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1 with a bimolecular rate constant similar to pyridostigmine.
3 nts with POTS underwent acute drug trials of pyridostigmine 30 mg orally and placebo, on separate mor
4 1 U/Kg i.v.), clonidine (0.25 mg orally), or pyridostigmine (60 mg orally) were measured in healthy y
5 nts, galantamine was far more effective than pyridostigmine, a peripherally acting AChE inhibitor, an
8 aried: few showed long-term improvement with pyridostigmine and some even deteriorated with treatment
9 ith a scale of advanced adverse effects from pyridostigmine bromide (chi2 for trend, P<.001), was gre
10 as performed, except all subjects were given pyridostigmine bromide (Mestinon; 30 mg orally) 1 hour b
11 ned exposure to a nerve gas prophylaxis drug pyridostigmine bromide (PB) and pesticides DEET and perm
13 holinesterase inhibitors (AChEis), including pyridostigmine bromide (PB), pesticides, and nerve agent
14 GWI was most strongly associated with using pyridostigmine bromide pills [odds ratio (OR) = 3.5; 95%
15 ive but worsens supine hypertension, whereas pyridostigmine can improve OH slightly but significantly
18 mice, the cholinergic ACh esterase inhibitor pyridostigmine increases ACh levels and bone mass appare
20 butamol and ephedrine alone or combined with pyridostigmine or 3,4-DAP is increasingly being reported
21 y reported that cholinergic stimulation with pyridostigmine (PY) induces anti-inflammatory cell recru
23 AH, and the potential therapeutic effects of pyridostigmine (PYR), an oral drug stimulating the paras
24 s have a reduced GH response to exercise, 2) pyridostigmine reverses this impaired response, and 3) d
27 ate was significantly lower at 2 hours after pyridostigmine than after placebo (100+/-16 versus 111+/
28 er study drug was significantly greater with pyridostigmine than placebo (-10.4+/-14.0 AU versus 0.6+
31 s exposure and advanced adverse effects from pyridostigmine were synergistic (Rothman S, 5.3; 95% CI,
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