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1 cuit between the red nucleus and the nucleus raphe magnus.
2 fos was significantly greater in the nucleus raphe magnus.
3 ular nucleus pars alpha, raphe obscurus, and raphe magnus.
4 current in brainstem neurons in the nucleus raphe magnus.
5 pha2-adrenoceptors on neurons in the nucleus raphe magnus.
6 nd just dorsal to the pyramidal tract in the raphe magnus.
7 nkephalin (DAMGO) was microinjected into the raphe magnus, a manipulation that selectively activates
8 ts located in the PAG itself, in the nucleus raphe magnus and adjacent structures, and in the spinal
9 ympathetic premotor neurons lying within the raphe magnus and gigantocellular reticular nucleus, pars
10 echolamine-containing neurons in the nucleus raphe magnus and locus coeruleus that project to the spi
11 d in the reticular formation adjacent to the raphe magnus and obscurus nuclei, in the gigantocellular
12 whereas it was significantly greater in the raphe magnus and pallidus nuclei in the double stimulati
13 , which includes the nucleus raphe obscurus, raphe magnus and raphe pallidus (NRP), is involved in re
14 ites of serotonergic cells are restricted to raphe magnus and the ventral part of nucleus reticularis
15 nificant respiratory effect when stimulating raphe magnus and/or locus ceruleus, but became critical
16 ntomedullary junction within raphe obscurus, raphe magnus, and gigantocellular nucleus pars alpha.
17 lateral amygdala, ventral cochlear nucleus, raphe magnus, and in the ventral horn of thoracic spinal
19 ucleus gigantocellularis pars alpha, nucleus raphe magnus, and nucleus raphe pallidus); 6) the ventro
21 phe obscurus-nucleus raphe pallidus, nucleus raphe magnus, and rostral and caudal ventrolateral medul
22 experiments demonstrate that raphe dorsalis, raphe magnus, and spinal dorsolateral funiculus lesions
28 ral ventrolateral medulla (RVLM) and nucleus raphe magnus (NRM) appear to originate from neurones wit
29 ed hyperalgesia, is dependent on the nucleus raphe magnus (NRM) but independent of the nucleus reticu
32 s and their interaction with MORs in nucleus raphe magnus (NRM) neurons important for opioid analgesi
33 jection of L-glutamate in either the nucleus raphe magnus (NRM) or the nucleus reticularis gigantocel
35 nd immunoreactivity of ACV/VI in the nucleus raphe magnus (NRM), a brainstem site critically involved
37 ects in neurons of the rat brainstem nucleus raphe magnus (NRM), a key supraspinal site for opioid an
38 ventral medulla (RVM), including the nucleus raphe magnus (NRM), nuclei reticularis gigantocellularis
43 orbital and infralimbic) regions and in the raphe magnus nucleus in quinpirole-sensitized rats (P<0.
45 ole alone (P<0.05), was not decreased in the raphe magnus nucleus, and was decreased in the piriform
46 TPH and SERT immunoreactivity in neurons of raphe magnus, obscurus, and pallidus and SERT in the neu
48 olateral and lateral PAG or into the nucleus raphe magnus or by direct application of naloxone (50 mi
49 r 3.0 micrograms of CGP 35348 in the nucleus raphe magnus or nucleus reticularis gigantocellularis pa
50 r CGP 35348 was microinjected in the nucleus raphe magnus or nucleus reticularis gigantocellularis pa
51 rbachol (5 microg) into sites in the nucleus raphe magnus or the nucleus gigantocellularis pars alpha
52 rigeminal nucleus, reticular nuclei, nucleus raphe magnus, pontine blink premotor area, and superior
53 etion of serotonergic neurons in the nucleus raphe magnus, raphe obscurus, raphe pallidus, and ventro
54 inal trigeminal, nucleus tractus solitarius, raphe magnus, raphe pallidus, and the rostral and caudal
55 y reticular formation, Barrington's nucleus, raphe magnus, raphe pallidus, subcoeruleus pars alpha, l
56 Serotonergic cells in the medullary nucleus raphe magnus (RM) and adjacent nucleus reticularis magno
57 diated through a relay in the pontomedullary raphe magnus (RM) and adjacent nucleus reticularis magno
59 ulation of medullary neurons, located within raphe magnus (RM) and the neighboring reticular nuclei,
62 whether serotonergic cells in the medullary raphe magnus (RM) receive baroreceptor input, cells were
63 chanism in the periaqueductal gray (PAG) and raphe magnus (RM) to stimulate descending, analgesic cir
64 wo neuronal populations within the medullary raphe magnus (RM): opioids silence pain-facilitatory ON
65 f the following sites: n. raphe obscurus, n. raphe magnus, rostral and caudal ventrolateral medulla,
66 within the periaqueductal gray, the nucleus raphe magnus, the gigantocellular reticular nucleus, and
67 ys or more, the locus coeruleus, the nucleus raphe magnus, the nucleus paragigantocellularis, pars al
68 show here that in the rat brainstem nucleus raphe magnus, which is important for central mechanisms
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