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1 stations included pathergy, cystic acne, and pyoderma gangrenosum.
2 ations are discussed regarding psoriasis and pyoderma gangrenosum.
3 d skin ulcers with a clinical resemblance to pyoderma gangrenosum.
4 abscesses and pretibial lesions diagnosed as pyoderma gangrenosum.
5 ic vasculitides such as Sweet's syndrome and pyoderma gangrenosum.
6 tion in a patient presenting with aggressive pyoderma gangrenosum.
7 samples confirmed the clinical suspicion of pyoderma gangrenosum.
8 port mapping of a disease locus for familial pyoderma gangrenosum-acne-arthritis to the long arm of c
9 e in which cutaneous manifestations, such as pyoderma gangrenosum and acne fulminans, predominated.
11 mic inflammatory disease; pyogenic arthritis pyoderma gangrenosum and acne; Muckle-Wells syndrome; fa
12 from patients with Sweet's syndrome (SW) and pyoderma gangrenosum and found numerous novel splice var
13 functions of PSTPIP1 in the pathogenesis of pyoderma gangrenosum and suggest that the cytoskeleton i
14 report a case of CB, initially diagnosed as pyoderma gangrenosum and treated with steroids, leading
17 to cause the syndrome of pyogenic arthritis, pyoderma gangrenosum, and acne (PAPA), a dominantly inhe
21 PAPA syndrome (pyogenic sterile arthritis, pyoderma gangrenosum, and acne, OMIM #604416) and famili
23 of the 157 consecutive patients treated for pyoderma gangrenosum at our institution (10 percent).
24 illation-associated disease and suggest that pyoderma gangrenosum can be classified as a dynamical di
25 gnosis of exclusion, and the misdiagnosis of pyoderma gangrenosum can result in substantial complicat
27 Among other entities, CB may be mistaken for pyoderma gangrenosum due to overlap of findings on histo
28 95 patients studied, 64 had been treated for pyoderma gangrenosum for a median of 10 months (range, 3
30 rma gangrenosum (PPG), an unusual variant of pyoderma gangrenosum, has been reported almost exclusive
32 required in all patients suspected of having pyoderma gangrenosum in order to rule out alternative di
37 Of the ulcers in the 64 patients treated for pyoderma gangrenosum, it was clear that those in 23 pati
38 ssibility that refractory celiac disease and pyoderma gangrenosum may be immunologically different.
39 s detected in all other patients with either pyoderma gangrenosum or SW, it was always associated wit
44 nt) did not respond to treatment directed at pyoderma gangrenosum, those in 8 (12 percent) were exace
46 e charts of 240 patients with a diagnosis of pyoderma gangrenosum who were evaluated at our instituti
47 nt, rarely occurring, extra-hepatic onset of pyoderma gangrenosum, with the AIH in remission, strengt
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