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1 also increases the loss of vitamin B-6 as 4-pyridoxic acid.
4 antifying pyridoxal 5'-phosphate (PLP) and 4-pyridoxic acid (4PA) in serum by high-performance liquid
6 tive was to evaluate the renal handling of 4-pyridoxic acid and the effects of renal dysfunction on v
7 mong intakes included urinary excretion of 4-pyridoxic acid and total vitamin B-6, pyridoxal 5'-phosp
9 PA:PL can distinguish between increases in 4-pyridoxic acid concentrations due to increased dietary i
10 the creatinine clearance, indicating that 4-pyridoxic acid is at least partially eliminated by tubul
11 ition of plasma [pyridoxal phosphate (PLP) > pyridoxic acid (PA) > pyridoxal] differed from that of C
12 e vitamin B-6 biomarkers PLP, pyridoxal, and pyridoxic acid (PA) and the pyridoxic acid:(pyridoxal +
15 vitamin B-6 required to normalize urinary 4-pyridoxic acid, plasma pyridoxal-P, erythrocyte pyridoxa
17 , pyridoxal, and pyridoxic acid (PA) and the pyridoxic acid:(pyridoxal + PLP) ratio (PAr), a proposed
18 We have observed high concentrations of 4-pyridoxic acid, the major catabolite of vitamin B-6 meta
19 aline phosphatase activity on the ratio of 4-pyridoxic acid to pyridoxal (PA:PL) in plasma in 10 men
20 were found between vitamin B-6 intake and 4-pyridoxic acid, total vitamin B-6, plasma pyridoxal 5'-p
21 of the subjects had inadequate values for 4-pyridoxic acid, total vitamin B-6, plasma pyridoxal 5'-p
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