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1 ), a protein involved in AMPA-type glutamate receptor endocytosis.
2 d levels of EEA1, a protein involved in AMPA receptor endocytosis.
3 ), a dominant negative mutant known to block receptor endocytosis.
4 io-ligand binding and recovery after maximal receptor endocytosis.
5 rin/activator protein 2 (AP2)-mediated GABAA receptor endocytosis.
6 d specific isoforms of endophilin to enhance receptor endocytosis.
7 ults in complete abolition of ligand-induced receptor endocytosis.
8 from the plasma membrane without concomitant receptor endocytosis.
9 e formation and trafficking, cytokinesis and receptor endocytosis.
10 ptor subunit NR2B at Tyr1472 correlated with receptor endocytosis.
11 n-2, a signal transducing GTPase involved in receptor endocytosis.
12 EGFR is essential and sufficient to support receptor endocytosis.
13 gesting that stargazin does not inhibit AMPA receptor endocytosis.
14 regulated kinases 1 and 2 (ERK1/2) and rapid receptor endocytosis.
15 zed, presumptively through clathrin-mediated receptor endocytosis.
16 H1R) results in several signaling events and receptor endocytosis.
17 bits Fz7 signaling, probably by blocking Fz7 receptor endocytosis.
18 not necessary for ERK1/2 phosphorylation or receptor endocytosis.
19 ptors, is mediated in part by agonist-driven receptor endocytosis.
20 he tyrosine kinase in the activity-dependent receptor endocytosis.
21 ating classic complement pathway and Fcgamma receptor endocytosis.
22 -95 ubiquitination prevent NMDA-induced AMPA receptor endocytosis.
23 E3 ligase that participates in cell surface receptor endocytosis.
24 teraction did not interfere with transferrin receptor endocytosis.
25 ithout any significant effect on transferrin receptor endocytosis.
26 lerance does not necessarily seem to involve receptor endocytosis.
27 nucleotide exchange factor, ARNO, to enhance receptor endocytosis.
28 time course of ligand-stimulated muscarinic receptor endocytosis.
29 plex, leading to inhibition of autophagy and receptor endocytosis.
30 ester but had no detectable effect on basal receptor endocytosis.
31 ing this lipase to participate in muscarinic receptor endocytosis.
32 esicle fraction may have a role in muscarine receptor endocytosis.
33 mobilization, chemotaxis, and ligand-induced receptor endocytosis.
34 a role for PLD in signaling that facilitates receptor endocytosis.
35 r FKBP12 as a negative regulator of TGF-beta receptor endocytosis.
36 tion is correlated with an increased rate of receptor endocytosis.
37 ling activities and cessation of others upon receptor endocytosis.
38 y to allow the normal rate of ligand-induced receptor endocytosis.
39 inhibits capping of both receptors, but not receptor endocytosis.
40 t Tyr992, reduces the rate of ligand-induced receptor endocytosis.
41 activation, we postulated a role for CAM in receptor endocytosis.
42 stin as the agonist-driven switch initiating receptor endocytosis.
43 ction in clathrin mediated G protein-coupled receptor endocytosis.
44 evels of P. carinii phagocytosis and mannose receptor endocytosis.
45 ARF proteins in regulating beta2-adrenergic receptor endocytosis.
46 de or be needed indirectly in events such as receptor endocytosis.
47 ly contain NPxY motifs that are required for receptor endocytosis.
48 suppressor mutant dynamin K44A, which blocks receptor endocytosis.
49 ere weaker in strains that were defective in receptor endocytosis.
50 K3 phosphorylation at Tyr980/Tyr981 followed receptor endocytosis.
51 ction for monoubiquitination in alpha-factor receptor endocytosis.
52 inal tail is crucial for glucagon-stimulated receptor endocytosis.
53 ated signal transduction properties, and for receptor endocytosis.
54 cAMP production, and St-Ht31 increased basal receptor endocytosis.
55 ition of cAMP production and increased basal receptor endocytosis.
56 eptor levels, and enhanced postsynaptic AMPA receptor endocytosis.
57 ons, which result in altered calcium-sensing receptor endocytosis.
58 n gephyrin is only reduced after the initial receptor endocytosis.
59 ng corticosterone-induced inhibition of NMDA receptor endocytosis.
60 s (5-HT, DA, DOI, and clozapine) bring about receptor endocytosis.
61 neuronal plasma membrane, and regulation of receptor endocytosis.
62 hape synaptic connectivity through localized receptor endocytosis.
63 adaptin protein-2 complex (AP2) and ensuing receptor endocytosis.
64 ell surface mGluR1a expression via decreased receptor endocytosis.
65 face heparan sulfate or a reduction in TRAIL receptor endocytosis.
66 D-95 may regulate NMDA receptor-induced AMPA receptor endocytosis.
67 een implicated in NMDA receptor-induced AMPA receptor endocytosis.
68 e molecular level, at least antibody induced receptor endocytosis.
69 orting protein Disabled-2 (Dab2) in TGF-beta receptor endocytosis.
70 estin SUMOylation mediates G protein-coupled receptor endocytosis.
71 and reversed by blocking ARF6 activation or receptor endocytosis.
72 and Dynamin regulate cell migration by PDGF receptor endocytosis.
73 by which cell migration is regulated by PDGF receptor endocytosis.
74 portant role in activity-dependent glutamate receptor endocytosis.
75 losely related isoform PLIC-1 did not affect receptor endocytosis.
76 cluding DNA repair, signal transduction, and receptor endocytosis.
77 wever, CIE treatment did not reduce the NMDA receptor endocytosis.
78 it promotes receptor insertion and decreases receptor endocytosis.
79 s can act as dominant negative inhibitors of receptor endocytosis.
80 receptor dimerization increases TRH-induced receptor endocytosis.
81 n growth factor-regulated actin assembly and receptor endocytosis, although the underlying mechanisms
82 ion stemmed from the known interplay between receptor endocytosis and actin filament formation, becau
83 tions of these molecules to include roles in receptor endocytosis and activation of MAP kinase signal
84 ptor cytoplasmic loop blocked PMA-stimulated receptor endocytosis and also prevented PMA inhibition o
85 f insulin are largely independent of insulin receptor endocytosis and are initiated by activation of
88 plicated Ruk in the regulation of apoptosis, receptor endocytosis and cytoskeletal rearrangements.
89 he channel through a mechanism that promotes receptor endocytosis and degradation and lend support to
91 and Ihog activity are mutually required for receptor endocytosis and degradation, triggered by Hedge
93 in surface AMPARs is accompanied by enhanced receptor endocytosis and dependent on proteasomal activi
94 for Vav family GEFs as regulators of ligand-receptor endocytosis and determinants of repulsive signa
97 ut report here that CD14 is not required for receptor endocytosis and downstream signaling mediated b
98 e PMM (phorbol 12-monomyristate), stimulated receptor endocytosis and inhibited glycine-gated chlorid
100 e endosomes that is involved in cell-surface receptor endocytosis and it also directly interacts with
102 dence that this form of memory requires AMPA receptor endocytosis and long-term depression of excitat
103 exposure of TRPV1 to agonists induces rapid receptor endocytosis and lysosomal degradation in both s
105 nternalization motif, resulting in defective receptor endocytosis and markedly increased TfR1 express
107 also suggest, conversely, that mechanisms of receptor endocytosis and molecular sorting may themselve
109 f spinophilin with Group I mGluRs attenuates receptor endocytosis and phosphorylation of ERK1/2, an e
110 F-dependent cell migration by promoting PDGF receptor endocytosis and Rac1 activation at the cell mem
111 of neuronal proteins implicated in synaptic receptor endocytosis and recycling, as well as in membra
114 R4 in rvs161 cells reinitiates Ste3 a-factor receptor endocytosis and requires Cdc55 function to do s
115 UDP-GlcNAc, a sugar-nucleotide that inhibits receptor endocytosis and signaling by promoting N-acetyl
117 ecursor protein modulates alpha2A-adrenergic receptor endocytosis and signaling through disrupting ar
118 gnaling pathway and demonstrated its role in receptor endocytosis and termination of the ERK1/2 signa
119 se results suggest a role for PIP5K-Ibeta in receptor endocytosis and that the truncated enzyme compe
122 actor receptor (EGFR) is mediated in part by receptor endocytosis and trafficking to the lysosomal de
123 mannose 6-phosphate receptors, we found that receptor endocytosis and transport to the trans-Golgi ne
126 sory deprivation most likely results in AMPA receptor endocytosis and/or lateral diffusion to the ext
127 In vivo Prk1p inhibition blocked pheromone receptor endocytosis, and caused cortical actin patches
131 of G proteins, while concurrently mediating receptor endocytosis, and some aspects of receptor signa
132 nd provide an in vivo link between arrestin, receptor endocytosis, and temporal recovery from adaptat
133 rrestin-stabilized receptor phosphorylation, receptor endocytosis, and the acceleration of mitogen-ac
135 sphorylation, beta-arrestin recruitment, and receptor endocytosis are all mediated primarily by GRK2/
136 The functional consequences of signaling receptor endocytosis are determined by the endosomal sor
137 determine whether beta-arrestin binding and receptor endocytosis are required for receptor dephospho
138 taxis assays, CCL22 showed dominance in both receptor endocytosis assays and heterologous competitive
139 The kinase activity of Ypk1 is required for receptor endocytosis because mutations in two residues i
140 ells results in an inhibition of transferrin receptor endocytosis because of a competition between AC
143 if accelerated CXCL12-induced wild-type (WT) receptor endocytosis but enabled CXCL12-mediated endocyt
144 3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor endocytosis but is reliably blocked by an endoc
145 hat is required for its clathrin binding and receptor endocytosis but not for its receptor binding an
146 necessary for 5-HT-mediated and DOI-mediated receptor endocytosis, but DA-mediated and clozapine-medi
147 II, WT treatment did not impair the rate of receptor endocytosis, but significantly reduced the init
148 differential regulation of G protein-coupled receptor endocytosis by different ligands, but also a di
150 ether phosphorylation is required for opioid receptor endocytosis by examining a functional, truncate
153 derived factor-1 (SDF-1), as demonstrated by receptor endocytosis, calcium mobilization, and actin po
156 ribe protein interactions that link zones of receptor endocytosis directly to the postsynaptic scaffo
157 2), consistent with its inability to promote receptor endocytosis, elastase did stimulate GRK6 recrui
158 xin (Bgt), facilitating the visualization of receptor endocytosis, exocytosis and delivery to synapti
159 ysosomal permeabilization and assess whether receptor endocytosis followed by trafficking to lysosome
162 that decreases the basal rate of IgM antigen receptor endocytosis in altering the threshold of B-cell
163 ized and quantified basal and regulated AMPA receptor endocytosis in cultured hippocampal neurons, in
166 f transforming growth factor-beta (TGF-beta) receptor endocytosis in signaling have been investigated
167 iency induces immunoglobulin M (IgM) antigen receptor endocytosis in the absence of immune stimulatio
168 n the amygdaloid complex, induction of NK(1) receptor endocytosis in the amygdala following immobilis
169 mber of dendritic processes undergoing NK(1) receptor endocytosis in the basolateral amygdala that wa
171 revision of our understanding of the role of receptor endocytosis in the biology of opiate drug actio
172 These data demonstrate a critical role for receptor endocytosis in the development of adverse side
175 RK1/2) activation, calcium mobilization, and receptor endocytosis] in the same cell background (human
176 ity binding of CCR5 chemokines but preserves receptor endocytosis, indicating that chemokines prefere
177 Morphine, a mu agonist triggering little mu receptor endocytosis, induced neither cross-desensitizat
178 tagonist (ifenprodil) or infusion of an AMPA receptor endocytosis inhibitor (GluA23Y) before rapamyci
179 us, our study demonstrated that Nef-mediated receptor endocytosis involves the ubiquitination motif a
182 igands that require PKC activation to effect receptor endocytosis is dependent on receptor dephosphor
185 urthermore, we find that mGluR-mediated AMPA receptor endocytosis is enhanced by CaM binding to group
186 our data indicate that the ligand-stimulated receptor endocytosis is required for CXCR2-mediated chem
189 amin mutant (Dyn/K44A) inhibited transferrin receptor endocytosis, it had no effect on phogringreen f
190 Furthermore, in both cases, inhibition of receptor endocytosis led to reduced N- and H-Ras activat
191 ated injury may involve facilitation of NMDA receptor endocytosis likely stimulated by DHPG-induced i
192 g integral to the process of cell migration, receptor endocytosis may be a terminal stop signal when
193 IP2 to EphA2 attenuates a positive signal to receptor endocytosis mediated by phosphatidylinositol 3-
194 the effect of immobilisation stress on NK(1) receptor endocytosis morphology, a marker of local subst
195 e high enough for chemotaxis but too low for receptor endocytosis, neutrophil CXCR1 and CXCR2 could b
196 vious studies have demonstrated that neither receptor endocytosis nor arrestin is required for ERK ac
197 t in hippocampal neurons revealed that GABAA receptor endocytosis occurred exclusively at extrasynapt
198 overexpression did not detectably influence receptor endocytosis or the stability of the receptor pr
199 minant-negative Dyn3 did not seem to inhibit receptor endocytosis, overexpression of a specific Dyn3
200 key structural elements important for EphA2 receptor endocytosis provide possible ways for the devel
201 fferent terminals and that agonist dependent receptor endocytosis provides evidence of a spatially an
202 hat the interplay between different modes of receptor endocytosis provides for segregation between di
203 ons and is a key player in postsynaptic AMPA receptor endocytosis, providing multiple ways of negativ
204 induction causes long-term increases in AMPA receptor endocytosis rate and dendritic synthesis of Arc
205 ntracellular domain shows similar effects on receptor endocytosis rate as that of the EpoR, but does
206 was partially explained by a 50% decrease in receptor endocytosis rate; however, at 37 degrees C, PDG
207 min after withdrawal results from increased receptor endocytosis rather than decreased exocytosis.
208 ediated CXCR2 internalization is crucial for receptor endocytosis, resensitization, and chemotaxis.
210 results indicate neither time-averaging nor receptor endocytosis significantly improves the cell's a
211 d the subunit-specific contributions to NMDA receptor endocytosis, specifically defining the endocyti
213 trate that PKC activation stimulates glycine receptor endocytosis, that both constitutive endocytosis
214 ion of NMDA receptors alone can trigger AMPA receptor endocytosis through calcium influx and activati
215 ch beta-arrestin orchestrates the process of receptor endocytosis through the activation of ADP-ribos
216 wnstream mechanisms, including autophagy and receptor endocytosis, through SCF (Skp1-Cul1-F-box)-medi
217 ges from healthy individuals reduced mannose receptor endocytosis to 53.2% (P < 0.05) and P. carinii
219 st, mutating only the Thr residues inhibited receptor endocytosis to the same extent as in the full m
220 tion, NMDA receptor activation enhances AMPA receptor endocytosis via a signaling mechanism required
222 ination is the predominant sorting signal in receptor endocytosis, we investigated whether Nef is ubi
223 cts of AFR1 in strains that are defective in receptor endocytosis were probably an indirect consequen
224 eins of the endocytic machinery and promotes receptor endocytosis, whereas C-terminally truncated ARR
226 promotion of random cell migration requires receptor endocytosis, whereas the chemotactic response t
227 arrestin1 functions as a clathrin adaptor in receptor endocytosis which is regulated by dephosphoryla
228 at the synaptic membrane and impairing AMPA receptor endocytosis, while leaving basal synaptic trans
229 aptor protein recruitment to the beta2AR and receptor endocytosis without affecting the internalizati
230 r abolished the effect of pp120 to stimulate receptor endocytosis, without affecting pp120 phosphoryl
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