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1 ciation from the receptor, or elimination of receptor reserve.
2 d for TPalpha-Galpha13 and in the context of receptor reserve.
3 tor apparently depends on the degree of P2Y1 receptor reserve.
4 g the SH interval indicated a small (10-20%) receptor reserve.
5 AMP accumulation revealed a relatively large receptor reserve (64%) for the maximal response.
6                               Modulating the receptor reserve also revealed acetylcholine's greater a
7  least two effectors with distinct levels of receptor reserve and that differentially reflect recepto
8              Therefore, our data demonstrate receptor reserve as a novel principle of T cell activati
9 HC variant weak agonists require significant receptor reserve, because decreasing the level of T cell
10 uggesting little difference in the levels of receptor reserve between the two assays.
11 ed in IP assays performed after reduction of receptor reserve by the alkylating agent, phenoxybenzami
12 knockdown of MORs revealed that depletion of receptor reserve does not account for presynaptic resist
13 ffects on CCL3L1 affinity, although possible receptor reserve effects obscure complete interpretation
14 c and postsynaptic responses suggests that a receptor reserve exists for presynaptic inhibition, but
15                  In these cells, there is no receptor reserve for 5-HT and 5-HT2C inverse agonists di
16                                          The receptor reserve for A2A-AdoRs to cause an increase of c
17 n in female rats such that there is a larger receptor reserve for morphine-mediated antinociception.
18 gonist and Furchgott analysis revealed a low receptor reserve for the activation of GIRK channels but
19 e to the existence of a greater A1 adenosine receptor reserve for the inhibition of beta-ICa,L than f
20 r the activation of GIRK channels but a >90% receptor reserve for the inhibition of Ca(2+) events.
21 t, these data suggest a substantial level of receptor reserve for the PI response in mouse hippocampu
22                                              Receptor reserves for 3 A2A-AdoR agonists were large.
23 e of assay incubation time and the degree of receptor reserve in applying the analytical model.
24                          An implication of a receptor reserve is the expansion of the number of ligan
25  assess whether differences in the degree of receptor reserve might explain this discrepancy of resul
26 acellular receptors, which could represent a receptor reserve, near the postsynaptic density.
27 that the IP response exhibited a much larger receptor reserve than the AA response, and reduction of
28          To determine whether T cells follow receptor reserve, we have characterized the effect of re
29          Some ligand-receptor systems have a receptor reserve where a maximal response can be achieve
30 tivity to low-potency agonists by decreasing receptor reserve without significantly altering receptor

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