ライフサイエンスコーパス: reannealing

1 te strand also disrupts transcription bubble reannealing.

2 placed single-stranded DNA tail and prevents reannealing.

3 by which FoxM1 regulates endothelial barrier reannealing.

4 ual subunits; they recompose by severing and reannealing.

5 e with one of the single strands can prevent reannealing.

6 lance between the ORF1 protein's melting and reannealing activities, thereby reducing its nucleic aci

7 ing at 95 degrees C for 5 min and subsequent reannealing after DIG-labeled probe synthesis eliminated

8 short length of duplex DNA followed by rapid reannealing and reinitiation of unwinding in several suc

9 be a function of the energetics of template reannealing and the relative strengths of the RNA:RNAP i

10 ures resulting from incomplete denaturation, reannealing, and self-annealing of target sequences.

11 eir complementary strands and inhibit duplex reannealing at a slippage site.

12 (VIFs) undergo rearrangement by severing and reannealing, but direct subunit exchange within the fila

13 Under dynamic force loading at reannealing cell-cell junctions, the R551A mutant bound

14 d passage, enabling important functions like reannealing denatured DNA and resolving recombination in

15 her suggest that template-nontemplate strand reannealing drives dissociation of abortive transcripts

16 depolymerization or, occasionally, to slowly reannealing holes in the microtubule wall.

17 Reannealing leads to a slightly relaxed trimeric structu

18 -cadherin and p120-catenin and stimulated AJ reannealing mediated by the Rap1 effector afadin.

19 PCR products are amplified in the same tube, reannealing occurs faster for the more abundant PCR prod

20 ility increase induced by PAR1, but prevents reannealing of adherens junctions (AJ), thereby persiste

21 at mediate both endothelial regeneration and reannealing of adherens junctions (AJs) to form a restri

22 the Rho GTPases, Rho, Rac, and Cdc42 in the reannealing of AJs.

23 ic fluctuations cause continuous melting and reannealing of base pairs so that DNA strands are able t

24 luding the slow release of RNA from rho, the reannealing of complementary DNA oligonucleotides to the

25 form large protein-DNA networks and promote reannealing of complementary DNA strands.

26 res that map within the repeat tracts during reannealing of complementary strands, containing equal l

27 (the UL29 gene product) presumably prevents reannealing of complementary strands.

28 t prior to loading, and a neutral gel allows reannealing of cross-linked DNA.

29 by the enzymes Mfd and Rho is facilitated by reannealing of DNA in the upstream region of the transcr

30 licase pathway" involving the separation and reannealing of DNA strands.

31 icrovessel leakage characterized by impaired reannealing of endothelial AJs after endothelial injury.

32 data demonstrate that FoxM1 is required for reannealing of endothelial AJs in order to form a restri

33 true fusion or from lysis and its attendant reannealing of membranes.

34 PNA prevents reannealing of single-stranded DNA products, but does no

35 their relative stabilities and the rates of reannealing of the daughter strand back to the parent.

36 he nascent lagging strand, thus facilitating reannealing of the parental strands.

37 otides by helicases is hampered due to rapid reannealing of the single-stranded DNA products.

38 are coated by SSB and thereby prevented from reannealing or promoting ongoing ATP hydrolysis.

39 After strand separation, rapid strand reannealing outcompetes slow non-enzymatic template copy

40 jacent mismatches were formed by melting and reannealing pairs of homologous 373 bp DNA fragments dif

41 eta-catenin rescued the defective AJ barrier-reannealing phenotype of FoxM1-deficient ECs.

42 ot a helix-destabilizing protein promoting a reannealing reaction but rather is a novel type of pairi

43 The reannealing reaction catalyzed by Prp24 proceeds more ef

44 w cooling in an aqueous environment, whereas reannealing the many single-stranded DNAs of complex gen

45 After denaturation and reannealing, the PCR product pool is subjected to MutH,

46 amically stable and have slower kinetics for reannealing to their complement than shorter analogues.

47 e, prevents hairpin formation and blocks DNA reannealing until the processing pathway is successfully

48 y which RAD52 presents the single strand for reannealing with complementary single-stranded DNA.

49 ssibly allowing the nascent RNA intermittent reannealing with the template strand, for prolonged acce

50 overy of barrier function through junctional reannealing within 2 hours.