ライフサイエンスコーパス: rauwolscine

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1 sitivities to the alpha2-AR antagonist [(3)H]rauwolscine.

2 higher after LPS in animals pretreated with rauwolscine.

3 pg/ml (p < 0.05) in animals pretreated with rauwolscine.

4 GS3 was reversed by the alpha2-AR antagonist rauwolscine.

5 on, since renal BF was markedly reduced with rauwolscine.

6 ckade with yohimbine (0.6-1.0 mg/kg i.v.) or rauwolscine (1.0 mg/kg i.v.) reversibly antagonized the

7 In old mice, inhibiting alpha2 ARs (rauwolscine; 10(-7) m) restored ROV more effectively in

8 tive alphaAR antagonists atipamezole (8.79), rauwolscine (7.75), 2-(2,6-dimethoxyphenoxyethyl)aminome

9 Rauwolscine, a highly specific antagonist of alpha-2 AR,

10 Rauwolscine administered after LPS had no protective eff

11 5) constriction in 1A (inhibited by 10(-7) m rauwolscine; alpha2AR antagonist).

12 by the alpha2-adrenergic receptor antagonist rauwolscine and by Clostridium botulinum toxin as well a

13 While the alpha2-antagonists, yohimbine, rauwolscine and idazoxan blocked NE-induced inhibition i

14 es on the dissociation of [3H]yohimbine, [3H]rauwolscine, and [3H]RX821002.

15 ha2 receptor antagonists, such as yohimbine, rauwolscine, and atipamezole.

16 Finally, in isolated rat gracilis vessels, rauwolscine completely inhibited the L-arginine-initiate

17 The highest rauwolscine dose decreased mortality from 100% to zero.

18 gonists with an order of inverse efficacy of rauwolscine > yohimbine > RX821002 > MK912, whereas phen

19 Molecular mechanistic studies indicate that rauwolscine interacts with the BiOctR, DmOctR, and alpha

20 her of the alpha 2-AR-selective antagonists, rauwolscine or atipamezole, reversed the functional effe

21 cells with the alpha2-adrenergic antagonist rauwolscine or with pertussis toxin increased membrane P

22 ediate level of alpha 1-AR (norepinephrine + rauwolscine) or alpha 2-AR (UK 14,304 + prazosin) tone w

23 uced (15-35 mmHg) with individual (prazosin, rauwolscine) or combined (phentolamine) alpha-receptor i

24 Rauwolscine pretreatment significantly reduced bowel hem

25 Furthermore, sodium enhanced [(3)H]rauwolscine's interactions with the BiOctR, but not at a

26 treated with doses of the alpha-2 antagonist rauwolscine up to 1 mg/kg, followed by 20 mg/kg LPS.

27 efficacious inverse agonists, yohimbine and rauwolscine, was 1.7- and 2.1-fold weaker.

28 pha(2)-adrenoceptor antagonists yohimbine or rauwolscine were co-administered, suggesting that the me

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