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1 the longest cDNA clones obtained by 5'-RACE (rapid amplification of cDNA ends).
2 ain reaction (RT-PCR) and by 5' and 3' RACE (rapid amplification of cDNA ends).
3 ts were detected by 5' and 3' RACE analysis (rapid amplification of cDNA ends).
4 sue growth factor) as a probe and by 5'RACE (rapid amplification of cDNA ends).
5 tained and verified through RT-PCR and RACE (rapid amplification of cDNA ends).
6 d mapped its transcription start sites by 5'-rapid amplification of cDNA ends.
7 MEC cDNA expression library as well as by 5' rapid amplification of cDNA ends.
8 equencing, reverse transcription-PCR, and 5'-rapid amplification of cDNA ends.
9 from the start codon, was determined by 5'- rapid amplification of cDNA ends.
10 eneration a full-length cDNA using 5' and 3' rapid amplification of cDNA ends.
11 on with degenerate primers and by 5'- and 3'-rapid amplification of cDNA ends.
12 IA (TFIIIA) was cloned by degenerate PCR and rapid amplification of cDNA ends.
13 ed by primer extension, as well as 5' and 3' rapid amplification of cDNA ends.
14 the expressed sequence tag and performing 5'-rapid amplification of cDNA ends.
15 nicum using reverse transcription PCR and 3'-rapid amplification of cDNA ends.
16 ranscription start site was determined by 5'-rapid amplification of cDNA ends.
17 ne and retrieval of the remaining cDNA by 5' rapid amplification of cDNA ends.
18 quence tags, PCR of human heart cDNA, and 5'-rapid amplification of cDNA ends.
19 se searching, polymerase chain reaction, and rapid amplification of cDNA ends.
20 ly spliced cDNA (U16+), was identified by 5' rapid amplification of cDNA ends.
21 use transcript sequence was determined using rapid amplification of cDNA ends.
22 DNA clones isolated by library screening and rapid amplification of cDNA ends.
23 as mapped by tiled array and confirmed by 5' rapid amplification of cDNA ends.
24 the missing 3' sequences were obtained by 3'-rapid amplification of cDNA ends.
25 PC-TP was cloned by library screening and 5'-rapid amplification of cDNA ends.
26 cription-polymerase chain reaction and 5'/3'-rapid amplification of cDNA ends.
27 thern blots (3.7 kb) was determined using 3' rapid amplification of cDNA ends.
28 anscriptase-polymerase chain reaction and 3' rapid amplification of cDNA ends.
29 human WIN cDNAs by library screening and 5'-rapid amplification of cDNA ends.
30 sozyme (RMPK) was cloned using the method of rapid amplification of cDNA ends.
31 from the purified protein and by 5'- and 3'-rapid amplification of cDNA ends.
32 nal regions of the genome were determined by rapid amplification of cDNA ends.
33 c fos clone using degenerate PCR followed by Rapid Amplification of cDNA Ends.
34 nt virus mRNA molecules was determined by 3' rapid amplification of cDNA ends.
35 using degenerate oligonucleotides, PCR, and rapid amplification of cDNA ends.
36 cDNA were subsequently captured by 5' and 3' rapid amplification of cDNA ends.
38 etermined by both S1 nuclease mapping and 5' rapid amplification of cDNA ends (5' RACE) to be located
46 on a partial amino acid sequence, 5'- and 3'-rapid amplification of cDNA ends allowed the generation
47 Furthermore, both primer extension and 5'-rapid amplification of cDNA ends analyses identified mul
56 rn blotting, phage library screening, and 5' rapid amplification of cDNA ends analysis in the region
63 a combination of cDNA library screening and rapid amplification of cDNA ends analysis, a novel human
64 the transcription start point (TSP) using 5' rapid amplification of cDNA ends analysis, the 5' promot
71 the gene trap transcripts are obtained by 5' rapid amplification of cDNA ends and are sequenced to de
72 The missing 5' sequences were obtained by 5'-rapid amplification of cDNA ends and by analysis of an N
74 homolog was isolated by a combination of 5'-rapid amplification of cDNA ends and direct polymerase c
75 h Mrf2 cDNA by cDNA library screening and 5' rapid amplification of cDNA ends and identified two isof
77 l-treated human THP-1 cells together with 5'-rapid amplification of cDNA ends and polymerase chain re
79 criptional start sites were identified by 5'-rapid amplification of cDNA ends and primer extension.
81 start site (+1), which was determined by 5' rapid amplification of cDNA ends and RNase protection as
82 upstream from the translation start site by rapid amplification of cDNA ends and RNase protection as
85 rent variants of the exchanger, we used both rapid amplification of cDNA ends and S1 nuclease protect
87 lobulin-like molecules, we utilized 3' RACE (rapid amplification of cDNA ends) and a highly conserved
88 flow designed to address this based on RACE (rapid amplification of cDNA ends) and long-read RNA sequ
89 of the K12 transcript was mapped by 5' RACE (rapid amplification of cDNA ends) and S1 nuclease protec
90 ript 1 (LT1) and LT2, by cDNA sequencing, 5' rapid amplification of cDNA ends, and primer extension a
91 the gene was mapped by RNase protection, 5' rapid amplification of cDNA ends, and primer extension e
92 Screening of a rat heart cDNA library, 5'-rapid amplification of cDNA ends, and RNase protection a
94 rge flgBCDEFGHIJKL operon were determined by rapid amplification of cDNA ends, and secretion of the F
95 nt study, mouse PAP7 cDNA was extended by 5'-rapid amplification of cDNA ends; and a 3432 bp sequence
100 ated from normal human melanocyte cDNA by 5'-rapid amplification of cDNA ends based on the homology t
102 onucleotide-directed RNase H cleavage and 5' rapid amplification of cDNA ends both indicate that the
103 and 237 bp, respectively) were amplified by rapid amplification of cDNA ends, cloned and sequenced.
106 the trapped LPS-response genes via 5' RACE (rapid amplification of cDNA ends) cloning identified nov
107 ewly discovered first exon, identified by 5'-rapid amplification of cDNA ends, consists of a 5'-untra
108 clone and subsequent analyses of products of rapid amplification of cDNA ends coupled with S1 nucleas
110 ethod described here comprises a modified 5'-rapid amplification of cDNA ends, deep sequencing of 3'
112 ecific oligonucleotide probes followed by 5' rapid amplification of cDNA ends detects an antisense sm
118 g portion of the coding sequence by 5' RACE (rapid amplification of cDNA ends), full-length cDNA was
119 quent reverse transcription-PCR (RT-PCR) and rapid amplification of cDNA ends generated a complete ge
120 a combination of cDNA library screening and rapid amplification of cDNA ends has permitted the isola
121 V5 outlier expression by RNA ligase-mediated rapid amplification of cDNA ends identified one case wit
125 cells and liver; however, 5'-RACE analysis (rapid amplification of cDNA ends) identified AS mRNA spe
127 the analysis of hsd2 transcripts by 5' RACE (rapid amplification of cDNA ends) indicates that inversi
128 HSCs and erythroid cells was confirmed by 5'-rapid amplification of cDNA ends, indicating that at lea
129 DeltarcrR-P and DeltarcrR-NP strains, and 5' Rapid Amplification of cDNA Ends mapped the 5' terminus
130 , reverse transcription-PCR (RT-PCR), the 5' rapid amplification of cDNA ends method (5'RACE), and im
132 gene (300 bp) was amplified by means of the rapid-amplification-of-cDNA-ends method, cloned and sequ
135 loning by a combination of sib selection and rapid amplification of cDNA ends of a cDNA encoding a ne
136 tained from an expressed sequence tag and 5'-rapid amplification of cDNA ends of a mouse liver cDNA l
140 searches of the GenBank databases and by 5' rapid amplification of cDNA ends of human brain cDNAs.
143 ntity of the clone, and after both 5' and 3' rapid amplification of cDNA ends, oligonucleotide primer
145 nspection of the Mct8 promoter region and 5'-rapid amplification of cDNA ends PCR analysis in F9 cell
147 ed using reverse transcription-PCR (RT-PCR), rapid amplification of cDNA ends PCR, RNA dot blotting,
149 mykiss) Mx cDNAs were cloned by using RACE (rapid amplification of cDNA ends) PCR and were designate
152 e divergent transcripts were mapped using 5' rapid amplification of cDNA ends-PCR (RACE-PCR), and str
153 ll-length cDNA of the enzyme was obtained by rapid amplification of cDNA ends-PCR amplification of cD
160 we cloned one of the trapped genes using 5' rapid amplification of cDNA ends polymerase chain reacti
161 g a rat lung expression library coupled with rapid amplification of cDNA ends polymerase chain reacti
162 nucleotides apart, were identified using 5'-rapid amplification of cDNA ends polymerase chain reacti
167 P mRNA was cloned after amplification by the rapid amplification of cDNA ends procedure, and a part o
173 ial Display Polymerase Chain Reaction and 5' Rapid Amplification of cDNA Ends protocols, we isolated
174 ssion of ETV4 using quantitative PCR, and by rapid amplification of cDNA ends, quantitative PCR, and
178 rmination sites, and splicing patterns using rapid amplification of cDNA ends (RACE) and full-length
179 is facilitated by cloning methods such as 5'-Rapid Amplification of cDNA Ends (RACE) and inverse poly
182 e correct cDNA sequence was determined by 5' rapid amplification of cDNA ends (RACE) and primer exten
184 th cDNA of the human hSmad5 (hSmad5) gene by rapid amplification of cDNA ends (RACE) and then determi
185 ovel combination of techniques including the rapid amplification of cDNA ends (RACE) and tiling array
190 Primer extension, RNase protection, and rapid amplification of cDNA ends (RACE) experiments indi
191 utational and experimental platform adapting rapid amplification of cDNA ends (RACE) for large-scale
192 se eosinophil and lung cDNA libraries and by rapid amplification of cDNA ends (RACE) from liver, sple
195 from complex mixtures of cellular mRNA using rapid amplification of cDNA ends (RACE) PCR as long as p
200 uencing multiple clones from each 5'- and 3'-rapid amplification of cDNA ends (RACE) product and asse
207 gene structures, a high-throughput method of rapid amplification of cDNA ends (RACE) was used to obta
208 by sequencing of EST clones and products of rapid amplification of cDNA ends (RACE), an mRNA that en
209 tion (PCR) amplification products, 5' and 3' rapid amplification of cDNA ends (RACE), and by helper p
210 HV) using next-generation RNA sequencing, 3' rapid amplification of cDNA ends (RACE), and tiled micro
211 ription-polymerase chain reactions (RT-PCR), rapid amplification of cDNA ends (RACE), and transient e
212 ing the GFP reporter system combined with 5' rapid amplification of cDNA ends (RACE), in vitro transc
216 tion (RT-PCR), in combination with 5' and 3' rapid amplification of cDNA ends (RACE), was used to clo
234 ts of Tap1 and Lmp2 mRNA in NOD mice, and 5'-rapid amplification of cDNA ends revealed the loss of a
236 fH transcriptional start sites by 5'RACE (5' rapid amplification of cDNA ends) revealed that these 5'
239 dentified by full-length RNA ligase-mediated rapid amplification of cDNA ends (RLM-RACE) between -61
241 (CAGE) tags, 1.2 million RNA ligase mediated rapid amplification of cDNA ends (RLM-RACE) reads, and 5
243 NA by a combination of techniques: 5' and 3' rapid amplification of cDNA ends, RT-PCR, and searching
246 are not present, and primer extension and 5'-rapid amplification of cDNA ends studies suggest that tr
248 onal start site has been mapped using the 5' rapid amplification of cDNA ends technique, and this sta
249 nd using the polymerase chain reaction-based rapid amplification of cDNA ends technique, three forms
250 ain reaction (RT-PCR) in conjunction with 5'-rapid amplification of cDNA ends technique, we have clon
254 full-length cDNA was cloned by the 5'-RACE (rapid amplification of cDNA ends) technique and sequence
259 rt site(s) of the IA-2 gene was mapped by 5' rapid amplification of cDNA ends to 97 bp upstream of th
260 f degenerate oligonucleotides and 5'- and 3'-rapid amplification of cDNA ends to clone two cDNAs of 2
266 ites are identified for the TREX genes using rapid amplification of cDNA ends to recover the 5'-flank
267 of a PC12 cDNA library, followed by 5' RACE (rapid amplification of cDNA ends) to determine the 5' en
268 c fusions, reverse transcriptase PCR, and 5' rapid amplification of cDNA ends, to localize and identi
274 transcription-polymerase chain reaction and rapid amplification of cDNA ends, we cloned a full-lengt
275 y, via both RNase protection analysis and 5'-rapid amplification of cDNA ends, we determined the tran
279 Using RPA, primer extension assay and 5' rapid amplification of cDNA ends, we were able to demons
283 of 69 rpoH-dependent genes using 5' RACE (5' rapid amplification of cDNA ends), which allowed us to d
284 IA and IB, were identified by performing 5'-rapid amplification of cDNA ends with human liver cDNA a
287 untranslated region (3'UTR), we performed 3' rapid amplification of cDNA ends with the S1 parental co
288 ronal channel SCN8A, we carried out 5'-RACE (rapid amplification of cDNA ends) with RNA from human an
289 Human brain cDNA library screening and 5' rapid amplification of cDNA ends yielded full-length DEN
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