ライフサイエンスコーパス: receptor blockade

1 spectively, with values of <0.5% ID/g during receptor blockade).

2 arized in nonrhythmic slices (following AMPA receptor blockade).

3 oduction in the presence or absence of IL-33 receptor blockade.

4 howed dose-dependent sensitivity to dopamine receptor blockade.

5 -dependent decline in cerebral binding after receptor blockade.

6 , neither group was affected by DLS dopamine receptor blockade.

7 ctance analysis during nicotinic and GABA(A) receptor blockade.

8 e raphe neurons were unaffected by ATP or P2-receptor blockade.

9 an effect attenuated by concomitant 5-HT2A/C receptor blockade.

10 ed in vaccinia virus-treated mice with IL-10 receptor blockade.

11 they were eliminated by systemic muscarinic receptor blockade.

12 -specific component acutely regulated by CB1 receptor blockade.

13 rability due to more selective dopamine (DA) receptor blockade.

14 tion, whereas SLEs were blocked by glutamate receptor blockade.

15 ts rely on the same mechanism: dopamine D(2) receptor blockade.

16 ustained attention was abolished by alpha(1)-receptor blockade.

17 opagation becomes stereotyped following GABA receptor blockade.

18 c-blocker administration or beta2-adrenergic receptor blockade.

19 response is sensitive to ET(B) but not ET(A) receptor blockade.

20 was attenuated by D2-like (but not D1-like) receptor blockade.

21 of GABAergic signaling and abolished by AMPA receptor blockade.

22 reased pro-nerve growth factor levels or p75 receptor blockade.

23 ercise ischaemia (PEI) and beta1 -adrenergic receptor blockade.

24 lating the solitary tract (ST) under GABA(A) receptor blockade.

25 based on differential extrasynaptic/synaptic receptor blockade.

26 ior and which are reversed by the muscarinic receptor blockade.

27 further antagonism by additional thromboxane receptor blockade.

28 ficacy is mediated by dopamine type 2 (D(2)) receptor blockade.

29 partial gamma-aminobutyric acid(A) (GABA(A)) receptor blockade.

30 from Cx30-deficient mice or with purinergic receptor blockade.

31 etains a bursting behaviour after ionotropic receptor blockade.

32 ut were dramatically reduced after Ang-(1-7) receptor blockade.

33 subdiaphragmatic vagotomy or corticosterone receptor blockade.

34 ptor system counteracts the effects of AT(4) receptor blockade.

35 t not its production) were sensitive to NMDA receptor blockade.

36 induces APOBEC3, inhibited by IFN-alpha/beta receptor blockade.

37 with inhibitors of caspases-1 and -5 or IL-1 receptor blockade.

38 cy calculations when considering therapeutic receptor blockade.

39 parable in extent to that obtained upon IL-1 receptor blockade.

40 or 8 weeks PI and was susceptible to 5-HT(3) receptor blockade.

41 reduced or not affected by chronic glutamate receptor blockade.

42 utamatergic activity after chronic glutamate receptor blockade.

43 potential that normally resulted from IP(3) receptor blockade.

44 iduals, men are more responsive to mu-opioid receptor blockade.

45 the resting membrane potential after GABA(A) receptor blockade.

46 iring is selectively attenuated following D1 receptor blockade.

47 improved clinical efficacy through extended receptor blockade.

48 rats, and this effect was prevented by ET(A) receptor blockade.

49 uginosa-infected mice with and without NKG2D receptor blockade.

50 iorated by A(1) receptor activation or A(2A) receptor blockade.

51 els and explore further the role of 5-HT(2A) receptor blockade.

52 The cardiac dysfunction is prevented by AT1 receptor blockade.

53 tes to calculate the net rate of reversal of receptor blockade.

54 re depletion or inositol 1,4,5-trisphosphate receptor blockade.

55 epression was prevented by MOR- but not GABA-receptor blockade.

56 ce indeed became hyperglycemic after insulin receptor blockade.

57 oid synthesis or highly selective prostanoid receptor blockade.

58 presence of adenosine A1 receptor and GABAB receptor blockade.

59 rome features may benefit from interleukin-1 receptor blockade.

60 the airways, which was reversed after IL-10 receptor blockade.

61 antly reduced by N-methyl-d-aspartate (NMDA) receptor blockade.

62 d hence lead to the potential for incomplete receptor blockade.

63 le ATR levels respond to GABA, but not NMDA, receptor blockade.

64 cells and were completely abrogated by IFN-I receptor blockade.

65 duration and failure rate during cholinergic receptor blockade.

66 refrontal cortex, were prevented by 5-HT2A/C receptor blockade.

67 logical reduction of fibrosis by angiotensin receptor blockade.

68 nditions in CGD that can be restored by IL-1 receptor blockade.

69 or AMI, possibly mediated by dopamine type 3 receptor blockades.

70 tive beneficial effects over beta-adrenergic receptor blockade, a current pharmacological heart failu

71 sponse to DBP-FITC was not affected by IFN-I receptor blockade, a finding consistent with the known d

72 gamma power is significantly reduced by NMDA receptor blockade, a treatment that paradoxically enhanc

73 IL-1beta receptor blockade abrogated aggravated NCGN in gp91(phox

74 Responses to selective receptor blockade, adenosine, nitroprusside, and verapam

75 beta-adrenergic receptor blockade after ACS is a measure of quality care

76 ognition 4 months after pharmacological NMDA receptor blockade already were affected by disrupted chr

77 IL-17A receptor blockade also abrogated the secondary effect of

78 Surprisingly, DA receptor blockade also altered the temperature fluctuati

79 NKG2D receptor blockade also resulted in decreased production

80 The effects of NMDA receptor blockade and ASIC1a blockade were compared.

81 This observation suggests that receptor blockade and axotomy interrupt the same signall

82 rats with neointima formation following VEGF receptor blockade and chronic hypoxia.

83 BA levels, consistent with the view that NOP receptor blockade and DOP receptor stimulation caused sy

84 d no effect on proliferation, concurrent RON receptor blockade and gemcitabine treatment increased ap

85 nsitivity in the mouse using pharmacological receptor blockade and genetic deletion of the channel.

86 nduced by vascular endothelial growth factor receptor blockade and hypoxia (Sugen/hypoxia) as well as

87 animals, vascular endothelial growth factor receptor blockade and hypoxia caused more severe pulmona

88 an be achieved by PRR stimulation, chemokine-receptor blockade and immune modulation of T cell functi

89 Angiotensin receptor blockade and inhibition of local AngII producti

90 O excitation occurs in the presence of GABAA receptor blockade and its EC50 value is two orders of ma

91 ctivation are sensitive to hippocampal D1/D5 receptor blockade and resistant to adrenoceptor blockade

92 nd that the synergistic effect of inhibitory receptor blockade and stimulation of costimulatory recep

93 from the recent clinical studies using IL-6 receptor blockade, and describe potential mechanisms by

94 baseline femoral neck T-score, interleukin-2 receptor blockade, and proteinuria (HR 2.02, 0.69, 0.4,

95 ich was prevented by concomitant angiotensin receptor blockade application and TRPC6 knockdown.

96 ety and beneficial effects of angiotensin II receptor blockade (ARB) in patients with structural hear

97 me 0 to 180 min; the response to angiotensin receptor blockade (ARB) was defined as the change from t

98 eir site of origin, important effects of CB1 receptor blockade are expressed via activation of periph

99 either 5-HT2C receptor activation nor 5-HT2A receptor blockade are sufficient to produce a therapeuti

100 l medical therapies, such as beta-adrenergic receptor blockade, are used to slow pathologic aortic gr

101 GABA(A) receptor blockade at the time of 3KA-SE or maternal sepa

102 ETA receptor blockade attenuated increases in macrophage inf

103 lthough we have known that mineralocorticoid receptor blockade attenuates cardiovascular and renal in

104 miting laboratory rodents) and that CeA NMDA receptor blockade attenuates cisplatin-induced anorexia

105 acological data showed that CeA AMPA/kainate receptor blockade attenuates cisplatin-induced pica (a p

106 study was to test the hypothesis that ET(A) receptor blockade attenuates superoxide production and i

107 scular diseases treated with beta-adrenergic receptor blockade (BB).

108 Mineralocorticoid receptor blockade before stress prevented the stress-ind

109 persisted following ipsilateral RVLM GABA(A) receptor blockade (bicuculline, BIC, 400 pmol, 100 nl; n

110 Mineralocorticoid receptor blockade blunted leptin-induced endothelial dys

111 an be activated in the presence of RVLM GABA receptor blockade, but sympathoinhibitory influences fro

112 ody weight gain; in contrast, intracore NMDA receptor blockade by AP-5 did not inhibit the energy bal

113 Angiotensin-(1-7) receptor blockade by D-Ala(7)-Ang-(1-7) prevented the AC

114 Receptor blockade by DHbetaE, an antagonist for heterome

115 al. now report that combining angiotensin II receptor blockade by losartan with beta-blocker treatmen

116 We have demonstrated that VEGF receptor blockade by SU5416 {3-[(2,4-dimethylpyrrol-5-yl

117 endent of glucocorticoids and glucocorticoid receptor blockade, by RU 486, increases striatal preproe

118 on of TGF-beta signaling through angiotensin receptor blockade can attenuate CS-induced lung injury i

119 ds in individuals with severe AH and if IL-2 receptor blockade can reverse this.

120 GABA(A) receptor blockade caused a decrease in the firing rate o

121 In contrast, adenosine receptor blockade caused a significant increase in CBF t

122 ms and brain regions through which adenosine receptor blockade causes arousal are incompletely unders

123 unusual human subject and in the monkey with receptor blockade decreased exposure to organs with high

124 GABAA and AMPA receptor activation but NMDA receptor blockade decreased oscillations only in the DP

125 ting enzyme inhibition nor mineralocorticoid receptor blockade decreased the primary outcome of posto

126 Endothelin (ET) receptor blockade delays the progression of diabetic nep

127 ient as a consequence of genetic deletion or receptor blockade demonstrated normal recruitment and se

128 se, suppressed replication is rescued by IFN-receptor blockade, demonstrating a role for IFN in restr

129 long-term bioluminescence recordings, GABAA receptor blockade desynchronized the Fbxl3(+/+) but not

130 Importantly, G-CSF receptor blockade did not adversely affect viral clearan

131 Based on rates of D-xylose absorption, GLP-1 receptor blockade did not affect gastric emptying of a s

132 Conversely, selective beta2 adrenergic receptor blockade did not affect MCFP responses to BMI.

133 ocal ATP application; however, purinergic P2-receptor blockade did not affect their CO2/H(+) responsi

134 Serotonin 1A receptor blockade did not alter PNFlx-evoked anxiety but

135 KORs in the LC together with beta-adrenergic receptor blockade did not potentiate KOR-induced reinsta

136 Dopamine receptor blockade did not prevent the MDPV-induced decre

137 In contrast, colony-stimulating factor 1 receptor blockade diminished C-C chemokine receptor type

138 Beta-receptor blockade does not alter the tachycardia phase t

139 Peritransplant IL-15 receptor blockade does not prolong allograft survival in

140 dy aimed to determine the impact of acute B2 receptor blockade during hemorrhagic shock in angiotensi

141 In fact, activin A receptor blockade during the M-CSF-driven differentiatio

142 neurons in nigral slices confirmed that NOP receptor blockade enhanced the DOP receptor-induced effe

143 These results suggest that D2-receptor blockade enhances content-specific representati

144 earchlight decoding approach we show that D2-receptor blockade enhances decoding of reward signals in

145 Epidermal growth factor receptor blockade enhances the effectiveness of RT alone

146 In contrast, adenosine receptor blockade fails to alter CBF or myocardial oxyge

147 y changes, we examined the effects of GABA-A receptor blockade, finding that picrotoxin (PTX) recapit

148 We also show that maintained darkness and D1 receptor blockade following maintained illumination and

149 The monkey with receptor blockade had an overall distribution qualitativ

150 5-HT7 receptor blockade had no effect on liver regeneration wh

151 S) that act by both CCR5 internalization and receptor blockade had not been reported until recently (

152 sympathetic drive to the heart through beta-receptor blockade has become a standard component of the

153 Dopamine D2 receptor blockade has been an obligate mechanism of acti

154 ntroduction of immunotherapy with checkpoint receptor blockade has changed the treatment of advanced

155 , including cancer vaccines and coinhibitory receptor blockade, have demonstrated clinical efficacy i

156 However, how acute NMDA receptor blockade impacts on brain functioning at a syst

157 ine-treated mice to elucidate how acute NMDA receptor blockade impacts on the properties of functiona

158 tantly, intra-LA NMDA (N-methyl-d-aspartate)-receptor blockade impaired reward-learning performance a

159 ized controlled trial, long-term aldosterone receptor blockade improved left ventricular diastolic fu

160 ently have we learned that mineralocorticoid receptor blockade improves pancreatic insulin release, i

161 cribe for the first time the effect of G-CSF receptor blockade in a therapeutic model of inflammatory

162 Moreover, NK cell receptor blockade in cytotoxicity assays demonstrates th

163 Prolonged AMPA-receptor blockade in hippocampal neuron cultures leads t

164 IL-15 receptor blockade in mouse cardiac and islet allotranspl

165 GABA(A) receptor blockade in post-sensitive period slices revert

166 showed that this compound provides 90% NK(1) receptor blockade in rhesus brain at a plasma level of 6

167 l function independent of weight loss or CB1 receptor blockade in the brain, suggesting that peripher

168 BA), neuropeptide Y (NPY), or beta-endorphin receptor blockade in the ipsilateral hypothalamic parave

169 NMDA receptor blockade in the LH-PeF nearly eliminated intra-

170 symptoms, which are caused by dopamine (DA) receptor blockade in the neostriatum.

171 reduced by EA and then restored with opioid receptor blockade in the PVN.

172 sensus on the relative importance of beta(2) receptor blockade in treating glaucoma may have to be re

173 and by examining of the impact of glutamate receptor blockade in two feeding-related targets of vmPF

174 AT(2) receptor blockade increased AT(1) receptor binding and m

175 tor were modest or absent, and selective EP2 receptor blockade increased cytokine release in some ins

176 GLP-1 receptor blockade increased postprandial glucagon in bot

177 udies provide novel evidence that VTA amylin receptor blockade increases food intake and attenuates t

178 e mice, while systemic 2-AG depletion or CB1 receptor blockade increases susceptibility in previously

179 Prasugrel P2Y(12) receptor blockade is associated with greater pharmacodyn

180 by prolonged gamma-aminobutyric acid type A receptor blockade is independent of brain-derived nerve

181 This beneficial effect of B2 receptor blockade is rapidly reached and sustained with

182 by either PA50 mAb or Fab and suggested that receptor blockade is the mechanism by which PA50 neutral

183 eficiency nor pharmacological glucocorticoid receptor blockade lowered elevated blood glucose levels.

184 In contrast, during beta1 -adrenergic receptor blockade, LV apical rotation, twist and untwist

185 Altogether, these results suggest that B2 receptor blockade may be a promising strategy to prevent

186 Alternatively, muscarinic receptor blockade may cause a negative hedonic state tha

187 Adenosine receptor blockade may improve glomerular filtration and

188 Behavioral impairment following nicotinic receptor blockade may not be due to the inability of the

189 IL-1 receptor blockade may provide a promising strategy in NC

190 oma cell interactions through specific Notch receptor blockade may represent a promising treatment st

191 Cannabinoid type 1 receptor blockade may thus counteract maladaptive altera

192 indings raise the possibility that A or A(A) receptors blockade might also confer a disease-modifying

193 DA receptor blockade mimics some, but not all, aspects of n

194 AMPA/kainate receptor blockade (NBQX, 100 microM) or Group II metabot

195 Neither angiotensin II receptors blockade nor alpha and beta adrenergic recepto

196 Ang II type 1 receptor blockade normalized the activation of the cycli

197 ther 5-HT2A/C receptor stimulation or 5-HT1A receptor blockade of naive control pups.

198 n=8) or bilateral (n=6) GABA(A) and GABA(B) receptor blockade of the RVLM (400 pmol BIC+400 pmol CGP

199 hysiologic data, the effects of spinal 5-HT3 receptor blockade on behavioral hypersensitivity and neu

200 degradation offsets the effects of oxytocin receptor blockade on both social place preference and cF

201 n VMH GABA levels and the effects of GABA(A) receptor blockade on counterregulatory responses to a st

202 e the dose-dependent effects of adenosine A1-receptor blockade on diuresis and renal function in pati

203 study was to investigate the effect of 5-HT7 receptor blockade on liver regeneration after partial he

204 s neutropenic, suggesting an effect of G-CSF receptor blockade on neutrophil homing to inflammatory s

205 Here, we examined the effects of muscarinic receptor blockade on rule-related activity in the macaqu

206 , we examined the effects of hindbrain 5-HT3 receptor blockade on suppression of intake by systemic C

207 quencing to explore the effects of H1 and H2 receptor blockade on the exercise transcriptome in human

208 uences of ventral tegmental area (VTA) CB(1) receptor blockade on the wheel-running performances of w

209 tional CB(1) receptor mutations and of CB(1) receptor blockade on wheel-running performance.

210 nd the antiproteinuric effect of angiotensin receptor blockade or angiotensin-converting enzyme inhib

211 nts, we studied the effect of adenosine A(1) receptor blockade or deletion on bone density.

212 diators, or cell types was assessed based on receptor blockade or depletion.

213 is similar to published effects of nicotinic receptor blockade or desensitization, and is mediated by

214 release evoked electrically during nicotinic receptor blockade or optogenetically by light activation

215 easurement of thrombus height after specific receptor blockade or use of altered proportions of pepti

216 s (P<0.001), which was reduced with thrombin receptor blockade (P=0.013).

217 PD-1 inhibitory receptor blockade partially reversed T cell unresponsive

218 ombined findings suggest that IFN-alpha/beta receptor blockade, particularly when started at early di

219 y paracrine inhibition, because somatostatin receptor blockade potently stimulated glucagon release w

220 discrete-trial procedures employed here, D1 receptor blockade preferential reduces Pavlovian and ope

221 The results suggest that D2 receptor blockade preferentially increases response dura

222 Aryl hydrocarbon receptor blockade prevented differentiation of the naive

223 Glucocorticoid receptor blockade prevented fasting-induced tissue Angpt

224 In addition, NMDA receptor blockade prevented memory-associated alteration

225 Postnatal 5-HT2A/C receptor blockade prevented PNFlx-evoked anxiety, attenu

226 CB cannabinoid receptor blockade prevented these effects.

227 erpolarizing current injection, but not AMPA receptor blockade, prevents synaptic stimulation from fa

228 was therefore of interest to ask whether DA receptor blockade produces extinction-like increases in

229 Where acute NMDA receptor blockade promotes hyperconnectivity in function

230 signals were mediated by B2 receptors and B2 receptor blockade protected against PAC necrosis evoked

231 Platelet-activating factor receptor blockade protected synapses in the mouse model,

232 inant-negative mutant of AMPKalpha1, and P2Y receptor blockade reduced DOR-stimulated glucose uptake.

233 D(1)-like receptor blockade reduced SNr GABA neuron firing frequen

234 Additionally, NKG2D receptor blockade reduced the epithelial cell sloughing

235 B2 receptor blockade reduced the extent of PAC necrosis evo

236 These data suggest that prenatal NMDA receptor blockade reduces the level of progenitors and t

237 IL-2 receptor blockade represents a possible mechanism to ove

238 ast, cardiac afferents, in the absence of TP receptor blockade responded consistently to repeated adm

239 Conversely, IFN-alphabeta receptor blockade restored Tfh and GC B cell phenotypes

240 alpha1-Receptor blockade resulted in a small decrease in ATP ov

241 Overall, our results suggest that muscarinic receptor blockade results in a bona fide learning impair

242 mma power is significantly reduced with NMDA receptor blockade, revealing a latent cortical network p

243 Bradykinin receptor blockade reversed the inhibitory effect of enal

244 Tetrodotoxin (TTX), but not postsynaptic receptor blockade, reversed depolarization-induced adapt

245 DA receptor blockade (SCH23390+eticlopride) fully eliminate

246 ne, recordings were conducted during full DA receptor blockade (SCH23390+eticloptide).

247 ET imaging of 22 patients to assess androgen-receptor blockade showed decreased (18)F-fluoro-5alpha-d

248 During hemorrhagic shock, acute B2 receptor blockade significantly attenuates the deleterio

249 Although N-methyl-d-aspartate (NMDA) receptor blockade stabilizes spines in the neocortex, in

250 smission also blunted central effects of CB1 receptor blockade, such as fear responses and anxiety-li

251 Furthermore, elements upstream of receptor blockade, such as hypoxia-inducible factor (HIF

252 Furthermore, the differential effects of receptor blockade suggest novel methods of immune respon

253 ased 6.5-fold and minimal responses to IL-10 receptor blockade suggested downregulated IL-10.

254 blockade in the NAc, and not local D(1)-like receptor blockade, suggesting a role for glutamate affer

255 sine-phosphate phosphatase and by purinergic receptor blockade (suramin).

256 they represent novel therapeutic targets for receptor blockade therapies.

257 These data suggest that ET-A receptor blockade therapy could serve as a coadjuvant in

258 immunoabsorption as well as subsequent IL-6 receptor blockade through tocilizumab, a complete and st

259 urophysiological recordings, and cholinergic receptor blockade to delineate the cholinergic contribut

260 This unique approach limits IL-1 receptor blockade to sites of inflammation, while sparin

261 igration assays, and antibody-mediated IL-10 receptor blockade to study plasmacytosis-associated IL-1

262 PS administration enhanced alpha1beta2gamma2 receptor blockade (to 98% in both cases).

263 We find that GABA(A) receptor blockade triggers fast changes in cellular exci

264 se results demonstrate that, spurred by AMPA-receptor blockade, up-regulation of betaCaMKII promotes

265 nverting enzyme inhibitors or angiotensin II receptor blockade use (OR, 0.42; [95% CI, 0.22-0.80]; P=

266 -converting enzyme inhibitors/angiotensin II receptor blockade use remained independent predictors fo

267 GD patients who had severe colitis with IL-1 receptor blockade using anakinra.

268 onist exenatide, with or without prior GLP-1 receptor blockade using exendin 9-39, on brain responses

269 effects were largely blocked by prior GLP-1 receptor blockade using exendin 9-39.

270 The hemodynamic effect of B2 receptor blockade using icatibant (B2 receptor antagonis

271 his study investigated the efficacy of IL-15 receptor blockade using Mut-IL-15/Fc in an outbred non-h

272 Cortisol response following mu-opioid receptor blockade using naltrexone in 119 of these subje

273 e (NHP) model of schizophrenia based on NMDA-receptor blockade using subanesthetic administration of

274 Delta-18 +/- 2%), an effect prevented by V1a receptor blockade (V2255), supporting local dendritic VP

275 s to adenosine and ATP were influenced by P1-receptor blockade via aminophylline.

276 (RR 0.45; 95% CI 0.24-0.83), and angiotensin receptor blockade vs placebo (RR 0.65; 95% CI 0.49-0.85)

277 sin II test showed that complete angiotensin receptor blockade was achieved only in the high-dosage g

278 5-HT7 receptor blockade was applied by intraperitoneal adminis

279 The P2Y12-adenosine diphosphate receptor blockade was assessed by the vasodilator-stimul

280 In this subgroup analysis, interleukin-1 receptor blockade was associated with significant improv

281 conditioned rats), intra-accumbens shell DA receptor blockade was found to prevent the expression of

282 Finally, by using ADP receptor blockade we confirm that NPP4 mediates platelet

283 isms underlying the hypophagic effect of CB1 receptor blockade, we combined the acute injection of th

284 Using bilateral local intracerebral hormone-receptor blockade, we found that ARs and ERs in the song

285 The major result was that effects of receptor blockade were most closely related to the first

286 -converting enzyme inhibitors/angiotensin II receptor blockade, were associated with the presence of

287 (both male morphs are sensitive to androgen receptor blockade, whereas females are not).

288 Vascular endothelial growth factor (VEGF) receptor blockade, which resulted in EC hyperproliferati

289 therapy with NEP-inhibitors and angiotensin-receptor-blockade, which has been shown being a promisin

290 enclamide (50 microg/kg/min ic) or adenosine receptor blockade with 8-phenyltheophylline (8-PT; 5 mg/

291 PD98059 and EP2 receptor blockade with AH6809 (6-isopropoxy-9-oxoxanthen

292 In contrast, cannabinoid type-1 (CB1) receptor blockade with AM251 (10 mum) prevented the acti

293 ission) (study 1) and 2) the effect of GLP-1 receptor blockade with exendin (9-39) on glucose toleran

294 ring a hyperglycemic clamp with either GLP-1 receptor blockade with exendin-(9-39) or saline.

295 Generalized glutamate receptor blockade with intracerebroventricular (i.c.v.)

296 One monkey was scanned after receptor blockade with PK 11195 (10.7 mg/kg intravenousl

297 Our data indicate that TP-receptor blockade with terutroban decreases portal press

298 Interleukin-6 receptor blockade with tocilizumab remains the mainstay

299 s largely antagonized by selective serotonin receptor blockade, with little further antagonism by add

300 We examined here whether D1 receptor blockade within striatal or frontal cortical DA