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1 ifferentiate recurrent GBM from EBRT-induced radiation necrosis.
2 tumor and treatment-induced changes such as radiation necrosis.
3 ver, none required further surgery to debulk radiation necrosis.
4 and for distinguishing tumor recurrence from radiation necrosis.
5 valence of debulking surgery for symptomatic radiation necrosis.
6 r distinguishing brain tumor recurrence from radiation necrosis.
7 nts with recurrent GBM than in patients with radiation necrosis.
8 nts with recurrent GBM than in patients with radiation necrosis.
9 up that received SRS alone were diagnosed as radiation necrosis.
10 ing glioblastoma from lower-grade tumors and radiation necrosis; (2) By what other investigators have
11 classification problems: distinguishing (1) radiation necrosis, a benign yet confounding effect of r
13 iating local recurrent brain metastasis from radiation necrosis after radiation therapy because the u
14 er for distinguishing viable malignancy from radiation necrosis and predicting tumor response to ther
17 ferentiate viable glioma (hyperintense) from radiation necrosis (hypointense to isointense) by APT MR
18 ng-detected abnormalities of the brain: pure radiation necrosis in 20 patients, a mixture of predomin
19 + glioma and 9L gliosarcoma) with a model of radiation necrosis in rats, we could clearly differentia
21 gnostic dilemma of recurrent neoplasm versus radiation necrosis is addressed in this study through a
22 nt metastasis (n = 19) than in patients with radiation necrosis (n = 21) (TBR(max), 3.2 +/- 0.9 vs. 2
23 than 20% of resected tissue) in 16 patients, radiation necrosis of the cranial nerves and/or their pa
25 edema had a minimal effect on MT ratio, and radiation necrosis showed prominent reductions in MT rat
26 e 2-year actuarial incidence of grade 3 to 5 radiation necrosis was 2.5% with low-dose RT and 5% with
28 s in 20 patients, a mixture of predominantly radiation necrosis with limited recurrent and/or residua
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