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1 apy to combat OP poisoning involves an oxime reactivator (2-PAM, obidoxime, TMB4, or HI-6) combined w
2 ata identify NSC319726 as a p53(R175) mutant reactivator and as a lead compound for p53-targeted drug
3 activity relationships of this new family of reactivators and shows that 1b-d are uncharged hAChE rea
4 ts mutant 17DeltaPst(LAT(-)) (low phenotypic reactivator) at 10(4) plaque-forming units/eye.
5 vator) or 17DeltaPst(LAT(-)) (low phenotypic reactivator) at 10(4) plaque-forming units/eye.
6 ed reactivation of the HSV-1 high phenotypic reactivator can upregulate gene expression involved in B
7 nhibition could be observed with HI-6 as the reactivator due to the extreme lability of the phosphory
8 ree yeast kinase families, nitrogen permease reactivator/halotolerance-5), polyamine transport kinase
9 is of acetylthiocholine reveal that distinct reactivator ionization states are involved in the reacti
10  poor nitrogen medium, the nitrogen permease reactivator kinase (Npr1) inhibits TORC1 activity and al
11 c property of the oximate anion, some of the reactivators may carry an accelerating determinant, as c
12 (2)), is superior to the reference uncharged reactivators monoisonitrosoacetone and 2,3-butanedione m
13  kinetics of the lead hydroxyimino acetamide reactivator of hAChE, when analyzed in terms of apparent
14 fficient than 2-PAM (up to 29000 times) as a reactivator of S(P)-phosphonates (k(r) ranged from 50 to
15                     Recently, a new class of reactivators of chemical warfare agent inhibited acetylc
16 thesis and activity of a new series of oxime reactivators of cholinesterases (ChEs) that contain tert
17 r structural refinement into three efficient reactivators of human butyrylcholinesterase (hBChE) inhi
18 o develop reversible inhibitors of AChE into reactivators of nerve agent-inhibited AChE.
19                 A new class of amidine-oxime reactivators of organophosphate (OP)-inhibited cholinest
20 bstituted 2-hydroxyiminoacetamido alkylamine reactivators of phosphylated human acetylcholinesterase
21  with either HSV-1 17Syn(+) (high phenotypic reactivator) or 17DeltaPst(LAT(-)) (low phenotypic react
22 culated with HSV-1 17Syn(+) (high phenotypic reactivator) or its mutant 17DeltaPst(LAT(-)) (low pheno
23                                   One of the reactivators showed improved in vivo efficacy compared t
24 tently infected with either the low- or high-reactivator strains.
25 )MMF(-) EBV reactivators, the 9 DZB(+)MMF(+) reactivators tended to have more prolonged viremia (11.4
26             Compared with 7 DZB(-)MMF(-) EBV reactivators, the 9 DZB(+)MMF(+) reactivators tended to
27 ated species at physiological pH enables the reactivators to cross the blood-brain barrier and distri
28 A small library of imidazole and imidazolium reactivators was successfully synthesized using this met
29 tors and shows that 1b-d are uncharged hAChE reactivators with a broad spectrum.

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