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1 ifferentiate recurrent GBM from EBRT-induced radiation necrosis.
2  tumor and treatment-induced changes such as radiation necrosis.
3 ver, none required further surgery to debulk radiation necrosis.
4 and for distinguishing tumor recurrence from radiation necrosis.
5 valence of debulking surgery for symptomatic radiation necrosis.
6 r distinguishing brain tumor recurrence from radiation necrosis.
7 nts with recurrent GBM than in patients with radiation necrosis.
8 nts with recurrent GBM than in patients with radiation necrosis.
9 up that received SRS alone were diagnosed as radiation necrosis.
10 ing glioblastoma from lower-grade tumors and radiation necrosis; (2) By what other investigators have
11  classification problems: distinguishing (1) radiation necrosis, a benign yet confounding effect of r
12 fficult to distinguish tumor recurrence from radiation necrosis after brain tumor therapy.
13 iating local recurrent brain metastasis from radiation necrosis after radiation therapy because the u
14 er for distinguishing viable malignancy from radiation necrosis and predicting tumor response to ther
15 the varying spatial and temporal patterns of radiation necrosis at MR imaging.
16                                      Delayed radiation necrosis at the treated site developed in 20 p
17 ferentiate viable glioma (hyperintense) from radiation necrosis (hypointense to isointense) by APT MR
18 ng-detected abnormalities of the brain: pure radiation necrosis in 20 patients, a mixture of predomin
19 + glioma and 9L gliosarcoma) with a model of radiation necrosis in rats, we could clearly differentia
20 er survival and slightly higher incidence of radiation necrosis in the high-dose RT arm.
21 gnostic dilemma of recurrent neoplasm versus radiation necrosis is addressed in this study through a
22 nt metastasis (n = 19) than in patients with radiation necrosis (n = 21) (TBR(max), 3.2 +/- 0.9 vs. 2
23 than 20% of resected tissue) in 16 patients, radiation necrosis of the cranial nerves and/or their pa
24 prior attempt at closure, trauma, infection, radiation necrosis, or tumor resection.
25  edema had a minimal effect on MT ratio, and radiation necrosis showed prominent reductions in MT rat
26 e 2-year actuarial incidence of grade 3 to 5 radiation necrosis was 2.5% with low-dose RT and 5% with
27                                              Radiation necrosis was generally distinguishable from tu
28 s in 20 patients, a mixture of predominantly radiation necrosis with limited recurrent and/or residua

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