ライフサイエンスコーパス: pyogenic

1 e was sequenced from 471 pharyngitis and 127 pyogenic and blood isolates recovered from 598 patients

2 controls competence in a parallel way in all pyogenic and bovis streptococci.

3 Alleles shared among the pharyngitis, pyogenic, and blood samples were identified in throat is

4 circuit linked to the sigX genes of mutans, pyogenic, and bovis streptococci that uses a novel small

5 ellulitis (OR = 1.75; CI, 1.1-2.8; P = .02), pyogenic arthritis (OR = 4.2; CI, 1.8-9.6; P = .001), di

6 mmon features of human PAPA syndrome such as pyogenic arthritis and skin inflammation were not recapi

7 sis of the synovium in patients with chronic pyogenic arthritis identified dramatic neovascularizatio

8 al onset multisystemic inflammatory disease; pyogenic arthritis pyoderma gangrenosum and acne; Muckle

9 ts with an autosomal dominant disease called pyogenic arthritis, pyoderma gangrenosum, and acne (PAPA

10 utations were shown to cause the syndrome of pyogenic arthritis, pyoderma gangrenosum, and acne (PAPA

11 Pyogenic Arthritis, Pyoderma Gangrenosum, and Acne Syndr

12 and tapetoretinal degeneration (MRST); and a pyogenic arthritis, pyoderma gangrenosum, and acne syndr

13 mic manifestations, skin cellulitis/abscess, pyogenic arthritis, tuberculosis, longer hospital stays,

14 s arise following parenchymal infection with pyogenic bacteria and are typified by inflammation and e

15 rm in response to a parenchymal infection by pyogenic bacteria, with Staphylococcus aureus representi

16 mutations in the TIR-MyD88 pathway underlie pyogenic bacterial diseases in childhood.

17 ad to severe immunodeficiency in response to pyogenic bacterial infection during childhood, little is

18 ogens is essential for successful control of pyogenic bacterial infection.

19 A case of pyogenic flexor tenosynovitis associated with C. cellula

20 lar, the mutant mice exhibited a substantial pyogenic form of the granulomatous response compared wit

21 al dystopia (3 cases), trichiasis (2 cases), pyogenic granuloma (2 cases), eyelid margin nodule (1 ca

22 pared for: 1) I-RL (n = 14), 2) peri-implant pyogenic granuloma (I-PG) (n = 5), 3) peri-implant perip

23 = 9), 4) T-RL (n = 44), 5) tooth-associated pyogenic granuloma (T-PG) (n = 21), and 6) tooth-associa

24 his is the first case in the literature of a pyogenic granuloma associated with a routine guided tiss

25 t dislocation in the Evicel group, 1 case of pyogenic granuloma in the Tisseel group, and no complica

26 Groesser et al. demonstrate that pyogenic granuloma is a RAS pathway-driven tumor.

27 A pyogenic granuloma is an exuberant growth of granulation

28 This case is interesting because the pyogenic granuloma was found associated with guided tiss

29 on average and included tearing, discomfort, pyogenic granuloma, and dacryocystitis.

30 Reviews of conjunctival myxoma, corneal pyogenic granuloma, and limbal juvenile xanthogranuloma

31 aluation of the tissue was consistent with a pyogenic granuloma.

32 Pyogenic granulomas generally are soft, painless, and de

33 s gingival lesions that clinically resembled pyogenic granulomas or periodontal abscesses, but with a

34 ntrol children, suggests that ocular surface pyogenic granulomas respond to topical timolol treatment

35 cutive children with acquired ocular surface pyogenic granulomas treated at Boston Children's Hospita

36 Pyogenic granulomas, acquired vascular lesions, form on

37 angiogenic response such as in psoriasis or pyogenic granulomas, the proliferating ECs markedly over

38 is, and species of the bovis, salivarius and pyogenic groups of streptococci, the pheromone XIP is se

39 have a protective role in murine immunity to pyogenic infection.

40 Most M1 GAS subclones recovered from pyogenic infections and blood were abundantly represente

41 ean of 9.8 months before their recovery from pyogenic infections and blood, which indicates that sele

42 rder of phagocyte function, characterized by pyogenic infections and granuloma formation caused by de

43 se (CGD), an immunodeficiency with recurrent pyogenic infections and granulomatous inflammation, resu

44 ciated with increased susceptibility to both pyogenic infections and to inflammatory disorders.

45 itment, mediated by beta2 integrins, combats pyogenic infections but also plays a key role in ischemi

46 Non-pyogenic infections of the spine are most frequently cau

47 Most patients die in childhood from pyogenic infections or an unusual lymphoma-like conditio

48 erican male presenting with severe recurrent pyogenic infections was found to have C2 deficiency (C2D

49 s a rare disorder characterized by recurrent pyogenic infections, defective neutrophil chemotaxis and

50 Although endophthalmitis secondary to pyogenic liver abscess (PLA) is becoming a globally emer

51 Although pyogenic liver abscess (PPLA) fatalities are decreasing

52 ng the viral hepatitides) include amebic and pyogenic liver abscess and cholangitis.

53 siella pneumoniae causing community-acquired pyogenic liver abscess complicated with metastatic menin

54 An emerging population of patients with pyogenic liver abscess includes those with complications

55 In contrast to amebic liver abscess, pyogenic liver abscess is associated with greater morbid

56 The overall prognosis for patients with pyogenic liver abscess is improving, although poor outco

57 Pyogenic liver abscess is most often cryptogenic in orig

58 Pyogenic liver abscess was predominantly managed by surg

59 Pyogenic liver abscess, although commonly occurring in p

60 ed by the same symptoms and signs as classic pyogenic liver abscess, but further distinguished by the

61 that of monomicrobial Klebsiella pneumoniae pyogenic liver abscess, characterized by the same sympto

62 phasized that the prognosis of patients with pyogenic liver abscess, who do not have underlying comor

63 leading to a new generation of patients with pyogenic liver abscess.

64 As of 1999, the vast majority of pyogenic liver abscesses should be approached therapeuti

65 s nitric oxide (.NO) in host defense against pyogenic microorganisms is unclear, and the actual inter

66 The type II ComRS system is shared by the pyogenic, mutans, and bovis streptococci, including the

67 ry episodes lead to accumulation of sterile, pyogenic, neutrophil-rich material within the affected j

68 Liver abscess is typically either of pyogenic or amebic origin.

69 Brain abscesses result from a pyogenic parenchymal infection commonly initiated by Gra

70 Common comorbidities with tuberculosis were pyogenic pneumonia in 26 patients (33%) and anaemia in 1

71 ison of MR images of patients diagnosed with pyogenic spondylodiscitis and tuberculous spondylodiscit

72 Characteristic features of pyogenic spondylodiscitis include: involvement of the lu

73 ish between tuberculous spondylodiscitis and pyogenic spondylodiscitis on MR images.

74 rentiate between features of tuberculous and pyogenic spondylodiscitis on MR images.

75 nts with confirmed spondylodiscitis (18 with pyogenic spondylodiscitis, and 16 with tuberculous spond

76 is the most common infectious agent causing pyogenic spondylodiscitis.

77 d autoinflammatory syndrome characterized by pyogenic sterile arthritis and less frequently accompani

78 disease, most notably in the PAPA syndrome: pyogenic sterile arthritis, pyoderma gangrenosum, and ac

79 Pyogenic sterile arthritis, pyoderma gangrenosum, and ac

80 bulinemia D with periodic fever syndrome and pyogenic sterile arthritis, pyoderma gangrenosum, and ac

81 PAPA syndrome (pyogenic sterile arthritis, pyoderma gangrenosum, and ac

82 r-X (fasX) were identified in four different pyogenic streptococcal species.

83 te proteins involved in haem uptake found in pyogenic streptococci and Clostridium novyi.

84 required for genetic transformation, yet no pyogenic streptococci are known to develop competence.

85 Patients with pyogenic vertebral osteomyelitis may have an increase in