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1 vated in mice with astrocyte-specific leptin receptor deficiency.
2 ypothesis using two separate models of GLP-1 receptor deficiency.
3 e ablated by angiotensin II type 1a (AT(1a)) receptor deficiency.
4 leptin cannot be used as a marker for leptin-receptor deficiency.
5 ological antagonism of AT1 receptors or AT1A receptor deficiency.
6 rapsyn expression and endplate acetylcholine receptor deficiency.
7 eptin receptor and mice with platelet leptin-receptor deficiency.
8 se models of neoplastic disease with TGFbeta receptor deficiencies.
9                            Whole body AT(1a) receptor deficiency ablated Ang II-induced ascending AAs
10                 Transplanting islets with Y1 receptor deficiency accelerates the normalization of hyp
11 rmore, the obesity induced by melanocortin 4 receptor deficiency also constricted the T-cell repertoi
12                                         P2X1 receptor deficiency also was associated with a marked re
13                                        G-CSF receptor deficiency and CXCL1 blockade suppressed myeloi
14 tment to alveolar air spaces was impaired by receptor deficiency, as was pulmonary expression of the
15  data show that IL-9 neutralization and IL-9 receptor deficiency attenuates disease, and this correla
16 s that was not affected by P2Y(12) or TXA(2) receptor deficiency, but was inhibited by the selective
17 nia, acetylcholinesterase deficiency and ACh-receptor deficiency; but genes encoding both structural
18                                         AT1A receptor deficiency caused a marked decrease in atherosc
19            Compounding the mutation with LDL receptor deficiency caused enhanced accumulation of both
20            Compounding the mutation with LDL receptor deficiency caused enhanced accumulation of both
21                 We report herein that 5-HT2B receptor deficiency confers a wide spectrum of antipsych
22 at C3a overexpression increased, whereas C3a receptor deficiency decreased post-stroke expression of
23 onic nitric oxide synthase I and thromboxane receptor deficiency did not change TGF responsiveness.
24                                   TNF type I receptor deficiency did not prevent obesity and SH.
25 mia caused either by apolipoprotein E or LDL receptor deficiency did not show convincing changes in t
26       While AngII infusion induced AAAs, AT2 receptor deficiency did not significantly affect either
27                                          AT2 receptor deficiency does not affect AngII-induced AAAs,
28 stinct neuronal populations and that ghrelin receptor deficiency does not affect sensitivity to the a
29 al and cellular levels demonstrate that NMDA receptor deficiency during prenatal development may unle
30 rosis and found that macrophage-specific AT1 receptor deficiency exacerbates kidney fibrosis induced
31 TATEMENT People and mice with growth hormone receptor deficiency (GHRD or Laron syndrome) are protect
32                               Growth hormone receptor deficiency (GHRD) results in short stature, enh
33       Introgression of leptin production and receptor deficiencies had separate effects from long-ter
34                                         AT1A receptor deficiency had a striking effect in reducing hy
35 red in AT1A receptor-deficient mice, and AT2 receptor deficiency had no effect on lesion area or cell
36                                          AT2 receptor deficiency had no significant effect on systoli
37                                 IFNalphabeta receptor deficiency in 129 mice decreases morbidity, lun
38                                        IL-33 receptor deficiency in Il4raF709 Il1rl1(-/-) mice protec
39 ovides a basis for the marked effect of AT1A receptor deficiency in reducing atherosclerosis.
40 etic linkage studies strongly implicate NMDA receptor deficiency in schizophrenia and suggest that re
41                          In addition, leptin receptor deficiency in T cells inhibits Th17 differentia
42 estations were caused by the offspring's own receptor deficiency, indicating that the genetic and non
43                                         AT1A receptor deficiency-induced reductions in atherosclerosi
44  myasthenic syndrome, in which acetylcholine receptor deficiency is due to mutations in rapsyn.
45 nzyme in cholesterol biosynthesis and that a receptor deficiency is responsible for a major genetic c
46 We hypothesized that pulmonary vascular ET-B receptor deficiency leads to increased lung ET, that exc
47                                          A2A receptor deficiency led to increased numbers of allogene
48                                    Scavenger receptor deficiency markedly impaired the recruitment of
49                                     Cytokine receptor deficiencies may contribute to immune deficienc
50 entify two distinct mechanisms by which NMDA receptor deficiency may disrupt frontal lobe function: a
51 s provide a physiological basis for the NMDA receptor deficiency model of schizophrenia and may clari
52                                  Neither AT2 receptor deficiency nor PD123319 had any significant eff
53 e have examined the effect of angiotensin II receptor deficiency on 4-(methylnitrosamino)-1-(3-pyridy
54  a rationale for the profound effect of AT1A receptor deficiency on atherogenesis.
55                           The effects of MT1 receptor deficiency on retinal morphology was investigat
56  this setting and tested the impact of IL-17 receptor deficiency or antibody-mediated neutralization
57  of renin production induced by global AT(1) receptor deficiency or by receptor blockade.
58 tations causing leptin production and leptin receptor deficiency, respectively, were introgressed ind
59                            Congenital leptin-receptor deficiency should be considered in the differen
60                          Animals with 5-HT2A receptor deficiency showed less immobility in the Porsol
61                                          C5a receptor deficiency, which also lessens myeloid-derived

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