ライフサイエンスコーパス: rad

1 el from an average 2.2 rad (0.022 Gy) to 1.0 rad (0.010 Gy) (P < .001).

2 ylactic cranial radiation to a dose of 3,000 rad.

3 er a broad range of frequencies (0.1-30, 000 rad/s), LTM provides a unique ability to noninvasively q

4 horacic radiation therapy consisted of 4,000 rad to the tumor and mediastinum with a 1,000-rad boost.

5 amster somatic hybrid cell panel and a 5,000 rad whole-genome radiation hybrid (WGRH) panel.

6 rel (millicurie) injected was 90 Gy/g (9,000 rad/g), of which 64% was from conversion electrons, 16%

7 ad to the tumor and mediastinum with a 1,000-rad boost.

8 This study shows that the 10,000-rad panel is appropriate for the generation of a high-re

9 A 10,000-rad radiation hybrid (RH) cell panel of the rhesus macaq

10 igh-resolution RH map of SSC12 on the 12,000-rad IMNpRH2 panel using CarthaGene software.

11 Analysis of the 5,000-rad horse x hamster radiation hybrid panel produced a ma

12 12 short tandem repeat markers on the 5,000-rad horse x hamster whole-genome radiation hybrid panel

13 ences were added to the Illinois-Texas 5,000-rad RH (RH, radiation hybrid) map.

14 The high-resolution Illinois-Texas 5,000-rad RH and comparative maps will facilitate identificati

15 A high-resolution SSC12 RH12,000-rad map will be useful in fine-mapping QTL and as a scaf

16 d 0.0020 mGy/MBq (0.0086, 0.0006, and 0.0074 rad/mCi) for the ovaries, testes, and red marrow, respec

17 The whole-body dose is 0.0111 rad/mCi, resulting in a 0.622 rad exposure with a maximu

18 (0.0295 rad/mCi) and 0.00709 mGy/MBq (0.0262 rad/mCi), respectively.

19 rrow and spleen were 0.00797 mGy/MBq (0.0295 rad/mCi) and 0.00709 mGy/MBq (0.0262 rad/mCi), respectiv

20 Ba2+-sensitive admittance was 3.09 +/- 0.03 rad at 10 Hz and 3.00 +/- 0.17 rad at 30 Hz.

21 the lipid tail rotation (D(rot_tail) = -0.04 rad(2)/ns) is slower then the head group rotation (D(rot

22 ps and an out of plane rate of Dtheta = 0.05 rad(2)/ps.

23 to the pancreas, and 0.0186 mGy/MBq (0.0688 rad/mCi) to the uterus.

24 ffusively at an in plane rate of Dphi = 0.07 rad(2)/ps and an out of plane rate of Dtheta = 0.05 rad(

25 ver (mean, 24.78 rad/mCi; range, 15.02-37.07 rad/mCi), with red marrow estimates on the order of 3.32

26 /mCi) to the kidneys, 0.0197 mGy/MBq (0.0728 rad/mCi) to the pancreas, and 0.0186 mGy/MBq (0.0688 rad

27 he critical organ receiving a dose of 0.0886 rad/mCi.

28 lags peak tidal extension by approximately 1 rad, suggestive of resonance.

29 nd 67Cu-BAT-2IT-1A3 were 586 and 1269 rad (1 rad = 0.01 Gy), respectively.

30 of the laser trap at frequencies less than 1 rad/s, indicating that at least locally, the gels behave

31 ived a radiation dose of 0.028 mGy/MBq (0.10 rad/mCi).

32 lated (>/=75.6 Gy to 90 Gy) EBRT (1 Gy = 100 rad).

33 The low-frequency (< or = 100 rad s-1) impedance of skin was much more significantly a

34 strain over a frequency range of 0.1 to 100 rad/sec.

35 The dose was 0.027 mGy/MBq (0.101 rad/mCi) to the kidneys, 0.0197 mGy/MBq (0.0728 rad/mCi)

36 64Cu- and 67Cu-BAT-2IT-1A3 were 586 and 1269 rad (1 rad = 0.01 Gy), respectively.

37 lungs, with a dose of 0.0393 mGy/MBq (0.1455 rad/mCi).

38 from 0.0002 to 0.0393 mGy/MBq (0.0006-0.1455 rad/mCi).

39 ation-associated LD(50) being reduced by 150 rad.

40 Tumor dosimetry ranged from 39 to 167 rad/mCi of 90Y (442-1887 rad/ dose).

41 3.09 +/- 0.03 rad at 10 Hz and 3.00 +/- 0.17 rad at 30 Hz.

42 h received an average of 0.046 mGy/MBq (0.17 rads/mCi, range 0.14-0.22 rad/mCi).

43 (0.59 rem) and a lung dose of 21.8 mGy (2.18 rad) in young, healthy subjects.

44 nged from 39 to 167 rad/mCi of 90Y (442-1887 rad/ dose).

45 ere the lower large intestine (1.43 +/- 0.19 rad/mCi) and upper large intestine (1.20 +/- 0.38 rad/mC

46 fferent frequencies ranging from omega = 0.2 rad/s to 6.9 rad/s.

47 yed and unenhanced (-1.6+/-0.8 to -2.3+/-1.2 rad/s).

48 sing the radiation level from an average 2.2 rad (0.022 Gy) to 1.0 rad (0.010 Gy) (P < .001).

49 hen the head group rotation (D(rot_hg) = 2.2 rad(2)/ns), which is slower than the overall in plane (D

50 ower than the overall in plane (D(rot) = 3.2 rad(2)/ns) for the lipid molecule.

51 t across the film is an odd multiple of pi/2 rad.

52 .046 mGy/MBq (0.17 rads/mCi, range 0.14-0.22 rad/mCi).

53 h received an average of 0.062 mGy/MBq (0.23 rad/mCi, range 0.08-0.30 rad/mCi), followed by the kidne

54 ctions were quantified to be as fast as 3.25 rad s(-1) and as large as 0.5 A, respectively.

55 .65 +/- 0.32 rad at 10 Hz and -2.79 +/- 0.26 rad at 30 Hz.

56 ts (n=51) underwent a single, rapid (117-266 rad/s) head rotation in the sagittal (n=13), coronal (n=

57 dose of 0.15 +/- 0.08 mGy/MBq (0.57 +/- 0.28 rad/mCi).

58 ovement velocities tested (ie, 4.71 and 6.28 rad/s).

59 ge decreases with a value of 7.5 mGy/MBq (28 rad/mCi) for a newborn.

60 ntwisting velocity, -2.0+/-0.7 to -5.6+/-2.3 rad/s) that was associated with an increase in IVPG (1.4

61 .3 and 2.2 +/- 0.5 mGy/MBq (2.4, 3.8 and 8.3 rad/mCi), respectively.

62 0.062 mGy/MBq (0.23 rad/mCi, range 0.08-0.30 rad/mCi), followed by the kidneys (0.051 mGy/MBq), liver

63 l mortality included radiation dose >/= 3000 rad ( >/= 30 Gy; supra-diaphragm: HR, 3.8; 95% CI, 1.1-1

64 Irradiation (3000 rads) of the i.v. donor splenocytes combined with RIB 5/

65 digrees informative for XLPRA, and on a 3000-rad canine-hamster radiation hybrid (RH) panel.

66 ap and 37 were positioned on the Roslin 3000-rad radiation hybrid (RH) map, with 20 assignments share

67 ve admittance in GP cells was -2.65 +/- 0.32 rad at 10 Hz and -2.79 +/- 0.26 rad at 30 Hz.

68 .07 mSv/MBq (0.64 +/- 0.18 and 0.55 +/- 0.32 rad/mCi) for females and males, respectively.

69 th red marrow estimates on the order of 3.32 rad/mCi (range, 1.24-5.55) of 90Y.

70 y bladder wall dose to 0.0885 mGy/MBq (0.327 rad/mCi) or 0.128 mGy/MBq (0.473 rad/mCi), respectively,

71 ich received an estimated 0.11 mGy/MBq (0.37 rad/mCi).

72 Ci) and upper large intestine (1.20 +/- 0.38 rad/mCi).

73 ceiving the highest dose, 0.11 mGy/MBq (0.39 rad/mCi).

74 s) but converge at high frequencies (> 10(4) rad/s).

75 g. the 'no-load' angular velocity is ca. 400 rad/s anticlockwise at 4 mM ATP.

76 was transferred to irradiated ACI rats (400 rad) with either purified T cells (4-10 x 10[7]) or seru

77 myeloma, SCID mice were irradiated with 400 rads and mice were injected either with 10(6) ARH-77 cel

78 ch received an average of 0.12 mGy/MBq (0.43 rad/mCi) (range, 0.098-0.15 mGy/MBq).

79 One species with omega(0) = 0.45 rad/ns and low eta is attributed to a trigonal planar Cd

80 /MBq (0.327 rad/mCi) or 0.128 mGy/MBq (0.473 rad/mCi), respectively, and the effective dose to 0.0149

81 ers of magnitude at low frequencies (0.5-1.5 rad/s) but converge at high frequencies (> 10(4) rad/s).

82 was 16 mGy (1.6 rad) (range, 4-45 mGy [4-4.5 rad]).

83 ccurately, giving a tumor dose of 81 +/- 7.5 rad/mCi.

84 n, it would have taken 22.7 mCi to deliver 5 rad to the liver.

85 injected dose at 56 mCi using the limit of 5 rads to a critical organ per study per year.

86 After sublethal radiation injury (500 rad), the infusion of purified CD4(+) T cells enhances h

87 Sublethal irradiation (500 rad) of mice prior to tumor inoculation abrogated the IL

88 immunosensitizing sublethal irradiation (500 rad) of the host before adoptive therapy.

89 Mice rendered pancytopenic with 500 rads and 1.2 mg of carboplatin had a nadir platelet coun

90 developed by testing DNA samples from a 5000 rad whole genome RH panel.

91 A second-generation 5000 rad radiation hybrid (RH) map of the cattle genome was c

92 as obtained using the recently produced 5000(rad) horse x hamster hybrid panel.

93 We recently constructed a 5000-rad cattle whole-genome radiation hybrid panel with the

94 ere mapped primarily by genotyping on a 5000-rad horse x hamster radiation hybrid panel, of which 28

95 A 5000-rad whole genome radiation hybrid panel is described for

96 enerated in this study and typed to our 5000-rad horse x hamster whole genome RH panel.

97 as the urinary bladder at 0.14 mGy/MBq (0.51 rad/mCi).

98 mCi]), followed by liver (0.14 mGy/MBq [0.52 rad/mCi]); the effective dose is 0.022 mSv/MBq (0.083 re

99 estion (ie, 12.48+/-0.95 versus 11.11+/-0.53 rad/s; P<0.05).

100 ssion occurs is approximately 100 g and 5500 rad/s/s.

101 ministered activity was 0.150 mGy/MBq (0.556 rad/mCi) with the realistic void schedule used in our st

102 and large intestines (161.26 muGy/MBq [0.597 rad/mCi] and 184.59 muGy/MBq [0.683 rad/mCi]).

103 average fetal radiation dose was 16 mGy (1.6 rad) (range, 4-45 mGy [4-4.5 rad]).

104 (66 mCi) to give the same dose rate of 43.6 rad/h in the lungs at 48 h.

105 rils is more mechanically rigid (517 Pa at 6 rad/s) than the gel composed of twisted fibrils (367 Pa

106 gel composed of twisted fibrils (367 Pa at 6 rad/s).

107 ls with peak reorientation speeds of up to 6 rad/s and returning to random after 5 min without stimul

108 Although the addition of 600 rad of total body irradiation delayed tumor growth, furt

109 e kidney capsule of diabetic irradiated (600 rads), naive C57BL/6 recipients (mean survival time = 48

110 dose is 0.0111 rad/mCi, resulting in a 0.622 rad exposure with a maximum single injection dose.

111 blood (131)I was 0.34 (range, 0.1 6 to 0.63) rad/mCi (0.09 mGy/MBq; range, 0.04 to 0.17 mGy/ MBq).

112 q [0.597 rad/mCi] and 184.59 muGy/MBq [0.683 rad/mCi]).

113 rgan is gallbladder wall (0.19 mGy/MBq [0.69 rad/mCi]), followed by liver (0.14 mGy/MBq [0.52 rad/mCi

114 f 2 less than the value of 0.45 mGy/MBq (1.7 rad/mCi) previously accepted.

115 nit of administered 131I was 1.0 Gy/GBq (3.7 rad/mCi) for patients with NHL whether in MTD or LD tria

116 , and gallbladder wall, 0.193 mGy/MBq (0.716 rad/mCi).

117 treatment absorbed dose limit of 75 cGy (75 rad), the administered 131I activity ranged from 2.1 to

118 lculated in this study of 0.21 mGy/MBq (0.77 rad/mCi) is approximately a factor of 2 less than the va

119 ses were delivered to the liver (mean, 24.78 rad/mCi; range, 15.02-37.07 rad/mCi), with red marrow es

120 ved the highest dose (229.50 muGy/MBq [0.849 rad/mCi]), followed by the small and large intestines (1

121 encies ranging from omega = 0.2 rad/s to 6.9 rad/s.

122 to amplitudes of approximately 1.5 x 10(-9) rad at the lowest frequency normal mode and the sensitiv

123 the target organ or tissue of 19.4 mGy (1.93 rad).

124 d, MHC class II +/+ mice and irradiated (950 rads) MHC class II -/- recipients.

125 e urinary bladder wall, 0.258 mGy/MBq (0.955 rad/mCi), and gallbladder wall, 0.193 mGy/MBq (0.716 rad

126 We propose that atl-1 and rad-5/clk-2 respond to single-stranded DNA generated by

127 Phenotypic comparison of Cebrc-2 and rad-51 mutants also revealed a role for CeBRC-2 in an er

128 c and cytological comparisons of Cebrc-2 and rad-51 mutants revealed fundamental phenotypic differenc

129 n DNA repair defects observed in Cebrc-2 and rad-51 mutants, and define mechanistic roles for CeBRC-2

130 that atl-1 (Caenorhabditis elegans ATR) and rad-5/clk-2 prevent mitotic catastrophe, function in the

131 e stress activates ataxia telangiectasia and rad-3-related (ATR) and Chk1 checkpoint kinases that inh

132 ndard prescribed absorbed dose of 7,000 cGy (rad) to the thyroid therefore was reduced by over 50% wi

133 hyperthyroidism, with a standard 7,000-cGy (rad) thyroid absorbed dose generally resulting in an inc

134 tal blood absorbed dose of less than 10 cGy (rad).

135 dental blood absorbed dose of up to 150 cGy (rad) could result.

136 ATDs, from 11.3 +/- 6.5 to 4.9 +/- 2.8 cGy (rad) (P < 0.001).

137 s exceeding the MTA safety limit of 200 cGy (rads) to the blood or bone marrow in many patients with

138 raction between hus1+ and another checkpoint rad gene, rad1+.

139 uires rad3+ and each of the other checkpoint rad genes.

140 combination machinery and the six checkpoint rad gene products plus the Cdsl kinase.

141 UV at a level equivalent to some checkpoint rad mutants.

142 9 acts in a process involving the checkpoint rad genes and that the rhp9 mutant is phenotypically ver

143 x fission yeast genes, termed the checkpoint rad genes, which are essential for both the S-M and DNA

144 process which is defective in the checkpoint rad mutants and which involves hus5 .

145 s complex is dependent on a third checkpoint rad gene, rad9+, suggesting that these three proteins ma

146 Whereas checkpoint rad mutants fail to arrest division in response to DNA d

147 to a less severe defect seen in rad9 delta, rad 17 delta, and rad24 delta.

148 ned to be 7.62 +/- 0.15 (standard deviation) rad/ms.

149 in PCMH[Y384F] which produced PchF[Y384F]FAD(rad) x PchC, the mutant enzyme containing the flavin rad

150 n of unassociated PchC(ox) by PchF[Y384F]FAD(rad) x PchC.

151 Fl(rad)(*-) undergoes a homogeneous proton transfer reactio

152 )H(-) (E(0)(f) = -1.05 V vs Fc/Fc(+)) and Fl(rad)(*2-) (E(0)(f) = -1.62 V vs Fc/Fc(+)), respectively.

153 y one electron to form the radical anion (Fl(rad)(*-)) at E(0)(f) = -1.22 V versus Fc/Fc(+) (E(0)(f)

154 n rates, the homogeneous reaction between Fl(rad)(*-) and Fl(ox) begins to be outrun, which leads to

155 ciated with the one-electron reduction of Fl(rad)(*-) to form Fl(red)(2-) (E(0)(f) = -1.98 V vs Fc/Fc

156 the starting material (Fl(ox)) to produce Fl(rad)H(*) and Fl(ox)(-), which are both able to undergo f

157 omolog of the S. pombe rad1 checkpoint gene, rad-5, and him-7-block both DNA damage-induced apoptosis

158 k markers previously mapped on the IMpRH7000-rad SSC12 and porcine genetic maps.

159 r, relocalization appears more frequently in rad-5 mutants, suggesting that RAD-5 plays a role in rep

160 ce tumor-to-nontumor ratios and may increase rad doses to tumor more selectively than directly labele

161 pressure-dependent radiative conductivity, k(rad), from these data is 2-5 times lower than previously

162 The blood absorbed dose (cGy/37 MBq [rad/mCi] administered) was reduced from 2.54 +/- 0.91 (m

163 nstant alpha to be (-1 +/- 0.34) x 10(-10) N/rad(2); and 3) the ciliary base may be an essential regu

164 calculated to be (4.6 +/- 0.62) x 10(-12) N/rad and nonlinear spring constant alpha to be (-1 +/- 0.

165 ich a torsional spring constant of ~23 pN.nm/rad(2) is estimated on average for all S1 types, similar

166 s of kinks and cross-links of ~100-200 pN.nm/rad.

167 alternative repair pathway in the absence of rad-51 and independent of nonhomologous end joining (NHE

168 rror-prone DSB repair pathway independent of rad-51 and non-homologous end joining, raising the possi

169 ng HR but can also function independently of rad-51 in DSB repair processes.

170 to DNA damage and can do so independently of rad-51.

171 ion forks by RPA-1 and functions upstream of rad-5/clk-2 in the S-phase checkpoint.

172 tient-specific mean 90Y dose (cGy/37 MBq, or rad/mCi) was 0.53 (0.32-0.78) to whole body, 3.75 (0.63-

173 cell cycle events and coordinates with other rad/hus checkpoint gene products in responding to radiat

174 1 +/- 183 pN.mum(2) and ks = 39.3 +/- 6.0 pN/rad under the same conditions.

175 al dynein interactions ( approximately 35 pN/rad) to interdoublet sliding resistance in these immobil

176 ibutions of the N-DRCs ( approximately 40 pN/rad) and residual dynein interactions ( approximately 35

177 0 +/- 280 pNmum(2) and ks = 79.6 +/- 10.5 pN/rad, respectively.

178 7 +/- 184 pN.mum(2) and ks = 43.3 +/- 7.7 pN/rad.

179 e identification of homologs of the S. pombe rad checkpoint genes from mammals, arthropods, and nemat

180 Silencing requires rad-2, gei-17, and the polh-1 translesion DNA polymerase

181 over the second-generation EST SSC12 RH7000-rad map of 103 ESTs and 15 framework markers covering ap

182 The protein products of several rad checkpoint genes of Schizosaccharomyces pombe (rad1+

183 iciency eDf25, is allelic to rfs-1, the sole rad-51 paralog group member in C. elegans.

184 net (Philips) with SSH MRCP 3DHR and SSHMRCP rad protocol.

185 t that mutations in the smk-1 gene cause the rad-2 phenotype.

186 with defects in the DNA damage aspect of the rad checkpoint control pathway had an increased frequenc

187 We recently found that the rad-2 mutation alleviates this checkpoint silent DNA dam

188 course, known to radiology residents as the "rad-path" course, was not a casualty of the BRAC, the Am

189 Unlike rad-51 mutants, Cebrc-2 mutants also accumulate RPA-1 at

190 gnated mec1-srf for synthetic lethality with rad-fifty-two) simultaneously cause two types of phenoty

191 In this study, a fission yeast rad mutant (slp1-362) was genetically investigated.