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1 hesis of NAD (+) and clearing the neurotoxin quinolinate.
2 he phthalate catabolic pathway cometabolizes quinolinate.
4 e enzymatic product subsequently cyclizes to quinolinate, an intermediate in the biosynthesis of nico
6 elis complex is approximated by PRPP and the quinolinate analogue phthalate bound to the active site.
7 formation of nicotinate mononucleotide from quinolinate and 5-phosphoribosyl-1-pyrophosphate (PRPP).
9 ntrastriatal injection of either malonate or quinolinate and systemic administration of 3-NP resulted
15 cted outside the needle track 10 hours after quinolinate injection and, on average, 20% of neurons ex
16 ced hippocampal cell death, are resistant to quinolinate, malonate, and 3-nitropropionic acid (3-NP).
18 of two metabolic pathways: the tryptophan to quinolinate pathway and the bacterial 2-nitrobenzoic aci
22 e biosynthetic pathway including NtPMT1a and quinolinate phosphoribosyltransferase (NtQPT2), and lowe
26 tructural similarities between phthalate and quinolinate: phthalate is a competitive inhibitor of thi
27 When supplied as the sole NAD precursor, quinolinate promoted B. bronchiseptica growth, and the a
36 study concludes that all currently annotated quinolinate synthases harbor a [4Fe-4S] cluster, that th
37 the biosynthetic pathway from tryptophan to quinolinate, the universal de novo precursor to the pyri
38 ratios of 3-sialyllactose to xanthosine and quinolinate to X-16397 and our previously reported urina
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