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1 activation in response to cold challenge or pyrogen.
2 ified after a long search for the endogenous pyrogen.
3 ator of host defenses and a major endogenous pyrogen.
4 mide fulfills the criteria for an endogenous pyrogen.
5 ibits the thermogenic response to endogenous pyrogens.
6 he febrile response to peripherally injected pyrogens.
7 hibitors suppress the fever induced by these pyrogens.
8 arities of signaling downstream of these two pyrogens.
9 and lyophilized preparation of Streptococcus pyrogenes.
10 e [LPS]) or endogenous (e.g., interleukin-1) pyrogen administration, while selective COX-2 inhibitors
12 popolysaccharide (LPS) is a potent exogenous pyrogen and produces anorexia via cytokine production.
13 erformance liquid chromatography (HPLC), and pyrogen and sterility assays, as well as determination o
15 s review, the discoveries made on endogenous pyrogens are revisited, with insights into the importanc
16 use, although it tested negative by standard pyrogen assays, it was associated with adverse events in
17 ignificantly reduced the febrile response to pyrogen, decreasing integrated tympanic membrane tempera
18 strate that IL-6 is a circulating endogenous pyrogen during LPS-induced fever, which acts in concert
19 duced by immune cells activated by exogenous pyrogens, e.g., lipopolysaccharides (LPS), released into
23 ed a phase I/II trial (IND#70627) of sterile pyrogen-free I-methylene blue to identify sentinel nodes
24 cted intraperitoneally at 1 microgram/mouse; pyrogen-free saline (PFS) was the vehicle and control so
25 ttle than in CD18(+) cattle by comparison to pyrogen-free saline (PFS)-inoculated control animals.
27 with either PGE2 (0, 10, 100, or 500 ng) in pyrogen-free saline with 0.5% ethanol (5 microl) or CCK-
28 (0.1, 0.4, and 1.0 microg) or vehicle alone (pyrogen-free saline, 0.2 microl) were administered durin
29 -sufficient rabbit knee joints with 10 mg of pyrogen-free urate crystals and analyzed IL-8 levels, le
30 The five treatments included: 1) distilled, pyrogen-free water; 2) 8.5% (w/v) SBHAN; 3) 4.3% (w/v) S
31 factor Junb and the gene Il1b, encoding the pyrogen IL-1beta, which macrophages express upon activat
33 n that the production of PGE2 in response to pyrogens is critically dependent on COX-2 expression.
34 ne whether this effect of IL-1beta, a potent pyrogen, is due to modification of body temperature.
35 polysaccharide (LPS), a well-known bacterial pyrogen, is recognized by several receptors, including t
39 cal was found to contain < or =4 +/- 1 EU/mL pyrogens (n = 66); all samples examined were sterile.
41 nce, as the principal endogenous circulating pyrogen responsible for activating CNS mechanisms in fev
42 ld less active than wild-type NOMV in rabbit pyrogen tests and in tumor necrosis factor alpha release
44 uced fevers to fevers produced by endogenous pyrogens, we studied the thermal responses of COX-1- and
46 Interleukin (IL)-1 is a potent endogenous pyrogen which causes fever when injected into a number o
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