ライフサイエンスコーパス: receptor for advanced glycation end products

1 h activation of Toll-like receptor-4 and the receptor for advanced glycation end products.

2 tion of hPA-SMC in a manner dependent on the receptor for advanced glycation end products.

3 mmatory cytokines and of S100A8, S100A9, and receptor for advanced glycation end products.

4 notype depended in part on expression of the receptor for advanced glycation end products.

5 on that S100A8 is an agonist for TLR4 or the receptor for advanced glycation end products.

6 roinflammatory by activating TLR4 and/or the receptor for advanced glycation end products.

7 rance (AFC) and intra-alveolar levels of the receptor for advanced glycation end-products.

8 e of mitochondrial DNA and involved TLR9 and receptors for advanced glycation end products.

9 tate dehydrogenase (5.7 [1.7-19.1]), soluble receptor for advanced glycation end products (3.5 [1.7-7

10 bodies directed against HMGB1 or against the receptor for advanced glycation end products, a putative

11 f a cytosolic signal transduction complex, a receptor for advanced glycation end products, a vacuolar

12 Key players are alpha-dicarbonyls and the receptor for advanced glycation end products (AGER).

13 The receptor for advanced glycation end products (AGEs) has

14 test the hypothesis that activation of RAGE (receptor for advanced glycation end products [AGEs]) by

15 That phenotype depended on expression of the receptor for advanced glycation end products and could b

16 no data exist regarding the role of soluble receptor for advanced glycation end products and recepto

17 Receptor for advanced glycation end products and soluble

18 plasma biomarkers of lung epithelial injury (receptor for advanced glycation end products and surfact

19 e 1 epithelial injury was assessed by plasma receptor for advanced glycation end products and systemi

20 Lower receptor for advanced glycation end products and tumor n

21 f chitinase-3-like 3 and accumulation of the receptor for advanced glycation end-products and its lig

22 d to carboxylated N-glycans expressed on the receptor for advanced glycation end-products and other c

23 type A, DNA indicated their engagement with receptors for advanced glycation end products and TLR9,

24 ression of chloride intracellular channel 4, receptor for advanced glycation end products, and perios

25 levels of cytokines and soluble form of the receptor for advanced glycation end-products, and safety

26 125I-sAbeta1-40 was inhibited by anti-RAGE (receptor for advanced glycation end products) antibody (

27 HMGB1 and its receptors TLR4/MD-2 and RAGE (receptor for advanced glycation end products) are not in

28 r characteristics and multivariate analysis (receptor for advanced glycation end products: area under

29 der the curve, 0.943+/-0.05; p<.001; soluble receptor for advanced glycation end products: area under

30 The involvement of TLR4 and the multiligand receptor for advanced glycation end products as receptor

31 Using integrin alphaIIb and RAGE (receptor for advanced glycation end products) as model t

32 We define RAGE (receptor for advanced glycation end products) as the hS1

33 adipokines, Toll-like receptor ligands, and receptor for advanced glycation end-products, as well as

34 Extracellular newly identified receptor for advanced glycation end products-binding pro

35 Activation of the receptor for advanced glycation end products by its liga

36 able evidence suggests that soluble forms of receptor for advanced glycation end products circulating

37 test the hypothesis that activation of RAGE (Receptor for Advanced Glycation End products) contribute

38 d using human and murine models of TLR4- and receptor for advanced glycation end products-dependent s

39 Thus, it is hypothesized that receptor for advanced glycation end products expression

40 Enhanced monocytic receptor for advanced glycation end products expression

41 Receptor for advanced glycation end products expression

42 Monocytic receptor for advanced glycation end products expression

43 Both receptor for advanced glycation end products>103.6 mean

44 independent of Toll-like receptors 2 and 4, receptor for advanced glycation end-products, high-mobil

45 ptor for advanced glycation end products and receptor for advanced glycation end products in humans w

46 Receptor for advanced glycation end products is targeted

47 s had significantly decreased plasma soluble receptor for advanced glycation end products levels (79.

48 ucts expression and decreased plasma soluble receptor for advanced glycation end products levels play

49 lycation end products expression and soluble receptor for advanced glycation end products levels were

50 er time in cytokines and soluble form of the receptor for advanced glycation end-products levels in t

51 ion of neutrophils with high glucose and the receptor for advanced glycation end products ligand S100

52 103.6 mean fluorescence intensity or soluble receptor for advanced glycation end products<76.88 arbit

53 advanced glycation end products and soluble receptor for advanced glycation end products may be prog

54 We propose that chronic hyperglycemia causes receptor for advanced glycation end products-mediated ep

55 ession might be deleterious, whereas soluble receptor for advanced glycation end products might be be

56 eseen function of the nuclear isoform of the Receptor for Advanced Glycation End-products (nRAGE) in

57 uPA/uPAR physically interacts with AGEs/receptor for advanced glycation end products on the cell

58 Receptor for advanced glycation end products (RAGE) and

59 We studied the expression of receptor for advanced glycation end products (RAGE) and

60 The receptor for advanced glycation end products (RAGE) and

61 Cs by transporting extracellular DNA through receptor for advanced glycation end products (RAGE) and

62 mulation and activation of monocytes through receptor for advanced glycation end products (RAGE) and

63 erentiation involved internalization of both receptor for advanced glycation end products (RAGE) and

64 Activation of the receptor for advanced glycation end products (RAGE) appe

65 Receptor for advanced glycation end products (RAGE) bind

66 The receptor for advanced glycation end products (RAGE) bind

67 aluated by assessing for the presence of the receptor for advanced glycation end products (RAGE) by i

68 Although the receptor for advanced glycation end products (RAGE) has

69 nd neurite outgrowth-promoting roles for the receptor for advanced glycation end products (RAGE) have

70 We demonstrate that overexpression of receptor for advanced glycation end products (RAGE) in i

71 Here, we investigated the role of the receptor for advanced glycation end products (RAGE) in n

72 Here, we have established a role for receptor for advanced glycation end products (RAGE) in R

73 enudation displayed striking upregulation of receptor for advanced glycation end products (RAGE) in t

74 We studied the function of the receptor for advanced glycation end products (RAGE) in t

75 The receptor for advanced glycation end products (RAGE) is a

76 The receptor for advanced glycation end products (RAGE) is a

77 The receptor for advanced glycation end products (RAGE) is a

78 The receptor for advanced glycation end products (RAGE) is a

79 The receptor for advanced glycation end products (RAGE) is a

80 Receptor for advanced glycation end products (RAGE) is a

81 The receptor for advanced glycation end products (RAGE) is a

82 The receptor for advanced glycation end products (RAGE) is a

83 The receptor for advanced glycation end products (RAGE) is a

84 Receptor for advanced glycation end products (RAGE) is a

85 The receptor for advanced glycation end products (RAGE) is a

86 The receptor for advanced glycation end products (RAGE) is a

87 The receptor for advanced glycation end products (RAGE) is a

88 The receptor for advanced glycation end products (RAGE) is a

89 The receptor for advanced glycation end products (RAGE) is a

90 The receptor for advanced glycation end products (RAGE) is a

91 The receptor for advanced glycation end products (RAGE) is a

92 The receptor for advanced glycation end products (RAGE) is e

93 The receptor for advanced glycation end products (RAGE) is h

94 The receptor for advanced glycation end products (RAGE) is h

95 The receptor for advanced glycation end products (RAGE) is p

96 In the kidney, the receptor for advanced glycation end products (RAGE) is p

97 The receptor for advanced glycation end products (RAGE) is p

98 The receptor for advanced glycation end products (RAGE) is u

99 The multiligand receptor for advanced glycation end products (RAGE) medi

100 The receptor for advanced glycation end products (RAGE) medi

101 Receptor for advanced glycation end products (RAGE) medi

102 s with robust and persistent upregulation of receptor for advanced glycation end products (RAGE) mess

103 Ligation of the receptor for advanced glycation end products (RAGE) occu

104 othesized that binding of TTR fibrils to the receptor for advanced glycation end products (RAGE) on c

105 proteins A8/A9 (S100A8/A9) interact with the receptor for advanced glycation end products (RAGE) on h

106 rotein A8/A9 (S100A8/A9), which binds to the receptor for advanced glycation end products (RAGE) on K

107 nt induced by HMGB1 required the presence of receptor for advanced glycation end products (RAGE) on n

108 After binding to the receptor for advanced glycation end products (RAGE) or t

109 deletion or pharmacological blockade of the receptor for advanced glycation end products (RAGE) prev

110 Activation of the receptor for advanced glycation end products (RAGE) repo

111 L-6 promoted by Toll-like receptor (TLR) and receptor for advanced glycation end products (RAGE) sign

112 domain (the A-box) that signals through the receptor for advanced glycation end products (RAGE) to r

113 r inhibit formation of AGEs and suppress the receptor for advanced glycation end products (RAGE) via

114 The receptor for advanced glycation end products (RAGE), a m

115 We report avid binding of LPA to the receptor for advanced glycation end products (RAGE), a m

116 ty of pattern recognition receptors, such as receptor for advanced glycation end products (RAGE), all

117 We show the major receptor for HMGB1, receptor for advanced glycation end products (RAGE), als

118 Receptor for advanced glycation end products (RAGE), an

119 appear partly mediated by activation of the receptor for advanced glycation end products (RAGE), as

120 With its receptor for advanced glycation end products (RAGE), col

121 enetic ablation of the receptor for S100A12, receptor for advanced glycation end products (RAGE), in

122 c ablation of its cell surface receptor, the receptor for advanced glycation end products (RAGE), in

123 helator of HMGB-1 and a blocking antibody to receptor for advanced glycation end products (RAGE), inh

124 es, including toll-like receptor (TLR)2, the receptor for advanced glycation end products (RAGE), mye

125 e expression of toll-like receptor 4 (TLR4), receptor for advanced glycation end products (RAGE), p-E

126 ilage, including S100/calgranulin ligands of receptor for advanced glycation end products (RAGE), pro

127 a and complexity, and enhanced expression of receptor for advanced glycation end products (RAGE), VCA

128 and S100A9, whereas B16F10 cells express the receptor for advanced glycation end products (RAGE), whi

129 ulated mice show that Abeta interaction with receptor for advanced glycation end products (RAGE)-bear

130 mgb1-induced effects in the ischemic retina, receptor for advanced glycation end products (Rage)-defi

131 se 13 (MMP-13) by articular chondrocytes via receptor for advanced glycation end products (RAGE)-medi

132 einase 13 (MMP-13) through activation of the receptor for advanced glycation end products (RAGE).

133 and heterodimers, and are secreted and bind receptor for advanced glycation end products (RAGE).

134 pleckstrin in response to the activation of receptor for advanced glycation end products (RAGE).

135 the extracellular matrix and by engaging the receptor for advanced glycation end products (RAGE).

136 Es bind cell-surface receptors including the receptor for advanced glycation end products (RAGE).

137 roinflammatory activity by mainly binding to receptor for advanced glycation end products (RAGE).

138 d medium was counteracted by blockade of the receptor for advanced glycation end products (RAGE)/nucl

139 like receptors, AIM2-like receptors, and the receptor for advanced glycation end products (RAGE)] to

140 tide polymorphisms in the human gene for the receptor for advanced glycation end-products (RAGE) are

141 that feed-forward signals downstream of the receptor for advanced glycation end-products (RAGE) can

142 Studies of the receptor for advanced glycation end-products (RAGE) indi

143 The receptor for advanced glycation end-products (RAGE) is a

144 The receptor for advanced glycation end-products (RAGE) is a

145 Expression of receptor for advanced glycation end-products (RAGE) is s

146 al of Experimental Medicine reports that the receptor for advanced glycation end-products (RAGE) prom

147 In this study, we show that the receptor for advanced glycation end-products (RAGE) prom

148 ition of toll-like receptor-4 (TLR4) and the receptor for advanced glycation end-products (RAGE) reve

149 ing the growth factor receptor CD114 and the receptor for advanced glycation end-products (RAGE), a C

150 Receptor for advanced glycation end-products (RAGE), and

151 tern molecules (or alarmins) by engaging the receptor for advanced glycation end-products (RAGE).

152 (HMGB1) is a major alarmin that binds to the receptor for advanced glycation end-products (RAGE).

153 ese chemoattractants and S100A9, through the receptor for advanced glycation end-products (RAGE; ie,

154 Receptor for advanced-glycation end products (RAGE) and

155 atory has been investigating the role of the receptor for advanced-glycation end products (RAGE) in m

156 However, in Receptors for Advanced Glycation End Products (RAGE) kno

157 A12 binding to its second proposed receptor (receptor for advanced glycation end products [RAGE]) has

158 eceptors (ie, Toll-like receptors [TLRs] and receptor for advanced glycation end products [RAGE]) pre

159 Here we present evidence that the 'receptor for advanced glycation end products' (RAGE) is

160 The receptor for advanced glycation end products, RAGE, is a

161 n this setting, reducible HMGB1 binds to the receptor for advanced glycation end products (RAGEs), bu

162 nd a synergistic collaboration with TLR2 and receptor for advanced glycation end products signaling,

163 Circulating levels of soluble receptor for advanced glycation end products (sRAGE) lik

164 r the emphysema subpopulation is the soluble receptor for advanced glycation end products (sRAGE).

165 Levels of the soluble form of the receptor for advanced glycation end-products (sRAGE) are

166 nce messenger ribonucleic acid expression of receptor for advanced glycation end products, surfactant

167 no significant differences were observed in receptor for advanced glycation end products, surfactant

168 Moreover, the receptor for advanced glycation end products that recogn

169 pathway is needed to maintain expression of receptor for advanced glycation end products, the ubiqui

170 s of heme oxygenase-1 (HO-1), TNF-alpha, the receptor for advanced glycation end products, vascular e

171 nflammatory gene induction by ligands of the receptor for advanced glycation end products was also at

172 Plasma receptor for advanced glycation end products was signifi

173 x metalloproteinase cleavage product soluble receptor for advanced glycation end products were signif

174 els of IL-1beta, MIP-2, TNF-alpha, TLR4, and receptor for advanced glycation end products, whereas mR