ライフサイエンスコーパス: receptor deficiency

1 vated in mice with astrocyte-specific leptin receptor deficiency.

2 ypothesis using two separate models of GLP-1 receptor deficiency.

3 e ablated by angiotensin II type 1a (AT(1a)) receptor deficiency.

4 leptin cannot be used as a marker for leptin-receptor deficiency.

5 ological antagonism of AT1 receptors or AT1A receptor deficiency.

6 rapsyn expression and endplate acetylcholine receptor deficiency.

7 eptin receptor and mice with platelet leptin-receptor deficiency.

8 obese mice or in mice with adipocyte insulin receptor deficiency.

9 ed in mice with Treg-specific glucocorticoid receptor deficiency.

10 resembling the phenotype of thyroid hormone receptor deficiency.

11 obesity, such as POMC deficiency and leptin receptor deficiency.

12 larmin and the genetic mouse model of CRF(1) receptor-deficiency.

13 se models of neoplastic disease with TGFbeta receptor deficiencies.

14 Whole body AT(1a) receptor deficiency ablated Ang II-induced ascending AAs

15 Transplanting islets with Y1 receptor deficiency accelerates the normalization of hyp

16 rmore, the obesity induced by melanocortin 4 receptor deficiency also constricted the T-cell repertoi

17 P2X1 receptor deficiency also was associated with a marked re

18 G-CSF receptor deficiency and CXCL1 blockade suppressed myeloi

19 Hepatocyte Il6 receptor deficiency and Stat3 deficiency both aggravate

20 del to mirror the treatment of acetylcholine receptor deficiency, and demonstrate improved muscle fat

21 tment to alveolar air spaces was impaired by receptor deficiency, as was pulmonary expression of the

22 data show that IL-9 neutralization and IL-9 receptor deficiency attenuates disease, and this correla

23 s that was not affected by P2Y(12) or TXA(2) receptor deficiency, but was inhibited by the selective

24 nia, acetylcholinesterase deficiency and ACh-receptor deficiency; but genes encoding both structural

25 Patients with acetylcholine receptor deficiency can also benefit from the addition o

26 AT1A receptor deficiency caused a marked decrease in atherosc

27 Compounding the mutation with LDL receptor deficiency caused enhanced accumulation of both

28 Compounding the mutation with LDL receptor deficiency caused enhanced accumulation of both

29 We report herein that 5-HT2B receptor deficiency confers a wide spectrum of antipsych

30 at C3a overexpression increased, whereas C3a receptor deficiency decreased post-stroke expression of

31 onic nitric oxide synthase I and thromboxane receptor deficiency did not change TGF responsiveness.

32 TNF type I receptor deficiency did not prevent obesity and SH.

33 mia caused either by apolipoprotein E or LDL receptor deficiency did not show convincing changes in t

34 While AngII infusion induced AAAs, AT2 receptor deficiency did not significantly affect either

35 AT2 receptor deficiency does not affect AngII-induced AAAs,

36 stinct neuronal populations and that ghrelin receptor deficiency does not affect sensitivity to the a

37 Interleukin-21 receptor deficiency does not lead to reduction in mucosa

38 al and cellular levels demonstrate that NMDA receptor deficiency during prenatal development may unle

39 Accordingly, genetic CRF(1) receptor-deficiency eliminated morphine-induced sociabil

40 tively correlate with muscle NAD, and TAS1R2 receptor deficiency enhances NAD responses across the gl

41 rosis and found that macrophage-specific AT1 receptor deficiency exacerbates kidney fibrosis induced

42 TATEMENT People and mice with growth hormone receptor deficiency (GHRD or Laron syndrome) are protect

43 Growth hormone receptor deficiency (GHRD) results in short stature, enh

44 Introgression of leptin production and receptor deficiencies had separate effects from long-ter

45 AT1A receptor deficiency had a striking effect in reducing hy

46 red in AT1A receptor-deficient mice, and AT2 receptor deficiency had no effect on lesion area or cell

47 AT2 receptor deficiency had no significant effect on systoli

48 Leptin receptor deficiency impairs T(FH) generation and antibod

49 IFNalphabeta receptor deficiency in 129 mice decreases morbidity, lun

50 IL-33 receptor deficiency in Il4raF709 Il1rl1(-/-) mice protec

51 ovides a basis for the marked effect of AT1A receptor deficiency in reducing atherosclerosis.

52 etic linkage studies strongly implicate NMDA receptor deficiency in schizophrenia and suggest that re

53 In addition, leptin receptor deficiency in T cells inhibits Th17 differentia

54 estations were caused by the offspring's own receptor deficiency, indicating that the genetic and non

55 AT1A receptor deficiency-induced reductions in atherosclerosi

56 This study investigates whether androgen receptor deficiency is associated with increased asthma

57 myasthenic syndrome, in which acetylcholine receptor deficiency is due to mutations in rapsyn.

58 nzyme in cholesterol biosynthesis and that a receptor deficiency is responsible for a major genetic c

59 Acetylcholine receptor deficiency is the most common form of the conge

60 vide conclusive evidence that TLR7 and IFN-I receptor deficiencies lead to severe disease in mice, re

61 We hypothesized that pulmonary vascular ET-B receptor deficiency leads to increased lung ET, that exc

62 A2A receptor deficiency led to increased numbers of allogene

63 Scavenger receptor deficiency markedly impaired the recruitment of

64 Cytokine receptor deficiencies may contribute to immune deficienc

65 entify two distinct mechanisms by which NMDA receptor deficiency may disrupt frontal lobe function: a

66 ons) and a Mendelian disease (melanocortin 4 receptor deficiency (MC4R)) that affect BMI; and two Men

67 s provide a physiological basis for the NMDA receptor deficiency model of schizophrenia and may clari

68 Neither AT2 receptor deficiency nor PD123319 had any significant eff

69 e have examined the effect of angiotensin II receptor deficiency on 4-(methylnitrosamino)-1-(3-pyridy

70 for a cohort of patients with acetylcholine receptor deficiency on anticholinesterase medication tha

71 a rationale for the profound effect of AT1A receptor deficiency on atherogenesis.

72 Importantly, in context of obesity, insulin receptor deficiency on CD8 T cells did not affect the fu

73 The effects of MT1 receptor deficiency on retinal morphology was investigat

74 this setting and tested the impact of IL-17 receptor deficiency or antibody-mediated neutralization

75 of renin production induced by global AT(1) receptor deficiency or by receptor blockade.

76 Likewise, genetic CRF(1) receptor-deficiency reduced morphine-induced firing of P

77 tations causing leptin production and leptin receptor deficiency, respectively, were introgressed ind

78 Most patients with acetylcholine receptor deficiency respond well to acetylcholinesterase

79 wever, neither insulin injection nor insulin receptor deficiency resulted in a difference in metaboli

80 In conclusion, M(1) receptor deficiency results in retinal ganglion cell los

81 We find that IL-21 receptor deficiency results in smaller GC that are profo

82 Thus, ALX receptor deficiency serves as an experimental model that

83 Congenital leptin-receptor deficiency should be considered in the differen

84 Animals with 5-HT2A receptor deficiency showed less immobility in the Porsol

85 y excessive hunger, including melanocortin-4 receptor deficiency, that present with low blood pressur

86 ed in an experimental model of acetylcholine receptor deficiency, the most common form of congenital

87 Wnt2b expression under conditions of leptin receptor deficiency, which also induced a delay in crypt

88 C5a receptor deficiency, which also lessens myeloid-derived