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1 nhibition of PPP canceled the UCHL1-mediated radioresistance.
2 M-MDSC infiltration, which results in tumor radioresistance.
3 of VEGF expression, tumor angiogenesis, and radioresistance.
4 whereas Nrf2-inducing drugs did not increase radioresistance.
5 s, invasion, metastasis, chemoresistance and radioresistance.
6 one Puma allele is sufficient to confer mice radioresistance.
7 ved macrophages (BMDMvarphi) increased tumor radioresistance.
8 ized by a high degree of chemoresistance and radioresistance.
9 epair, implicating the PI3K/Akt-1 pathway in radioresistance.
10 been suggested to play a major role in tumor radioresistance.
11 enic progeny, which may contribute to tumour radioresistance.
12 nisms that mediate HIF-1 activation and thus radioresistance.
13 osphorylate downstream substrates and confer radioresistance.
14 to de novo and acquired chemoresistance and radioresistance.
15 mined its role in mesenchymal transition and radioresistance.
16 esis and HSP70 expression is associated with radioresistance.
17 and tensin homologue (PTEN), correlates with radioresistance.
18 n to inhibit the development of LDIR-induced radioresistance.
19 eath, whereas decreasing their levels causes radioresistance.
20 , but these tumors are often associated with radioresistance.
21 a, and cyclin B1 and diminished the adaptive radioresistance.
22 Many melanomas exhibit high levels of radioresistance.
23 of Akt phosphorylation, a known pathway for radioresistance.
24 n of HDJ-2, whereas overexpression conferred radioresistance.
25 eptor (EGFR) activity and is associated with radioresistance.
26 iotherapy remains only palliative because of radioresistance.
27 combine with BCR ligands to promote B-1 cell radioresistance.
28 ing a cytoprotective response and subsequent radioresistance.
29 ivation of ATM, Chk1, and Chk2, and impaired radioresistance.
30 be responsible for the observed increase in radioresistance.
31 SCs are believed to contribute to chemo- and radioresistance.
32 a mimicked the effects of Ras-CM to increase radioresistance.
33 cells, transformed fibroblasts and mediated radioresistance.
34 e to that of miR-24 in NPC tumorigenesis and radioresistance.
35 effectors, was sufficient to confer partial radioresistance.
36 ctors, was also sufficient to confer partial radioresistance.
37 ne with mutant BRCA2 was linked to increased radioresistance.
38 n-mediated cytotoxicity and thereby to tumor radioresistance.
39 emonstrating that IGF-IR directly influences radioresistance.
40 xtranuclear mechanism for the development of radioresistance.
41 large part, responsible for the deinococci's radioresistance.
42 ising therapeutic strategy for reversing NPC radioresistance.
43 ma is an essential mediator of FGFR1-induced radioresistance.
44 ic responses exerts an independent effect on radioresistance.
45 in, RNF8, which is required for ATDC-induced radioresistance.
46 ting this interaction abrogated ATDC-induced radioresistance.
47 king advantage of the dynamic instability of radioresistance.
48 ing biological processes that promote glioma radioresistance.
49 H1299 and NCI-H460, with different levels of radioresistance.
50 subset of hypoxic cells that determine tumor radioresistance.
51 inhibited DNA damage responses, and enhanced radioresistance.
52 hich are often characterized by a pronounced radioresistance.
53 pregulated by IR and have been implicated in radioresistance.
54 ing axis in NPC tumor cell growth as well as radioresistance.
55 itically contributes to NPC tumor growth and radioresistance.
56 nt samples and its roles in tumor growth and radioresistance.
57 rticles to understand possible mechanisms in radioresistance.
58 ions reveal that in addition to their higher radioresistance, a shift from asymmetric to symmetric di
60 cinoma MCF+FIR cells that showed a transient radioresistance after exposure chronically to fractionat
61 cellular proliferation, carcinogenesis, and radioresistance, all known to be regulated by the activa
62 ession of c-JUN in MDA-MB-231 cells promoted radioresistance and abrogated miR-125-mediated radiosens
63 adhesion to fibronectin enhanced tumor cell radioresistance and attenuated the cytotoxic and radiose
64 ver, whether specific miRNAs regulate tumour radioresistance and can be exploited as radiosensitizing
66 the cellular population that confers glioma radioresistance and could be the source of tumour recurr
67 ng radiation therapy may contribute to their radioresistance and could be therapeutically targeted.
68 ur cells associated with therapy resistance (radioresistance and drug resistance) are likely to give
70 X phosphorylation and is required for normal radioresistance and efficient accumulation of DNA-damage
71 the importance of PI3K in mediating enhanced radioresistance and have implicated PI3K as a potential
75 end-joining (NHEJ) pathway is essential for radioresistance and lymphocyte-specific V(D)J (variable
76 cell lines suggests a distinct mechanism of radioresistance and may represent a critical factor in d
77 urthermore, the previously observed enhanced radioresistance and mutability in WTK1 cells must be att
78 ing radiation (IR) is essential for cellular radioresistance and nonhomologous-end-joining-mediated D
79 f rectal cancer miRs may yield biomarkers of radioresistance and offer treatment targets for resensit
80 0-Gy IR course was associated with increased radioresistance and proliferation, suggesting a role for
81 ponse in cancer stem cells may overcome this radioresistance and provide a therapeutic model for mali
82 herefore drives a pathway that enhances HSPC radioresistance and radiation-induced B-cell malignancie
83 ence for the influence of IGF-IR on cellular radioresistance and response to therapy and raise the po
84 damage signaling pathways contribute to GSC radioresistance and that combined inhibition of cell-cyc
86 icate that ALDH(+) cells contribute to tumor radioresistance and their molecular targeting may enhanc
88 We examined whether PTEN deficiency leads to radioresistance and whether this can be reversed by nelf
90 High levels of wild-type IGF-IR conferred radioresistance, and mutational analysis revealed that t
91 autocrine signaling through EGFR to increase radioresistance, and the EGFR KI GW572016 acts as a radi
93 elomere stability, population doubling time, radioresistance, and tumorigenicity in a mouse xenograft
96 though not all of the signaling pathways for radioresistance are well defined, it is now clear that R
98 ed a specific role for FGFR1 in glioblastoma radioresistance as modeled by U87 and LN18 glioblastomas
99 , by using mutant I kappa B alpha, inhibited radioresistance as well as reducing steady-state levels
100 embryo fibroblast cell line, shows much more radioresistance associated with a much stronger G(2) arr
101 offers a facile approach for overcoming the radioresistance associated with poorly oxygenated cells.
102 emical inhibitors substantially reversed the radioresistance-associated increase in cell migration.
103 cted to elucidate the molecular mechanism of radioresistance-associated metastatic potential of cervi
104 ctive biomarkers and molecular mechanisms of radioresistance bears promise to improve cancer therapie
105 n taxonomic classification, genome size, and radioresistance between cell types studied here support
106 ransformation, in that PI3-K is required for radioresistance but not transformation, whereas MEK and
107 To test this hypothesis, we investigated radioresistance by treating primary BALB/c mouse mammary
109 Finally, we demonstrate that Ras-mediated radioresistance can be uncoupled from Ras-mediated trans
111 cued the decreased checkpoint activation and radioresistance caused by L1CAM knockdown, demonstrating
114 adder cancer cells increased tumorigenicity, radioresistance, degeneration of reactive oxygen species
115 HK18-IR cells showed increased clonogenic radioresistance [dose-modifying factor (DMF), 1.47], red
116 Since these responses contribute to cellular radioresistance, ERBB1, the most extensively studied ERB
122 t, tumor cell fate, DNA damage response, and radioresistance in a subset of HOXB9-overexpressing brea
124 ed approach to investigate the mechanisms of radioresistance in cancer cells and help guide improveme
126 that high Wnt signalling is associated with radioresistance in colorectal cancer (CRC) cells and int
127 olecular mechanism of Wnt signalling-induced radioresistance in CRC and ISCs, and further unveils the
128 y uncovers a previously unknown mechanism of radioresistance in CRPC, which can be therapeutically re
130 termine the mechanisms by which Ras mediates radioresistance in epithelial cells, we assessed the imp
132 up of NF-kappaB target genes are involved in radioresistance in FIR-treated tumor cells with inactive
133 of concept that FGFR1 targeting can degrade radioresistance in glioblastoma, a widespread problem in
135 s mesenchymal (MES) transdifferentiation and radioresistance in glioma stem cells (GSCs), but molecul
138 hondrial MKP1 was responsible for conferring radioresistance in HER2-overexpressing breast cancer cel
141 ular target to overcome a novel mechanism of radioresistance in KRAS-mutant tumor cells, which stands
143 as necessary or sufficient for Ras-mediated radioresistance in matched pairs of RIE-1 rat intestinal
149 anism was further confirmed by the increased radioresistance in PrxII-overexpressing MCF+FIS4 cells w
152 sion and activity frequently correlates with radioresistance in several cancers, including non-small
159 Ras-dependent signaling pathways involved in radioresistance include those mediated by phosphatidylin
160 mechanisms, which link tumor progenitors to radioresistance, including activation of the WNT/beta-ca
167 ly be used to radiosensitize tumors in which radioresistance is dependent on Ras-driven autocrine sig
172 es show that the ability of BRCA1 to promote radioresistance is restricted to the late S and G2 phase
175 eptor fusion protein (Enbrel), revealed that radioresistance mediated by BMDMvarphi required intact T
176 aB is responsible for a major portion of the radioresistance observed in a cell population (HK18-IR)
179 ed as a novel HIF-1 activator, increased the radioresistance of cancer cells by producing an antioxid
180 taple approach for cancer treatment, whereas radioresistance of cancer cells remains a substantial cl
186 proliferation may contribute to the clinical radioresistance of GBM tumors, the combination of rapamy
189 ugments DNA damage checkpoint activation and radioresistance of GSCs through L1-ICD-mediated NBS1 upr
190 that selective cellular quiescence increases radioresistance of human cell lines in vitro and mice in
192 for cell-fibronectin interactions to induce radioresistance of human non-small cell lung cancer cell
194 mice has two components: an increase in the radioresistance of individual cells and, to a lesser ext
197 s self-renewal, motility, tumorigenesis, and radioresistance of MES GSCs via a loss of the MES signat
199 he genesis, malignant progression, and chemo/radioresistance of multiple human malignancies, includin
200 icine (TOM) compound was found to reduce the radioresistance of NSCLC by inhibiting the nuclear facto
202 t may partly account for the relatively high radioresistance of plants versus yeast and mammals.
203 cells, which is consistent with the relative radioresistance of S-phase cells as measured with cell s
205 anged balance and mechanisms of differential radioresistance of T cell subsets after graded doses of
207 ) gene substantially increased the intrinsic radioresistance of the DNA-PKcs(-/-) tumor cells and sub
208 antation experiments show that the increased radioresistance of the transgenic animals is provided by
210 astoma and locally advanced lung cancer, but radioresistance of these two types of cancer remains a s
212 t meiosis, which contrasts with its reported radioresistance of up to 1,000 Gy in survival assays.
214 mutant I kappa B alpha was unable to inhibit radioresistance or reduce 14-3-3 zeta, GADD153, cyclin A
215 tion of NF-kappaB with other key elements in radioresistance, particularly with respect to extracellu
216 on-induced Notch-1 signaling associated with radioresistance possibly via miR-34a-mediated pathways.
217 hat YAP accelerates tumor growth and confers radioresistance, promoting ongoing proliferation after r
222 ray) in human keratinocytes with an adaptive radioresistance that can be inhibited by short interferi
224 molecular pathways of LDIR-induced adaptive radioresistance, the transcription factor nuclear factor
225 Despite the implication of Wnt signalling in radioresistance, the underlying mechanisms are unknown.
226 ecause quiescence is usually associated with radioresistance, these cell kinetic changes suggest that
227 hus, whereas Raf contributes to Ras-mediated radioresistance, this is accomplished through a MEK-inde
228 e development of in vitro isogenic models of radioresistance through exposure to fractionated radiati
229 that cancer stem cells contribute to glioma radioresistance through preferential activation of the D
230 lular heterogeneity and dynamically acquired radioresistance to predict the effectiveness of differen
231 ome a prerequisite for overcoming chemo- and radioresistance using radiosensitizing drugs or hypoxia-
233 ncreased radiosensitivity, whereas increased radioresistance was observed in human breast cancer MCF-
235 ated with radiotherapy, can induce increased radioresistance, we have asked which Ras effector pathwa
236 of cIAP1/cIAP2 in lymph node metastasis and radioresistance, we use an in vitro pre-clinical model a
237 tly test the link between Akt activation and radioresistance, we utilized PTEN-deficient U251 gliobla
238 ntaining BRCA1 showed both increased TCR and radioresistance, whereas cells without BRCA1 showed decr
239 nner, indicating that PI3-K is necessary for radioresistance, whereas inhibition of NF-kappaB with th
240 e the CRAF/CHK2 association enhancing tumour radioresistance, while an allosteric CRAF inhibitor sens
241 verexpression of wild-type Artemis increased radioresistance, while D165N overexpression conferred pa
243 Caffeine or UCN-01 abolishes the extreme radioresistance with the strong G(2) arrest and at the s
244 demonstrate an association of acquired tumor radioresistance with up-regulation of components of the
245 he development of drugs that can reduce that radioresistance would potentiate the efficacy of radiati
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