ライフサイエンスコーパス: radiation damage

1 mall sublineages (one to four cells) without radiation damage.

2 is accelerated and/or synchronized following radiation damage.

3 ffract x-rays efficiently while withstanding radiation damage.

4 s limited by the thickness of the window and radiation damage.

5 ace images are obtained without irreversible radiation damage.

6 with micron-sized X-ray beams the effects of radiation damage.

7 photoelectrons are the predominant source of radiation damage.

8 tive stress, and regulation of recovery from radiation damage.

9 ses the recovery of Dr from ultraviolet (UV) radiation damage.

10 tantially alleviates the growth defect after radiation damage.

11 sample or limiting exposure times to prevent radiation damage.

12 could increase the expression and degree of radiation damage.

13 residue is required for effective repair of radiation damage.

14 RecA-dependent process during recovery from radiation damage.

15 e dimensions with resolution limited only by radiation damage.

16 owed that flavopiridol inhibited repair from radiation damage.

17 tization for several phenotypic endpoints of radiation damage.

18 may have related roles in the toleration of radiation damage.

19 unit, crystal variability and sensitivity to radiation damage.

20 have similar bone-protective effects against radiation damage.

21 d vessels did not show histological signs of radiation damage.

22 represents a novel defense mechanism against radiation damage.

23 S) that may constitute one-third of ionizing radiation damage.

24 d are defective at the G1/S checkpoint after radiation damage.

25 ATM might interact with c-Abl in response to radiation damage.

26 to predict the role of STGBs in annihilating radiation damage.

27 sten films plays a dominant role in reducing radiation damage.

28 e protein in the droplets and the absence of radiation damage.

29 new materials capable of withstanding severe radiation damage.

30 =O vibrational signatures with no observable radiation damage.

31 with X-rays, including the phase problem and radiation damage.

32 probe protein interactions while minimizing radiation damage.

33 on into materials with significantly reduced radiation damage.

34 igh temperatures, corrosive environments and radiation damage.

35 ue that exploits the susceptibility of NC to radiation damage.

36 acts form after injury to the eye, including radiation damage.

37 dD, reflecting its role in the DNA repair of radiation damage.

38 for 'aloof' spectroscopy that largely avoids radiation damage.

39 ucceeded by cryo-crystallography to mitigate radiation damage.

40 ructures under native conditions and without radiation damage.

41 hotons, better statistics, and lower overall radiation damage.

42 from hypoxia-reoxygenation, and against UVA radiation damage.

43 he only source of energy for the recovery of radiation-damage.

44 l understanding of the processes controlling radiation damage accumulation is necessary.

45 he development of agents that can ameliorate radiation damage after exposure to radiation has occurre

46 into the map during refinement and shows how radiation damage alters scattering from negatively charg

47 cell death is important for both mitigating radiation damage and alleviating the side effects of ant

48 Theoretical simulations of radiation damage and charge transport in DNA depend on a

49 TrpRS II is induced after radiation damage and contains an N-terminal extension si

50 FEL structure shows little to no evidence of radiation damage and is more complete than a model deter

51 ues of DJ-1, shows an extreme sensitivity to radiation damage and may be subject to other forms of ox

52 nces between Bijvoet mates despite extensive radiation damage and multi-crystal scaling; the pre-scre

53 emtosecond-duration x-ray pulses to minimize radiation damage and obtained a high-resolution room-tem

54 a viable solution, though many are prone to radiation damage and plagued with temperature instabilit

55 We investigated the effects of radiation damage and sample preparation on the bacteria

56 somatostatin analogs, both by mitigation of radiation damage and the currently observed reduction of

57 rtant underlying biological processes in the radiation damage and tissue repair process.

58 g sufficiently large crystals that withstand radiation damage and yield high-resolution data at synch

59 d as having an unspecified role in repair of radiation damage and, more specifically, DNA double-stra

60 Grand Canyon apatites of differing He date, radiation damage, and U-Th zonation yield a self-consist

61 ing that the in vivo macrophage responses to radiation damage are genetically modified processes.

62 a dose of 33 MGy, no signs of X-ray-induced radiation damage are visible in this integral membrane p

63 polar condensed media as well as biological radiation damage arising from dissociative electron atta

64 ce of reliable structural information due to radiation damage artifacts caused by the intense synchro

65 physicochemical and chemical stages of early radiation damage at the scale of an entire human genome

66 Here, we examine trends in radiation damage behaviour for families of compounds rel

67 diation, most likely by reducing exposure of radiation-damaged breast cells to stimulating effects of

68 nd avoid aggregation, concentration effects, radiation damage, buffer mismatch and other common probl

69 parameters with experimental observations of radiation damage buildup remains elusive.

70 lineated the cellular response of MR-1 to UV radiation damage by analyzing the transcriptional profil

71 Lattice defects generated by radiation damage can diffuse to grain boundaries (GBs) a

72 us targeting and phosphorylation in ionizing radiation-damaged cells, whereas UV light-induced 53BP1

73 Ionizing radiation damages chromosomal DNA and activates p53-depe

74 -phosphatidylcholines were most sensitive to radiation damage compared to the ester- and ether-linked

75 rinciple of protein's greater sensitivity to radiation damage compared with that of nucleic acid.

76 e study of biological samples, provided that radiation damage could be prevented.

77 ronology and U-Th-Pb dating is the effect of radiation damage, created by alpha-recoils from alpha-de

78 However, radiation damage currently limits the application of SAX

79 Ionizing radiation damages DNA in several ways, including through

80 is required for expression of an ultraviolet radiation-damaged DNA binding activity and is disrupted

81 f several factors also involved in repair of radiation-damaged DNA, including the DNA-dependent prote

82 e fibre layer following occipital lobe/optic radiation damage due to stroke.

83 -Mn and Mn-Ca distances are less affected by radiation damage due to the their heavy masses, while on

84 bacterial cell wall membrane did not sustain radiation damage during STEM imaging at low electron dos

85 Here we use molecular modeling to map the radiation damage during the 10-50 fs to the intensity, t

86 Surprisingly, radiation damage effects revealed by x-ray diffraction w

87 e highly durable and especially resistant to radiation damage effects.

88 Ion beams create radiation damage efficiently without material activation

89 Understanding and predicting radiation damage evolution in complex materials is cruci

90 mplex oxides is critical to ionic transport, radiation damage evolution, sintering, and aging.

91 Preliminary radiation damage experiments substantiate the prediction

92 contrast to proteins, there is no spread of radiation damage far from the primary site of ionization

93 al questions about the detailed mechanism of radiation damage formation remain largely unanswered.

94 of SiC and Si, are analyzed with a model of radiation damage formation which accounts for the fracta

95 uction nature of XFEL experiments provides a radiation-damage-free view of the functionally important

96 nsible for eliminating most ultraviolet (UV) radiation damage from DNA, as well as base alterations c

97 dues arise from their greater sensitivity to radiation damage from electron irradiation as determined

98 high-resolution data with minimal effects of radiation damage from sub-10-mum crystals of membrane an

99 Radiation damage from the alpha-particle decay of Pu cre

100 We postulate that this effect is caused by radiation damage from the tracer dose during dosimetry.

101 ional, elementally selective imaging without radiation damage, has had a revolutionary impact in many

102 Efforts to prevent or mitigate radiation damage have included development of antioxidan

103 We propose a novel mechanism of radiation damage healing in metals, which may guide furt

104 Ultraviolet radiation damages human skin and results in an old and w

105 ff of high-resolution information content by radiation damage in a dose-dependent manner.

106 We propose that radiation damage in a few mitochondria is transmitted vi

107 lectrons are the most important component of radiation damage in biological environments because they

108 The formation of stable radiation damage in crystalline solids often proceeds vi

109 The formation of radiation damage in Ge above room temperature is dominat

110 The buildup of radiation damage in ion-irradiated crystals often depend

111 giant surface-to-volume ratio may alleviate radiation damage in irradiated metallic materials as fre

112 ring meiotic recombination, during repair of radiation damage in mature oocytes, and in proliferating

113 direct experimental demonstration of reduced radiation damage in protein crystals with small beams, d

114 Effects of the collision cascade density on radiation damage in SiC remain poorly understood.

115 e, fused Purkinje neurons increase following radiation damage in the developing cerebellum.

116 Structural changes induced by radiation damage in X-ray crystallography hinder the abi

117 p21(-/-) mice had significantly less radiation damage, including 6-fold less scarring, 40% in

118 ng a modified linear quadratic model for the radiation damage, incorporating the effects of hypoxia a

119 ation was aided by the novel technique of UV radiation damage-induced phasing.

120 Radiation damage is a major cause of failure in macromol

121 Radiation damage is a major limitation in crystallograph

122 By contrast, below 35 K the radiation damage is frozen in place, permitting the evol

123 These results demonstrate that substantial radiation damage is likely to have occurred during X-ray

124 cal stretcher, no focusing is required, thus radiation damage is minimized and the surface forces are

125 us checkpoint gene products in responding to radiation damage is proposed.

126 undamental property in predicting cumulative radiation damage is the number of atoms permanently disp

127 e microscopy is intrinsically limited by the radiation damage it causes and the degree to which it al

128 The elimination of ultraviolet (UV) radiation-damaged keratinocytes via apoptosis is an impo

129 Radiation damage led to disruption of inter-ring contact

130 Histological analyses demonstrated that radiation damage led to less bone regeneration.

131 Graphene's ability to reduce radiation damage levels to hydrogen bond breakage is dem

132 an experimental strategy that optimizes the radiation damage lifetime of the crystal, or to assign a

133 mode and an exposure beyond the traditional radiation damage limit.

134 n be used to achieve high resolution, beyond radiation damage limits for biological samples.

135 The radiation damage may induce conformational changes of th

136 t-enhanced CT had an increased amount of DNA radiation damage (mean increase +/- standard error of th

137 s that measure close to 5,000 atoms/alpha in radiation-damaged natural zircons.

138 The x-ray exposure at which significant radiation damage occurs has been quantified for frozen c

139 ought that the induced charge and associated radiation damage of atoms in polyatomic molecules can be

140 This methodology minimizes the radiation damage of beam-sensitive materials, such as mi

141 some mechanistic insight into the effects of radiation damage on DNA, and (2) overcomes specific tech

142 lectron lasers (XFELs) reduce the effects of radiation damage on macromolecular diffraction data and

143 and destroy" approach before the effects of radiation damage on the data become significant.

144 Using an alternate test of DNA repair, i.e., radiation-damaged or undamaged reporter DNA, we introduc

145 cer clonal evolution (potentially induced by radiation damage), or is due to an innately aggressive t

146 Besides fundamental limits imposed by radiation damage, poor detectors and beam-induced sample

147 accharomyces cerevisiae to tolerate ionizing radiation damage requires many DNA-repair and checkpoint

148 atom structural and chemical analysis of all radiation-damage-resistant atoms present in, and on top

149 sis, sensing, actuation, supercapacitors and radiation-damage-resistant materials.

150 In maize (Zea mays) leaves, UV-B radiation damages ribosomes by crosslinking cytosolic ri

151 ylation is catalyzed by PP1, we asked if the radiation damage signal to Nek2 was mediated by PP1.

152 prevents autoimmune responses to ultraviolet-radiation damaged skin.

153 nificantly and consistently downregulated in radiation-damaged skin.

154 lacements per atom (DPA) unit in quantifying radiation damage (such as inadequacy in quantifying degr

155 zation, require extremely high resistance to radiation damage, such as resistance to amorphization or

156 onization of heavy atoms increases the local radiation damage that is seen in the diffraction pattern

157 low symmetry, and that the residue-specific radiation damage that occurs with increasing electron do

158 this beta-emitting scout dose could inflict radiation damage, the extent of which we aimed to quanti

159 tructural and nanoscale features to mitigate radiation damage, this study demonstrates enhancement of

160 -dihydrothymidine (dHT), formed via ionizing radiation damage to 2'-deoxycytidine and thymidine, resp

161 ribose sugar in the polymer chain restricts radiation damage to a small region and prevents major en

162 omposition, and sufficient stability against radiation damage to allow for multiple images to be obta

163 se recruitment of BMDCs into the cerebellum, radiation damage to cerebellar cells, or both, increase

164 9 of 9 assessable animals without detectable radiation damage to critical organs, including bone marr

165 -2'-deoxyuridine (dHdU), formed via ionizing radiation damage to cytosine under anoxic conditions and

166 produced in significant amounts by ionizing radiation damage to cytosine under anoxic conditions.

167 f potential synergistic interactions between radiation damage to DNA and oxidative stress-induced dam

168 ex protein signalling systems that recognise radiation damage to DNA and plasma membrane lipids.

169 SC-'236 did not appreciably alter radiation damage to jejunal crypt cells and tissue invol

170 nd therapy as it substantially minimises the radiation damage to non-tumour cells of healthy tissues.

171 vivo half-lives result in poor contrast and radiation damage to normal tissue.

172 ovides a basis for new approaches to prevent radiation damage to the bowel.

173 rs in the irradiated group suggests possible radiation damage to the pituitary, with consequent reduc

174 bound water molecules in the early stages of radiation damage to the protein crystal.

175 on, meiosis, and repair of gaps and ionizing radiation damage to the same extent as rad51.

176 Radiation damage to the whole lung is a serious risk in

177 tudy, we examined whether the enhancement of radiation damage to tumors by TPZ can be predicted from

178 ute to our understanding of the mechanism of radiation damage, to our appreciation of the importance

179 Nanomaterials have been proposed as radiation damage tolerant materials, due to the hypothes

180 cells (BMDCs) into the tumors, restoring the radiation-damaged vasculature by vasculogenesis and ther

181 Susceptibility to radiation damage was determined by irradiating operators

182 based on detecting gas bubbles generated by radiation damage was used to localize internal proteins

183 on and subsequent proton motion can mitigate radiation damage when heavier atoms absorb X-rays.

184 Biological specimens suffer radiation damage when imaged in an electron microscope,

185 may undergo loss of immunoreactivity due to radiation damage when labeled with large amounts of 131I

186 Low-energy electrons do not induce radiation damage, which enables acquiring subnanometer r

187 -ray free electron laser sources to overcome radiation damage, while sample consumption is dramatical

188 sed MultiScale Approach to the assessment of radiation damage with ions gives a positive answer to th

189 diffraction information before the onset of radiation damage, yet the majority of structures solved