ライフサイエンスコーパス: rapid eye movement

1 ept from the jumpy images resulting from our rapid eye movements.

2 increases in theta activity during both non-rapid eye movement and rapid eye movement sleep and a re

3 EG spectra averaged across the night for non-rapid eye movement and rapid eye movement sleep separate

4 gnificant effects of pattern A human IgGs on rapid eye movement and slow-wave sleep time parameters i

5 ally similar to that of wake, accompanied by rapid eye movements and muscle atonia.

6 phalogram (EEG), hippocampal theta activity, rapid eye movements, autonomic activation and loss of po

7 necessary for the generation of the accurate rapid eye movements called saccades.

8 bserved between the surge in ATP and EEG non-rapid eye movement delta activity (0.5-4.5 Hz) during sp

9 cle with fluctuating vigilance, intrusion of rapid eye movement dream imagery into wakefulness and em

10 the suppression of visual perception during rapid eye movements in primates, demonstrating common fu

11 NT Patients with strabismus are able to make rapid eye movements, known as saccades, toward visual ta

12 There was an inverse correlation between the rapid eye movement latency and the peak of the pupillary

13 mised if the sensor moves rapidly, as during rapid eye movements, making the period immediately after

14 t one of the prominent EEG signatures of non-rapid eye movement (non-REM) sleep and are thought to pl

15 ncephalogram (EEG) slow-wave activity in non-rapid eye movement (non-REM) sleep and theta and alpha a

16 determined for consolidated episodes of non-rapid eye movement (non-REM) sleep of minimal common len

17 mal sleep architecture (undifferentiated non-rapid-eye-movement [non-REM] sleep or poorly structured

18 omote the transition to wakefulness from non-rapid eye movement (NREM) and rapid eye movement (REM) s

19 tituted of two global behavioral states, non-rapid eye movement (NREM) and rapid eye movement (REM),

20 p cycles: delta (0-3 Hz) activity during non-rapid eye movement (NREM) associated stages was greater

21 dinal study of the adolescent decline in non-rapid eye movement (NREM) delta (1-4 Hz) and theta (4-8

22 dic junctions may be instantiated during non-rapid eye movement (NREM) sleep after hippocampal associ

23 lectroencephalographic (EEG) hallmark of non-rapid eye movement (NREM) sleep and are believed to medi

24 reduced slow wave activity (SWA) during non-rapid eye movement (NREM) sleep and impaired long-term r

25 g/kg) at 21:00 decreased the latency to non-rapid eye movement (NREM) sleep and increased the durati

26 replay of task-related ensembles during non-rapid eye movement (NREM) sleep and temporal shifts that

27 during hypercapnia but instead increased non-rapid eye movement (NREM) sleep by approximately 43% dur

28 ociates beta-amyloid pathology with both non-rapid eye movement (NREM) sleep disruption and memory im

29 n contrast, rapid eye movement (REM) and non-rapid eye movement (NREM) sleep enhance previously induc

30 larify the synergistic role of different non-rapid eye movement (NREM) sleep stages (stages 2 and 3-4

31 ighttime), XX males had more spontaneous non-rapid eye movement (NREM) sleep than XX females.

32 nNOS/NK1 neurons is directly related to non-rapid eye movement (NREM) sleep time, NREM bout duration

33 nstance, compared to wakefulness, during non-rapid eye movement (NREM) sleep TMS elicits a larger pos

34 During non-rapid eye movement (NREM) sleep, a global decrease in sy

35 Deep non-rapid eye movement (NREM) sleep, also known as slow-wave

36 ne in CMR(glc(ox)) during anesthesia and non-rapid eye movement (NREM) sleep, and another, the invers

37 During non-rapid eye movement (NREM) sleep, cortical neurons altern

38 reased EEG delta (0.5-4 Hz) power during non-rapid eye movement (NREM) sleep, increased time spent in

39 electroencephalogram (EEG) signature of non-rapid eye movement (NREM) sleep, is generally viewed as

40 he EEG power between 0.5 and 4 Hz during non-rapid eye movement (NREM) sleep, is one of the best char

41 dles are thalamocortical oscillations in non-rapid eye movement (NREM) sleep, that play an important

42 nal neuronal oscillations characterizing non-rapid eye movement (NREM) sleep.

43 electroencephalogram (EEG) power during non-rapid eye movement (NREM) sleep.

44 cally-dominant consolidated trace during non-rapid eye movement (NREM) sleep.

45 e connectivity is disrupted during early non-rapid eye movement (NREM) sleep.

46 curs in hippocampus and neocortex during non-rapid eye movement (NREM) sleep.

47 ontent of stimuli was conserved in light non-rapid eye movement (NREM) sleep.

48 e that episodic memory is dependent upon non-rapid eye movement (NREM), rather than REM, sleep.

49 cal slow waves are a defining feature of non-rapid eye-movement (NREM) sleep and are thought to be im

50 During non-rapid eye-movement (NREM) sleep, cortical and thalamic n

51 iations and plausible mechanisms linking non-rapid-eye-movement (NREM) sleep disruption, Abeta, and A

52 e oscillations and sleep spindles during non-rapid-eye-movement (NREM) sleep has been proposed to sup

53 wer density in the 0.75-4.5 Hz range) in non-rapid-eye-movement (NREM) sleep is the primary marker of

54 e interaction of the cardinal rhythms of non-rapid-eye-movement (NREM) sleep-the thalamo-cortical spi

55 nd network junction instantiation during non-rapid eye movement [NREM] periods) brings greater specif

56 Rapid eye movement occurred in only five patients (14%).

57 ments in wakefulness, without disrupting non-rapid eye movement or rapid eye movement sleep.

58 ime, sleep fragmentation, abnormal short non-rapid eye movement - rapid eye movement (REM) cycling, R

59 conscious patients showed an alternating non-rapid eye movement/rapid eye movement sleep pattern and

60 In contrast, rapid eye movement (REM) and non-rapid eye movement (NRE

61 y, MK-1064 promotes sleep and increases both rapid eye movement (REM) and non-REM (NREM) sleep in rat

62 imately 90-min ultradian oscillation between rapid eye movement (REM) and non-REM (NREM) sleep stages

63 cant increase in wakefulness and decrease in rapid eye movement (REM) and non-REM (NREM) sleep.

64 in nocturnal activity (siesta), a period of rapid eye movement (REM) and non-REM sleep, was absent i

65 We propose that rapid eye movement (REM) and slow-wave sleep contribute

66 ion, abnormal short non-rapid eye movement - rapid eye movement (REM) cycling, REM sleep reduction or

67 Llewellyn's claim that rapid eye movement (REM) dream imagery may be related to

68 This article argues that rapid eye movement (REM) dreaming is elaborative encodin

69 Llewellyn's proposal that rapid eye movement (REM) dreaming reflects elaborative e

70 Rapid eye movement (REM) dreaming results in "emotionall

71 llyn has written a fascinating article about rapid eye movement (REM) dreams and how they promote the

72 Unlike mneomotechnically encoded material, rapid eye movement (REM) dreams are inherently difficult

73 ellyn develops the more specific thesis that rapid eye movement (REM) dreams, because of their simila

74 mories undergo "elaborative encoding" during rapid eye movement (REM) dreams, generating novel associ

75 was translated into sustained inhibition of rapid eye movement (REM) in vivo.

76 of memories, and features of sleep, such as rapid eye movement (REM) or sleep spindles, have been sh

77 vely during waking (0.329 +/- 0.06%/min) and rapid eye movement (REM) sleep (0.349 +/- 0.13%/min).

78 ons, were more active during wakefulness and rapid eye movement (REM) sleep (wake/REM active) than du

79 The absence of atonia during rapid eye movement (REM) sleep and dream-enactment behav

80 usals, by using established criteria, during rapid eye movement (REM) sleep and non-REM (NREM) sleep

81 The expression of the states of rapid eye movement (REM) sleep and non-REM (NREM) sleep

82 lso outline key models for the regulation of rapid eye movement (REM) sleep and non-REM sleep, how mu

83 , oral administration of glycine-induced non-rapid eye movement (REM) sleep and shortened NREM sleep

84 It also induced a reduction in time spent in rapid eye movement (REM) sleep and slow-wave sleep and a

85 Although quiet wake and rapid eye movement (REM) sleep are characterized by simi

86 Rapid eye movement (REM) sleep behavior disorder (RBD) i

87 The presence of probable rapid eye movement (REM) sleep behavior disorder was str

88 Rapid eye movement (REM) sleep behaviour disorder (RBD)

89 estation of prodromal Parkinson's disease is rapid eye movement (REM) sleep behaviour disorder, which

90 ory aversive conditioning during stage 2 and rapid eye movement (REM) sleep but not following aversiv

91 ta oscillations in the waking rat and during rapid eye movement (REM) sleep by simultaneously recordi

92 -p44/42 MAPK, and phospho-CREB are higher in rapid eye movement (REM) sleep compared with awake mice

93 uch patterns was significantly larger during rapid eye movement (REM) sleep compared with non-REM sta

94 Rapid eye movement (REM) sleep constitutes a distinct "t

95 ing Parkinson's disease, Lewy body dementia, rapid eye movement (REM) sleep disorder and/or multiple

96 The hypothesis that rapid eye movement (REM) sleep disturbances are the hall

97 his response enhancement was proportional to rapid eye movement (REM) sleep duration.

98 Rapid eye movement (REM) sleep enhances hippocampus-depe

99 Although wake and rapid eye movement (REM) sleep exhibit long timescales,

100 (PPT) nucleus for the regulation of recovery rapid eye movement (REM) sleep following REM sleep depri

101 induce an immediate transition to waking or rapid eye movement (REM) sleep from slow-wave sleep (SWS

102 Initial theories of rapid eye movement (REM) sleep generation posited that i

103 Rapid eye movement (REM) sleep has been considered a par

104 o wakefulness, and the developmental role of rapid eye movement (REM) sleep in children.

105 ough rodent models suggest the importance of rapid eye movement (REM) sleep in spatial navigational m

106 one region identified in the brainstem as a rapid eye movement (REM) sleep induction zone.

107 Rapid eye movement (REM) sleep is a distinct brain state

108 Rapid eye movement (REM) sleep is a recurring part of th

109 Rapid eye movement (REM) sleep is an important component

110 -wave sleep, whereas during the second half, rapid eye movement (REM) sleep is more predominant.

111 results of this study suggest that baseline rapid eye movement (REM) sleep may serve a protective fu

112 The functions and underlying mechanisms of rapid eye movement (REM) sleep remain unclear.

113 ysiological, and neurobiological features of rapid eye movement (REM) sleep suggest more functions th

114 finches, we observed slow wave sleep (SWS), rapid eye movement (REM) sleep, an intermediate sleep (I

115 similar levels during quiet waking (QW), and rapid eye movement (REM) sleep, and minimal release duri

116 hing about equally during quiet wake and non-rapid eye movement (REM) sleep, and to a lesser degree d

117 ved in the regulation of wakefulness (W) and rapid eye movement (REM) sleep, but our understanding of

118 as long been implicated in the generation of rapid eye movement (REM) sleep, but the underlying circu

119 Narcolepsy, a disorder of rapid eye movement (REM) sleep, is characterized by exce

120 ond half of the night, which is dominated by rapid eye movement (REM) sleep, led to better discrimina

121 When dreaming during rapid eye movement (REM) sleep, we can perform complex m

122 However, during rapid eye movement (REM) sleep, when cortical activity i

123 The hypothesized role of rapid eye movement (REM) sleep, which is rich in dreams,

124 nspatial behaviors such as wheel running and rapid eye movement (REM) sleep.

125 egions and are integral to the regulation of rapid eye movement (REM) sleep.

126 latter structure shows decreased activity in rapid eye movement (REM) sleep.

127 eep onset (WASO), slow-wave sleep (SWS), and rapid eye movement (REM) sleep.

128 lness from non-rapid eye movement (NREM) and rapid eye movement (REM) sleep.

129 ed by high apnea and hypopnea indices during rapid eye movement (REM) sleep.

130 ximally active immediately before and during rapid eye movement (REM) sleep.

131 phase of firing to the peak of theta during rapid eye movement (REM) sleep.

132 to the cat pontine tegmentum rapidly induces rapid eye movement (REM) sleep.

133 tion (PRF) promotes wakefulness and inhibits rapid eye movement (REM) sleep.

134 pocampal activity during awake behaviour and rapid eye movement (REM) sleep.

135 rtical motor activity was related to time in rapid eye movement (REM) sleep.

136 e and significant increase in NREM sleep and rapid eye movement (REM), and a similar, albeit less rob

137 al states, non-rapid eye movement (NREM) and rapid eye movement (REM), characterized by quiescence an

138 hat Llewellyn's hypothesis about the lack of rapid eye movement (REM)-sleep dreaming leading to loss

139 tion chamber, whereas slow-wave sleep (SWS), rapid eye movement (REM)-sleep, total sleeping time (TST

140 during NREM states was lower than Awake and rapid eye movement (REM).

141 g the function of dreams that is premised on rapid eye movement (REM)/dream isomorphism is unsupporta

142 sleep, in particular during paradoxical [or rapid eye movement (REM)] sleep and sleep state transiti

143 itionally, dreaming has been identified with rapid eye-movement (REM) sleep, characterized by wake-li

144 al activity and behavioral states, including rapid eye-movement (REM) sleep.

145 IEDs also induce spindles during rapid-eye movement (REM) sleep and wakefulness-behaviora

146 otoneurons necessary for the motor atonia of rapid-eye movement (REM) sleep in cats.

147 function of awake, slow-wave sleep (SWS) and rapid-eye movement (REM) sleep states and prenatal choli

148 Interestingly, IIS primarily occurred during rapid-eye movement (REM) sleep, which is notable because

149 ned fear, increased both slow-wave sleep and rapid-eye movement (REM) sleep.

150 ring sleep abnormalities, commonly involving rapid-eye movement (REM) sleep.

151 phase, SB-334867 produced small increases in rapid-eye-movement (REM) and non-REM (NR) sleep.

152 Recently, restless rapid-eye-movement (REM) sleep emerged as a robust signa

153 rstanding of the mechanisms and functions of rapid-eye-movement (REM) sleep have occurred over the pa

154 eduction in nonrapid-eye-movement (NREM) and rapid-eye-movement (REM) sleep, as well as increased sle

155 role for acetylcholine in the regulation of rapid-eye-movement (REM) sleep.

156 g active association between memories during rapid eye movement [REM] dreams followed by indexation a

157 leep time, sleep efficiency, sleep onset and rapid eye movement [REM] sleep latencies, non-REM and RE

158 Are rapid eye movements (REMs) in sleep associated with visu

159 are also described, including impairment of rapid eye movements (saccades) and the fixations intersp

160 However, this encoding of action for rapid eye movements (saccades) has remained unclear: Pur

161 explore static visual scenes by alternating rapid eye movements (saccades) with periods of slow and

162 isions are naturally served by attention and rapid eye movements (saccades), but little is known abou

163 na and visual cortex into motor commands for rapid eye movements (saccades).

164 Human observers make large rapid eye movements-saccades-to bring behaviorally relev

165 olute and relative total sleep time, and non-rapid eye movement sleep (N1, N2, and N3).

166 phic (EEG) delta power during subsequent non-rapid eye movement sleep (NREMS) and is associated with

167 ormone-releasing hormone (GHRH) promotes non-rapid eye movement sleep (NREMS), in part via a well cha

168 Phase-delayed misalignment increased rapid eye movement sleep (P < 0.001) and the sleeping me

169 ced sleep efficiency (P = .008), and reduced rapid eye movement sleep (P = .02).

170 Rapid eye movement sleep (REM) and exploratory wake, bot

171 Contextual fear significantly reduces rapid eye movement sleep (REM) during post-exposure slee

172 LY379268 (LY37), dose-dependently suppresses rapid eye movement sleep (REM) whereas systemic administ

173 Sleep, and particularly rapid eye movement sleep (REM), has been implicated in t

174 shown fear conditioning disrupts subsequent rapid eye movement sleep (REM).

175 process governs tightly the manifestation of rapid eye movement sleep (REMS) [1, 4].

176 ss produces a sexually dimorphic increase in rapid eye movement sleep (REMS) amount in mice that is g

177 ular mechanisms that determine the amount of rapid eye movement sleep (REMS) and non-REMS (NREMS) rem

178 primary brain vigilance states (waking, non-rapid eye movement sleep [NREM] and REM sleep) within an

179 ivity during both non-rapid eye movement and rapid eye movement sleep and a reduction of delta power

180 ttern of alterations was not observed during rapid eye movement sleep and could not be easily explain

181 ot) over water method for SD that eliminated rapid eye movement sleep and greatly reduced non-rapid e

182 < 0.05) as a result of greater slow wave and rapid eye movement sleep and lower fragmentation.

183 e relative contribution of two sleep states, rapid eye movement sleep and slow-wave sleep, to offline

184 Among other OSA-related variables, AHI in rapid eye movement sleep and time spent with oxygen satu

185 tification pattern with a 400% increase from rapid eye movement sleep and wake, to light and deep sle

186 disease (PD), in 15 subjects with idiopathic rapid eye movement sleep behavior disorder (iRBD) and co

187 sporter (DAT) imaging to identify idiopathic rapid eye movement sleep behavior disorder (IRBD) patien

188 pical Parkinson syndromes (n=11), idiopathic rapid eye movement sleep behavior disorder (n=10), and h

189 Rapid eye movement sleep behavior disorder (RBD) is asso

190 Rapid eye movement sleep behavior disorder (RBD) is comm

191 The dream enactment of rapid eye movement sleep behavior disorder (RBD) is ofte

192 atic hypotension, mild cognitive impairment, rapid eye movement sleep behavior disorder (RBD), depres

193 is downregulated in patients with idiopathic rapid eye movement sleep behavior disorder and antedates

194 Rapid eye movement sleep behavior disorder is distinguis

195 eline, 0.65; mean follow-up, 2.88; P = .01), rapid eye movement sleep behavior disorder scores were s

196 eflected by supranuclear ophtalmoparesis and rapid eye movement sleep behavior disorder with underlyi

197 Some non-motor symptoms such as hyposmia, rapid eye movement sleep behavior disorder, and constipa

198 ve daytime sleepiness, insomnia, narcolepsy, rapid eye movement sleep behavior disorder, and restless

199 rum samples from 56 patients with idiopathic rapid eye movement sleep behavior disorder, before and a

200 kers using standardized scales for hyposmia, rapid eye movement sleep behavior disorder, depression,

201 ients (age 65.0+/-5.6 years) with idiopathic rapid eye movement sleep behaviour disorder and 21 age/g

202 All cases with rapid eye movement sleep behaviour disorder and mild cog

203 glia network dysfunction differentiated both rapid eye movement sleep behaviour disorder and Parkinso

204 nnectivity, and for loss of tracer uptake in rapid eye movement sleep behaviour disorder and Parkinso

205 lso elevated (P<0.0001) in the patients with rapid eye movement sleep behaviour disorder but lower th

206 Rapid eye movement sleep behaviour disorder has been eva

207 depression, excessive daytime sleepiness, or rapid eye movement sleep behaviour disorder in early Par

208 sy-proven Lewy body disease, indicating that rapid eye movement sleep behaviour disorder plus mild co

209 Rapid eye movement sleep behaviour disorder preceded cog

210 m baseline was observed, as reflected in the Rapid Eye Movement Sleep Behaviour Disorder Questionnair

211 roved by addition of clinical scores (UPSIT, Rapid Eye Movement Sleep Behaviour Disorder Screening Qu

212 c networks may provide markers of idiopathic rapid eye movement sleep behaviour disorder to identify

213 Rapid eye movement sleep behaviour disorder was indistin

214 The presence of rapid eye movement sleep behaviour disorder was specific

215 In addition, eight patients with rapid eye movement sleep behaviour disorder, 10 with Par

216 tients with polysomnographically-established rapid eye movement sleep behaviour disorder, 48 patients

217 h sensitivity (96%) and specificity (74% for rapid eye movement sleep behaviour disorder, 78% for Par

218 changes are present in so-called idiopathic rapid eye movement sleep behaviour disorder, a condition

219 ange) for specific features were: seven with rapid eye movement sleep behaviour disorder-60 years (27

220 We identified a rapid eye movement sleep behaviour disorder-related meta

221 itive functions and were more likely to have rapid eye movement sleep behaviour disorder.

222 cations for future neuroprotective trials in rapid eye movement sleep behaviour disorder.

223 detect basal ganglia network dysfunction in rapid eye movement sleep behaviour disorder.

224 t determinants of visual hallucinations were rapid eye movement sleep behavioural disorder (P = 0.026

225 ical features of Parkinson's disease such as rapid eye movement sleep behavioural disorder and the po

226 particular excessive daytime somnolence and rapid eye movement sleep behavioural disorder, disorders

227 Hypersomnia, rapid eye movement sleep disorder and/or narcolepsy were

228 sis.RECENT FINDINGS: Narcolepsy is a chronic rapid eye movement sleep disorder.

229 orexant were observed in wakefulness and non-rapid eye movement sleep during both dark and light phas

230 as clonazepam did the opposite, reducing non-rapid eye movement sleep EEG instability without effects

231 ng a paradigm designed to mimic intermittent rapid eye movement sleep epochs, we show that applicatio

232 RO5256390 profoundly reduced rapid eye movement sleep in wild-type mice; these effect

233 In many patients, a short rapid eye movement sleep latency (REML) during the NPSG

234 evidence suggests that the slow waves of non-rapid eye movement sleep may function as markers to trac

235 Cholinergic activity associated with rapid eye movement sleep may function to facilitate long

236 in abnormalities in the brainstem disinhibit rapid eye movement sleep motor activity, leading to drea

237 aking that are thought to be an intrusion of rapid eye movement sleep muscle atonia into wakefulness.

238 ncreased slow wave activity power during non-rapid eye movement sleep over widespread, bilateral scal

239 trast, frequent interictal spikes during non-rapid eye movement sleep predicted a reduced homeostatic

240 lectroencephalogram (delta power) during non-rapid eye movement sleep reflects homeostatic sleep need

241 oss the night for non-rapid eye movement and rapid eye movement sleep separately were classified usin

242 p period, sleep efficiency, or slow-wave and rapid eye movement sleep stage duration (P > .30).

243 No slow wave sleep or rapid eye movement sleep stages could be identified and

244 uish wake, non-rapid eye movement sleep, and rapid eye movement sleep states.

245 d intact, males had more total sleep and non-rapid eye movement sleep than females during the active

246 in slow wave sleep time (45 min vs 28 min), rapid eye movement sleep time (11 min vs 3 min), or the

247 pportunities and failed to increase NREM and rapid eye movement sleep times, despite accumulating a s

248 ranial magnetic stimulation (TMS) during non-rapid eye movement sleep to examine whether the spontane

249 y clinical features and the demonstration of rapid eye movement sleep without atonia on polysomnograp

250 roencephalographic (EEG) activity during non-rapid eye movement sleep, a highly heritable trait with

251 ulting in chronic sleepiness, fragmented non-rapid eye movement sleep, and cataplexy.

252 r task are reactivated during subsequent non-rapid eye movement sleep, and disrupting this neuronal r

253 n the spectral profile, observed only during rapid eye movement sleep, and only at the highest dose t

254 recordings are used to distinguish wake, non-rapid eye movement sleep, and rapid eye movement sleep s

255 delta power spectrum, produced low-delta non-rapid eye movement sleep, and slightly increased wakeful

256 akefulness promotes slow-wave sleep, but not rapid eye movement sleep, during a period of low sleep p

257 ith the phenomenology and neurophysiology of rapid eye movement sleep, the early and acute psychotic

258 During rapid eye movement sleep, the spontaneous drift of the a

259 as well as a theta power (4-7Hz) decrease in rapid eye movement sleep, were associated with disease b

260 ifted progressively from 7 Hz to 6 Hz during rapid eye movement sleep, whereas slow wave activity dec

261 sleep and a reduction of delta power in non-rapid eye movement sleep.

262 efulness and had an increased propensity for rapid eye movement sleep.

263 ncephalogram (EEG) signatures of stage 2 non-rapid eye movement sleep.

264 wave activity decreased gradually during non-rapid eye movement sleep.

265 rat hippocampus during maze exploration and rapid eye movement sleep.

266 SFAs decreased wakefulness and increased non-rapid eye movement sleep.

267 y wakefulness at the expense of both SWS and rapid eye movement sleep.

268 are bursts of 11-15 Hz that occur during non-rapid eye movement sleep.

269 d eye movement sleep and greatly reduced non-rapid eye movement sleep.

270 are commonly observed during stage 2 of non-rapid eye movement sleep.

271 without disrupting non-rapid eye movement or rapid eye movement sleep.

272 kening event during both slow-wave sleep and rapid eye movement sleep.

273 ne of them regulates the circadian rhythm of rapid eye movement sleep.

274 ring wakefulness and to a lesser extent, non-rapid eye movement sleep.

275 eye movement sleep, and decreased or absent rapid eye movement sleep.

276 2 h, and did not occur during wakefulness or rapid eye movement sleep.

277 derlying mechanism may be the suppression of rapid eye movement sleep.

278 rns characterized by increased percentage of rapid eye movement sleep.

279 with skeletal muscle paralysis characterizes rapid eye movement sleep.

280 gonist, induced significant increases in non-rapid-eye movement sleep (NREMS) lasting for 4-10 h.

281 pression, apathy, sleep disorders (including rapid-eye movement sleep behaviour disorder), and erecti

282 ation were present during both slow-wave and rapid-eye movement sleep, were repeatedly observed over

283 ) only in the dark period with no changes in rapid-eye-movement sleep (REMS).

284 sis of V1, activation enhancement during non-rapid-eye-movement sleep after training was observed spe

285 ch was associated with time in slow-wave and rapid-eye-movement sleep after training.

286 Rapid-eye-movement sleep and non-rapid-eye-movement slee

287 igue, insomnia, anosmia, hypersalivation and rapid-eye-movement sleep behaviour disorder) in the year

288 Rapid-eye-movement sleep and non-rapid-eye-movement sleep contributed about equally to th

289 In contrast, during rapid-eye-movement sleep, the neocortical tone is sustai

290 w-frequency (0.5-2.0 Hz) oscillations in non-rapid-eye-movement sleep, was significantly larger in th

291 the cortical states resembling slow-wave and rapid-eye-movement sleep.

292 on structure but only during wakefulness and rapid-eye-movement sleep.

293 ons across wakefulness, slow-wave sleep, and rapid-eye-movement sleep.

294 me functional differences between waking and rapid-eye-movement sleep.

295 mes in Parkinson's Disease (SCOPA-AUT), REM (Rapid Eye Movement) Sleep Behavior Disorder Single-Quest

296 ed between periods of presumed slow-wave and rapid-eye-movement-sleep/active-state, which were charac

297 It has been demonstrated that in sleep onset rapid eye movement (SOREM) periods in BIISS, REM sleep t

298 defining thalamocortical oscillation of non-rapid eye movement stage 2 sleep, correlate with IQ and

299 ctions for sleep time, sleep cycle time, and rapid eye movement time as functions of body and brain m

300 ure were used, including total sleep period, rapid eye movement, wake after sleep onset, absolute and