ライフサイエンスコーパス: pyramidal

1 eology to compare the soma volume of layer V pyramidal and gigantopyramidal neurons in primary motor

2 (T-channels) in the firing activity of both pyramidal and inhibitory interneurons in the subiculum.

3 rate-and-fire model incorporating excitatory pyramidal and inhibitory interneurons indicated that tAC

4 These labeled neurons were identified as pyramidal based either by expression of SMI32 (a pyramid

5 rsistent sodium current (INa) density in CA1 pyramidal but not bipolar neurons.

6 at nicotine can differentially influence PFC pyramidal cell activity by nAChR modulation of layer II/

7 ng O-GlcNAcylation decreases spontaneous CA3 pyramidal cell activity under basal and hyperexcitable c

8 tion of adult neurogenesis led to atrophy of pyramidal cell apical dendrites in dorsal CA3 and to neu

9 ns, thereby contributing to the selection of pyramidal cell assemblies at the theta trough via disinh

10 n microscopic (EM) reconstruction of rat CA3 pyramidal cell axon terminals revealed approximately 1.7

11 Intriguingly, CA3 pyramidal cell backprojections that specifically target

12 (HCN1) channels are predominantly located in pyramidal cell dendrites within the cortex.

13 ts with schizophrenia show evidence of dlPFC pyramidal cell dendritic atrophy, likely reductions in c

14 When a single cortical pyramidal cell establishes a synapse onto a somatostatin

15 eurons in cortical processing (regulation of pyramidal cell excitatory input) and behavioral control

16 es a novel subclass of layer 5 glutamatergic pyramidal cell in mouse PFC (either sex).

17 sitive (PV(+)) neurons control the timing of pyramidal cell output in cortical neuron networks.

18 min-positive (PV(+)) neurons tightly control pyramidal cell output.

19 We demonstrate that, at connected layer 5 pyramidal cell pairs of developing mouse visual cortex,

20 rward inhibition of spike transfer at mf-CA3 pyramidal cell synapses.

21 In turn, pyramidal cell targets of SST(+) neurons showed an incre

22 er 3 (L3) via reciprocal connections between pyramidal cells (PCs) and parvalbumin (PV)-containing in

23 streams mediated by subsets of glutamatergic pyramidal cells (PCs) that receive diverse inputs and pr

24 lar recordings of CA3 neurons in vivo In CA3 pyramidal cells (PCs), mf-CA3 synaptic responses consist

25 from other DGCs as well as from CA3 and CA1 pyramidal cells after pilocarpine treatment, changes tha

26 synaptic inputs from SST(+) interneurons to pyramidal cells and corresponding chronic reductions in

27 d, as characterized by c-Fos upregulation in pyramidal cells and GABA interneurons coexpressing vasoa

28 g-term potentiation (LTP) in hippocampal CA1 pyramidal cells and hippocampal-dependent cognitive func

29 stant optogenetic stimulation targeting both pyramidal cells and inhibitory interneurons has recently

30 ed by a complex interplay between excitatory pyramidal cells and inhibitory interneurons.

31 Pyramidal cells and interneurons expressing parvalbumin

32 amp and juxtacellular configuration from CA3 pyramidal cells and interneurons.

33 in laser-microdissected DLPFC layer 3 and 5 pyramidal cells and layer 3 parvalbumin interneurons, an

34 ing mature dentate granule cells, area CA1-3 pyramidal cells and mossy cells.

35 xperimentally: interconnected populations of pyramidal cells and parvalbumin-positive inhibitory cell

36 itic spines, produced spine loss in cortical pyramidal cells and striatal hyperdopaminergia in mice.

37 the hippocampus in humans and atrophy of CA3 pyramidal cells and suppression of adult neurogenesis in

38 -term plasticity of excitatory inputs to CA3 pyramidal cells combines with robust feedforward inhibit

39 Although dorsal and ventral CA1 pyramidal cells exhibit markedly different gene expressi

40 cordings from morphologically identified CA3 pyramidal cells from control and complex IV-deficient mi

41 Thus, in rat motor cortex, pyramidal cells have long duration action potentials, wh

42 function in young normal mice, that old CA1 pyramidal cells have reduced excitability and increased

43 tic brain, whereas axons of interneurons and pyramidal cells in CA1 appear to sprout across the hippo

44 , in part, on neural circuitry that includes pyramidal cells in layer 3 (L3) and layer 5 (L5) of the

45 age clamp recordings were then made from CA1 pyramidal cells in normal aCSF.

46 between putative inhibitory interneurons and pyramidal cells in PFC and VIP.

47 e significantly lower in DLPFC layer 3 and 5 pyramidal cells in schizophrenia.

48 erlying increase in input resistance in deep pyramidal cells in temporal and prefrontal association n

49 Pools of L3 and L5 pyramidal cells in the DLPFC were individually captured

50 Lower dendritic spine density on layer 3 pyramidal cells in the dorsolateral prefrontal cortex (D

51 subunit of SK-type K(+) channels in ventral pyramidal cells is associated with an enhanced activatio

52 profiles exhibited by dorsal and ventral CA1 pyramidal cells is unclear.

53 tyric acid (GABA) interneurons disinhibiting pyramidal cells may be relevant to this hyperglutamaterg

54 hat molecular alterations in DLPFC L3 and L5 pyramidal cells might be characteristic of the disease p

55 bers (MFs) corelease glutamate and GABA onto pyramidal cells of CA3 during development, until the end

56 ransiently corelease glutamate and GABA onto pyramidal cells of CA3.

57 Tests in CA1 hippocampal pyramidal cells reveal that a slow AHP is reduced by blo

58 cortical microcircuit motif: populations of pyramidal cells that are densely interconnected with inh

59 nal models of developing and mature cortical pyramidal cells that express NaV1.2.

60 Rat pyramidal cells typically lack these channels, while the

61 rved was not restricted to synapses with CA3 pyramidal cells via large mossy-fibre boutons, but rathe

62 A slow afterhyperpolarization in pyramidal cells was reduced by a selective block of CaV1

63 a(N1768D/+) CA1, but not CA3 or neocortical, pyramidal cells was significantly reduced compared with

64 lls and the inhibitory drive they exerted on pyramidal cells were reduced in conditional mice lacking

65 In hippocampal pyramidal cells, a prominent elevation of CaV1 activity

66 n intricate circuit with granule cells, CA3c pyramidal cells, and local interneurons, but the influen

67 by postsynaptic AMPAR internalization in PFC pyramidal cells, and we observed a profound impairment i

68 lecular properties of excitatory synapses on pyramidal cells, comparatively little is known about exc

69 ry postsynaptic responses in the majority of pyramidal cells, spiny stellate cells, and interneurons

70 In DLPFC L3 and L5 pyramidal cells, transcriptome alterations were numerous

71 intrinsic firing and evoked NMDA currents in pyramidal cells, whereas D2 receptor function was unalte

72 and suppress long-range amygdala-projecting pyramidal cells.

73 mulus in both tsA-201 cells and male rat CA1 pyramidal cells.

74 tributes to persistent firing in neocortical pyramidal cells.

75 n (PV) expressing interneurons and layer 2/3 pyramidal cells.

76 e they prolong the duration of slow IPSCs in pyramidal cells.

77 n transfer at synapses, is weaker in ventral pyramidal cells.

78 tic integration along the dendritic arbor of pyramidal cells.

79 ation and maintenance of axonal terminals on pyramidal cells.

80 xert some form of divisive inhibition on the pyramidal cells.

81 nels are significantly higher in ventral CA1 pyramidal cells.

82 ork atoms are not in tetrahedral or trigonal-pyramidal coordination.

83 midal hippocampal and adjacent somatosensory pyramidal cortical neurons from male and female postnata

84 The square pyramidal Fe(0)(N2)(P4N2) complex catalyzes the conversi

85 Me3 (6), a compound with an unusual trigonal pyramidal geometry at a Nb(III) center, a Nb identical w

86 p, we quantified dendritic morphology in CA1 pyramidal hippocampal and adjacent somatosensory pyramid

87 Mature dendritic spines on CA1 pyramidal hippocampal neurons decreased 4 days after the

88 ic <c>, prismatic-II [Formula: see text] and pyramidal-II <c + a>, which are distinct from the ground

89 in- or somatostatin-positive interneurons on pyramidal layer 5 neurons in the medial prefrontal corte

90 statin-immunoreactive (-ir) cells in the CA1 pyramidal layer and robust morphological sprouting in th

91 midal based either by expression of SMI32 (a pyramidal marker), or by their shape and size, and lack

92 The device consists of an assembly of pyramidal microneedle structures integrated with Pt and

93 act structure in bilayer conical, as well as pyramidal, MN, as confirmed by scanning electron microsc

94 midal neurons, with deep (primarily layer V) pyramidal (n = 203) and superficial (primarily layer III

95 = 203) and superficial (primarily layer III) pyramidal (n = 233) neurons quantified for comparative p

96 we report a persistent elevation of layer V pyramidal neuron activity in the somatosensory cortex of

97 This enhanced pyramidal neuron activity was caused in part by increase

98 This resulted in the expected loss of pyramidal neuron AIS voltage-gated sodium and potassium

99 red dendritic spines within layer II and III pyramidal neuron dendrites in Brodmann area 46 dorsolate

100 to hippocampus, which target the very distal pyramidal neuron dendrites, provide an unusually strong

101 ession, disrupted rhythmicity, and increased pyramidal neuron excitability.

102 t not males, by increasing ERK signaling and pyramidal neuron excitability.

103 ug technology to specifically manipulate CA1 pyramidal neuron excitatory activity, electrophysiology,

104 ion of somatostatin-expressing cells reduced pyramidal neuron hyperactivity and reversed mechanical a

105 networks shifted their activity in favor of pyramidal neuron hyperactivity: somatostatin-expressing

106 hese channels counteracts the increased L2/3 pyramidal neuron hyperexcitability of Kcnq2-null neurons

107 tional development of the PFC by quantifying pyramidal neuron morphology and cognitive performance.

108 reated Scn1a (+/-) mice exhibited normalized pyramidal neuron sodium current density and reduced hipp

109 amotrigine treatment had no effect on either pyramidal neuron sodium current or hippocampal NaV1.6 le

110 However, do different pyramidal neuron subtypes also receive synaptic inputs t

111 Pyramidal neuron subtypes differ in intrinsic electrophy

112 Via realistic simulations of a CA1 pyramidal neuron, we further demonstrate that theta-driv

113 ngs implicate failure of adaptive control of pyramidal neuron-PV circuits as a pathophysiological mec

114 -expression is limited to a specific type of pyramidal neuron: CT.

115 chelators or Ca(2+) entry blockers to delay pyramidal neuronal death in both regions.

116 is sufficient to drive large changes in L2/3 pyramidal neuronal excitability even in the presence of

117 cally, electrically, and chemically detailed pyramidal neuronal models based on rat data.

118 nous neurodynamics of hand M1's subgroups of pyramidal neuronal pools in each of our subjects by appl

119 oss cortical areas, whereas many lower-layer pyramidal neurons (i.e., layers V-VI) favor connections

120 es along the apical dendrites of layer (L) 5 pyramidal neurons (PNs) in the mouse barrel cortex, and

121 er collateral (SC) inputs to hippocampal CA1 pyramidal neurons (PNs) produces a long-term enhancement

122 cal pathway onto mouse CA2 compared with CA1 pyramidal neurons (PNs).

123 ontribute to the degeneration of hippocampal pyramidal neurons after recurrent seizures and brain isc

124 Large (Betz cells), medium, and small pyramidal neurons all expressed Kv3.1b.

125 d abnormal mitochondrial distribution in the pyramidal neurons along with mitochondrial dysfunction i

126 olves the coordinated activity of excitatory pyramidal neurons and a specific population of inhibitor

127 a significant decrease in the firing of PrL pyramidal neurons and did not seem to propagate to other

128 ite arbors and spines in Cornu Ammonis (CA)1 pyramidal neurons and exacerbated behavioral defects.

129 iform discharges were recorded in layer V-VI pyramidal neurons and fast-spiking interneurons in slice

130 e cellular level by excitatory glutamatergic pyramidal neurons and inhibitory gamma-aminobutyric acid

131 KO mice revealed higher E/I ratio in layer 5 pyramidal neurons and lower general protein synthesis.

132 mice lacking ITSN1 suffer from dispersion of pyramidal neurons and malformation of the radial glial s

133 Inhibitory transmission in hippocampal pyramidal neurons and striatal dopamine receptor D1-expr

134 d a more uniform subcellular distribution in pyramidal neurons and supported inhibitory postsynaptic

135 napses between the entorhinal cortex and CA2 pyramidal neurons and the persistence of long-term socia

136 cipal site of communication between cortical pyramidal neurons and their targets, a key locus of acti

137 D and BD on synaptic connectivity of layer 5 pyramidal neurons and underscore the persistent impact o

138 inhibitory basket interneurons connected to pyramidal neurons and used cluster analysis to classify

139 did suppress aberrant spontaneous firing of pyramidal neurons and was associated with significantly

140 find that the effects of D2Rs on prefrontal pyramidal neurons are actually mediated by pathways asso

141 D3-receptor-expressing pyramidal neurons are electrophysiologically and anatomi

142 ng global cerebral ischemia, hippocampal CA1 pyramidal neurons are more vulnerable to injury than the

143 est that GIRK channels in dorsal hippocampal pyramidal neurons are necessary for normal learning invo

144 ated the consequences for E/I balance in PFC pyramidal neurons as well as cognition, social interacti

145 of Cre-recombinase in hippocampal CA1 region pyramidal neurons at postnatal day 0 (P0) or day 21 (P21

146 ilateral optogenetic stimulation of cortical pyramidal neurons both prevented and reduced pain-like b

147 y not essential for synapse formation in CA1 pyramidal neurons but shape synaptic properties and that

148 nduced in tandem in cultured rat neocortical pyramidal neurons by chronic manipulations of firing, bu

149 Inhibiting pyramidal neurons by optogenetically activating somatost

150 patial range of inhibitory input provided to pyramidal neurons by PV interneurons in layers 2/3, 4 an

151 in the LFP and the discharge of a subset of pyramidal neurons called "place cells" is spatially orga

152 Remarkably, upregulation of Dcc in mouse PFC pyramidal neurons causes vulnerability to stress-induced

153 ed saccade direction selectivity in putative pyramidal neurons due to nonspecific increases in activi

154 neurotransmission, increases onto cingulate pyramidal neurons during peri-pubertal development and t

155 ecording synaptic responses in dmPFC layer V pyramidal neurons elicited by repeated 5 Hz electrical s

156 In cortical pyramidal neurons EPSP suppression by preceding APs depe

157 ction potentials, while in the macaque, some pyramidal neurons exhibit short duration "thin" spikes.

158 Excitatory pyramidal neurons exhibited inter- and intralaminar hete

159 In rat motor cortex, SMI32-postive pyramidal neurons expressing Kv3.1b were very rare and w

160 ade dendritic spine loss at directly injured pyramidal neurons followed by retrograde presynaptic hyp

161 Here, using recordings from LIII pyramidal neurons from acute slices of mouse medial ento

162 ole-cell recording revealed that hippocampal pyramidal neurons from B6.Q54 and F1.Q54 animals exhibit

163 ls and of native synaptic NMDARs in cortical pyramidal neurons from mice of either sex increased in c

164 Pyramidal neurons from the hippocampus of Celsr3 knockou

165 oth their PPI and learning defects, cortical pyramidal neurons from Upf3b-null mice display deficient

166 round potassium current observed in cortical pyramidal neurons from wild type mice was conspicuously

167 w that one-third of the thick-tufted layer 5 pyramidal neurons have an axon originating from a dendri

168 postsynaptic current frequency, measured on pyramidal neurons in acute hippocampal slices at 270 DAT

169 ned the intrinsic excitability of PRL and IL pyramidal neurons in adolescent rats 24 h following a br

170 Using whole-cell recordings of layer V pyramidal neurons in an ex vivo brain slice preparation,

171 ion potential (AP) firing in Scn8a(N1768D/+) pyramidal neurons in brain slices revealed early afterde

172 Using dynamic clamp in hippocampal CA1 pyramidal neurons in brain slices, we showed that the ef

173 nded with reduced dendritic spine density on pyramidal neurons in layer 1 of the medial PFC.

174 ns between apical and basal dendrites of CA1 pyramidal neurons in mouse hippocampal slices.

175 By analyzing CA1 pyramidal neurons in mutant hippocampal slice cultures t

176 current clamp recording, we found that L2/3 pyramidal neurons in prefrontal cortex of fmr1(-/y) mous

177 xpression to identify Fos-expressing layer V pyramidal neurons in prelimbic cortex (PLC) of FosGFP-tr

178 cium imaging with single-spine resolution in pyramidal neurons in rat hippocampal slices from either

179 el current in nucleated patches from layer 5 pyramidal neurons in rat neocortex, in physiological ext

180 ity of long/thin dendritic spines of layer 5 pyramidal neurons in the adult PrL-C.

181 Here, we show that pyramidal neurons in the barrel cortex of BC1 knock out

182 med postsynaptic dendritic spines of layer 5 pyramidal neurons in the mouse motor cortex during devel

183 ged postsynaptic dendritic spines of layer V pyramidal neurons in the mouse primary motor cortex usin

184 ery on dendritic spine remodeling of layer 5 pyramidal neurons in the mouse primary visual cortex.

185 pses in the apical dendritic tuft of layer V pyramidal neurons in the mPFC.

186 icated GABAergic deficits onto layer (L) 2/3 pyramidal neurons in the pathogenesis of neocortical hyp

187 R activation can enhance the excitability of pyramidal neurons in the PFC.

188 ic and dendritic excitability of layer (L) 5 pyramidal neurons in the prefrontal cortex of the fmr1(-

189 Moreover, activation of pyramidal neurons in this cortical region was sufficient

190 It is not known if the other major types of pyramidal neurons in this layer also express this enzyme

191 ncy adaptation - split layer 5 barrel cortex pyramidal neurons into two clusters: one of adapting cel

192 ergic excitation of commissural layer 5 mPFC pyramidal neurons is abolished in neuropathic pain rats

193 ecrease in the intrinsic excitability of CA1 pyramidal neurons is believed to contribute to age-relat

194 , daily cocaine injections, t-LTP in layer V pyramidal neurons is induced at +30 ms, a normally ineff

195 a result of KCNQ2 dysfunction in neocortical pyramidal neurons is still unknown.

196 fferential vulnerabilities of CA1 versus CA3 pyramidal neurons is unclear.

197 Hippocampal pyramidal neurons lacking CDKL5 show decreased dendritic

198 se of the CA1 microcircuit, we find that CA1 pyramidal neurons lacking CDKL5 show hyperexcitability i

199 IRK2c) expressed individually in hippocampal pyramidal neurons lacking GIRK2.

200 the excitatory projections of glutamatergic pyramidal neurons located in layer 3, whose activity is

201 Here, in hippocampal pyramidal neurons of both primary cultures and slices, w

202 detected coexpression of DCC and miR-218 in pyramidal neurons of human and mouse PFC.

203 that AMPAR-mediated synaptic transmission in pyramidal neurons of prefrontal cortex (PFC) was diminis

204 ic plasticity between dorsal and ventral CA1 pyramidal neurons of rat hippocampus.

205 model with conditional disruption of ANK3 in pyramidal neurons of the adult forebrain (Ank-G cKO).

206 -seq to identify mistranslating mRNAs in CA1 pyramidal neurons of the FX mouse model (Fmr1(-/y)) hipp

207 Here we show that, in the pyramidal neurons of the hippocampal CA1 region in mice,

208 cial role for excitatory synapses connecting pyramidal neurons of the hippocampus and cortex with fas

209 In CA1 pyramidal neurons of the hippocampus two types of AMPARs

210 ies from dentate gyrus granule cells and CA1 pyramidal neurons of the hippocampus.

211 Dendritic spines on the principal pyramidal neurons of the orbitofrontal prefrontal cortex

212 cs in male Sprague Dawley rats to silence IL pyramidal neurons optically for 20 s immediately after u

213 In addition to L5 pyramidal neurons, L2/3 pyramidal neurons play an important role in prefrontal c

214 Deletion of HDAC2 in forebrain pyramidal neurons prevented the negative effects of anti

215 nhibitory and excitatory responses to ACh in pyramidal neurons represent complementary mechanisms gov

216 r-expression of either isoform in dorsal CA1 pyramidal neurons restored contextual fear learning in a

217 Recordings from layer II/III pyramidal neurons revealed action potential widening tha

218 lular integrity in the motor cortex, and CA1 pyramidal neurons show abnormalities predominantly withi

219 Patch clamp recordings from layer V pyramidal neurons showed that optogenetic stimulation no

220 orsomedial prefrontal cortex, with different pyramidal neurons signaling Go and No-Go action plans.

221 -cAMP/protein kinase A dopamine signaling in pyramidal neurons that in turn pathologically recruits l

222 tion of excitatory synaptic responses in CA2 pyramidal neurons that relied on N-methyl-d-aspartate re

223 the authors construct biophysical models of pyramidal neurons that reproduce observed plasticity gra

224 n phosphatase 1 regulatory subunit 1B(+) BLA pyramidal neurons to dopamine receptor 1(+) CeA neurons

225 petitive behaviors, while R-spondin 2(+) BLA pyramidal neurons to dopamine receptor 2(+) CeA neurons

226 cells regulate surround suppression to allow pyramidal neurons to optimally encode visual information

227 our models using the responses of layer 2/3 pyramidal neurons to simulated presynaptic input with di

228 s-of-function variant KCNQ2(I205V) into L2/3 pyramidal neurons using in utero electroporation also re

229 rons; VIP cells also indirectly excite these pyramidal neurons via parallel disinhibition.

230 Increased excitability of layer II/III pyramidal neurons was accompanied by consistent reductio

231 In drinking monkeys, evoked firing of OFC pyramidal neurons was reduced, whereas the amplitude and

232 jury, cholinergic modulation of layer 5 (L5) pyramidal neurons was severely impaired.

233 In macaque motor cortex, a large sample of pyramidal neurons were nearly all found to express Kv3.1

234 idea, EPSPs in both cortical and hippocampal pyramidal neurons were suppressed by preceding APs in an

235 Activation of PFC pyramidal neurons with a CaMKII-driven Gq-coupled design

236 ions (ADPs) in subcortically projecting (SC) pyramidal neurons within L5 of the PFC.

237 MDA EPSCs in mouse layer 5 prefrontal cortex pyramidal neurons without affecting AMPA EPSC currents.

238 prelimbic medial prefrontal cortex (PL-mPFC) pyramidal neurons, a phenomenon that correlates with the

239 omato-dendritic GIRK currents in Girk2 (-/-) pyramidal neurons, although GIRK2c achieved a more unifo

240 ii) BigLEN-mediated hyperpolarization of BLA pyramidal neurons, and (iii) feeding induced by DREADD-m

241 e, presented characteristics of non-adapting pyramidal neurons, and also had higher IPSC and EPSC fre

242 t required for scaling up in CA1 hippocampal pyramidal neurons, and found that the GluA2 subunit is b

243 lity and action potential properties of L2/3 pyramidal neurons, and identifies Nav1.6 as a new potent

244 ference in the immunoreactivity of Kv3.1b in pyramidal neurons, and this may be one of the factors ex

245 complexity of basal dendritic arbors of CA2 pyramidal neurons, but caused no alteration in the densi

246 inently expressed in hippocampal CA2 and CA3 pyramidal neurons, but little is known about its physiol

247 uced KCa2 channel currents in layer V IL-PFC pyramidal neurons, confirming functional downregulation

248 xpression of c-fos was increased in cortical pyramidal neurons, consistent with increased neuronal ac

249 ads to hyperexcitability of layer 2/3 (L2/3) pyramidal neurons, exhibiting an increased input resista

250 ls (ChCs) target the axon initial segment of pyramidal neurons, forming an array of boutons termed a

251 In addition to L5 pyramidal neurons, L2/3 pyramidal neurons play an import

252 Like neocortical pyramidal neurons, neurons in our model receive sensory

253 nterneurons and decreases the firing rate of pyramidal neurons, phenomena mimicked by exogenously app

254 previously described thin- and thick-tufted pyramidal neurons, respectively.

255 -term potentiation (sLTP) in hippocampal CA1 pyramidal neurons, revealing that the activation of thes

256 duces dendritic lengths of mPFC layer II/III pyramidal neurons, S-SDS increases arborization and spin

257 conditional Lis1 inactivation in excitatory pyramidal neurons, starting in juvenile mouse brain, wer

258 synaptic currents (mEPSCs) onto EC layer III pyramidal neurons, suggesting that these channels decrea

259 on, cholinergic stimulation excited putative pyramidal neurons, whereas the activity of putative inte

260 tic excitation and inhibition in hippocampal pyramidal neurons, which affects 'Hebbian' long-term syn

261 -cAMP/protein kinase A dopamine signaling in pyramidal neurons, which then pathologically recruits vo

262 tion is much higher in ACC than LPFC layer 3 pyramidal neurons, with a significantly higher frequency

263 y strong excitatory drive at the soma of CA2 pyramidal neurons, with EPSPs that are 5-6 times larger

264 xamine ion dynamics in spines in hippocampal pyramidal neurons.

265 mined using whole-cell recordings in layer V pyramidal neurons.

266 and postsynaptic transmission in rat PL-mPFC pyramidal neurons.

267 reased length and number of intersections of pyramidal neurons.

268 elp to explain the morphology of neocortical pyramidal neurons.

269 napses between the entorhinal cortex and CA2 pyramidal neurons.

270 ger optogenetic silencing of a subset of CA1 pyramidal neurons.

271 the function of these stores in hippocampal pyramidal neurons.

272 in mitochondria occurring in CA1 but not CA3 pyramidal neurons.

273 al circuitry of excitatory inputs to Layer 3 pyramidal neurons.

274 ibited egocentric spatial maps comparable to pyramidal neurons.

275 ramidal neurons than in superficial and deep pyramidal neurons.

276 ous excitatory synaptic inputs onto cortical pyramidal neurons.

277 CA3, INa,P was increased in both bipolar and pyramidal neurons.

278 terneuron inhibitory drive over layer II/III pyramidal neurons.

279 ological differences between rat and macaque pyramidal neurons.

280 pontaneous firings of somatosensory cortical pyramidal neurons.

281 nditional inactivation of Lis1 targeting CA1 pyramidal neurons.

282 excitability of neocortical layer 2/3 (L2/3) pyramidal neurons.

283 ed the slow AHP component (sAHP) in cortical pyramidal neurons.

284 ndritic Ca(2+) spikes in neocortical layer 5 pyramidal neurons.

285 that are 5-6 times larger than those in CA1 pyramidal neurons.

286 e synaptogenic immediate early gene NPTX2 by pyramidal neurons.

287 ACh exerts two opposing actions in cortical pyramidal neurons: transient inhibition and longer-lasti

288 t electrophysiological responses in cortical pyramidal neurons: transient inhibition driven by calciu

289 ility of prelimbic (PL) and infralimbic (IL) pyramidal neurons; a cocaine-induced increase in PL exci

290 usal, VIP cells rapidly and directly inhibit pyramidal neurons; VIP cells also indirectly excite thes

291 The pyramidal polymer microneedle in this study was fully re

292 tion-coupled intrinsic oscillator framework, pyramidal resonance interneuron network gamma (PRING), b

293 o 2 subtypes: semilunar (SL) and superficial pyramidal (SP) cells.

294 The IPSC difference between pyramidal subtypes was activity independent.

295 sponsive parkinsonism, cerebellar ataxia and pyramidal symptoms.

296 he major output cell type of the neocortex - pyramidal tract neurons (PTs) - send axonal projections

297 Pyramidal tract neurons (PTs) represent the major output

298 Pyramidal tract signs were described in 13 patients and

299 e first being the rostral decussation of the pyramidal tract, which instead of occurring at the spino

300 This reduced activation was strong in pyramidal tract-type neurons (-50%) but essentially abse