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1 rCBF changes were assessed with a three-dimensional voxe
2 rCBF changes were evaluated with statistical parametric
3 rCBF data were recorded with a flow-sensitive alternatin
4 rCBF decreases in limbic/paralimbic areas, temporal and
5 rCBF was measured in 38 control subjects, 29 MCI patient
6 i) declined from 6.95+/-0.04 to 6.60+/-0.05, rCBF declined from 48+/-7 to 10+/-3 ml/100 g/min, and NA
7 to conditionally express recombinant CBF-A (rCBF-A) activate the transcription of FSP1 and undergo E
8 e scanning interval, most of the accelerated rCBF changes seen in the subsequently impaired group occ
10 lyses revealed regional abnormalities in all rCBF and rCMR measurements that were consistent in locat
12 epression Rating Scale were administered and rCBF was measured by means of single photon emission com
13 nverse relationship between 11C-PIB BPND and rCBF MR imaging in the voxel-based analysis that was abs
14 emory experiment, both FCS-rCBF coupling and rCBF/FCS ratio were modulated by task load in the ECN an
17 n functional connectivity strength (FCS) and rCBF during resting and an N-back working-memory task.
18 llected at each of these glycemic levels and rCBF measurements within the previously described networ
19 s between selective motor manifestations and rCBF in specific regions suggest possible regional selec
21 ed with significant emotional reactivity and rCBF reductions in the ventromedial prefrontal cortex, s
22 Associations between extraversion scores and rCBF in each olfactory stimulus condition were assessed
24 cipital perfusion deficits but with anterior rCBF deficits in a pattern often described in the litera
27 ent in the frontal and temporal regions, but rCBF changes in men with ADHD were more widespread and p
30 was calculated as a percentage of cerebellar rCBF, analysis of covariance found decreases in HD cauda
36 cingulate and dorsolateral prefrontal cortex rCBF is significantly related to decision making, as ass
37 late and left dorsolateral prefrontal cortex rCBF significantly correlated with performance on the Ga
44 BF bilaterally in the thalamus and decreased rCBF in the left occipital lobe, right cerebellum, and t
45 teral thalami, right midbrain, and decreased rCBF in the right premotor cortex (P < 0.05, corrected).
46 etween improvement of rigidity and decreased rCBF in the SMA (r(s) = -0.4, P < 0.02) and between impr
51 r model analyses were performed to determine rCBF differences between OA and control groups, rCBF dif
53 ndritic arborization, we complimented direct rCBF comparisons with connectivity analyses to determine
54 hip was connection-distance dependent; i.e., rCBF correlated stronger with long-range hubs than short
55 pinal neurons in RVLM to reflexively elevate rCBF and slow the EEG as part of the oxygen-conserving (
56 s relay signals from the MCVA, which elevate rCBF in response to hypoxia, and (3) the SVA is a functi
57 jection of NaCN (300 pmol in 20 nl) elevated rCBF (17 +/- 5 %) and synchronized the EEG from RVLM, bu
58 rats electrical stimulation of RVLM elevated rCBF (laser-Doppler flowmetry) by 31 +/- 6 %, reduced ce
59 rs, ultra-high-risk subjects showed elevated rCBF in the hippocampus, basal ganglia, and midbrain.
60 nchronizes the EEG and coordinately elevates rCBF; (b) the responses are mediated by excitation of ne
61 ly restricted region of subthalamus elevates rCBF, (2) these neurons relay signals from the MCVA, whi
62 he hypothesis that whisker movement elicited rCBF changes are input frequency dependent and are most
63 wise, whole-brain analysis revealed enhanced rCBF only in the left posterior hippocampus (pHp) in the
69 ring the working-memory experiment, both FCS-rCBF coupling and rCBF/FCS ratio were modulated by task
70 ative cerebral blood volume (rCBV) and flow (rCBF) maps were acquired before treatment and after 1 an
71 nd measures of regional cerebral blood flow (rCBF) (a marker of neuronal activity) to describe the fu
72 elevations of regional cerebral blood flow (rCBF) and synchronization of the electroencephalogram (E
76 s, we measured regional cerebral blood flow (rCBF) as a marker of synaptic activity at rest and durin
78 dependent from regional cerebral blood flow (rCBF) changes during moderate focal cerebral ischemia.
79 f longitudinal regional cerebral blood flow (rCBF) changes in the years preceding measurement of amyl
80 was to compare regional cerebral blood flow (rCBF) changes related to working memory in adults with a
81 n to determine regional cerebral blood flow (rCBF) changes representing ongoing pain experienced by p
83 ps resulted in regional cerebral blood flow (rCBF) decreases in medial orbitofrontal cortex Brodmann'
84 ith concurrent regional cerebral blood flow (rCBF) determination before and after administration of b
86 ygenation, and regional cerebral blood flow (rCBF) did not reach significance, but a trend for a pron
87 if changes in regional cerebral blood flow (rCBF) during hypoglycemia relative to euglycemia are sim
88 resting state regional cerebral blood flow (rCBF) during normal aging and investigated its influence
89 thors measured regional cerebral blood flow (rCBF) during performance of a task that required unmedic
90 PET to assess regional cerebral blood flow (rCBF) during rest and tested for between-genotype differ
91 ant changes in regional cerebral blood flow (rCBF) during states of cardiovascular arousal common to
92 hange in tumor relative cerebral blood flow (rCBF) from baseline and area under the plasma concentrat
93 sed to examine regional cerebral blood flow (rCBF) in 30 patients with chronic schizophrenia and 30 n
95 easure resting regional cerebral blood flow (rCBF) in 52 individuals at ultra-high risk for psychosis
99 l volumes, and regional cerebral blood flow (rCBF) in healthy controls (HC) (n = 24), patients diagno
100 DBS increases regional cerebral blood flow (rCBF) in immediate downstream targets but does not revea
102 SPECT to study regional cerebral blood flow (rCBF) in patients with HD during rest and maze testing.
103 ET) to measure regional cerebral blood flow (rCBF) in sighted and congenitally blind subjects perform
104 ect of Alos on regional cerebral blood flow (rCBF) in the absence and presence of rectal or sigmoid s
106 covaried with regional cerebral blood flow (rCBF) in the dorsal medial prefrontal cortex, rostral an
107 owed increased regional cerebral blood flow (rCBF) in the vmPFC on both versions of the IGT compared
108 frontal cortex regional cerebral blood flow (rCBF) is lower in cocaine-dependent subjects than in non
109 cerebral blood volume (rCBV) and blood flow (rCBF) maps were acquired before chemoradiotherapy and at
110 to placebo on regional cerebral blood flow (rCBF) measured by SPECT in healthy volunteers to charact
111 lectrode and changes in cerebral blood flow (rCBF) measured with a laser Doppler probe placed over th
113 identified on regional cerebral blood flow (rCBF) SPECT scans of adolescent children and young adult
115 less change in regional cerebral blood flow (rCBF) to the anterior cingulate and supplementary motor
117 We measured regional cerebral blood flow (rCBF) using pseudo-continuous arterial spin labelling.
123 phy (SPECT) of regional cerebral blood flow (rCBF) was used to compare the central nervous system res
124 in normalized regional cerebral blood flow (rCBF) were assessed while participants repetitively wrot
125 traversion and regional cerebral blood flow (rCBF) while participants were exposed to olfactory stimu
126 ing to measure regional cerebral blood flow (rCBF) while they performed kinematically matched sequenc
127 activation of regional cerebral blood flow (rCBF) with PASAT in patients with mild TBI to explore me
129 nal changes in regional cerebral blood flow (rCBF), assessed by (15)O-water PET, over a mean 7 year p
131 s a measure of regional cerebral blood flow (rCBF), we investigated the relationship between reduced
137 ts of lesional relative cerebral blood flow (rCBF): 1.89 +/- 0.32 (0.72 mg E2) vs. 1.32 +/- 0.19 (P),
138 dated measure (relative cerebral blood flow [rCBF], <30%), thrombectomy patients had a smaller median
139 ed as a percentage of cerebellar blood flow, rCBF in the striatum and orbital cortex in patients with
142 hronic MJ users showed significantly greater rCBF than controls in the vmPFC on the standard IGT and
144 F differences between OA and control groups, rCBF differences between sessions within each group, and
145 several DMN and ECN regions exhibited higher rCBF per unit connectivity strength (rCBF/FCS ratio); wh
146 ith Val homozygotes, Met carriers had higher rCBF in prefrontal (BA25 extending into BA10) and hippoc
147 rtex) in which Val homozygotes showed higher rCBF in females than males, but Met carriers showed the
148 stigmine significantly decreased hippocampal rCBF in control subjects (P < .0005) and veterans with s
149 .05) but significantly increased hippocampal rCBF in veterans with syndrome 2 (P < .005) and veterans
150 a longitudinal reduction in left hippocampal rCBF that was not evident in subjects who remained in a
152 ASL MR imaging examination of hippocampal rCBF in a cholinergic challenge experiment may be useful
153 found for working memory-related hippocampal rCBF change, which was uniquely attenuated in Met allele
156 effects of L-NAME inhibition on brain pH(i), rCBF, and NADH redox state during 3 h of severe focal ce
157 ss BBB disruption; PHD3-/- mice had impaired rCBF upon early reperfusion but comparable functional ou
158 t whether visually apparent abnormalities in rCBF constitute statistically significant differences be
159 activity, this study assessed alterations in rCBF and related resting state functional connectivity (
162 ss expensive method for assessing changes in rCBF during hypoglycemia without radiation exposure.
165 d quantification of IN-OT-induced changes in rCBF in the living human brain unaffected by cognitive,
168 )C-PIB show greater longitudinal declines in rCBF in certain areas, representing regions with greater
169 ual cortex and cerebellum show a decrease in rCBF, in a dose range of 0.2-1 minimum alveolar concentr
170 e produced statistically robust decreases in rCBF in bilateral orbitofrontal cortex, thalamus, opercu
171 ine induces focal increases and decreases in rCBF in healthy volunteers in areas primarily innervated
173 efrontal cortex and significant decreases in rCBF in the vicinity of the limbic/paralimbic areas (i.e
174 Areas of relative increases or decreases in rCBF were measured by using the [(15)O]H(2)O method.
176 for age was used to test for differences in rCBF after the cholinergic challenge across the four gro
178 icantly reduced, by over 59 %, elevations in rCBF and, by 78 %, changes in EEG evoked from RVLM.
179 and subthalamus revealed that elevations in rCBF were elicited only from a limited area, which encom
180 rats had less regulatory rebound increase in rCBF after the initial drop in rCBF at the onset of MCAO
184 sitively with NO(x) production; increases in rCBF during HBO2 exposure were associated with large inc
185 maging techniques found similar increases in rCBF in the thalamus, medial prefrontal cortex, and glob
186 obese men produced significant increases in rCBF in the vicinity of the ventromedial and dorsolatera
187 was indication of compensatory increases in rCBF of the occipital cortex during incremental learning
192 ne, there is a consistent pattern of rise in rCBF in the anterior cingulate cortex and insula while t
193 s expected, bilateral STN DBS also increased rCBF in the bilateral thalami, right midbrain, and decre
198 while nicotine (1.2 nmol in 40 nl) increased rCBF by 13 +/- 5 % and synchronized the EEG from MCVA.
199 rametric mapping analysis revealed increased rCBF bilaterally in the thalamus and decreased rCBF in t
201 exercise and mental stress tasks, increased rCBF in cerebellar vermis, right anterior cingulate and
202 tion initiates CNS O2 toxicity by increasing rCBF, which allows excessive O2 to be delivered to the b
204 us study showing significant STN DBS-induced rCBF change in the thalamus, midbrain and supplementary
205 y involved in mechanical stimulation-induced rCBF changes and thus may represent therapeutic targets
207 study the authors prospectively investigated rCBF and clinical response to venlafaxine, a novel antid
211 nt showed significantly greater longitudinal rCBF increases in orbitofrontal, medial frontal, and ant
213 e modified suture technique produced a lower rCBF, larger infarct size, smaller variance of infarct s
214 aine-addicted subjects showed markedly lower rCBF in the bilateral orbitofrontal cortex than the comp
215 ingulate and right insula covaried with MAP; rCBF in pons, cerebellum and right insula covaried with
216 significantly decreased cortical grey matter rCBF in the occipital lobe (mean difference -11.1 mL/100
219 resence of hypertension and related the mean rCBF in those clusters to the presence of MCI and AD.
221 oton emission computed tomography to measure rCBF, after which they completed the Gambling Task.
230 h as ion channel manipulation, and observing rCBF changes may help our understanding of migraine aura
231 l stimulus conditions revealed activation of rCBF in the left medial prefrontal and left anterior cin
234 hy subjects: there was a greater decrease of rCBF in lateral and medial premotor areas, putamen, and
235 ncentration causes a predominant decrease of rCBF in the cortical regions and increase of rCBF in the
239 ha-smooth muscle actin, and the induction of rCBF-A appropriately alters their expression as well.
240 with automated three-dimensional matching of rCBF images was used to coregister and quantify results.
241 positron emission tomography measurements of rCBF at baseline and up to eight annual follow-up visits
244 tion, higher values and rapid restoration of rCBF were observed in group 2, while rCBF in both hemisp
250 Neither the percentage change of rCBV or rCBF predicted survival, whereas the regional response e
251 n-of-interest analysis for change in rCBV or rCBF to the change in perfusion parameters on the basis
252 contrast, change in average percent rCBV or rCBF, MR tumor volume changes, age, extent of resection,
255 eters on the basis of PRM (PRM(rCBV) and PRM(rCBF)) for their accuracy in predicting overall survival
257 h placebo, improved IBS symptoms and reduced rCBF in 5-HT3R containing regions of the EMS, but not in
258 llow-up, symptomatic improvement and reduced rCBF in the hippocampus and ventral striatum were observ
260 minating the confounding effect that reduced rCBF has on assessment of BBB P-glycoprotein activity an
263 tients with HD showed an increase in resting rCBF for all brain regions measured except the caudate n
264 xploratory analyses of interregional resting rCBF covariation revealed a specific and significant dia
269 s within each group, and whether sessionwise rCBF differences were related to variability in perceive
270 dent longitudinal increases in resting state rCBF in brain regions intrinsic to memory processes.
271 higher rCBF per unit connectivity strength (rCBF/FCS ratio); whereas, this index was lower in poster
272 ts who achieve rapid reperfusion, a stricter rCBF threshold to estimate the ischemic core should be c
273 auditory localization in the blind subjects, rCBF activity in the right posterior parietal cortex was
279 of pramipexole's effects suggest that these rCBF responses indicate functional effects of a D3-prefe
282 , symptom severity was positively related to rCBF in the right amygdala and negatively related to rCB
284 e ischemic core in thrombectomy patients was rCBF <20% (area under the curve [AUC], 0.89; 95% CI, 0.8
287 se men than in lean men (P < 0.005), whereas rCBF decreases in the hypothalamus and thalamus were att
288 tion of rCBF were observed in group 2, while rCBF in both hemispheres was significantly decreased in
291 frontal gyrus were inversely correlated with rCBF changes in the left amygdala and the right amygdala
292 s sessions was significantly correlated with rCBF decreases in the 5-HT3R-rich amygdala, ventral stri
294 condition, extraversion was correlated with rCBF in the occipital cortex and inferior temporal gyrus
295 S showed a striking spatial correlation with rCBF, and the correlation was stronger in the default-mo
297 Prestimulus cortisol levels covaried with rCBF responses in the rostral anterior cingulate cortex.
298 een-genotype group differences covaries with rCBF in other nodes throughout the brain in a genotype-
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