ライフサイエンスコーパス: receptor aggregation

1 irement for multivalent pathogen binding for receptor aggregation.

2 main of the Fc epsilonRI beta subunit before receptor aggregation.

3 2 with Lyn and PLC gamma were enhanced after receptor aggregation.

4 esynaptic vesicle tethering and postsynaptic receptor aggregation.

5 stal lattice, offering intriguing models for receptor aggregation.

6 ellular free calcium levels, as occurs after receptor aggregation.

7 signals are generated only after TNF-induced receptor aggregation.

8 Grb2 becomes augmented twofold on FcgammaRI-receptor aggregation.

9 nalling pathway recruited following Fc gamma receptor aggregation.

10 coprecipitated with Fc epsilonRI only after receptor aggregation.

11 cells was also appropriately upregulated by receptor aggregation.

12 silon-RI) were determined following chimeric receptor aggregation.

13 ce by using histamine, as well as IgE or IgG receptor aggregation.

14 n evaluating the extent of hormone-dependent receptor aggregation.

15 I model suggests two possible ligand-induced receptor aggregations.

16 yrosines that may become phosphorylated upon receptor aggregation and bind distinct effectors by virt

17 2 domains was not tyrosine phosphorylated by receptor aggregation and failed to transduce intracellul

18 ains, we demonstrate that MHCII prevents Fas receptor aggregation and inhibits Fas-mediated signaling

19 or to cross-linking by multivalent allergen, receptor aggregation and mast cell activation.

20 Binding to the ligand results in receptor aggregation and recruitment of adaptor proteins

21 V radiation can activate the Fas pathway via receptor aggregation and subsequent recruitment of the d

22 o investigate the kinetics of ligand-induced receptor aggregation and to study how the kinetics and e

23 Steric effects that limit receptor aggregation and transient formation of small re

24 phorylation of SHIP was an early event after receptor aggregation and was present in cells deficient

25 However, this model predicts extensive receptor aggregation at antigen concentrations that yiel

26 at the neuromuscular junction () or glycine receptor aggregation at central inhibitory synapses ().

27 There is no simple relationship between receptor aggregation at equilibrium and the degranulatio

28 To test whether dystroglycan plays a role in receptor aggregation at the neuromuscular junction, we o

29 s a passive action of "sensitization" before receptor aggregation by Ag.

30 Although anti-BCR treatment leads to receptor aggregation by immature stage B cells, the aggr

31 ys statistical resampling to measure the Eph receptor aggregation distribution within each pixel of a

32 ly dissociates from the receptor and induces receptor aggregation in long lasting clusters that are e

33 tive to monovalent hapten, which can prevent receptor aggregation induced by IgE, whereas other activ

34 Receptor aggregation induces multiple signalling pathway

35 ure of the insulin receptor and incorporates receptor aggregation into the kinetic model.

36 Ligand-induced receptor aggregation is a well-known mechanism for initi

37 cted, indicating that Jak2 activation during receptor aggregation is dependent on Jak2 and not anothe

38 e nature of the myeloid response to Fc gamma receptor aggregation is highly variable and depends on t

39 creased receptor clustering, indicating that receptor aggregation is sensitive to the lipid compositi

40 nd associated with Fc epsilon RI gamma after receptor aggregation, it was not tyrosine phosphorylated

41 Mathematical modeling provided estimates of receptor aggregation kinetics based on FcepsilonRI occup

42 InsP3 receptor aggregation may be a characteristic cellular re

43 ons reproduced positive cooperativity in the receptor aggregation model when the aggregation equilibr

44 ionship between tyrosine phosphorylation and receptor aggregation nonlinear.

45 In addition, insulin receptor aggregation occurs in response to ligand bindin

46 crystal form offers an intriguing model for receptor aggregation on the cell surface.

47 and negative regulation of neurotransmitter receptor aggregation on the postsynaptic membrane is a c

48 Our measurements reveal changes in receptor aggregation that are consistent with netrin-1-i

49 of acquired Fas resistance is inhibition of receptor aggregation via a major histocompatibility comp

50 Receptor aggregation was associated with the redistribut