戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                                              rCBF changes were assessed with a three-dimensional voxe
2                                              rCBF changes were evaluated with statistical parametric
3                                              rCBF data were recorded with a flow-sensitive alternatin
4                                              rCBF decreases in limbic/paralimbic areas, temporal and
5                                              rCBF was measured in 38 control subjects, 29 MCI patient
6 i) declined from 6.95+/-0.04 to 6.60+/-0.05, rCBF declined from 48+/-7 to 10+/-3 ml/100 g/min, and NA
7  to conditionally express recombinant CBF-A (rCBF-A) activate the transcription of FSP1 and undergo E
8 e scanning interval, most of the accelerated rCBF changes seen in the subsequently impaired group occ
9                                 In addition, rCBF measurement indicated that alcohol-fed rats had les
10 lyses revealed regional abnormalities in all rCBF and rCMR measurements that were consistent in locat
11     The main effects in healthy subjects, an rCBF increase in subgenual cingulate Brodmann's area 25
12 epression Rating Scale were administered and rCBF was measured by means of single photon emission com
13 nverse relationship between 11C-PIB BPND and rCBF MR imaging in the voxel-based analysis that was abs
14 emory experiment, both FCS-rCBF coupling and rCBF/FCS ratio were modulated by task load in the ECN an
15  positive association of between craving and rCBF in the left orbitofrontal cortex.
16     Finally, task-induced changes of FCS and rCBF in the lateral-parietal lobe positively correlated
17 n functional connectivity strength (FCS) and rCBF during resting and an N-back working-memory task.
18 llected at each of these glycemic levels and rCBF measurements within the previously described networ
19 s between selective motor manifestations and rCBF in specific regions suggest possible regional selec
20 d for in-depth evaluation of amyloid PET and rCBF data.
21 ed with significant emotional reactivity and rCBF reductions in the ventromedial prefrontal cortex, s
22 Associations between extraversion scores and rCBF in each olfactory stimulus condition were assessed
23  to movement were relatively unaffected, and rCBF did not change in cerebellum or visual cortex.
24 cipital perfusion deficits but with anterior rCBF deficits in a pattern often described in the litera
25                                  We assessed rCBF during baseline, rectal distention, and anticipatio
26 en-group differences in correlations between rCBF and response time.
27 ent in the frontal and temporal regions, but rCBF changes in men with ADHD were more widespread and p
28  in regional synaptic activity as indexed by rCBF.
29  cortex, and a larger increase in cerebellar rCBF.
30 was calculated as a percentage of cerebellar rCBF, analysis of covariance found decreases in HD cauda
31                  For each hormone condition, rCBF was measured with [15O]H2O PET, and BOLD signal was
32                 In this region, a consistent rCBF response to stimulation was observed across subject
33                                  Conversely, rCBF in the cerebellum increased.
34  the Gambling Task, but orbitofrontal cortex rCBF did not.
35 ated with lower resting orbitofrontal cortex rCBF in cocaine-dependent subjects.
36 cingulate and dorsolateral prefrontal cortex rCBF is significantly related to decision making, as ass
37 late and left dorsolateral prefrontal cortex rCBF significantly correlated with performance on the Ga
38          Left dorsolateral prefrontal cortex rCBF was lower in the cocaine-dependent subjects than in
39                                     Cortical rCBF was extracted using a 3-dimensional stereotactic su
40           These data confirm limbic cortical rCBF changes associated with effective antidepressant tr
41                                    Decreased rCBF to the frontotemporal region characterized the cere
42                        Amphetamine decreased rCBF to motor cortex, visual cortex, fusiform gyrus, pos
43 CBF in the left frontal region and decreased rCBF in the left amygdala.
44 BF bilaterally in the thalamus and decreased rCBF in the left occipital lobe, right cerebellum, and t
45 teral thalami, right midbrain, and decreased rCBF in the right premotor cortex (P < 0.05, corrected).
46 etween improvement of rigidity and decreased rCBF in the SMA (r(s) = -0.4, P < 0.02) and between impr
47            MCI and AD patients had decreased rCBF in the posterior cingulate gyrus (P = .01) with ext
48                    AD patients had decreased rCBF relative to that in control subjects and MCI patien
49  postural reflexes correlated with decreased rCBF in the PPN (r(s) = -0.38, P < 0.03).
50 icance, but a trend for a pronounced delayed rCBF rise was seen for surface seizures (p = 0.08).
51 r model analyses were performed to determine rCBF differences between OA and control groups, rCBF dif
52 ups of children with significantly different rCBF behavior were identified.
53 ndritic arborization, we complimented direct rCBF comparisons with connectivity analyses to determine
54 hip was connection-distance dependent; i.e., rCBF correlated stronger with long-range hubs than short
55 pinal neurons in RVLM to reflexively elevate rCBF and slow the EEG as part of the oxygen-conserving (
56 s relay signals from the MCVA, which elevate rCBF in response to hypoxia, and (3) the SVA is a functi
57 jection of NaCN (300 pmol in 20 nl) elevated rCBF (17 +/- 5 %) and synchronized the EEG from RVLM, bu
58 rats electrical stimulation of RVLM elevated rCBF (laser-Doppler flowmetry) by 31 +/- 6 %, reduced ce
59 rs, ultra-high-risk subjects showed elevated rCBF in the hippocampus, basal ganglia, and midbrain.
60 nchronizes the EEG and coordinately elevates rCBF; (b) the responses are mediated by excitation of ne
61 ly restricted region of subthalamus elevates rCBF, (2) these neurons relay signals from the MCVA, whi
62 he hypothesis that whisker movement elicited rCBF changes are input frequency dependent and are most
63 wise, whole-brain analysis revealed enhanced rCBF only in the left posterior hippocampus (pHp) in the
64  basal forebrain failed to modify SVA-evoked rCBF increase.
65 e carpometacarpal (CMC) joint and to examine rCBF variability across sessions.
66                         The authors examined rCBF in 17 healthy volunteers and 18 schizophrenia patie
67                     The PTSD group exhibited rCBF decreases in medial frontal gyrus in the traumatic
68                          Only MCVs exhibited rCBF increases in the left amygdala.
69 ring the working-memory experiment, both FCS-rCBF coupling and rCBF/FCS ratio were modulated by task
70 ative cerebral blood volume (rCBV) and flow (rCBF) maps were acquired before treatment and after 1 an
71 nd measures of regional cerebral blood flow (rCBF) (a marker of neuronal activity) to describe the fu
72  elevations of regional cerebral blood flow (rCBF) and synchronization of the electroencephalogram (E
73                Regional cerebral blood flow (rCBF) and volume (rCBV) were measured with gadolinium-ba
74          Using regional cerebral blood flow (rCBF) as a marker of basal neuronal activity, this study
75 erformed using regional cerebral blood flow (rCBF) as a marker of neuronal activity.
76 s, we measured regional cerebral blood flow (rCBF) as a marker of synaptic activity at rest and durin
77 ; P<0.01), and elevates cerebral blood flow (rCBF) by 18+/-5% (P<0.05).
78 dependent from regional cerebral blood flow (rCBF) changes during moderate focal cerebral ischemia.
79 f longitudinal regional cerebral blood flow (rCBF) changes in the years preceding measurement of amyl
80 was to compare regional cerebral blood flow (rCBF) changes related to working memory in adults with a
81 n to determine regional cerebral blood flow (rCBF) changes representing ongoing pain experienced by p
82            The regional cerebral blood flow (rCBF) changes that occur during cortical spreading depre
83 ps resulted in regional cerebral blood flow (rCBF) decreases in medial orbitofrontal cortex Brodmann'
84 ith concurrent regional cerebral blood flow (rCBF) determination before and after administration of b
85                Regional cerebral blood flow (rCBF) determinations by positron emission tomography and
86 ygenation, and regional cerebral blood flow (rCBF) did not reach significance, but a trend for a pron
87  if changes in regional cerebral blood flow (rCBF) during hypoglycemia relative to euglycemia are sim
88  resting state regional cerebral blood flow (rCBF) during normal aging and investigated its influence
89 thors measured regional cerebral blood flow (rCBF) during performance of a task that required unmedic
90  PET to assess regional cerebral blood flow (rCBF) during rest and tested for between-genotype differ
91 ant changes in regional cerebral blood flow (rCBF) during states of cardiovascular arousal common to
92 hange in tumor relative cerebral blood flow (rCBF) from baseline and area under the plasma concentrat
93 sed to examine regional cerebral blood flow (rCBF) in 30 patients with chronic schizophrenia and 30 n
94 ges in resting regional cerebral blood flow (rCBF) in 32 healthy men.
95 easure resting regional cerebral blood flow (rCBF) in 52 individuals at ultra-high risk for psychosis
96 H treatment on regional cerebral blood flow (rCBF) in ADHD patients.
97 gnosis on glutamate and cerebral blood flow (rCBF) in adults with SZ and healthy controls.
98 ne craving and regional cerebral blood flow (rCBF) in distributed sites.
99 l volumes, and regional cerebral blood flow (rCBF) in healthy controls (HC) (n = 24), patients diagno
100  DBS increases regional cerebral blood flow (rCBF) in immediate downstream targets but does not revea
101 normalities of regional cerebral blood flow (rCBF) in major depression.
102 SPECT to study regional cerebral blood flow (rCBF) in patients with HD during rest and maze testing.
103 ET) to measure regional cerebral blood flow (rCBF) in sighted and congenitally blind subjects perform
104 ect of Alos on regional cerebral blood flow (rCBF) in the absence and presence of rectal or sigmoid s
105 lution, and of regional cerebral blood flow (rCBF) in the brain of transgenic APP23 mice.
106  covaried with regional cerebral blood flow (rCBF) in the dorsal medial prefrontal cortex, rostral an
107 owed increased regional cerebral blood flow (rCBF) in the vmPFC on both versions of the IGT compared
108 frontal cortex regional cerebral blood flow (rCBF) is lower in cocaine-dependent subjects than in non
109 cerebral blood volume (rCBV) and blood flow (rCBF) maps were acquired before chemoradiotherapy and at
110  to placebo on regional cerebral blood flow (rCBF) measured by SPECT in healthy volunteers to charact
111 lectrode and changes in cerebral blood flow (rCBF) measured with a laser Doppler probe placed over th
112 fy D3-mediated regional cerebral blood flow (rCBF) responses in living primates.
113  identified on regional cerebral blood flow (rCBF) SPECT scans of adolescent children and young adult
114             In regional cerebral blood flow (rCBF) studies with isoflurane and sevoflurane, there is
115 less change in regional cerebral blood flow (rCBF) to the anterior cingulate and supplementary motor
116 ess changes in regional cerebral blood flow (rCBF) upon awakening from stage 2 sleep.
117    We measured regional cerebral blood flow (rCBF) using pseudo-continuous arterial spin labelling.
118                Regional cerebral blood flow (rCBF) was assessed by using the bolus (15)O-labeled wate
119                Regional cerebral blood flow (rCBF) was measured within and outside the perfusion defi
120                Regional cerebral blood flow (rCBF) was monitored by Laser-Doppler flowmetry.
121                Regional cerebral blood flow (rCBF) was monitored through a Laser-Doppler flow probe a
122 e experiments, regional cerebral blood flow (rCBF) was recorded by laser Doppler flowmetry.
123 phy (SPECT) of regional cerebral blood flow (rCBF) was used to compare the central nervous system res
124  in normalized regional cerebral blood flow (rCBF) were assessed while participants repetitively wrot
125 traversion and regional cerebral blood flow (rCBF) while participants were exposed to olfactory stimu
126 ing to measure regional cerebral blood flow (rCBF) while they performed kinematically matched sequenc
127  activation of regional cerebral blood flow (rCBF) with PASAT in patients with mild TBI to explore me
128 of hippocampal regional cerebral blood flow (rCBF) with pulsed ASL.
129 nal changes in regional cerebral blood flow (rCBF), assessed by (15)O-water PET, over a mean 7 year p
130 ology, such as regional cerebral blood flow (rCBF), remains incompletely understood.
131 s a measure of regional cerebral blood flow (rCBF), we investigated the relationship between reduced
132 ET) imaging of regional cerebral blood flow (rCBF).
133 hich increases regional cerebral blood flow (rCBF).
134 obally elevate regional cerebral blood flow (rCBF).
135 d reduction in regional cerebral blood flow (rCBF).
136 ed hippocampal regional cerebral blood flow (rCBF).
137 ts of lesional relative cerebral blood flow (rCBF): 1.89 +/- 0.32 (0.72 mg E2) vs. 1.32 +/- 0.19 (P),
138 dated measure (relative cerebral blood flow [rCBF], <30%), thrombectomy patients had a smaller median
139 ed as a percentage of cerebellar blood flow, rCBF in the striatum and orbital cortex in patients with
140                         The finding of focal rCBF abnormalities in the right hemisphere of 2 right-ha
141 n) and arterial spin labeling evaluation for rCBF.
142 hronic MJ users showed significantly greater rCBF than controls in the vmPFC on the standard IGT and
143                             In the OA group, rCBF increases representing ongoing pain were identified
144 F differences between OA and control groups, rCBF differences between sessions within each group, and
145 several DMN and ECN regions exhibited higher rCBF per unit connectivity strength (rCBF/FCS ratio); wh
146 ith Val homozygotes, Met carriers had higher rCBF in prefrontal (BA25 extending into BA10) and hippoc
147 rtex) in which Val homozygotes showed higher rCBF in females than males, but Met carriers showed the
148 stigmine significantly decreased hippocampal rCBF in control subjects (P < .0005) and veterans with s
149 .05) but significantly increased hippocampal rCBF in veterans with syndrome 2 (P < .005) and veterans
150 a longitudinal reduction in left hippocampal rCBF that was not evident in subjects who remained in a
151 sociated with significantly less hippocampal rCBF.
152    ASL MR imaging examination of hippocampal rCBF in a cholinergic challenge experiment may be useful
153 found for working memory-related hippocampal rCBF change, which was uniquely attenuated in Met allele
154                                     However, rCBF increases in the prefrontal cortex were significant
155                 In response to hypoglycemia, rCBF was significantly increased in the thalamus, medial
156 effects of L-NAME inhibition on brain pH(i), rCBF, and NADH redox state during 3 h of severe focal ce
157 ss BBB disruption; PHD3-/- mice had impaired rCBF upon early reperfusion but comparable functional ou
158 t whether visually apparent abnormalities in rCBF constitute statistically significant differences be
159 activity, this study assessed alterations in rCBF and related resting state functional connectivity (
160              However, we noted no changes in rCBF and flux in presymptomatic carriers compared with c
161                                   Changes in rCBF correlated positively with NO(x) production; increa
162 ss expensive method for assessing changes in rCBF during hypoglycemia without radiation exposure.
163                                   Changes in rCBF in the caudate nucleus predicted gene status (P = 0
164            There were significant changes in rCBF in the dorsal rostral pons, anterior cingulate cort
165 d quantification of IN-OT-induced changes in rCBF in the living human brain unaffected by cognitive,
166                      Task-related changes in rCBF in the men without ADHD were more prominent in the
167          SPECT was used to assess changes in rCBF induced by amphetamine in 16 healthy volunteers.
168 )C-PIB show greater longitudinal declines in rCBF in certain areas, representing regions with greater
169 ual cortex and cerebellum show a decrease in rCBF, in a dose range of 0.2-1 minimum alveolar concentr
170 e produced statistically robust decreases in rCBF in bilateral orbitofrontal cortex, thalamus, opercu
171 ine induces focal increases and decreases in rCBF in healthy volunteers in areas primarily innervated
172            Greater longitudinal decreases in rCBF in the high-(11)C-PIB group than in the low-(11)C-P
173 efrontal cortex and significant decreases in rCBF in the vicinity of the limbic/paralimbic areas (i.e
174  Areas of relative increases or decreases in rCBF were measured by using the [(15)O]H(2)O method.
175 t were greater than or equal to decreases in rCBF.
176  for age was used to test for differences in rCBF after the cholinergic challenge across the four gro
177 d increase in rCBF after the initial drop in rCBF at the onset of MCAO.
178 icantly reduced, by over 59 %, elevations in rCBF and, by 78 %, changes in EEG evoked from RVLM.
179  and subthalamus revealed that elevations in rCBF were elicited only from a limited area, which encom
180 rats had less regulatory rebound increase in rCBF after the initial drop in rCBF at the onset of MCAO
181                     The abnormal increase in rCBF was found to have progressed to the left hippocampu
182  differences such as the delayed increase in rCBF.
183            Greater longitudinal increases in rCBF are also observed in those with higher amyloid load
184 sitively with NO(x) production; increases in rCBF during HBO2 exposure were associated with large inc
185 maging techniques found similar increases in rCBF in the thalamus, medial prefrontal cortex, and glob
186  obese men produced significant increases in rCBF in the vicinity of the ventromedial and dorsolatera
187  was indication of compensatory increases in rCBF of the occipital cortex during incremental learning
188                         Greater increases in rCBF over time in the high-(11)C-PIB group were found in
189                           These increases in rCBF precede the onset of O2-induced convulsions.
190                                 Increases in rCBF suggest a cellular and vascular compensatory proces
191 reflected by both decreases and increases in rCBF.
192 ne, there is a consistent pattern of rise in rCBF in the anterior cingulate cortex and insula while t
193 s expected, bilateral STN DBS also increased rCBF in the bilateral thalami, right midbrain, and decre
194                        Amphetamine increased rCBF in two mesial prefrontal zones (Brodmann's areas 8
195 en improvement in bradykinesia and increased rCBF in the thalamus (r(s) = 0.31, P < 0.05).
196                   MCI patients had increased rCBF in the left hippocampus (P < .001), right amygdala
197                    AD patients had increased rCBF in the right anterior cingulate gyrus (P = .02) com
198 while nicotine (1.2 nmol in 40 nl) increased rCBF by 13 +/- 5 % and synchronized the EEG from MCVA.
199 rametric mapping analysis revealed increased rCBF bilaterally in the thalamus and decreased rCBF in t
200 n (10 sec train) at an active site increased rCBF by 25 +/- 6%.
201  exercise and mental stress tasks, increased rCBF in cerebellar vermis, right anterior cingulate and
202 tion initiates CNS O2 toxicity by increasing rCBF, which allows excessive O2 to be delivered to the b
203 ects of electrical stimulation by increasing rCBF.
204 us study showing significant STN DBS-induced rCBF change in the thalamus, midbrain and supplementary
205 y involved in mechanical stimulation-induced rCBF changes and thus may represent therapeutic targets
206 dium ion channel blocker was able to inhibit rCBF changes in both the cat and rats.
207 study the authors prospectively investigated rCBF and clinical response to venlafaxine, a novel antid
208 verapamil brain distributional clearance, K1/rCBF).
209                                  Voxel-level rCBF was compared among groups by using an analysis of v
210                                 Longitudinal rCBF changes differed significantly between high- (n=10)
211 nt showed significantly greater longitudinal rCBF increases in orbitofrontal, medial frontal, and ant
212                  Differences in longitudinal rCBF changes between high- and low-(11)C-PIB groups were
213 e modified suture technique produced a lower rCBF, larger infarct size, smaller variance of infarct s
214 aine-addicted subjects showed markedly lower rCBF in the bilateral orbitofrontal cortex than the comp
215 ingulate and right insula covaried with MAP; rCBF in pons, cerebellum and right insula covaried with
216 significantly decreased cortical grey matter rCBF in the occipital lobe (mean difference -11.1 mL/100
217                                         Mean rCBF (ml/100 g/min) for S1BF were: S1BF [0 s] left corte
218                                         Mean rCBF levels were compared by using random effects regres
219 resence of hypertension and related the mean rCBF in those clusters to the presence of MCI and AD.
220 overnight and underwent PET scans to measure rCBF responses to bilateral STN DBS.
221 oton emission computed tomography to measure rCBF, after which they completed the Gambling Task.
222  (11)C-verapamil, and (15)O-water to measure rCBF.
223                                  We measured rCBF and EEG responses in rats exposed at 4 to 6 atmosph
224       When the subjects were not taking MPH, rCBF was higher in the motor, premotor, and the anterior
225              In rats pretreated with L-NAME, rCBF remained maximally decreased throughout 75 min of H
226 eived placebo at both times to assess normal rCBF variability.
227 e degree of "left-handedness" and normalized rCBF during right-hand writing.
228                Nicotine increased normalized rCBF in the left frontal region and decreased rCBF in th
229                                     Observed rCBF changes potentially indicate dysregulated CNS appra
230 h as ion channel manipulation, and observing rCBF changes may help our understanding of migraine aura
231 l stimulus conditions revealed activation of rCBF in the left medial prefrontal and left anterior cin
232           A principal-components analysis of rCBF data pooled from the two studies identified three f
233              Fully quantified assessments of rCBF and rCMR for glucose were obtained while subjects w
234 hy subjects: there was a greater decrease of rCBF in lateral and medial premotor areas, putamen, and
235 ncentration causes a predominant decrease of rCBF in the cortical regions and increase of rCBF in the
236 apping was used to analyze the PET images of rCBF changes.
237 rCBF in the cortical regions and increase of rCBF in the subcortical regions.
238 ected by AD is reduced and is independent of rCBF.
239 ha-smooth muscle actin, and the induction of rCBF-A appropriately alters their expression as well.
240 with automated three-dimensional matching of rCBF images was used to coregister and quantify results.
241 positron emission tomography measurements of rCBF at baseline and up to eight annual follow-up visits
242 e amyloid plaques, and the quantification of rCBF.
243                                 Reduction of rCBF appeared greatest in the absence of visceral stimul
244 tion, higher values and rapid restoration of rCBF were observed in group 2, while rCBF in both hemisp
245 mic region and allowing rapid restoration of rCBF.
246                We reinvestigated a series of rCBF SPECT scans obtained several years ago on drug-naiv
247        Our results support the usefulness of rCBF SPECT imaging as a diagnostic aid in PPA.
248               The effects of mecamylamine on rCBF were generally opposite to those of nicotine.
249                       Pattern recognition on rCBF maps indicated that IN-OT-induced changes were sust
250     Neither the percentage change of rCBV or rCBF predicted survival, whereas the regional response e
251 n-of-interest analysis for change in rCBV or rCBF to the change in perfusion parameters on the basis
252  contrast, change in average percent rCBV or rCBF, MR tumor volume changes, age, extent of resection,
253        During exposures to hyperbaric oxygen rCBF decreased at 4 ATA, decreased for the initial 30 mi
254                 Together, both increased pHp rCBF and strengthened pHp-PCC rsFC predicted relapse wit
255 eters on the basis of PRM (PRM(rCBV) and PRM(rCBF)) for their accuracy in predicting overall survival
256       However, for both subgroups with PTSD, rCBF changes in medial frontal gyrus were inversely corr
257 h placebo, improved IBS symptoms and reduced rCBF in 5-HT3R containing regions of the EMS, but not in
258 llow-up, symptomatic improvement and reduced rCBF in the hippocampus and ventral striatum were observ
259       Correlational analysis between reduced rCBF and BNT was performed.
260 minating the confounding effect that reduced rCBF has on assessment of BBB P-glycoprotein activity an
261                 Analysis of learning-related rCBF in network regions revealed improvement in baseline
262 the optimal ischemic core threshold remained rCBF <30% (AUC, 0.83; 95% CI, 0.77, 0.85).
263 tients with HD showed an increase in resting rCBF for all brain regions measured except the caudate n
264 xploratory analyses of interregional resting rCBF covariation revealed a specific and significant dia
265 evidence of a distinctive pattern of resting rCBF abnormalities associated with CFS.
266                                    Resultant rCBF changes were evaluated parametrically through the f
267                                  Sessionwise rCBF differences in the OA group in the postcentral, ros
268                   No significant sessionwise rCBF differences were observed in controls.
269 s within each group, and whether sessionwise rCBF differences were related to variability in perceive
270 dent longitudinal increases in resting state rCBF in brain regions intrinsic to memory processes.
271  higher rCBF per unit connectivity strength (rCBF/FCS ratio); whereas, this index was lower in poster
272 ts who achieve rapid reperfusion, a stricter rCBF threshold to estimate the ischemic core should be c
273 auditory localization in the blind subjects, rCBF activity in the right posterior parietal cortex was
274                                          The rCBF data were acquired 15 min before and up to 78 min a
275                                          The rCBF in the right hemisphere reticular system was relate
276 n inter-regional correlation analysis on the rCBF data set.
277                                        Their rCBF data were automatically normalized to whole-brain c
278                  Although the cause of these rCBF changes in HD patients is unclear, nitric oxide syn
279  of pramipexole's effects suggest that these rCBF responses indicate functional effects of a D3-prefe
280 dy, and clinical response appears related to rCBF changes.
281 the right amygdala and negatively related to rCBF in medial frontal gyrus.
282 , symptom severity was positively related to rCBF in the right amygdala and negatively related to rCB
283 ed group, mood provocation produced a unique rCBF decrease in pregenual anterior cingulate 24a.
284 e ischemic core in thrombectomy patients was rCBF <20% (area under the curve [AUC], 0.89; 95% CI, 0.8
285                                         When rCBF was calculated as a percentage of cerebellar blood
286                                         When rCBF was calculated as a percentage of cerebellar rCBF,
287 se men than in lean men (P < 0.005), whereas rCBF decreases in the hypothalamus and thalamus were att
288 tion of rCBF were observed in group 2, while rCBF in both hemispheres was significantly decreased in
289 posed to pleasant and unpleasant odors while rCBF was measured using [(15)O] water PET.
290 , postural stability and gait correlate with rCBF responses in a priori determined regions.
291 frontal gyrus were inversely correlated with rCBF changes in the left amygdala and the right amygdala
292 s sessions was significantly correlated with rCBF decreases in the 5-HT3R-rich amygdala, ventral stri
293  condition, extraversion was correlated with rCBF in the amygdala and occipital cortex.
294  condition, extraversion was correlated with rCBF in the occipital cortex and inferior temporal gyrus
295 S showed a striking spatial correlation with rCBF, and the correlation was stronger in the default-mo
296 y, prestimulus cortisol levels covaried with rCBF in the subgenual anterior cingulate cortex.
297    Prestimulus cortisol levels covaried with rCBF responses in the rostral anterior cingulate cortex.
298 een-genotype group differences covaries with rCBF in other nodes throughout the brain in a genotype-
299                                   Glu and WM rCBF decreased linearly with age while Gln and Gln/Glu i
300                                   Glu and WM rCBF were correlated with the UCSD Performance-Based Ski
301  Glu, Gln, Gln/Glu, and AC white matter (WM) rCBF.

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top