryzicola drives expression of a target with an EBE
99 cognizes a rice pathogen, Xanthomonas oryzae pv. oryzicola, which causes bacterial streak disease.
101 rice pathogens Xanthomonas oryzae pathovar (pv.) oryzae and pv. oryzicola produce numerous transcrip
102 ana pathogens Pseudomonas syringae pathovar (pv.) tomato and pv. maculicola were made by insertion of
103 mal conductivity kappa of MgSiO3 perovskite (pv) by ab initio lattice dynamics calculations combined
104 their control proteins HrpV and HrpG from Ps pv. tomato DC3000 in vitro establish that HrpRS forms a
106 ene production and increases growth of P. s. pv. tomato and severity of speck disease on susceptible
109 st response to the large collection of P. s. pv. tomato strains that express different combinations o
110 the host Pto kinase, which recognizes P. s. pv. tomato strains that express the effector AvrPto.
113 n both the olive tree pathogen P. savastanoi pv. savastanoi and the human opportunistic pathogen Pseu
114 Deletion of the dgcP gene in P. savastanoi pv. savastanoi NCPPB 3335 and P. aeruginosa PAK reduced
115 e olive tree pathogen Pseudomonas savastanoi pv. savastanoi is dependent on the integrity of its GGDE
116 fforts allowed overproduction of P. syringae pv. glycinea PG4180 CmaA in P. syringae pv. syringae FF5
118 phaseolicola, and pPMA4326A from P. syringae pv. maculicola encoded a type IVA T4SS (VirB-VirD4 conju
119 owth of the universally virulent P. syringae pv. maculicola ES4326 among more than 100 Arabidopsis ec
122 P. syringae pv. syringae 61 and P. syringae pv. phaseolicola 1448A restored HR elicitation and patho
123 report on the genome sequence of P. syringae pv. phaseolicola isolate 1448A, which encodes 5,353 open
125 ngae pv. syringae, pPh1448B from P. syringae pv. phaseolicola, and pPMA4326A from P. syringae pv. mac
128 P. syringae pathovars, hrpP from P. syringae pv. syringae 61 and P. syringae pv. phaseolicola 1448A r
129 g a plasmid-encoded T3SS and the P. syringae pv. syringae 61 effector gene hopA1 increased in planta
132 Here we characterize a gene in P. syringae pv. syringae B728a and P. syringae pv. tomato DC3000, te
133 tor gene composition; the EEL of P. syringae pv. syringae B728a is the largest and most complex.
134 e supporting the hypothesis that P. syringae pv. syringae B728a produces both of these siderophores.
138 ngae pv. glycinea PG4180 CmaA in P. syringae pv. syringae FF5 as a FLAG-tagged protein and overproduc
139 welve PFPs along with pPSR1 from P. syringae pv. syringae, pPh1448B from P. syringae pv. phaseolicola
141 ana did not confer resistance to P. syringae pv. tabaci (Pta) expressing avrPto or avrPtoB, but recog
143 fect was suppressed by wild-type P. syringae pv. tabaci and P. fluorescens heterologously expressing
144 esistant to a virulent strain of P. syringae pv. tabaci and showed an accelerated hypersensitive resp
145 cens, a TTSS-deficient mutant of P. syringae pv. tabaci, or flg22 (a flagellin-derived peptide elicit
147 AD(P) leaking into the ECC after P. syringae pv. tobacco DC3000/avrRpt2 infection is sufficient for i
148 y related to the tomato pathogen P. syringae pv. tomato (Pto), including strains isolated from snowpa
149 ficantly when co-inoculated with P. syringae pv. tomato but not when co-inoculated with a type III se
150 ed protein and overproduction of P. syringae pv. tomato CmaA in Escherichia coli as a His-tagged prot
152 not prevent the HR activated by P. syringae pv. tomato DC3000 + avrB, avrRpm1, avrRps4 or avrRpt2, b
153 and related T3SS substrates for P. syringae pv. tomato DC3000 and three other sequenced strains.
154 728a, P. syringae strain 61, and P. syringae pv. tomato DC3000 differ in size and effector gene compo
156 d to test the ability of several P. syringae pv. tomato DC3000 effectors for their ability to suppres
158 ated with the Hrp T3SS system in P. syringae pv. tomato DC3000 have predicted lytic transglycosylase
161 variance of basal resistance to P. syringae pv. tomato DC3000 in the Col-0 x Fl-1 F(2) population.
163 the loss of swarming motility of P. syringae pv. tomato DC3000 on medium containing a low percentage
165 repertoire of the model pathogen P. syringae pv. tomato DC3000 were deleted to produce polymutant DC3
166 ogenetically divergent pathovar, P. syringae pv. tomato DC3000, revealed a strong degree of conservat
167 syringae pv. syringae B728a and P. syringae pv. tomato DC3000, termed phcA, that has homology to a f
170 1-1, hopS1, and hopS2 operons in P. syringae pv. tomato DC3000; these operons encode three homologous
172 a virulence protein by promoting P. syringae pv. tomato growth and enhancing symptoms associated with
175 ells to promote the virulence of P. syringae pv. tomato strain DC3000 (PstDC3000) on Arabidopsis thal
177 ine family transporter (BCCT) in P. syringae pv. tomato strain DC3000 that mediates the transport of
178 In this study, we found that P. syringae pv. tomato strain DC3000 was distinct from most bacteria
179 hat were susceptible to virulent P. syringae pv. tomato strain DC3000, but resistant to DC3000 expres
180 efined 29 type III proteins from P. syringae pv. tomato, and 19 from P. syringae pv. phaseolicola rac
182 The introduction of Pseudomonas syringae pv. actinidiae (Psa) severely damaged the New Zealand ki
184 the bacterial pathogen Pseudomonas syringae pv. maculicola (Pma) ES4326 and activation of SA synthes
185 nced resistance to both Pseudomonas syringae pv. maculicola (Psm) ES4326 and Hyaloperonospora parasit
190 real bacterial pathogen Pseudomonas syringae pv. oryzae, transgenic EFR wheat lines had reduced lesio
191 from the bean pathogen Pseudomonas syringae pv. phaseolicola (Pph) is driven by exposure to the stre
193 with the bean pathogen Pseudomonas syringae pv. phaseolicola illustrate how exposure to resistance m
194 opsis is a non-host for Pseudomonas syringae pv. phaseolicola NPS3121 (Pph), a bacterial pathogen of
195 rming enzyme (EFE) from Pseudomonas syringae pv. phaseolicola PK2 is a member of the mononuclear nonh
196 non-host resistance to Pseudomonas syringae pv. phaseolicola strain 3121 (Psp), suggesting that resi
197 with the plant pathogen Pseudomonas syringae pv. phaseolicola where isolates that have lost the genom
200 n effector protein from Pseudomonas syringae pv. pisi, triggers RPS4-dependent immunity in resistant
201 ete hrp/hrc region from Pseudomonas syringae pv. syringae 61 into the genome of the soil bacterium Ps
202 ated as a phytotoxin by Pseudomonas syringae pv. syringae B301D contains a 4-Cl-L-Thr-9 moiety where
203 ete genomic sequence of Pseudomonas syringae pv. syringae B728a (Pss B728a) has been determined and i
208 transcript profiles of Pseudomonas syringae pv. syringae B728a support a model in which leaf surface
209 surfactant produced by Pseudomonas syringae pv. syringae B728a that was not detected by traditional
212 in and syringopeptin by Pseudomonas syringae pv. syringae is controlled by the regulatory genes salA
215 lowing inoculation with Pseudomonas syringae pv. tabaci carrying avrPto, aconitase-silenced N. bentha
217 mplement recognition of Pseudomonas syringae pv. tomato (Pst) bacteria expressing either avrPto or av
218 ility of Arabidopsis to Pseudomonas syringae pv. tomato (Pst) DC3000 independently of the phyB/PIF th
219 the bacterial pathogen Pseudomonas syringae pv. tomato (Pst) DC3000, including increased disease res
222 lent bacterial pathogen Pseudomonas syringae pv. tomato (Pst) DC3000/avrRpt2, and renders plants susc
223 the bacterial pathogen Pseudomonas syringae pv. tomato (Pst) in the cultivated tomato, Lycopersicon
224 retion system (TTSS) of Pseudomonas syringae pv. tomato (Pst) injects into the plant cell effector pr
225 PS2, RPM1, and RPS5, to Pseudomonas syringae pv. tomato (Pst) strain DC3000 containing the cognate ef
226 The bacterial pathogen Pseudomonas syringae pv. tomato (Pst) strain DC3000 infects tomato and Arabid
227 n effector protein from Pseudomonas syringae pv. tomato (Pst), behaves as an avirulence factor that a
233 th a virulent strain of Pseudomonas syringae pv. tomato also up-regulated LeARG2 expression and argin
236 acterial plant pathogen Pseudomonas syringae pv. tomato DC3000 (DC3000) causes disease in Arabidopsis
237 The plant pathogen Pseudomonas syringae pv. tomato DC3000 (DC3000) is found in a wide variety of
239 sigma factor encoded by Pseudomonas syringae pv. tomato DC3000 (DC3000), a plant pathogen that is lik
240 on after infection with Pseudomonas syringae pv. tomato DC3000 (Pst DC3000), demonstrating that the t
241 st avirulent strains of Pseudomonas syringae pv. tomato DC3000 (Pst) carrying AvrRpt2, AvrB, or AvrPp
243 g22 or inoculation with Pseudomonas syringae pv. tomato DC3000 (PstDC3000) hrcC mutant, which is defi
246 allenge with strains of Pseudomonas syringae pv. tomato DC3000 allowing differentiation of basal resi
247 the biotrophic bacteria Pseudomonas syringae pv. tomato DC3000 and for susceptibility to the necrotro
249 stance to the bacterium Pseudomonas syringae pv. tomato DC3000 and the fungal pathogen Botrytis ciner
250 ll as non-host pathogen Pseudomonas syringae pv. tomato DC3000 and the general elicitor cryptogein-in
252 al sensed by individual Pseudomonas syringae pv. tomato DC3000 cells during infection of Arabidopsis
253 to "mine" the genome of Pseudomonas syringae pv. tomato DC3000 for the metabolic potential to biosynt
255 fection with pathogenic Pseudomonas syringae pv. tomato DC3000 lacking hopQ1-1 [PtoDC3000(DeltahQ)] w
256 The bacterial pathogen Pseudomonas syringae pv. tomato DC3000 must detoxify plant-produced hydrogen
257 odel plant pathosystem, Pseudomonas syringae pv. tomato DC3000 on tomato and Arabidopsis thaliana hos
258 ut not to the bacterium Pseudomonas syringae pv. tomato DC3000 or to the oomycete Hyaloperonospora ar
259 signalling, for example Pseudomonas syringae pv. tomato DC3000 produces coronatine (COR), a jasmonic
261 how the plant pathogen Pseudomonas syringae pv. tomato DC3000 responds to iron limitation and have f
262 The bacterial pathogen Pseudomonas syringae pv. tomato DC3000 suppresses the two-tiered plant innate
264 nse, the plant pathogen Pseudomonas syringae pv. tomato DC3000 uses the virulence factor coronatine t
267 a during infection with Pseudomonas syringae pv. tomato DC3000, corn (Zea mays) under herbivory by co
268 ne were challenged with Pseudomonas syringae pv. tomato DC3000, which is a bacterial pathogen of bras
272 resistance response to Pseudomonas syringae pv. tomato expressing avrRps4 and was cloned based on th
273 in tomato to strains of Pseudomonas syringae pv. tomato expressing the (a)virulence proteins AvrPto o
274 eraction of tomato with Pseudomonas syringae pv. tomato is an established model system for understand
275 ersicum), resistance to Pseudomonas syringae pv. tomato is elicited by the interaction of the host Pt
276 tor protein AvrPto from Pseudomonas syringae pv. tomato is secreted into plant cells where it promote
277 nomical relevant tomato-Pseudomonas syringae pv. tomato pathosystem is widely used to explore and und
280 ity to the hemibiotroph Pseudomonas syringae pv. tomato strain DC3000 (Pto DC3000) was attenuated in
281 t the annotation of the Pseudomonas syringae pv. tomato strain DC3000 genome sequence and is easily p
283 cted a mutant screen of Pseudomonas syringae pv. tomato strain DC3000 to identify genes that contribu
284 se has been cloned from Pseudomonas syringae pv. tomato strain DC3000, Pseudomonas putida KT2440, and
285 genic bacteria, such as Pseudomonas syringae pv. tomato strain DC3000, the causative agent of tomato
288 ses upon recognition of Pseudomonas syringae pv. tomato strains expressing the AvrPto or AvrPtoB prot
289 acterial speck disease, Pseudomonas syringae pv. tomato, by recognizing the pathogen effector protein
290 d by the plant pathogen Pseudomonas syringae pv. tomato, has a carboxy-terminal domain that is an E3
291 enhanced resistance to Pseudomonas syringae pv. tomato, which is consistent with a previous study sh
299 t the bacterial pathogen Pseudomonas syringe pv. tomato (Pst) DC3000 in the salicylic acid (SA)- and
300 resistance to the pathogen Xanthomonasoryzae pv. oryzae (Xoo), suggesting the presence of a related d
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