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1 midal, nonpyramidal multipolar, and inverted pyramidal neurons).
2 ic modeling of signaling pathways in the CA1 pyramidal neuron.
3 ger optogenetic silencing of a subset of CA1 pyramidal neurons.
4 the function of these stores in hippocampal pyramidal neurons.
5 in mitochondria occurring in CA1 but not CA3 pyramidal neurons.
6 al circuitry of excitatory inputs to Layer 3 pyramidal neurons.
7 ibited egocentric spatial maps comparable to pyramidal neurons.
8 ramidal neurons than in superficial and deep pyramidal neurons.
9 ous excitatory synaptic inputs onto cortical pyramidal neurons.
10 CA3, INa,P was increased in both bipolar and pyramidal neurons.
11 terneuron inhibitory drive over layer II/III pyramidal neurons.
12 ological differences between rat and macaque pyramidal neurons.
13 pontaneous firings of somatosensory cortical pyramidal neurons.
14 nditional inactivation of Lis1 targeting CA1 pyramidal neurons.
15 excitability of CA1 or basolateral amygdala pyramidal neurons.
16 F to dendrites and spines of hippocampal CA1 pyramidal neurons.
17 that IK1 channels do not mediate the sAHP in pyramidal neurons.
18 excitability of neocortical layer 2/3 (L2/3) pyramidal neurons.
19 lutamate release in hippocampal and cortical pyramidal neurons.
20 icity of inhibitory neurotransmission on CA1 pyramidal neurons.
21 despite impairing tonic inhibition onto L2/3 pyramidal neurons.
22 ut decreasing GABAergic inhibition onto L2/3 pyramidal neurons.
23 acilitated long-term depression (LTD) in PFC pyramidal neurons.
24 tional postsynaptic KAR complexes to the CA3 pyramidal neurons.
25 ing spine density in apical dendrites of CA1 pyramidal neurons.
26 urons and affecting inhibition onto cortical pyramidal neurons.
27 nnel, IK1 (KCNN4) as the sAHP channel in CA1 pyramidal neurons.
28 ty of evoked NMDAR currents on layer 2/3 PFC pyramidal neurons.
29 ed the slow AHP component (sAHP) in cortical pyramidal neurons.
30 ndritic Ca(2+) spikes in neocortical layer 5 pyramidal neurons.
31 that are 5-6 times larger than those in CA1 pyramidal neurons.
32 e synaptogenic immediate early gene NPTX2 by pyramidal neurons.
33 xamine ion dynamics in spines in hippocampal pyramidal neurons.
34 and postsynaptic transmission in rat PL-mPFC pyramidal neurons.
35 reased length and number of intersections of pyramidal neurons.
36 napses between the entorhinal cortex and CA2 pyramidal neurons.
37 mined using whole-cell recordings in layer V pyramidal neurons.
38 elp to explain the morphology of neocortical pyramidal neurons.
39 ) is synthesized by diacylglycerol lipase in pyramidal neurons; 2) travels retrogradely to nearby inh
42 prelimbic medial prefrontal cortex (PL-mPFC) pyramidal neurons, a phenomenon that correlates with the
43 ility of prelimbic (PL) and infralimbic (IL) pyramidal neurons; a cocaine-induced increase in PL exci
44 found that native SK channel distribution in pyramidal neurons, across the somatodendritic domain, de
45 prefrontal cortex (PFC) is necessary for the pyramidal neuron activity believed to enable working mem
46 we report a persistent elevation of layer V pyramidal neuron activity in the somatosensory cortex of
50 ontribute to the degeneration of hippocampal pyramidal neurons after recurrent seizures and brain isc
53 d abnormal mitochondrial distribution in the pyramidal neurons along with mitochondrial dysfunction i
54 omato-dendritic GIRK currents in Girk2 (-/-) pyramidal neurons, although GIRK2c achieved a more unifo
55 ny in the firing activity of hippocampal CA1 pyramidal neurons, an impaired homeostatic response to p
56 olves the coordinated activity of excitatory pyramidal neurons and a specific population of inhibitor
57 or dendritic recordings from rat hippocampal pyramidal neurons and demonstrate that a majority of NMD
58 a significant decrease in the firing of PrL pyramidal neurons and did not seem to propagate to other
59 inhibitory neurons, also surround mouse CA2 pyramidal neurons and envelop their excitatory synapses.
60 ite arbors and spines in Cornu Ammonis (CA)1 pyramidal neurons and exacerbated behavioral defects.
61 cells convey strong excitatory drive to CA3 pyramidal neurons and express presynaptic, PKA-dependent
62 iform discharges were recorded in layer V-VI pyramidal neurons and fast-spiking interneurons in slice
63 This effect was observed in both putative pyramidal neurons and in interneurons and was stronger i
64 e cellular level by excitatory glutamatergic pyramidal neurons and inhibitory gamma-aminobutyric acid
66 cess that controls excitatory inputs to both pyramidal neurons and interneurons can balance excitatio
67 KO mice revealed higher E/I ratio in layer 5 pyramidal neurons and lower general protein synthesis.
68 mice lacking ITSN1 suffer from dispersion of pyramidal neurons and malformation of the radial glial s
71 d a more uniform subcellular distribution in pyramidal neurons and supported inhibitory postsynaptic
72 napses between the entorhinal cortex and CA2 pyramidal neurons and the persistence of long-term socia
73 cipal site of communication between cortical pyramidal neurons and their targets, a key locus of acti
74 fic gating), tested using a network model of pyramidal neurons and three classes of interneurons with
75 D and BD on synaptic connectivity of layer 5 pyramidal neurons and underscore the persistent impact o
76 inhibitory basket interneurons connected to pyramidal neurons and used cluster analysis to classify
77 did suppress aberrant spontaneous firing of pyramidal neurons and was associated with significantly
78 ii) BigLEN-mediated hyperpolarization of BLA pyramidal neurons, and (iii) feeding induced by DREADD-m
79 e, presented characteristics of non-adapting pyramidal neurons, and also had higher IPSC and EPSC fre
80 t required for scaling up in CA1 hippocampal pyramidal neurons, and found that the GluA2 subunit is b
81 lity and action potential properties of L2/3 pyramidal neurons, and identifies Nav1.6 as a new potent
82 s from soma and dendrites of rat hippocampal pyramidal neurons, and measured spectral tuning before a
83 ference in the immunoreactivity of Kv3.1b in pyramidal neurons, and this may be one of the factors ex
84 numbers, dynamics, and morphology of layer 5 pyramidal neuron apical dendritic spines in the primary
85 find that the effects of D2Rs on prefrontal pyramidal neurons are actually mediated by pathways asso
87 nd firing rate, number of active hippocampal pyramidal neurons are critical for reliable representati
89 ng global cerebral ischemia, hippocampal CA1 pyramidal neurons are more vulnerable to injury than the
90 est that GIRK channels in dorsal hippocampal pyramidal neurons are necessary for normal learning invo
91 ated the consequences for E/I balance in PFC pyramidal neurons as well as cognition, social interacti
92 of Cre-recombinase in hippocampal CA1 region pyramidal neurons at postnatal day 0 (P0) or day 21 (P21
93 of dendritic measures differentiated typical pyramidal neurons between the Siberian tiger and the clo
94 -intrinsic neuronal excitability of cortical pyramidal neurons both in vitro and in vivo as shown by
96 ilateral optogenetic stimulation of cortical pyramidal neurons both prevented and reduced pain-like b
97 y not essential for synapse formation in CA1 pyramidal neurons but shape synaptic properties and that
98 complexity of basal dendritic arbors of CA2 pyramidal neurons, but caused no alteration in the densi
99 inently expressed in hippocampal CA2 and CA3 pyramidal neurons, but little is known about its physiol
100 nduced in tandem in cultured rat neocortical pyramidal neurons by chronic manipulations of firing, bu
102 patial range of inhibitory input provided to pyramidal neurons by PV interneurons in layers 2/3, 4 an
103 y promotes the excitation of hippocampal CA1 pyramidal neurons by suppressing feedforward inhibition.
104 in the LFP and the discharge of a subset of pyramidal neurons called "place cells" is spatially orga
105 Remarkably, upregulation of Dcc in mouse PFC pyramidal neurons causes vulnerability to stress-induced
106 uced KCa2 channel currents in layer V IL-PFC pyramidal neurons, confirming functional downregulation
107 xpression of c-fos was increased in cortical pyramidal neurons, consistent with increased neuronal ac
113 theta-frequency resonance properties of CA1 pyramidal neurons, deficits in synaptic transmission at
114 inputs from different pathways cluster on a pyramidal neuron dendrite, a pathway can be gated-on by
115 red dendritic spines within layer II and III pyramidal neuron dendrites in Brodmann area 46 dorsolate
116 to hippocampus, which target the very distal pyramidal neuron dendrites, provide an unusually strong
117 Baseline electrophysiologic function of CA1 pyramidal neurons does not differ between Ndufs4(KO) and
118 ed saccade direction selectivity in putative pyramidal neurons due to nonspecific increases in activi
119 neurotransmission, increases onto cingulate pyramidal neurons during peri-pubertal development and t
121 mTOR-dependent spinogenesis on CA1 and mPFC pyramidal neurons, effects that may support the memory-e
122 ecording synaptic responses in dmPFC layer V pyramidal neurons elicited by repeated 5 Hz electrical s
127 ug technology to specifically manipulate CA1 pyramidal neuron excitatory activity, electrophysiology,
128 ction potentials, while in the macaque, some pyramidal neurons exhibit short duration "thin" spikes.
130 ads to hyperexcitability of layer 2/3 (L2/3) pyramidal neurons, exhibiting an increased input resista
135 ade dendritic spine loss at directly injured pyramidal neurons followed by retrograde presynaptic hyp
136 ls (ChCs) target the axon initial segment of pyramidal neurons, forming an array of boutons termed a
137 dendritic arborization in isolated cortical pyramidal neurons from a mouse model of 16p11.2 duplicat
139 ole-cell recording revealed that hippocampal pyramidal neurons from B6.Q54 and F1.Q54 animals exhibit
141 ls and of native synaptic NMDARs in cortical pyramidal neurons from mice of either sex increased in c
144 oth their PPI and learning defects, cortical pyramidal neurons from Upf3b-null mice display deficient
145 round potassium current observed in cortical pyramidal neurons from wild type mice was conspicuously
146 w that one-third of the thick-tufted layer 5 pyramidal neurons have an axon originating from a dendri
147 ion of somatostatin-expressing cells reduced pyramidal neuron hyperactivity and reversed mechanical a
148 networks shifted their activity in favor of pyramidal neuron hyperactivity: somatostatin-expressing
149 hese channels counteracts the increased L2/3 pyramidal neuron hyperexcitability of Kcnq2-null neurons
150 oss cortical areas, whereas many lower-layer pyramidal neurons (i.e., layers V-VI) favor connections
151 re, CaMKv knockdown in mouse hippocampal CA1 pyramidal neurons impairs synaptic transmission and plas
152 s in mouse organotypic slices, and layer 2/3 pyramidal neuron in somatosensory cortex of living mice.
153 postsynaptic current frequency, measured on pyramidal neurons in acute hippocampal slices at 270 DAT
154 ned the intrinsic excitability of PRL and IL pyramidal neurons in adolescent rats 24 h following a br
155 of apical dendrites of layer 5 (L5) and L2/3 pyramidal neurons in adult V1 of Lynx1 knock-out (KO) mi
156 rareal excitation was stronger in PV than in pyramidal neurons in all layer 2/3 pathways, we observed
158 ion potential (AP) firing in Scn8a(N1768D/+) pyramidal neurons in brain slices revealed early afterde
160 B activity in single dendritic spines of CA1 pyramidal neurons in cultured murine hippocampal slices.
162 inputs to the dendritic spines of individual pyramidal neurons in layer 2/3 of ferret visual cortex.
165 aSyn were injected directly into the soma of pyramidal neurons in mouse neocortical brain slices duri
168 n "in mind," requires persistent activity of pyramidal neurons in prefrontal cortex (PFC) mediated by
169 current clamp recording, we found that L2/3 pyramidal neurons in prefrontal cortex of fmr1(-/y) mous
170 xpression to identify Fos-expressing layer V pyramidal neurons in prelimbic cortex (PLC) of FosGFP-tr
171 cium imaging with single-spine resolution in pyramidal neurons in rat hippocampal slices from either
172 el current in nucleated patches from layer 5 pyramidal neurons in rat neocortex, in physiological ext
173 tween hippocampal astrocytes and neighboring pyramidal neurons in rodent in vitro models of epilepsy.
176 was accompanied by a marked preservation of pyramidal neurons in the CA3 area and increased expressi
177 pping of the probabilistic spiking nature of pyramidal neurons in the cortex to the stochastic switch
178 ary visual cortex by measuring the tuning of pyramidal neurons in the joint orientation and spatial f
180 gnificantly increased basal spine density on pyramidal neurons in the medial prefrontal cortex (mPFC)
181 med postsynaptic dendritic spines of layer 5 pyramidal neurons in the mouse motor cortex during devel
182 ged postsynaptic dendritic spines of layer V pyramidal neurons in the mouse primary motor cortex usin
183 ery on dendritic spine remodeling of layer 5 pyramidal neurons in the mouse primary visual cortex.
185 icated GABAergic deficits onto layer (L) 2/3 pyramidal neurons in the pathogenesis of neocortical hyp
187 ic and dendritic excitability of layer (L) 5 pyramidal neurons in the prefrontal cortex of the fmr1(-
189 ains in a randomly chosen group of layer 2/3 pyramidal neurons in the somatosensory cortex triggered
190 -term potentiation of excitatory synapses on pyramidal neurons in the stratum radiatum rarely occurs
193 It is not known if the other major types of pyramidal neurons in this layer also express this enzyme
195 ncy adaptation - split layer 5 barrel cortex pyramidal neurons into two clusters: one of adapting cel
196 ergic excitation of commissural layer 5 mPFC pyramidal neurons is abolished in neuropathic pain rats
197 ecrease in the intrinsic excitability of CA1 pyramidal neurons is believed to contribute to age-relat
198 , daily cocaine injections, t-LTP in layer V pyramidal neurons is induced at +30 ms, a normally ineff
199 nd that postnatal development of PNNs on CA2 pyramidal neurons is modified by early-life enrichment,
200 uppress anxiety, while the inhibition of CA1 pyramidal neurons is required to suppress fear responses
205 se of the CA1 microcircuit, we find that CA1 pyramidal neurons lacking CDKL5 show hyperexcitability i
207 the excitatory projections of glutamatergic pyramidal neurons located in layer 3, whose activity is
208 tional development of the PFC by quantifying pyramidal neuron morphology and cognitive performance.
213 that AMPAR-mediated synaptic transmission in pyramidal neurons of prefrontal cortex (PFC) was diminis
215 model with conditional disruption of ANK3 in pyramidal neurons of the adult forebrain (Ank-G cKO).
216 -seq to identify mistranslating mRNAs in CA1 pyramidal neurons of the FX mouse model (Fmr1(-/y)) hipp
218 cial role for excitatory synapses connecting pyramidal neurons of the hippocampus and cortex with fas
222 e-pulse TMS on dendritic activity in layer 5 pyramidal neurons of the somatosensory cortex using an o
223 cs in male Sprague Dawley rats to silence IL pyramidal neurons optically for 20 s immediately after u
224 nterneurons and decreases the firing rate of pyramidal neurons, phenomena mimicked by exogenously app
225 In addition to L5 pyramidal neurons, L2/3 pyramidal neurons play an important role in prefrontal c
226 ophysiological assessments were made of mPFC pyramidal neurons (PN) from adult male HIV-1 transgenic
227 es along the apical dendrites of layer (L) 5 pyramidal neurons (PNs) in the mouse barrel cortex, and
228 er collateral (SC) inputs to hippocampal CA1 pyramidal neurons (PNs) produces a long-term enhancement
232 ngs implicate failure of adaptive control of pyramidal neuron-PV circuits as a pathophysiological mec
233 nhibitory and excitatory responses to ACh in pyramidal neurons represent complementary mechanisms gov
236 increases in parenchymal arteriole tone, the pyramidal neuron response was unaffected by blockers of
238 r-expression of either isoform in dorsal CA1 pyramidal neurons restored contextual fear learning in a
240 -term potentiation (sLTP) in hippocampal CA1 pyramidal neurons, revealing that the activation of thes
241 duces dendritic lengths of mPFC layer II/III pyramidal neurons, S-SDS increases arborization and spin
242 creases spine density and length in cortical pyramidal neurons, selective re-expression of FMRP in as
243 lular integrity in the motor cortex, and CA1 pyramidal neurons show abnormalities predominantly withi
245 ing to SWRs in rTg4510 mice, whilst putative pyramidal neurons showed increased temporal and phase lo
246 ll voltage-clamp recordings of mEPSCs in CA1 pyramidal neurons showed that E2 increases both mEPSC fr
248 nd locations along the dendritic tree of CA1 pyramidal neurons, showed diversity characterised by the
249 orsomedial prefrontal cortex, with different pyramidal neurons signaling Go and No-Go action plans.
250 reated Scn1a (+/-) mice exhibited normalized pyramidal neuron sodium current density and reduced hipp
251 amotrigine treatment had no effect on either pyramidal neuron sodium current or hippocampal NaV1.6 le
253 conditional Lis1 inactivation in excitatory pyramidal neurons, starting in juvenile mouse brain, wer
257 synaptic currents (mEPSCs) onto EC layer III pyramidal neurons, suggesting that these channels decrea
258 uts onto postsynaptic specializations of CA3 pyramidal neurons termed thorny excrescences and these s
260 -cAMP/protein kinase A dopamine signaling in pyramidal neurons that in turn pathologically recruits l
261 tion of excitatory synaptic responses in CA2 pyramidal neurons that relied on N-methyl-d-aspartate re
262 the authors construct biophysical models of pyramidal neurons that reproduce observed plasticity gra
264 n phosphatase 1 regulatory subunit 1B(+) BLA pyramidal neurons to dopamine receptor 1(+) CeA neurons
265 petitive behaviors, while R-spondin 2(+) BLA pyramidal neurons to dopamine receptor 2(+) CeA neurons
267 cells regulate surround suppression to allow pyramidal neurons to optimally encode visual information
268 our models using the responses of layer 2/3 pyramidal neurons to simulated presynaptic input with di
269 s in response reliability and selectivity of pyramidal neurons to visual stimuli, as confirmed by two
270 ACh exerts two opposing actions in cortical pyramidal neurons: transient inhibition and longer-lasti
271 t electrophysiological responses in cortical pyramidal neurons: transient inhibition driven by calciu
272 t electrophysiological responses in cortical pyramidal neurons: transient inhibition driven by calciu
273 s-of-function variant KCNQ2(I205V) into L2/3 pyramidal neurons using in utero electroporation also re
274 fluence synaptic currents in hippocampal CA1 pyramidal neurons using patch clamp recordings in acute
276 usal, VIP cells rapidly and directly inhibit pyramidal neurons; VIP cells also indirectly excite thes
278 In drinking monkeys, evoked firing of OFC pyramidal neurons was reduced, whereas the amplitude and
281 patch clamp recordings from infralimbic mPFC pyramidal neurons, we found that chronic stress selectiv
284 In macaque motor cortex, a large sample of pyramidal neurons were nearly all found to express Kv3.1
285 idea, EPSPs in both cortical and hippocampal pyramidal neurons were suppressed by preceding APs in an
286 e targeting proximal and distal dendrites of pyramidal neurons, where the defining feature of a beta
287 on, cholinergic stimulation excited putative pyramidal neurons, whereas the activity of putative inte
288 tic excitation and inhibition in hippocampal pyramidal neurons, which affects 'Hebbian' long-term syn
289 arization increased baseline firing rates of pyramidal neurons, which altered their susceptibility to
290 -cAMP/protein kinase A dopamine signaling in pyramidal neurons, which then pathologically recruits vo
292 a rigid, predominantly fast-firing subset of pyramidal neurons with low spatial specificity and limit
294 ore target touch; suppression of activity in pyramidal neurons with the ArchT silencing opsin resulte
295 tion is much higher in ACC than LPFC layer 3 pyramidal neurons, with a significantly higher frequency
296 y strong excitatory drive at the soma of CA2 pyramidal neurons, with EPSPs that are 5-6 times larger
297 ty (M2R-ir) was absent from the perikarya of pyramidal neurons, with the exception of the Golgi compl
299 ng this period, parvalbumin interneurons and pyramidal neurons within the same layers receive weaker
300 MDA EPSCs in mouse layer 5 prefrontal cortex pyramidal neurons without affecting AMPA EPSC currents.
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