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1 ressed relative to myocardial oxygen demand (rate-pressure product).
2 k work rate, maximal oxygen consumption, and rate pressure product.
3                   Rest MBF was normalized to rate-pressure product.
4  present after correcting rest flows for the rate-pressure product.
5 d shift in the relationship between MVO2 and rate-pressure product.
6 n increase in heart rate, blood pressure and rate-pressure product.
7 did not demonstrate the expected increase in rate-pressure product.
8 y (p < 0.02) larger with O3 than with FA for rate-pressure product (1,353 beats/min/mm Hg) and for he
9  flow (3.3+/-2.5 x rest) exceeded normalized rate-pressure product (1.9+/-0.8 x rest; P<.05).
10 line donor vessel absolute flow adjusted for rate pressure product: 177.5 to 139.9 mL/min (difference
11 ats/min, p=0.6) and a comparable increase in rate-pressure product (2.2+/-0.9 vs. 1.9+/-1.2 beats/min
12 /min; P = not significant [ns]) and peak Dob rate.pressure product (20,738 +/- 3,947 vs. 20,047 +/- 3
13 oad after NTG, as indicated by a higher peak rate-pressure product (24 492+/-1054 versus 22 536+/-101
14 and perfusate Ca2+ with similar increases in rate-pressure product (48+/-5%, 42+/-6%, and 51+/-6%, re
15                                  The resting rate pressure product (6628 +/- 504 versus 5225 +/- 807)
16 sed heart rate (79+/-22 to 115+/-28 bpm) and rate-pressure product (9748+/-2862 to 15,157+/-3433 mm H
17 esting MBF and increase in CVR corrected for rate-pressure product after AVR, although these changes
18                                 However, the rate pressure product, an index of myocardial work, tend
19           Rest blood flows normalized to the rate-pressure product, an index of cardiac work, were si
20 atine:ATP ratio that correlated with reduced rate pressure product and increased diastolic pressure.
21 ease in systolic blood pressure, heart rate, rate-pressure product and CO and a fall in PVR were simi
22                In controls, during ischemia, rate-pressure product and oxygen consumption decreased b
23 dobutamine is in excess of that predicted by rate-pressure product and reflects the unmeasured inotro
24 spite similar values for peak heart rate and rate-pressure product and similar severity of CAD.
25 butamine significantly increased heart rate, rate-pressure product and the first derivative of left v
26                                              Rate-pressure product and wall motion score index, simil
27       Changes in heart rate, blood pressure, rate-pressure product, and cardiac output were greater w
28 001), blood pressure (P < .005) at rest, and rate-pressure product at peak exercise (P < .003), but e
29     Heart rates, systolic blood pressure and rate-pressure product at peak exercise were not differen
30                                     The mean rate-pressure product at peak pacing was 22,313+/-5,357
31 e-EN, the ischemic threshold, defined as the rate-pressure product at the onset of 1-mm ST depression
32                                              Rate pressure product both at baseline and at peak adeno
33  by increases in blood pressure, heart rate, rate-pressure product, cardiac index, and stroke work in
34  relatively minor increase in heart rate and rate-pressure product compared with ischemia induced by
35 ed with lower baseline and postmental stress rate-pressure product compared with nifedipine or placeb
36    Over all segments, mean MBF normalized by rate-pressure product ("corrected MBF") was 1.2+/-0.3 mL
37                    Similarly, heart rate and rate pressure product declined from the baseline dipyrid
38 he isolated heart dose-dependently increased rate pressure product, dP/dt, -dP/dt, and myocardial oxy
39                                          The rate-pressure product for the group as a whole increased
40 ; 95% confidence interval [CI], 0.3 to 0.9), rate pressure product (HR, 0.7; 95% CI, 0.5 to 0.9), lef
41 HR, 2.6; 95% CI, 1.2 to 5.4), lower exercise rate-pressure product (HR, 0.6; 95% CI, 0.5 to 0.8), res
42 c complications, organ dysfunction, or heart rate pressure product identified.
43   Normalization of resting blood flow to the rate pressure product improved the reproducibility signi
44                                 In addition, rate-pressure product improved suggesting decreased card
45 uring stress, there were equivalent rises in rate pressure product in both groups (DCM, 76+/-15% and
46 assessed by measuring developed pressure and rate pressure product in Langendorff mode.
47 og x kg-1 x min-1 IV) caused doubling of the rate-pressure product in both normal and LVR hearts and
48                                          The rate-pressure product in postischemic hearts was 46% of
49                               In CHF hearts, rate-pressure product increased only 40% in response to
50 ated (p < 0.001) with a 2.2-fold increase in rate-pressure product induced by dobutamine.
51                 They also correlated with RV rate-pressure product (K(mono), R(2) = 0.41, P = 0.0005;
52  at a left ventricular pressure of 50 mm Hg, rate-pressure product, left ventricular tension-time ind
53 icant (P < 0.01) and comparable increases in rate-pressure product, measured in beats per minute x mm
54   Ischemic threshold, measured either as the rate-pressure product or the duration of exercise at the
55                 Endothelial NO decreased the rate-pressure product (P<0.05), upregulated cytochrome c
56 mand were seen (tension-time index, P=0.024; rate-pressure product, P=0.046), as well as an increase
57 obutamine in normal segments correlated with rate-pressure product (r=.81, P<.05), the slope of the l
58 mpaired recovery of systolic (24% versus 53% rate pressure product recovery; P<0.05) and diastolic fu
59 did not alter blood pressure, heart rate, or rate-pressure product relative to placebo.
60          Heart rate, mean arterial pressure, rate-pressure product, respiratory rate, and catecholami
61         HYP showed a 13% to 26% reduction in rate pressure product (RPP) and impaired dP/dt versus SH
62                                     Baseline rate-pressure product (RPP) (6559+/-1590 versus 7144+/-1
63             Exercise BP, heart rate (HR) and rate-pressure product (RPP) at maximal and submaximal wo
64 onship between ST-segment depression and the rate-pressure product (RPP) during exercise to determine
65            Cold induced similar increases in rate-pressure product (RPP) in smokers and nonsmokers.
66  E) were repeated at 2.5 and 5 minutes for a rate-pressure product (RPP) less than 20% baseline.
67                    Measurements were made of rate-pressure product (RPP=LV developed pressure x heart
68               MBF was also normalized to the rate.pressure product (RPP).
69 damole caused an increase (mean, 21%) in the rate-pressure product secondary to reflex tachycardia re
70                              The increase in rate-pressure product tended to be greater in patients w
71   The initial high Ca2+ challenge caused the rate-pressure product to increase by 74 +/- 5% in all he
72 educed mechanical inefficiency (the ratio of rate-pressure product to MVO(2)) and myofilament sensiti
73 tion fraction, myocardial scar and ischemia, rate-pressure product, type of radiotracer or stress age
74                                       The RV rate-pressure product was also determined by means of ri
75  were maintained relatively constant and the rate-pressure product was relatively unchanged, but lact
76      Left ventricular developed pressure and rate pressure product were significantly depressed in th
77 itude of ST segment depression, and exercise rate-pressure product-were found to be independent predi
78 -diastolic pressure, developed pressure, and rate-pressure product, which was also blocked by DPCPX a

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