ライフサイエンスコーパス: recrudescence

1 an note or discharge summary stated the term recrudescence.

2 tify any genetic changes underlying parasite recrudescence.

3 ncentrations at baseline, 7 days, and day of recrudescence.

4 re rates were low (45.9%), owing to parasite recrudescence.

5 r these drugs do not represent biomarkers of recrudescence.

6 enched, 37 (46%) of 81 patients had parasite recrudescence.

7 and plasma obtained at or just before viral recrudescence.

8 to compare seasonality in EoE diagnosis and recrudescence.

9 to onward transmission and subsequent patent recrudescence.

10 on beyond 6 mo, ensuring vernal reproductive recrudescence.

11 acute viral control and in preventing viral recrudescence.

12 den is one of the early hallmarks of disease recrudescence.

13 d reinfection could either precede or follow recrudescence.

14 genotyping to distinguish new infection and recrudescence.

15 alizing Ab delayed but did not prevent virus recrudescence.

16 ls altered CNS cellular tropism during viral recrudescence.

17 ed Abs as key effectors in suppressing virus recrudescence.

18 cell responses but experienced low-level HCV recrudescence 12 weeks later, when HCV-specific T cells

19 arasites from entering latency and abolishes recrudescence after ART treatment of infected mice.

20 Posttreatment recrudescence also did not develop in animals deficient

21 Rapid recrudescence also occurred despite broad CD8(+) T-cell

22 reased T cell proliferation with rapid viral recrudescence and a decrease in TREC levels.

23 ce of CD4 T cell help, mice succumb to viral recrudescence and disease.

24 ce is emerging, ten (16%) of 63 patients had recrudescence and in Ratanakiri, where artemisinin resis

25 Both recrudescence and reinfection could occur in the same in

26 spite appropriate antimicrobial therapy, but recrudescence and reinfection have previously been diffi

27 and asymptomatic infection and during viral recrudescence and SIV encephalitis.

28 immunity is ineffective in controlling virus recrudescence and suggest that the continued presence of

29 sults show that there is a lag between viral recrudescence and the boosting of ADCC antibodies, which

30 inguish this re-infection from drug failure (recrudescence) and to adjust final drug efficacy estimat

31 combined population attributable fraction of recrudescence associated with K13 mutation and Pfmdr1 am

32 Complete clearance of parasites with no recrudescence by day 14.

33 ciparum, reinfections are distinguished from recrudescences by polymerase chain reaction analysis of

34 llance failure followed by gamma-herpesvirus recrudescence can be modeled using murine gamma-herpesvi

35 llance failure followed by gamma-herpesvirus recrudescence can be modeled using murine gamma-herpesvi

36 Recrudescence could be prevented by passive transfer of

37 x, and nucleocapsid proteins did not prevent recrudescence, demonstrating that neutralization is crit

38 Recrudescence did not occur in any volunteer.

39 ed with pretreatment VL, the amount of viral recrudescence during the treatment interruptions, and th

40 o longer suppressed reproduction and gonadal recrudescence ensued.

41 ated eIF2alpha correlates with high rates of recrudescence following ART, and inhibiting eIF2alpha de

42 ing an exonuclease, associates with parasite recrudescence following dihydroartemisinin-piperaquine t

43 ndidate molecular markers predicted parasite recrudescence following dihydroartemisinin-piperaquine t

44 xistence of a genetic change responsible for recrudescence following fosmidomycin-clindamycin treatme

45 sic interval timer to initiate physiological recrudescence following the winter solstice.

46 Parasite clearance and recrudescence for up to 63 days of follow-up were assess

47 intact Ab responses, resulted in significant recrudescence from latency, both in the spleen and the l

48 ated antibody was likely indicative of viral recrudescence from latency.

49 ould enable prompt diagnosis and distinguish recrudescence from mimics, such as transient ischemic at

50 ed and adjusted by genotyping to distinguish recrudescence from new infection) for each episode of un

51 s, with or without adjustment to distinguish recrudescence from new infection.

52 SR group (patients who were hospitalized for recrudescence) had more women, African American individu

53 the seasonality of the initial diagnosis or recrudescence (i.e., food bolus impaction) of EoE.

54 Following viral recrudescence in muMT mice, total CD8(+) T cell numbers

55 By pi day 56, viral recrudescence in the brain was detected in 2 of 6 infect

56 The estimates for rates of malaria recrudescence in the three treatment groups were similar

57 ing responses to treatment and in predicting recrudescence is important.

58 Viral recrudescence is prevented by the transfer of antiviral

59 -compliance with long treatment regimens and recrudescence) issues that limit their therapeutic poten

60 Recrudescence occurred a mean (SD) of 3.9 (0.6) years af

61 Viral recrudescence occurred in four of the five Mamu-A*01-pos

62 Viral recrudescence occurred within 4 to 9 days, and mice succ

63 f trench fever shows how war can lead to the recrudescence of an infectious disease and how medicine

64 dose on day 100 or after 1 year resulted in recrudescence of anti-pig antibody and graft failure.

65 D8(+) immune T cells significantly inhibited recrudescence of brain cysts during chronic ME49 infecti

66 golimod, drug-induced remission was lost and recrudescence of clinical disease was observed.

67 letion of the full RTX infusion, substantial recrudescence of CLL cells occurs, and these cells have

68 ce of initial NK cell control, there is late recrudescence of disease and mortality in immunodeficien

69 o latent cysts (bradyzoites) responsible for recrudescence of disease.

70 ribution of either the diagnosis or clinical recrudescence of EoE throughout the year.

71 ion between seasonality and diagnosis and/or recrudescence of eosinophilic esophagitis (EoE) remains

72 g virus in other body compartments to permit recrudescence of EVD, has shaken our thinking of what it

73 recurrent, disseminated infection was due to recrudescence of his initial infection rather than reinf

74 ents to clear chronic infection and stop the recrudescence of infection after therapy.

75 lying mechanisms that control latency or the recrudescence of infection that occurs during subsequent

76 n the absence of anti-viral Ab, resulting in recrudescence of infectious virus.

77 Recrudescence of latent virus was investigated in blood,

78 e infection, but suffer an ultimately lethal recrudescence of lytic viral replication in the respirat

79 spinal fluid of a patient who then developed recrudescence of meningitis despite treatment with vanco

80 Recrudescence of neurotropic coronavirus due to loss of

81 siological mechanism that has been linked to recrudescence of parasites after monotherapy with ART an

82 t failure was associated with reinfection or recrudescence of preexisting infection.

83 e patients develop worsening oxygenation and recrudescence of pulmonary hypertension but, unexpectedl

84 The recrudescence of severe invasive group A streptococcal (

85 A recrudescence of testes and body mass occurred from mid-

86 f refractoriness to melatonin (Mel) triggers recrudescence of the atrophied reproductive apparatus of

87 Genotyping confirmed recrudescence of the initial parasite populations and dr

88 the DNA vaccine combination did not prevent recrudescence of the latent infection after injection of

89 hich persistence of infection in the mother, recrudescence of the parasite during pregnancy, and the

90 t cessation of heparin therapy can lead to a recrudescence of thrombosis and acute ischemia.

91 pheral blood mononuclear cells suggests that recrudescence of viral replication in brain occurs by re

92 baseline by day 15 and failed to rebound at recrudescence or after reinfection.

93 recurrence (23-486 d) was not different for recrudescence or reinfection.

94 mutation alone was a strong risk factor for recrudescence (P = .009).

95 vious stroke-related deficits (or poststroke recrudescence [PSR]) is an underrecognized and inadequat

96 Kaplan-Meier estimates of the recrudescence rate in the intention-to-treat population

97 Recrudescence rates by day 28, after correction by genot

98 ) but not IgA anti-spike Ab suppressed virus recrudescence, reduced viral antigen in most cell types

99 Onset of disease recrudescence results in decreased parasite cyst burden

100 Recrudescence subsequently developed 1) in animals defic

101 MHC expression on glial cells at the time of recrudescence suggested that memory T cells, although fu

102 ntrol group (patients who did not experience recrudescence), the PSR group (patients who were hospita

103 a homogenous distribution of episodes of EoE recrudescence throughout the year was noted, with no sig

104 n complete clearance of the parasite with no recrudescence up to 122 d after discontinuation of thera

105 ted Kaplan-Meier risk of P falciparum 42 day recrudescence was 54% (95% CI 45-63) in the modified ITT

106 Although parasitemia levels were similar, recrudescence was more frequent and prolonged, and anemi

107 The number of days to recrudescence was unrelated to the number of SNPs accumu

108 Recrudescences were uncommon, and all occurred after AL

109 ion may thus play a role in preventing viral recrudescence, while increasing the risk of pathological

110 Recrudescence with identical or highly phylogenetically