ライフサイエンスコーパス: rapid eye movement sleep

1 tion of active sleep (AS; also called REM or rapid eye movement sleep).

2 firing during active sleep (AS, also called rapid-eye-movement sleep).

3 derlying mechanism may be the suppression of rapid eye movement sleep.

4 kening event during both slow-wave sleep and rapid eye movement sleep.

5 ne of them regulates the circadian rhythm of rapid eye movement sleep.

6 ring wakefulness and to a lesser extent, non-rapid eye movement sleep.

7 eye movement sleep, and decreased or absent rapid eye movement sleep.

8 2 h, and did not occur during wakefulness or rapid eye movement sleep.

9 o wakefulness from either slow wave sleep or rapid eye movement sleep.

10 ility in thalamocortical networks during non-rapid eye movement sleep.

11 sponse are similar in wakefulness and in non-rapid eye movement sleep.

12 slow wave activity ipsilaterally during non-rapid eye movement sleep.

13 and not in a familiar environment or during rapid eye movement sleep.

14 s that occur during exploratory behavior and rapid eye movement sleep.

15 ents) or without (7 patients) CSA during non-rapid eye movement sleep.

16 tic phenomenon of paradoxical sleep (PS), or rapid eye movement sleep.

17 d a regionally selective reactivation during rapid eye movement sleep.

18 rns characterized by increased percentage of rapid eye movement sleep.

19 ncephalogram (EEG) signatures of stage 2 non-rapid eye movement sleep.

20 with skeletal muscle paralysis characterizes rapid eye movement sleep.

21 sleep and a reduction of delta power in non-rapid eye movement sleep.

22 efulness and had an increased propensity for rapid eye movement sleep.

23 wave activity decreased gradually during non-rapid eye movement sleep.

24 rat hippocampus during maze exploration and rapid eye movement sleep.

25 SFAs decreased wakefulness and increased non-rapid eye movement sleep.

26 y wakefulness at the expense of both SWS and rapid eye movement sleep.

27 are bursts of 11-15 Hz that occur during non-rapid eye movement sleep.

28 d eye movement sleep and greatly reduced non-rapid eye movement sleep.

29 are commonly observed during stage 2 of non-rapid eye movement sleep.

30 without disrupting non-rapid eye movement or rapid eye movement sleep.

31 the cortical states resembling slow-wave and rapid-eye-movement sleep.

32 on structure but only during wakefulness and rapid-eye-movement sleep.

33 ons across wakefulness, slow-wave sleep, and rapid-eye-movement sleep.

34 ear conditioning had less theta power during rapid-eye-movement sleep.

35 n, can disrupt sleep structure, particularly rapid-eye-movement sleep.

36 eep and (b) tonic activity during waking and rapid-eye-movement sleep.

37 me functional differences between waking and rapid-eye-movement sleep.

38 ically involved in the generation of active (rapid eye movement) sleep.

39 cataplexy or any indication of abnormal REM (rapid eye movement) sleep.

40 roencephalographic (EEG) activity during non-rapid eye movement sleep, a highly heritable trait with

41 ed between periods of presumed slow-wave and rapid-eye-movement-sleep/active-state, which were charac

42 y had an increase in NREMS and a decrease in rapid eye movement sleep after interleukin-1beta treatme

43 sis of V1, activation enhancement during non-rapid-eye-movement sleep after training was observed spe

44 ch was associated with time in slow-wave and rapid-eye-movement sleep after training.

45 ivity during both non-rapid eye movement and rapid eye movement sleep and a reduction of delta power

46 global deactivation of the brain during non rapid eye movement sleep and a regionally selective reac

47 ttern of alterations was not observed during rapid eye movement sleep and could not be easily explain

48 ot) over water method for SD that eliminated rapid eye movement sleep and greatly reduced non-rapid e

49 sms, which may underlie the promotion of non-rapid eye movement sleep and have implications for the u

50 < 0.05) as a result of greater slow wave and rapid eye movement sleep and lower fragmentation.

51 e relative contribution of two sleep states, rapid eye movement sleep and slow-wave sleep, to offline

52 Among other OSA-related variables, AHI in rapid eye movement sleep and time spent with oxygen satu

53 tification pattern with a 400% increase from rapid eye movement sleep and wake, to light and deep sle

54 Rapid-eye-movement sleep and non-rapid-eye-movement slee

55 p) states; the down states are absent during rapid-eye-movement sleep and waking.

56 ulting in chronic sleepiness, fragmented non-rapid eye movement sleep, and cataplexy.

57 r task are reactivated during subsequent non-rapid eye movement sleep, and disrupting this neuronal r

58 n the spectral profile, observed only during rapid eye movement sleep, and only at the highest dose t

59 recordings are used to distinguish wake, non-rapid eye movement sleep, and rapid eye movement sleep s

60 delta power spectrum, produced low-delta non-rapid eye movement sleep, and slightly increased wakeful

61 ches and hypnic headache are associated with rapid eye movement sleep, as illustrated by recent polys

62 disease (PD), in 15 subjects with idiopathic rapid eye movement sleep behavior disorder (iRBD) and co

63 sporter (DAT) imaging to identify idiopathic rapid eye movement sleep behavior disorder (IRBD) patien

64 pical Parkinson syndromes (n=11), idiopathic rapid eye movement sleep behavior disorder (n=10), and h

65 Rapid eye movement sleep behavior disorder (RBD) is asso

66 Rapid eye movement sleep behavior disorder (RBD) is comm

67 The dream enactment of rapid eye movement sleep behavior disorder (RBD) is ofte

68 atic hypotension, mild cognitive impairment, rapid eye movement sleep behavior disorder (RBD), depres

69 is downregulated in patients with idiopathic rapid eye movement sleep behavior disorder and antedates

70 Rapid eye movement sleep behavior disorder is distinguis

71 Rapid eye movement sleep behavior disorder is intricatel

72 eline, 0.65; mean follow-up, 2.88; P = .01), rapid eye movement sleep behavior disorder scores were s

73 eflected by supranuclear ophtalmoparesis and rapid eye movement sleep behavior disorder with underlyi

74 Some non-motor symptoms such as hyposmia, rapid eye movement sleep behavior disorder, and constipa

75 ve daytime sleepiness, insomnia, narcolepsy, rapid eye movement sleep behavior disorder, and restless

76 rum samples from 56 patients with idiopathic rapid eye movement sleep behavior disorder, before and a

77 kers using standardized scales for hyposmia, rapid eye movement sleep behavior disorder, depression,

78 arly prone to hallucinations, delusions, and rapid eye movement sleep behavior disorder.

79 mes in Parkinson's Disease (SCOPA-AUT), REM (Rapid Eye Movement) Sleep Behavior Disorder Single-Quest

80 ients (age 65.0+/-5.6 years) with idiopathic rapid eye movement sleep behaviour disorder and 21 age/g

81 All cases with rapid eye movement sleep behaviour disorder and mild cog

82 glia network dysfunction differentiated both rapid eye movement sleep behaviour disorder and Parkinso

83 nnectivity, and for loss of tracer uptake in rapid eye movement sleep behaviour disorder and Parkinso

84 lso elevated (P<0.0001) in the patients with rapid eye movement sleep behaviour disorder but lower th

85 Rapid eye movement sleep behaviour disorder has been eva

86 depression, excessive daytime sleepiness, or rapid eye movement sleep behaviour disorder in early Par

87 sy-proven Lewy body disease, indicating that rapid eye movement sleep behaviour disorder plus mild co

88 Rapid eye movement sleep behaviour disorder preceded cog

89 m baseline was observed, as reflected in the Rapid Eye Movement Sleep Behaviour Disorder Questionnair

90 roved by addition of clinical scores (UPSIT, Rapid Eye Movement Sleep Behaviour Disorder Screening Qu

91 c networks may provide markers of idiopathic rapid eye movement sleep behaviour disorder to identify

92 Rapid eye movement sleep behaviour disorder was indistin

93 The presence of rapid eye movement sleep behaviour disorder was specific

94 In addition, eight patients with rapid eye movement sleep behaviour disorder, 10 with Par

95 tients with polysomnographically-established rapid eye movement sleep behaviour disorder, 48 patients

96 h sensitivity (96%) and specificity (74% for rapid eye movement sleep behaviour disorder, 78% for Par

97 changes are present in so-called idiopathic rapid eye movement sleep behaviour disorder, a condition

98 ange) for specific features were: seven with rapid eye movement sleep behaviour disorder-60 years (27

99 We identified a rapid eye movement sleep behaviour disorder-related meta

100 itive functions and were more likely to have rapid eye movement sleep behaviour disorder.

101 cations for future neuroprotective trials in rapid eye movement sleep behaviour disorder.

102 detect basal ganglia network dysfunction in rapid eye movement sleep behaviour disorder.

103 pression, apathy, sleep disorders (including rapid-eye movement sleep behaviour disorder), and erecti

104 igue, insomnia, anosmia, hypersalivation and rapid-eye-movement sleep behaviour disorder) in the year

105 t determinants of visual hallucinations were rapid eye movement sleep behavioural disorder (P = 0.026

106 ical features of Parkinson's disease such as rapid eye movement sleep behavioural disorder and the po

107 particular excessive daytime somnolence and rapid eye movement sleep behavioural disorder, disorders

108 (PET(CO2)) was gradually reduced during non-rapid eye movement sleep by increasing tidal volume with

109 Rapid-eye-movement sleep and non-rapid-eye-movement sleep contributed about equally to th

110 hr, orexin KO mice recovered their NREM and rapid eye movement sleep deficits at comparable rates an

111 Hypersomnia, rapid eye movement sleep disorder and/or narcolepsy were

112 sis.RECENT FINDINGS: Narcolepsy is a chronic rapid eye movement sleep disorder.

113 orexant were observed in wakefulness and non-rapid eye movement sleep during both dark and light phas

114 akefulness promotes slow-wave sleep, but not rapid eye movement sleep, during a period of low sleep p

115 as clonazepam did the opposite, reducing non-rapid eye movement sleep EEG instability without effects

116 All children had at least two sleep-onset rapid eye movement sleep episodes in this test.

117 ng a paradigm designed to mimic intermittent rapid eye movement sleep epochs, we show that applicatio

118 It has been hypothesized that REM (rapid eye movement) sleep has an important role in memor

119 ographic (EEG) slow wave activity during non-rapid eye movement sleep in rats.

120 ring by lights-off and redistribution of non-rapid eye movement sleep in short light-dark cycles.

121 taplectic arrests and other abnormalities of rapid eye movement sleep in the absence of endogenous or

122 RO5256390 profoundly reduced rapid eye movement sleep in wild-type mice; these effect

123 Sleep, and particularly deep non-rapid-eye-movement sleep, increase interictal epileptifo

124 Non-rapid eye movement sleep is associated with decreases in

125 use orexin promotes wakefulness and inhibits rapid eye movement sleep, its absence may permit inappro

126 In many patients, a short rapid eye movement sleep latency (REML) during the NPSG

127 o microinjection of cholinomimetics causes a rapid eye movement sleep-like state.

128 evidence suggests that the slow waves of non-rapid eye movement sleep may function as markers to trac

129 Cholinergic activity associated with rapid eye movement sleep may function to facilitate long

130 in abnormalities in the brainstem disinhibit rapid eye movement sleep motor activity, leading to drea

131 aking that are thought to be an intrusion of rapid eye movement sleep muscle atonia into wakefulness.

132 olute and relative total sleep time, and non-rapid eye movement sleep (N1, N2, and N3).

133 ripples (80-200 Hz) chiefly occur during non-rapid eye movement sleep (NREM), and that ripple oscilla

134 primary brain vigilance states (waking, non-rapid eye movement sleep [NREM] and REM sleep) within an

135 phic (EEG) delta power during subsequent non-rapid eye movement sleep (NREMS) and is associated with

136 EEG slow wave power ipsilaterally during non-rapid eye movement sleep (NREMS) but not during REMS or

137 fusion induced a progressive decrease in non-rapid eye movement sleep (NREMS) during the 4-week perio

138 TNFalpha dose-dependently increased non-rapid eye movement sleep (NREMS) in the controls but did

139 tivity of thalamocortical origin; during non-rapid eye movement sleep (NREMS), activity in the spindl

140 ormone-releasing hormone (GHRH) promotes non-rapid eye movement sleep (NREMS), in part via a well cha

141 gonist, induced significant increases in non-rapid-eye movement sleep (NREMS) lasting for 4-10 h.

142 dent reductions in respiratory events in non-rapid-eye-movement sleep (NREMS) and rapid-eye-movement

143 ection of the immunotoxin, the amount of non-rapid-eye-movement sleep (NREMS) and rapid-eye-movement

144 ine cholinergic neurones, which control REM (rapid eye movement) sleep or dreaming, is likely to cont

145 ncreased slow wave activity power during non-rapid eye movement sleep over widespread, bilateral scal

146 Phase-delayed misalignment increased rapid eye movement sleep (P < 0.001) and the sleeping me

147 ced sleep efficiency (P = .008), and reduced rapid eye movement sleep (P = .02).

148 eep quality, increased percentage of time in rapid eye movement sleep, pain at night at least three t

149 showed an alternating non-rapid eye movement/rapid eye movement sleep pattern and a homoeostatic decl

150 trast, frequent interictal spikes during non-rapid eye movement sleep predicted a reduced homeostatic

151 lectroencephalogram (delta power) during non-rapid eye movement sleep reflects homeostatic sleep need

152 ntain wakefulness and abnormal intrusions of rapid eye movement sleep-related phenomena into wakefuln

153 Rapid eye movement sleep (REM) and exploratory wake, bot

154 mic peptides Hypocretin/Orexin (Hcrt/Orx) to rapid eye movement sleep (REM) control and the sleep dis

155 Contextual fear significantly reduces rapid eye movement sleep (REM) during post-exposure slee

156 C) produce relatively selective decreases in rapid eye movement sleep (REM) in mice that vary with st

157 LY379268 (LY37), dose-dependently suppresses rapid eye movement sleep (REM) whereas systemic administ

158 Sleep, and particularly rapid eye movement sleep (REM), has been implicated in t

159 n. subcoeruleus (SubC) in the generation of rapid eye movement sleep (REM).

160 shown fear conditioning disrupts subsequent rapid eye movement sleep (REM).

161 siological states, slow-wave sleep (SWS) and rapid-eye-movement sleep (REM), often identified by the

162 process governs tightly the manifestation of rapid eye movement sleep (REMS) [1, 4].

163 ss produces a sexually dimorphic increase in rapid eye movement sleep (REMS) amount in mice that is g

164 Consistent alterations in rapid eye movement sleep (REMS) and in brain temperature

165 ular mechanisms that determine the amount of rapid eye movement sleep (REMS) and non-REMS (NREMS) rem

166 a 30% reduction in time spent in spontaneous rapid eye movement sleep (REMS) as a consequence of a re

167 Impaired rapid eye movement sleep (REMS) is commonly observed in

168 The behavioral state of active or rapid eye movement sleep (REMS) is dominant during fetal

169 evidence that NO plays an important role in rapid eye movement sleep (REMS) regulation.

170 implicated in the modulation of spontaneous rapid eye movement sleep (REMS).

171 Br-cGMP increased wakefulness and suppressed rapid-eye-movement sleep (REMS) and non-REMS (NREMS) in

172 of non-rapid-eye-movement sleep (NREMS) and rapid-eye-movement sleep (REMS) increased during the dar

173 ) only in the dark period with no changes in rapid-eye-movement sleep (REMS).

174 in non-rapid-eye-movement sleep (NREMS) and rapid-eye-movement sleep (REMS).

175 generalization of partial seizures, however rapid-eye-movement sleep seems to suppress seizures.

176 oss the night for non-rapid eye movement and rapid eye movement sleep separately were classified usin

177 p period, sleep efficiency, or slow-wave and rapid eye movement sleep stage duration (P > .30).

178 No slow wave sleep or rapid eye movement sleep stages could be identified and

179 uish wake, non-rapid eye movement sleep, and rapid eye movement sleep states.

180 into wakefulness of physiological aspects of rapid eye movement sleep such as cataplexy and hallucina

181 d intact, males had more total sleep and non-rapid eye movement sleep than females during the active

182 ith the phenomenology and neurophysiology of rapid eye movement sleep, the early and acute psychotic

183 During non-rapid eye movement sleep, the initial response was stron

184 During rapid eye movement sleep, the spontaneous drift of the a

185 In contrast, during rapid-eye-movement sleep, the neocortical tone is sustai

186 in slow wave sleep time (45 min vs 28 min), rapid eye movement sleep time (11 min vs 3 min), or the

187 dopamine transporter (DAT) gene reduced non-rapid eye movement sleep time and increased wakefulness

188 e movement (NREM) delta power] and increased rapid eye movement sleep time compared with baseline.

189 pportunities and failed to increase NREM and rapid eye movement sleep times, despite accumulating a s

190 ranial magnetic stimulation (TMS) during non-rapid eye movement sleep to examine whether the spontane

191 iring is prominent, whereas during waking or rapid eye movement sleep, tonic, single-spike activity d

192 the effects of light on behavior, including rapid eye movement sleep triggering by lights-off and re

193 The lowest oxygen saturation measured during rapid eye movement sleep was 78 +/- 5% preoperatively an

194 characteristic change in PET,CO2 during non-rapid eye movement sleep was shown to be independent of

195 w-frequency (0.5-2.0 Hz) oscillations in non-rapid-eye-movement sleep, was significantly larger in th

196 Latencies of late RVLMS responses during rapid eye movement sleep were significantly longer than

197 Theta amplitude and frequency during rapid eye movement sleep were virtually identical.

198 as well as a theta power (4-7Hz) decrease in rapid eye movement sleep, were associated with disease b

199 ons, recorded during either wheel running or rapid eye movement sleep, were not different either.

200 ation were present during both slow-wave and rapid-eye movement sleep, were repeatedly observed over

201 ifted progressively from 7 Hz to 6 Hz during rapid eye movement sleep, whereas slow wave activity dec

202 e predatorylike attack sometimes observed in rapid eye movement sleep without atonia (REM-A), created

203 y clinical features and the demonstration of rapid eye movement sleep without atonia on polysomnograp