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1 ressed relative to myocardial oxygen demand (rate-pressure product).
2 k work rate, maximal oxygen consumption, and rate pressure product.
3 Rest MBF was normalized to rate-pressure product.
4 present after correcting rest flows for the rate-pressure product.
5 d shift in the relationship between MVO2 and rate-pressure product.
6 n increase in heart rate, blood pressure and rate-pressure product.
7 did not demonstrate the expected increase in rate-pressure product.
8 y (p < 0.02) larger with O3 than with FA for rate-pressure product (1,353 beats/min/mm Hg) and for he
10 line donor vessel absolute flow adjusted for rate pressure product: 177.5 to 139.9 mL/min (difference
11 ats/min, p=0.6) and a comparable increase in rate-pressure product (2.2+/-0.9 vs. 1.9+/-1.2 beats/min
12 /min; P = not significant [ns]) and peak Dob rate.pressure product (20,738 +/- 3,947 vs. 20,047 +/- 3
13 oad after NTG, as indicated by a higher peak rate-pressure product (24 492+/-1054 versus 22 536+/-101
14 and perfusate Ca2+ with similar increases in rate-pressure product (48+/-5%, 42+/-6%, and 51+/-6%, re
16 sed heart rate (79+/-22 to 115+/-28 bpm) and rate-pressure product (9748+/-2862 to 15,157+/-3433 mm H
17 esting MBF and increase in CVR corrected for rate-pressure product after AVR, although these changes
20 atine:ATP ratio that correlated with reduced rate pressure product and increased diastolic pressure.
21 ease in systolic blood pressure, heart rate, rate-pressure product and CO and a fall in PVR were simi
23 dobutamine is in excess of that predicted by rate-pressure product and reflects the unmeasured inotro
25 butamine significantly increased heart rate, rate-pressure product and the first derivative of left v
28 001), blood pressure (P < .005) at rest, and rate-pressure product at peak exercise (P < .003), but e
29 Heart rates, systolic blood pressure and rate-pressure product at peak exercise were not differen
31 e-EN, the ischemic threshold, defined as the rate-pressure product at the onset of 1-mm ST depression
33 by increases in blood pressure, heart rate, rate-pressure product, cardiac index, and stroke work in
34 relatively minor increase in heart rate and rate-pressure product compared with ischemia induced by
35 ed with lower baseline and postmental stress rate-pressure product compared with nifedipine or placeb
36 Over all segments, mean MBF normalized by rate-pressure product ("corrected MBF") was 1.2+/-0.3 mL
38 he isolated heart dose-dependently increased rate pressure product, dP/dt, -dP/dt, and myocardial oxy
40 ; 95% confidence interval [CI], 0.3 to 0.9), rate pressure product (HR, 0.7; 95% CI, 0.5 to 0.9), lef
41 HR, 2.6; 95% CI, 1.2 to 5.4), lower exercise rate-pressure product (HR, 0.6; 95% CI, 0.5 to 0.8), res
43 Normalization of resting blood flow to the rate pressure product improved the reproducibility signi
45 uring stress, there were equivalent rises in rate pressure product in both groups (DCM, 76+/-15% and
47 og x kg-1 x min-1 IV) caused doubling of the rate-pressure product in both normal and LVR hearts and
52 at a left ventricular pressure of 50 mm Hg, rate-pressure product, left ventricular tension-time ind
53 icant (P < 0.01) and comparable increases in rate-pressure product, measured in beats per minute x mm
54 Ischemic threshold, measured either as the rate-pressure product or the duration of exercise at the
56 mand were seen (tension-time index, P=0.024; rate-pressure product, P=0.046), as well as an increase
57 obutamine in normal segments correlated with rate-pressure product (r=.81, P<.05), the slope of the l
58 mpaired recovery of systolic (24% versus 53% rate pressure product recovery; P<0.05) and diastolic fu
64 onship between ST-segment depression and the rate-pressure product (RPP) during exercise to determine
69 damole caused an increase (mean, 21%) in the rate-pressure product secondary to reflex tachycardia re
71 The initial high Ca2+ challenge caused the rate-pressure product to increase by 74 +/- 5% in all he
72 educed mechanical inefficiency (the ratio of rate-pressure product to MVO(2)) and myofilament sensiti
73 tion fraction, myocardial scar and ischemia, rate-pressure product, type of radiotracer or stress age
75 were maintained relatively constant and the rate-pressure product was relatively unchanged, but lact
77 itude of ST segment depression, and exercise rate-pressure product-were found to be independent predi
78 -diastolic pressure, developed pressure, and rate-pressure product, which was also blocked by DPCPX a
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