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1 rophages, and these effects are prevented by pyrrolidinedithiocarbamate.
2 a and was blocked by the NF-kappaB inhibitor pyrrolidinedithiocarbamate.
3 teine, dimethyl- and tetramethylthiourea, or pyrrolidinedithiocarbamate abrogated the increase in mRN
4 Herein, we demonstrate that the antioxidant pyrrolidinedithiocarbamate activates cAMP-dependent prot
6 stimulation of ICAM-1 could be suppressed by pyrrolidinedithiocarbamate, an inhibitor of NF-kappaB ac
7 as signaling agents in HPV, the antioxidants pyrrolidinedithiocarbamate and ebselen and the Cu,Zn sup
10 ated HASMCs was inhibited by the antioxidant pyrrolidinedithiocarbamate and the serine protease inhib
13 chemical inhibitors such as lactacystin and pyrrolidinedithiocarbamate as well as with an I kappa B
15 ulation of NF-kappa B by the immunomodulator pyrrolidinedithiocarbamate enhanced the cytopathogenicit
16 NF-kappaB inhibitors Bay11-7082 and ammonium pyrrolidinedithiocarbamate inhibited an early postentry
17 inhibited by the commonly used antioxidants pyrrolidinedithiocarbamate (PDTC) and butylated hydroxya
20 We report here that the known antioxidant pyrrolidinedithiocarbamate (PDTC) leads to time and dose
21 kappaB (IkappaB)-alpha, or with low doses of pyrrolidinedithiocarbamate (PDTC) selectively inhibited
26 by the antioxidants N-acetyl-L-cysteine and pyrrolidinedithiocarbamate, suggesting the involvement o
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