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1 vated in mice with astrocyte-specific leptin receptor deficiency.
2 ypothesis using two separate models of GLP-1 receptor deficiency.
3 e ablated by angiotensin II type 1a (AT(1a)) receptor deficiency.
4 leptin cannot be used as a marker for leptin-receptor deficiency.
5 ological antagonism of AT1 receptors or AT1A receptor deficiency.
6 rapsyn expression and endplate acetylcholine receptor deficiency.
7 eptin receptor and mice with platelet leptin-receptor deficiency.
8 se models of neoplastic disease with TGFbeta receptor deficiencies.
11 rmore, the obesity induced by melanocortin 4 receptor deficiency also constricted the T-cell repertoi
14 tment to alveolar air spaces was impaired by receptor deficiency, as was pulmonary expression of the
15 data show that IL-9 neutralization and IL-9 receptor deficiency attenuates disease, and this correla
16 s that was not affected by P2Y(12) or TXA(2) receptor deficiency, but was inhibited by the selective
17 nia, acetylcholinesterase deficiency and ACh-receptor deficiency; but genes encoding both structural
22 at C3a overexpression increased, whereas C3a receptor deficiency decreased post-stroke expression of
23 onic nitric oxide synthase I and thromboxane receptor deficiency did not change TGF responsiveness.
25 mia caused either by apolipoprotein E or LDL receptor deficiency did not show convincing changes in t
28 stinct neuronal populations and that ghrelin receptor deficiency does not affect sensitivity to the a
29 al and cellular levels demonstrate that NMDA receptor deficiency during prenatal development may unle
30 rosis and found that macrophage-specific AT1 receptor deficiency exacerbates kidney fibrosis induced
31 TATEMENT People and mice with growth hormone receptor deficiency (GHRD or Laron syndrome) are protect
35 red in AT1A receptor-deficient mice, and AT2 receptor deficiency had no effect on lesion area or cell
40 etic linkage studies strongly implicate NMDA receptor deficiency in schizophrenia and suggest that re
42 estations were caused by the offspring's own receptor deficiency, indicating that the genetic and non
45 nzyme in cholesterol biosynthesis and that a receptor deficiency is responsible for a major genetic c
46 We hypothesized that pulmonary vascular ET-B receptor deficiency leads to increased lung ET, that exc
50 entify two distinct mechanisms by which NMDA receptor deficiency may disrupt frontal lobe function: a
51 s provide a physiological basis for the NMDA receptor deficiency model of schizophrenia and may clari
53 e have examined the effect of angiotensin II receptor deficiency on 4-(methylnitrosamino)-1-(3-pyridy
56 this setting and tested the impact of IL-17 receptor deficiency or antibody-mediated neutralization
58 tations causing leptin production and leptin receptor deficiency, respectively, were introgressed ind
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