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1 n epidemic of rabies virus in North American raccoons.
2 tectable in nontumor tissue or in unaffected raccoons.
3 ntified in neuroglial tumors of free-ranging raccoons.
4 th 100% of neuroglial tumors in free-ranging raccoons.
5 ore than 20 years and including 35,387 rabid raccoons.
6 ct of potential behavioural changes in rabid raccoons.
7 lates induced pathological findings of AD in raccoons.
8 ts; 88 (4%) to rodents/rabbits; 10 (0.5%) to raccoons; 5 (0.2%) to bats; and 24 (1.2%) to other anima
10 nine parvovirus (CPV) variants isolated from raccoons-a newly recognized CPV host-to different carniv
11 (GABA) in the somatosensory thalamus of the raccoon and to compare these features to those of other
12 sily observed and/or common mammals, such as raccoons and bobcats, bode poorly for species of conserv
14 uences from puma, coyote, gray wolf, bobcat, raccoon, and striped skunk revealed two major groups rel
15 infections with Baylisascaris procyonis, the raccoon ascarid, have been increasingly recognized in do
17 e that selection favors oral transmission to raccoons but classical stercorarian transmission to wood
19 n this study, we investigated the effects of raccoon contact patterns on rabies spread using network
20 ng detailed data describing the variation in raccoon contact rates into a network modelling approach,
26 Himalayan palm civets (Paguma larvata), and raccoon dogs (Nyctereutes procyonoides), sold in exotic
27 hina and spread to humans via civet cats and raccoon dogs in the wet markets before spreading to 37 c
28 of zoonotic strains found in palm civets and raccoon dogs, as well as isolates spanning the early, mi
30 duced in Aleutian and non-Aleutian mink, but raccoons failed to show serological or pathological evid
32 iant virus that has circulated undetected in raccoons for at least 24 years, with transfers to and fr
36 neurons in the somatosensory thalamus of the raccoon is less than that in the cat and monkey, but the
37 preparation of ADV-TR prepared from infected raccoon lymph nodes was inoculated into mink and raccoon
39 om fecal samples collected from anesthetized raccoons (n = 738) trapped in six Pennsylvania counties
40 umented a 99.3% decrease in the frequency of raccoon observations, decreases of 98.9% and 87.5% for o
41 s identified by PCR in samples from mink and raccoons on commercial ranches during an outbreak of Ale
42 rent trade-off scenarios in cycles involving raccoons or woodrats, including a proper three-way trade
45 proximity logging collars on a wild suburban raccoon population and then simulated rabies spread acro
46 monstrate that when rabies enters a suburban raccoon population, the likelihood of a disease outbreak
47 of dumb and furious behaviours in the rabid raccoon population, there are similar outbreak sizes and
48 provide new insights into rabies dynamics in raccoon populations and have important implications for
49 approach, we were able to show that suburban raccoon populations are highly susceptible to rabies out
50 t patterns influence the spread of rabies in raccoon populations in order to design effective control
51 assumed low levels of immunity (1-5%) within raccoon populations, suggesting that raccoons develop li
55 ticks (Ixodes texanus; n=718) collected from raccoons (Procyon lotor; n=91) and genotyped at 11 micro
57 e epizoology of rabies with the expansion of raccoon rabies from a small pocket around northern Virgi
60 ed kidney recipient were consistent with the raccoon rabies virus variant and were more than 99.9% id
62 each host species but, with the exception of raccoons, relatively little evidence for onward transmis
63 e rabies virus outbreak among North American raccoons reveals the long lasting effect of the initial
65 ibutions of PV+ and CaBP+ cell bodies in the raccoon somatosensory thalamus are very similar to those
66 terozygote deficits (HDs) at the CP scale in raccoon ticks (Ixodes texanus; n=718) collected from rac
70 nic tumor antigen genes, we cultured primary raccoon tumor cells and passaged them in mice, confirmin
71 oon lymph nodes was inoculated into mink and raccoons, typical AD was induced in Aleutian and non-Ale
73 y, in capsid protein (VP2) phylogenies, most raccoon viruses fell as evolutionary intermediates betwe
74 , and periodicity of rabies epizootics among raccoons were compared with predictions derived from a s
75 protein of ADV, indicated that both mink and raccoons were infected by a new isolate of ADV, designat
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