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1 ts in motoneurons innervating muscles of the quadriceps.
2 ning, and increased muscle fiber loss in the quadriceps.
3 maximum (1RM) per exercise) emphasizing the quadriceps.
4 ex I defect, with 85% A3243G mutation in his quadriceps.
5 ith the oxidative capacity and volume of the quadriceps.
6 cerol for triglyceride glycerol synthesis in quadriceps.
7 exors and adductors and hamstrings more than quadriceps.
8 atrix genes than did those from the mdx(5cv) quadriceps.
9 igher in mdx(5cv) diaphragm than in mdx(5cv) quadriceps.
10 emities' muscles, with marked sparing of the quadriceps.
11 art, diaphragm, lung, fat, gastrocnemius and quadriceps.
15 eive surface coil was placed under the right quadriceps, allowing determination of intramuscular [PCr
17 excitation was produced by afferents of the quadriceps and deep peroneal muscle nerves (which discha
20 of wild-type (WT) and Ccr2(-/-) mice, and in quadriceps and diaphragm muscles of mdx(5cv) mice, a mou
22 glycerol ([U-(13)C]glycerol) was complete in quadriceps and gastrocnemius, but not soleus, within 2 h
23 rmance on the balance board (P < 0.001), and quadriceps and hamstring strength also increased signifi
24 ance was assessed using a balance board, and quadriceps and hamstring strength were measured by an is
27 s also attenuated atrophy in the nonoperated quadriceps and in the hamstring and adductor muscles of
29 t contraction and weak co-contraction of the quadriceps and its antagonists did not lead to any signi
30 was reduced during postural tasks involving quadriceps and soleus co-contraction (standing and late-
31 task, which also involved co-contraction of quadriceps and soleus, was greater than during matched v
32 ion, no tissue disease in the foot/ankle and quadriceps, and no evidence of viral persistence in foot
34 nd for girls, Cox regression revealed that a quadriceps angle of >or=20 degrees and an injury during
35 de various skeletal abnormalities, increased quadriceps angle, generalized ligamentous laxity, and fa
40 ng protein-delta, in 3-month-old A/J(dys-/-) quadriceps (before marked histopathology is evident), in
41 on of [1,2-(1)(3)C]leucine with intermittent quadriceps biopsies for determination of MPS and anaboli
43 ng freshly isolated CD31(+) cells from human quadriceps biopsies, we found that the down-regulation o
44 torque in WT (39 +/- 6%) and mdx (76 +/- 8%) quadriceps, but significant changes in NMJ morphology, n
47 sessed at a given (weaker than 30%) level of quadriceps contraction was reduced during postural tasks
48 ely matching target forces during submaximal quadriceps contractions and in the same tasks, OA patien
49 mass (dual-energy X-ray absorptiometry) and quadriceps cross-sectional area (CSA; computed tomograph
53 owing 12 wk of combination therapy in heart, quadriceps, diaphragm, and soleus, compared with vector
54 ralis obliquus, the iliotibial band, and the quadriceps during strenuous activity have yet to be meas
58 s were observed and accompanied by sustained quadriceps electromyographic activity often lasting > 2s
59 prepared with arterial and venous cannulae, quadriceps EMG electrodes and EEG electrodes in the late
61 y males who performed repeated constant-load quadriceps exercise of a moderate intensity in a whole-b
63 for interventions such as aerobic exercise, quadriceps exercises, footwear modification, education,
64 moter E-box mutation decreased expression in quadriceps, extensor digitorum longus, and soleus approx
65 ficant decrease in distal tongue, but not in quadriceps, extensor digitorum longus, soleus, or ventri
66 ed during voluntary tonic contraction of the quadriceps, falling to zero at around one-third of maxim
67 The purpose of this study was to compare quadriceps fatigability in patients with varying severit
69 ion during the control test had the greatest quadriceps fatigue attenuation with hyperoxia (r(2) = 0.
77 /- 6% control, P < 0.05), the attenuation of quadriceps fatigue was similar between the sexes (36 +/-
84 a hip pain associated with abnormalities in quadriceps femoris muscle and ipsilaterally reduced dist
89 sessed via changes in pre- vs. post-exercise quadriceps force in response to supramaximal magnetic fe
92 necrosis was also observed in diaphragms and quadriceps from mdx:mIgf+/+ mice when compared with age-
93 and 5 +/- 2 nmol/micromol triglyceride/h for quadriceps, gastrocnemius, and soleus muscle, respective
94 4-fold, caused a reduction in weight of the quadriceps, gastrocnemius, soleus, and even the heart it
95 eficits in the sensorimotor functions of the quadriceps have been identified and proposed as possible
98 ng in COPD, and low frequency fatigue of the quadriceps is an infrequent feature of incremental walki
99 le young p110beta-mKO mice possessed a lower quadriceps mass and exhibited less strength than control
100 3; P = 0.038); the same pattern was true for quadriceps maximal voluntary force (30.8 +/- 10.4 and 36
104 esulted in a decrease in dendritic length of quadriceps motoneurons in untreated animals, and this de
105 erone treatment, the activation of remaining quadriceps motoneurons was assessed using peripheral ner
106 d in decreased dendritic length in remaining quadriceps motoneurons, and this atrophy was attenuated
108 ronymous recurrent inhibition from soleus to quadriceps motor neurones is under at least two types of
110 n was a heteronymous recurrent inhibition of quadriceps motor neurons mediated by Renshaw cells which
113 MRS) transmit-receive surface coil under the quadriceps muscle allowed estimation of [PCr]; .V(O2) wa
114 -receive surface coil placed under the right quadriceps muscle allowed estimation of intramuscular [P
117 patients receiving placebo exhibited greater quadriceps muscle atrophy, with a -14.3 +/- 3.6% change
118 d increase in UCP3 mRNA levels occurs in rat quadriceps muscle between 12 and 24 h of food removal.
120 see text]o2max) and microcirculation loss on quadriceps muscle biopsy (in CD31(+) immunofluorescence
121 ial occlusion pressures; cardiac output; and quadriceps muscle blood flow and oxygen tension (PMo2).
122 ts in significant contractile fatigue of the quadriceps muscle but not of the diaphragm in healthy, r
124 ed significantly improved motor performance, quadriceps muscle contractility, and sciatic nerve condu
126 onymous recurrent inhibition during isolated quadriceps muscle contraction, but to a much lesser exte
128 easured by dual-energy x-ray absorptiometry, quadriceps muscle cross-sectional area measured by magne
129 a phosphorus magnetic resonance spectroscopy quadriceps muscle exercise-recovery study before and aft
130 y muscles (IRL) exacerbated exercise-induced quadriceps muscle fatigue (Q(tw) = -12 +/- 8% IRL-CTRL v
132 en fluorescent protein (EGFP) in transfected quadriceps muscle fibers in living mice subjected to con
135 ation (NMES), applied to the surgical limb's quadriceps muscle for the first 6 weeks following surger
137 he IC/MS assay was used to quantitate OAs in quadriceps muscle from sedentary mice compared to fatigu
139 describe the biomechanical properties of the quadriceps muscle in ICU survivors 12 months after ICU d
140 kinase activation) and GLUT4-GFP-transfected quadriceps muscle in living, anesthetized mice either mu
144 n, exercise training significantly increased quadriceps muscle mitochondrial capacity by 24% and VO2
145 tudy was to determine whether fatigue of the quadriceps muscle occurs after high intensity cycle exer
146 In conclusion, contractile fatigue of the quadriceps muscle occurs after high intensity cycle exer
147 Muscle biopsy samples were obtained from the quadriceps muscle of both the control and exercised legs
148 r 2 kinase than were observed in Mtm1 p.R69C quadriceps muscle or in muscles from wild-type littermat
149 injection of pMCKhLPL into the peritoneum or quadriceps muscle results in plasma triglyceride reducti
152 aximal exercise performance, respiratory and quadriceps muscle strength and endurance and quality-of-
154 e measures included demographic information, quadriceps muscle strength, and QA using a burst-superim
156 idation in myocytes cultured from human male quadriceps muscle taken from subjects with varied BMI, f
158 s, CP-640186 lowered hepatic, soleus muscle, quadriceps muscle, and cardiac muscle malonyl-CoA with E
159 onths of age, there was gross atrophy of the quadriceps muscle, and other hindlimb muscles were varia
160 and ear), heart (heart base and ventricle), quadriceps muscle, and the tibiotarsal joint at 1, 2, 4,
162 ne of B. burgdorferi in the ear, heart base, quadriceps muscle, skin, and tibiotarsal joint tissue of
166 y (wild-type, WT) and dystrophic (mdx) mouse quadriceps muscles and evaluated transcript levels of th
170 ulate in obesity, we performed lipidomics on quadriceps muscles from obese mice with impaired glucose
171 be more severely affected by weakness of the quadriceps muscles in individuals with knee OA who have
172 wed many lipid droplets within the psoas and quadriceps muscles of dysferlin-deficient A/J(dys-/-) mi
174 quadriceps muscles, and WSCV for gluteal and quadriceps muscles was 2.2% and 3.6%, respectively.
175 rmine the kinetics of IL-6 secretion, intact quadriceps muscles were transfected with enhanced green
176 6 (0.82-0.99), respectively, for gluteal and quadriceps muscles, and WSCV for gluteal and quadriceps
179 ngs in permitting a large involvement of the quadriceps musculature in closed chain lower limb extens
180 ificant increases in body mass, muscle mass, quadriceps myofiber size, and survival, but other measur
184 3p/5p (miR-542-3p/5p) were quantified in the quadriceps of patients with chronic obstructive pulmonar
185 icroRNAs (miRNAs) that were increased in the quadriceps of patients with muscle wasting and to determ
187 rpose of this study was to determine whether quadriceps or diaphragmatic fatigue or both occur after
188 with a weaker relationship between strength (quadriceps or hamstring) and physical functioning (chair
190 f oxidative capacity and muscle volume - the quadriceps oxidative capacity - was 36 % of the adult va
193 y fitness (CRF, VO(2peak)), muscle strength (quadriceps peak torque), body composition (%fat), and PA
194 ance spectroscopy to measure whole-body VO2, quadriceps phosphate metabolism and pH during continuous
195 in both solei (SOL(R) and SOL(L)) and right quadriceps (QD(R)), and stimulating cuffs on both poster
196 e (OR, 12.4; 95% CI, 4.53-33.77; P < 0.001), quadriceps (rectus femoris) cross-sectional area (Qcsa)
197 subjects with the slope indicating that the quadriceps represented 36 % of the VO(2) increase during
198 : muscle mass using bioelectrical impedance, quadriceps, respiratory muscle and handgrip strength, an
199 After exercise, a microarray analysis in quadriceps revealed ATF3 affects genes modulating chemot
202 onfirmed the homogenous pattern of selective quadriceps sparing but noted additional features like as
204 e immunostained muscle biopsy specimens from quadriceps-sparing autosomal-recessive and autosomal-dom
205 nergy balance but had significantly impaired quadriceps strength (-21.9 +/- 6.0 Newton-meters [Nm]/kg
208 chniques) with appendicular lean mass (ALM), quadriceps strength (QS), and bone mineral density (BMD)
212 indicated that subjects with lower levels of quadriceps strength and higher levels of QAF had lower l
216 number of muscle deficits (low muscle mass, quadriceps strength and physical performance) and 6-minu
221 ed by a burst superimposition test to assess quadriceps strength and volitional activation 3 and 12 m
222 aseline QA was significantly associated with quadriceps strength at baseline (rho = 0.30, P < 0.01) a
224 ise therapy, the level of QA did not predict quadriceps strength at the 2-month followup (beta = -0.0
232 smI polymorphism was associated with greater quadriceps strength in patients-37.0 +/- 13.3, 33.8 +/-
233 e deletion allele is associated with greater quadriceps strength independent of confounding factors.
235 s no significant between-group difference in quadriceps strength or health-related quality of life.
237 We report for the first time that greater quadriceps strength protected against cartilage loss at
239 d to determine whether baseline QA predicted quadriceps strength scores at the 2-month followup.
241 e C allele of the FokI polymorphism had less quadriceps strength than did those with > or =1 T allele
248 Osteoarthritis Index, had isometric tests of quadriceps strength, and underwent weight-bearing radiog
249 ty, malalignment, proprioceptive inaccuracy, quadriceps strength, hamstring strength), activity level
252 arthritis (OA) to determine the influence of quadriceps strength, medial knee laxity, limb alignment,
253 s predicted knee motion after accounting for quadriceps strength, underscoring the importance of addr
262 allele was associated with greater isometric quadriceps strength; mean (SD) 31.4 (10.8) kg for insert
265 t 75 +/- 7 W for 21.9 +/- 1.8 min during the quadriceps studies, and for 17.7 +/- 1.9 min during the
267 asuring unpotentiated and potentiated twitch quadriceps tensions before and 30 minutes after incremen
269 (Polistes canadensis) and an ant (Dinoponera quadriceps) that live in simple eusocial societies.
271 alized (31)P spectra were collected from the quadriceps throughout using a dual-tuned ((1)H and (31)P
272 le nerves, listed in order of effectiveness: quadriceps, tibialis posterior (throughout L6 and L7), g
273 d potentials evoked by group II afferents of quadriceps, tibialis posterior, and flexor digitorum lon
275 igue was assessed via changes in potentiated quadriceps twitch force (DeltaQ(tw,pot)) as measured pre
276 igue was assessed via changes in potentiated quadriceps twitch force (DeltaQ(tw,pot)) from pre- to po
279 ed with a figure-of-eight magnetic coil, and quadriceps twitch force (TwQ) was measured before and at
281 .05).Maximum voluntary force and potentiated quadriceps twitch force were decreased below baseline af
282 pre- to post-exercise changes in potentiated quadriceps twitch torque (DeltaQTsingle ) evoked by elec
287 In EL subjects, our results show that (i) quadriceps volume, maximum voluntary contraction isometr
288 itioning of the stretch reflex of biceps and quadriceps was abnormal in both hemizygous males and car
292 ing an established cohort, we tested whether quadriceps weakness in patients with COPD is influenced
295 symptoms were excluded, the relationship of quadriceps weakness to OA was attenuated, with only the
299 nervating the vastus lateralis muscle of the quadriceps were labeled with cholera toxin-conjugated ho
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