ライフサイエンスコーパス: reabsorption

1 regulating calcium and phosphate absorption/reabsorption.

2 rapid renal clearance followed by high renal reabsorption.

3 tion via increased proximal tubule phosphate reabsorption.

4 glucose stimulates NHE3-mediated bicarbonate reabsorption.

5 (DCT) plays a critical role in renal sodium reabsorption.

6 etion and increases fractional renal calcium reabsorption.

7 mechanisms that ensure proper salt and water reabsorption.

8 subunits expression and ENaC-mediated sodium reabsorption.

9 fundamental for renal and intestinal sodium reabsorption.

10 NaPi-IIa is responsible for most P(i) renal reabsorption.

11 absorption of vitamin B12 and renal protein reabsorption.

12 lation of renal HCO(3(-)) secretion and salt reabsorption.

13 protecting Na(+) balance to promoting water reabsorption.

14 pathway permeability but did not change NaCl reabsorption.

15 provides the driving force for renal sodium reabsorption.

16 order membrane of RPTCs, and reduced glucose reabsorption.

17 ted that SGLT2 mediates 90% of renal glucose reabsorption.

18 nclude alterations in NKCC2-dependent sodium reabsorption.

19 nal stimuli suggests different roles in P(i) reabsorption.

20 s in an increase in ENaC activity and sodium reabsorption.

21 ties of vitamin D action and renal phosphate reabsorption.

22 sium secretion and less favorable for sodium reabsorption.

23 which subsequently mediates salt and volume reabsorption.

24 s arginine vasopressin-dependent renal water reabsorption.

25 tubular fluid, which inhibits salt and water reabsorption.

26 uptake transporters and inherent low passive reabsorption.

27 metabolism, including hepatic synthesis and reabsorption.

28 tubule in mice resulted in impaired renal Pi reabsorption.

29 %) for pseudouridine, indicating partial net reabsorption.

30 n exchange rather than the effect on glucose reabsorption.

31 was related to the changes in tubular Na(+) reabsorption.

32 ular filtration rate (GFR) and tubular Na(+) reabsorption.

33 novel method of regulation for distal sodium reabsorption.

34 V5 endocytosis and increases urinary calcium reabsorption.

35 nduced sodium release did not undergo distal reabsorption.

36 l tubule, which is the site of renal glucose reabsorption.

37 dney, claudin-2 mediates paracellular sodium reabsorption.

38 epithelia, thus contributing to renal Ca(2+) reabsorption.

39 ubule is the major pathway for renal glucose reabsorption.

40 bone resorption and increasing renal calcium reabsorption.

41 AT in the regulation of intestinal bile acid reabsorption..

42 es in glomerular permselectivity and tubular reabsorption account, at least in part, for the proteins

43 in the proximal tubule and Henle's loop from reabsorption (acid load) to secretion (base load).

44 anilide (GW9662), were found to decrease Na+ reabsorption across mpkCCDc14 cell layers.

45 mediates the effect of vasopressin on sodium reabsorption along the distal nephron.

46 delivery are the main determinants of Na(+) reabsorption along the kidney tubule.

47 Lymphatic reabsorption also may contribute to UFF, but little is k

48 MAGE-D2 is essential for fetal renal salt reabsorption, amniotic fluid homeostasis, and the mainte

49 Given the critical role of TRPM6 in Mg(2+) reabsorption, an inducible kidney-specific Adcy3 deletio

50 d a 36% decrease in ouabain-sensitive sodium reabsorption and a significantly attenuated response to

51 The alveolar ridge undergoes reabsorption and atrophy subsequent to tooth removal and

52 ns (CV) are required for cerebrospinal fluid reabsorption and brain homeostasis, but mechanisms that

53 The tubular nephron is responsible for reabsorption and catabolism of filtered low molecular we

54 e inner ear, where they are involved in NaCl reabsorption and endolymph production, respectively.

55 hways involved in regulation of tubular salt reabsorption and enzymatic pathways for organ developmen

56 bute to calcium homeostasis by adjusting the reabsorption and excretion of filtered calcium through p

57 l absorption, tissue accumulation, and renal reabsorption and excretion.

58 trators free of toxic elements, with reduced reabsorption and extended coverage of the solar spectrum

59 Thanks to the suppressed reabsorption and high emission efficiencies of the quant

60 ese changes associated with less bicarbonate reabsorption and higher lithium clearance in females.

61 lemia resulting from constitutive renal salt reabsorption and impaired K(+) secretion.

62 not fully understood, increased renal sodium reabsorption and impaired pressure natriuresis play key

63 and cubilin, mediates renal proximal-tubular reabsorption and is decreased in Dent disease because of

64 orchestrating the balance between renal salt reabsorption and K(+) and H(+) excretion.

65 -glucose cotransport enhance proximal tubule reabsorption and make the GFR supranormal through the ph

66 The FcRn pathway facilitates reabsorption and mediates transcytosis by its pH-depende

67 tical TAL, CaSR inhibitors increased calcium reabsorption and paracellular pathway permeability but d

68 nsing receptor (CaR) modulates renal calcium reabsorption and parathyroid hormone (PTH) secretion and

69 ride pores to facilitate electrogenic sodium reabsorption and potassium and acid secretion.

70 eceptor, that differentially controls sodium reabsorption and potassium secretion in the late distal

71 vide insights into the mechanisms of protein reabsorption and potential targets for treating diabetic

72 This dye has significant probability of reabsorption and re-emission in concentrated solutions w

73 ivation, which is relevant for kidney sodium reabsorption and salt-sensitive hypertension.

74 cellular pH (pHo), and contributes to solute reabsorption and secretion in many epithelia.

75 tes to the regulation of AQP2-mediated water reabsorption and suggest new potential therapeutic strat

76 s regulating hypoxia signalling, lung liquid reabsorption and surfactant maturation, which may be an

77 Thus, the mechanisms of albumin reabsorption and transcytosis are undergoing intense stu

78 glomerular filtration, with partial tubular reabsorption and transient translocation into the proxim

79 ated water channel that controls renal water reabsorption and urine concentration.

80 ells express homologous proteins involved in reabsorption and waste modification.

81 ving rise to abnormal kidney proximal tubule reabsorption, and additional nervous system and ocular d

82 osphaturia from decreased proximal phosphate reabsorption, and decreased activity and protein of the

83 en KLHL3 and CUL3 mutations, increased Na-Cl reabsorption, and disease pathogenesis.

84 rmation of angiotensinogen, increased sodium reabsorption, and increased renal fibrosis.

85 that vascular flow, tubular dilation, water reabsorption, and intratubular flow all play important r

86 vated serum FGF23, decreased renal phosphate reabsorption, and low serum concentrations of phosphate

87 ts high photoluminescence quantum yield, low reabsorption, and relatively low refractive indices for

88 ncreased urine cAMP levels, water and sodium reabsorption, and urine osmolality and decreased urine o

89 ecreased urine cAMP levels, water and sodium reabsorption, and urine osmolality and increased urine o

90 Ab), decreased urine cAMP levels, free water reabsorption, and urine osmolality and increased urine o

91 renal hemodynamics, inhibits salt and fluid reabsorption, antagonizes the renin-angiotensin system,

92 a regulated paracellular pathway for calcium reabsorption, approaches to regulate this transport path

93 O2 consumption and the efficiency of sodium reabsorption are dependent on sodium diet.

94 egment, luminal delivery and transepithelial reabsorption are directly coupled, a phenomenon called g

95 f transporters mediating renal tubular Na(+) reabsorption are well established causes of hypertension

96 ely filtered at the renal glomerulus without reabsorption at the tubule.

97 lly attributed to primary impairments in CSF reabsorption, but little experimental evidence supports

98 DR due to compensatory distal tubular sodium reabsorption, but whether this translates to human DR is

99 wth factor (FGF) 23 inhibits renal phosphate reabsorption by activating FGF receptor (FGFR) 1c in a K

100 NK4) inhibits electroneutral sodium chloride reabsorption by attenuating the cell surface expression

101 in, the two major hormones regulating sodium reabsorption by CD, generate a coordinated stimulation o

102 l Na/K/2Cl cotransporter NKCC2 regulate NaCl reabsorption by epithelial cells of the renal thick asce

103 for physiological regulation of renal Ca(2+) reabsorption by parathyroid hormones or the tissue kalli

104 Impaired albumin reabsorption by proximal tubular epithelial cells (PTECs

105 receptor (CSR), in the regulation of Ca(2+) reabsorption by salivary gland ducts.

106 Prevention of renal glucose reabsorption by SGLT2 deletion reduced HFD- and obesity-

107 Inhibiting glucose reabsorption by sodium glucose co-transporter proteins (

108 Klotho, a protein that supports renal Ca(2+) reabsorption by stabilizing the transient receptor poten

109 glomerular filtration barrier and subsequent reabsorption by the downstream proximal tubule, causing

110 arly proximal tubule and most of the glucose reabsorption by the kidney, overall.

111 s prevented the impairment of alveolar fluid reabsorption caused by hypoxia.

112 The results indicate that the reabsorption corrections, applied on molecular emission

113 tical modelling predicted that tubular Na(+) reabsorption decreased in the proximal tubule but increa

114 ll facilitate studies of mechanisms in renal reabsorption, demonstrate that Dent disease-causing CLC-

115 an oxidative stress and AQP2-dependent water reabsorption disturbs.

116 sed 2.8-fold, suggesting enhanced intestinal reabsorption due to induction of ileal transporters (Slc

117 lar mechanisms underlying AVP-mediated water reabsorption, evidenced by our identification of 4-acety

118 FABP in the liver, and renal elimination and reabsorption facilitated by OAT proteins.

119 lucocorticoid by 11betaHSD1 stimulates Na(+) reabsorption; failure to downregulate the enzyme during

120 tive distal nephron where it performs sodium reabsorption from the lumen.

121 or which targets the kidney to block glucose reabsorption, has the potential to improve kidney diseas

122 porin-2 trafficking and the consequent water reabsorption, however, are not completely understood.

123 Increased renal tubular sodium reabsorption impairs pressure natriuresis and plays an i

124 ansporter-2 inhibitor, reduces renal glucose reabsorption in an insulin-independent manner.

125 n in thick ascending limbs via NO, and water reabsorption in collecting ducts.

126 ivation of beta1-AR stimulates active Ca(2+) reabsorption in DCT2/CNT; an increase in TRPV5 activity

127 y, proteins involved in transcellular Ca(2+) reabsorption in DCTs were not decreased.

128 r excretion is achieved by continuous sodium reabsorption in distal nephron segments with low water p

129 the nephron and suggest that lower proximal reabsorption in female rats expedites excretion of a sal

130 hy are they unable to inhibit 90% of glucose reabsorption in humans?

131 -)-cotransporter (NKCC2) is crucial for NaCl reabsorption in kidney thick ascending limb (TAL) and dr

132 tion and electroneutral transepithelial NaCl reabsorption in microperfused CCDs of wild-type mice but

133 usually associated with defective HCO(3)(-) reabsorption in proximal tubule cells) and hypokalaemic

134 asolateral step of transepithelial HCO(3)(-) reabsorption in proximal tubule epithelia, contributing

135 e was reabsorbed in WT mice compared with no reabsorption in Sglt2(-/-) mice.

136 e regulation of HCO(3)(-) secretion and NaCl reabsorption in the CNT/CCD under acid-base stress and e

137 ensitive rats, promoting ENaC-mediated Na(+) reabsorption in the collecting duct and the development

138 hanisms underlying the paracellular chloride reabsorption in the collecting duct are not understood.

139 Vasopressin modulates sodium reabsorption in the collecting duct through adenylyl cyc

140 Vasopressin regulates water reabsorption in the collecting duct, but extracellular n

141 r coupling chloride reabsorption with sodium reabsorption in the collecting duct.

142 ithelial Na+ channels (ENaCs) mediate sodium reabsorption in the cortical collecting duct (CCD), but

143 activity of which is instrumental in Mg(2+) reabsorption in the DCT.

144 This channel is crucial for calcium reabsorption in the distal convoluted tubule (DCT).

145 porter (NCC) is the primary mediator of salt reabsorption in the distal convoluted tubule and is a ke

146 10 is required in the kidney for normal salt reabsorption in the distal convoluted tubule because of

147 Ca(2+) excretion by mediating active Ca(2+) reabsorption in the distal convoluted tubule of the kidn

148 Transcellular magnesium (Mg(2+)) reabsorption in the distal convoluted tubule represents

149 (ENaC) is the limiting entry point for Na(+) reabsorption in the distal kidney nephron and is regulat

150 s epithelial Na+ channel (ENaC)-mediated Na+ reabsorption in the distal nephron by affecting status o

151 l cotransporter (NCC, SLC12A3) mediates salt reabsorption in the distal nephron of the kidney and is

152 5) is the gatekeeper of transcellular Ca(2+) reabsorption in the distal nephron.

153 orters NKCC2 and NCC (key components of salt reabsorption in the distal renal tubule), possibly throu

154 a(+)/Cl(-) cotransporter (NCC) mediated salt reabsorption in the distal tubules of the kidney.

155 ults demonstrate that SGLT2 mediates glucose reabsorption in the early proximal tubule and most of th

156 (OH)2D3; calcitriol) formation and phosphate reabsorption in the kidney and counteracts vascular calc

157 ates both basal and agonist-stimulated Na(+) reabsorption in the kidney collecting duct, acting to en

158 iltrate in wild-type mice and the absence of reabsorption in the kidney in Glut2(-/-) mice confirm th

159 tasis and blood pressure by enhancing sodium reabsorption in the kidney's distal nephron (DN).

160 as been attributed to enhanced distal sodium reabsorption in the kidney, the structural defects have

161 (NKCC2) co-transporters, which regulate salt reabsorption in the kidney.

162 o may play important roles regarding calcium reabsorption in the kidney.

163 muscle cells, neuronal signalling and Ca(2+) reabsorption in the kidney.

164 g limb (TAL) of Henle is critical for Ca(++) reabsorption in the kidney.

165 te the physiological role of CNNM2 in Mg(2+) reabsorption in the kidney.

166 lay a key role in mediating paracellular ion reabsorption in the kidney.

167 pithelial Ca(2+) channel critical for Ca(2+) reabsorption in the kidney.

168 hormonal cAMP-dependent regulation of Mg(2+) reabsorption in the kidney.

169 Active Ca(2+) reabsorption in the late distal convoluted and connectin

170 nsporter (NCC) is the major pathway for salt reabsorption in the mammalian distal convoluted tubule.

171 Low dietary Na(+) intake increased Na(+) reabsorption in the proximal tubule and decreased it in

172 High dietary Na(+) intake decreased Na(+) reabsorption in the proximal tubule and increased it in

173 may explain why hypercalcemia inhibits Na(+) reabsorption in the proximal tubule of the kidney.

174 fusion pressure can directly regulate sodium reabsorption in the proximal tubule.

175 nless (AMN), two major receptors for protein reabsorption in the proximal tubule.

176 Arginine vasopressin (AVP) enhances water reabsorption in the renal collecting duct by vasopressin

177 lucose uptake regulates NHE3-mediated NaHCO3 reabsorption in the renal proximal tubule.

178 ignaling through Na/K-ATPase regulate sodium reabsorption in the renal proximal tubule.

179 ldren) and is the result of impaired cystine reabsorption in the renal proximal tubule.

180 the ClC-Kb chloride channel involved in NaCl reabsorption in the renal tubule.

181 dins Cldn10b, -16, and -19 facilitate cation reabsorption in the TAL, and their absence leads to a se

182 e of claudin-16 and -19, critical for Ca(++) reabsorption in the TAL.

183 d transporters involved in mediating NH4 (+) reabsorption in the thick ascending limb of the loop of

184 ta suggest that renal Casr regulates calcium reabsorption in the thick ascending limb, independent of

185 Blocking ileal CBA reabsorption in transferred Rag1(-/-) mice restored Mdr1

186 This is physiologically relevant to Ca(2+) reabsorption in vivo, as short hairpin RNA knockdown of

187 inhibits SGLT2 in vitro and urinary glucose reabsorption in vivo.

188 tical modelling predicted that tubular Na(+) reabsorption increased in the proximal tubule but decrea

189 cate that distal tubular compensatory sodium reabsorption is a primary driver of DR.

190 P(i) reabsorption is a transcellular process that occurs alon

191 The 3-->1 transformation through acetone reabsorption is also demonstrated.

192 Renal water reabsorption is controlled by arginine vasopressin (AVP)

193 Our data show that fractional Na(+) reabsorption is distributed differently according to die

194 owed that flow-dependent Na(+) and HCO(3)(-) reabsorption is due to a modulation of both NHE3 and vac

195 Renal Ca(2+) reabsorption is essential for maintaining systemic Ca(2+

196 Reabsorption is primarily handled by SGLT2, and SGLT2-sp

197 Water reabsorption is regulated by AQP2 trafficking between in

198 a novel mechanism by which TRPV5 and Ca(2+) reabsorption is regulated by the kidney and support the

199 , we found that Cubilin/AMN-mediated protein reabsorption is required for the maintenance of nephrocy

200 ion (nearly the same as the rate of HCO3 (-) reabsorption, JHCO3 ) in response to changes in blood [C

201 lecting duct, mineralocorticoids drive Na(+) reabsorption, K(+) secretion, and H(+) secretion through

202 s natriuresis with correspondent renal water reabsorption, limits natriuretic osmotic diuresis, and r

203 large Stokes shift necessary for suppressing reabsorption losses in large-area devices.

204 fication of the integrated compensatory NaCl reabsorption mechanisms provides insight into thiazide d

205 embrane of salivary gland ducts and regulate reabsorption of [Ca(2+)] from the saliva via TRPC3, thus

206 an be reversed to the original material upon reabsorption of acetone.

207 However, high renal reabsorption of Affibody molecules prevents the use of r

208 essed by proximal tubular cells mediates the reabsorption of ALA, and variants of PEPT2 have differen

209 f megalin, a protein involved in the tubular reabsorption of albumin and lipid-bound proteins.

210 syndrome caused by abnormal proximal tubule reabsorption of bicarbonate resulting in metabolic acido

211 ASBT, SLC10A2) is responsible for intestinal reabsorption of bile acids and plays a key role in chole

212 rolaemia, because inhibition of ASBT reduces reabsorption of bile acids, thus increasing bile acid sy

213 esterol and triglycerides as a result of the reabsorption of biliary lipids from the intestine.

214 which is primarily mediated by altering the reabsorption of Ca(2+) filtered by the glomerulus.

215 the ductal tree into the oral cavity, ductal reabsorption of Ca(2+) remains enigmatic.

216 cending limb, claudins are important for the reabsorption of calcium and magnesium and are tightly re

217 es a transepithelial voltage that drives the reabsorption of calcium and magnesium.

218 crease in intestinal absorption and/or renal reabsorption of calcium.

219 or approximately 25% of Abeta clearance, and reabsorption of cerebrospinal fluid Abeta accounts for a

220 h as Na(+) and a spatially distinct site for reabsorption of divalent cations such as Ca(2+) and Mg(2

221 permits proximal tubule luminal exposure and reabsorption of fatty acid/albumin complexes, we hypothe

222 relatively narrow range by control of renal reabsorption of filtered inorganic phosphate (P(i)).

223 large part through its role in the regulated reabsorption of filtered Na(+).

224 In the kidney, bulk reabsorption of filtered proteins occurs in the proximal

225 Reabsorption of filtered sodium by CD cells occurs via a

226 Tubular reabsorption of filtered sodium is tightly controlled to

227 olume decline is consistent with atrophy and reabsorption of globally sclerotic glomeruli and hypertr

228 ine to absorb glucose and contributes to the reabsorption of glucose filtered by the kidney.

229 is important in glucose liver transport and reabsorption of glucose in the kidney along with SGLT2 a

230 ted physiologic functions, including tubular reabsorption of macromolecules, that gained access to th

231 ve shown that OAT1 mediates the secretion or reabsorption of many important metabolites, including in

232 Complete reabsorption of Me-4FDG from the glomerular filtrate in

233 SGLT2 is responsible for reabsorption of most of the glucose filtered by the kidn

234 otensin II (ANG II) stimulates renal tubular reabsorption of NaCl by targeting Na(+)/H(+) exchanger N

235 plasma at the glomerulus followed by active reabsorption of nearly 99% of that filtrate by the tubul

236 a and NaPi-2c play a major role in the renal reabsorption of P(i).

237 I], 0.59 to 0.96; P=0.02), decreased tubular reabsorption of phosphate (OR, 0.41; 95% CI, 0.23 to 0.7

238 vitamin D (VITD) supplementation on tubular reabsorption of phosphate (TRP), parathyroid hormone (PT

239 (FGF23) axis, creatinine, and renal tubular reabsorption of phosphate (TRP).

240 al metabolism by directly modulating tubular reabsorption of phosphate and calcium and by acting as a

241 Intestinal absorption and renal reabsorption of phosphate are mediated by members of the

242 plasma phosphate concentration, and tubular reabsorption of phosphate increased during the proteinur

243 ing the fractional sodium excretion, tubular reabsorption of phosphate, and gamma-glutamyltransferase

244 12)-intrinsic factor complex and the tubular reabsorption of protein in the proximal tubule.

245 s, suggesting a role of Oatp1a1 in the renal reabsorption of rosuvastatin.

246 mal tubule, claudins have a role in the bulk reabsorption of salt and water.

247 excretion resulting in a decrease in distal reabsorption of sodium.

248 Half-dose PDT induced a more rapid reabsorption of the fluid, a more lasting effect, and eq

249 for phase matching and a rapidly decreasing reabsorption of the generated X-rays.

250 as probably due to glomerular filtration and reabsorption of the protein tracer in proximal tubular c

251 ed that SGLT2 is responsible for >80% of the reabsorption of the renal filtered glucose load.

252 Reabsorption of water from the luminal fluid of the neph

253 is ensured by the selective transepithelial reabsorption of water into the hypertonic medullary inte

254 channels creates the major driving force for reabsorption of water through the alveolar epithelium in

255 proximal tubules are very important for the reabsorption of water, ions and organic solutes from the

256 tatic organ required for waste excretion and reabsorption of water, salts and other macromolecules.

257 ribe and reproduce the distortions caused by reabsorption on emission spectra and quantum yields.

258 tribute to pH(i) regulation, and promote ion reabsorption or secretion by many epithelia.

259 ermined by production and the net balance of reabsorption or secretion by the kidney and intestine.

260 maintain stable fractional solute and fluid reabsorption over a wide range of glomerular filtration

261 Salt reabsorption pathways were created by the coordinate ind

262 its renal vasoconstriction, increased sodium reabsorption, proliferation, fibrosis and renal injury.

263 hat link aldosterone to NCC-mediated Na+/Cl- reabsorption remain elusive.

264 DCT2/CNT region, their role in active Ca(2+) reabsorption remains elusive.

265 The physiological relevance of PiT-2 to P(i) reabsorption remains to be elucidated.

266 ge can lead to increased or decreased sodium reabsorption, respectively, through the Na(+)/Cl(-) cotr

267 inhibits K(+) secretion and stimulates Na(+) reabsorption, respectively.

268 eptually, modest inhibition of renal tubular reabsorption should provide effective relief for the mil

269 s a VP-like effect on electrolytes and water reabsorption, suggesting that it may affect AQP2 traffic

270 directly or indirectly reduces renal calcium reabsorption, suggesting the presence of a novel calcium

271 s reabsorbed transcellularly, whereas sodium reabsorption takes transcellular and paracellular routes

272 edominant route for transepithelial chloride reabsorption that determines the extracellular NaCl cont

273 r characterized by defective urinary cystine reabsorption that results in the formation of cystine-ba

274 potassium by increasing electroneutral NaCl reabsorption, therefore reducing Na(+)/K(+) exchange.

275 Aldosterone promotes electrogenic sodium reabsorption through the amiloride-sensitive epithelial

276 he kidney that underlies paracellular Ca(++) reabsorption through the tight junction.

277 ass vasopressin signaling and increase water reabsorption through two different intracellular signali

278 aximum renal tubular threshold for phosphate reabsorption (TmP/GFR), serum Pi, and 1,25(OH)2D compare

279 In contrast, the bile salt reabsorption transporters Ostalpha and Ostbeta were up-r

280 urine output (P </= 0.04, except for sodium reabsorption under constant pressure [P = 0.17]).

281 cular volume expansion, increased free water reabsorption, urinary prostaglandin E2 excretion, and re

282 nephrocyte combines filtration with protein reabsorption, using evolutionarily conserved genes and s

283 ole of SOCE in the regulation of renal water reabsorption, using the inbred rat strain SHR-A3 as an a

284 asopressin stimulates renal water and sodium reabsorption via increased tubular cell cAMP levels, we

285 epithelial sodium channel (ENaC) and sodium reabsorption via phosphorylation and sequestering of the

286 Altered glucose reabsorption via the facilitative glucose transporter 2

287 ineralocorticoid release promoted free water reabsorption via the renal concentration mechanism.

288 rives paracellular Na(+), Ca(2+), and Mg(2+) reabsorption via the tight junction (TJ).

289 y develop from impaired transcellular Ca(2+) reabsorption via TRPV5 in the distal convoluted tubule (

290 ate proximal collections, fractional glucose reabsorption was 93 +/- 1% in WT and 21 +/- 6% in Sglt2(

291 Salt reabsorption was also activated by induction of an alpha

292 In this case, PT albumin reabsorption was markedly increased.

293 Renal glucose reabsorption was measured with the stepped hyperglycemic

294 n of efferent ductal genes involved in fluid reabsorption was significantly lower in AF2ERKI males.

295 rance studies showed that fractional glucose reabsorption was significantly lower in Sglt2(-/-) mice

296 cells of the collecting duct regulate water reabsorption, we used Cre-Loxp technology to specificall

297 alocorticoid-coupled increases in free water reabsorption were counterbalanced by rhythmical glucocor

298 suggesting a mechanism for coupling chloride reabsorption with sodium reabsorption in the collecting

299 hrotoxicity, are the site of oligonucleotide reabsorption within the kidney.

300 vascular volume depletion (promotion of salt reabsorption without K(+) secretion), a condition that i