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1                                              Recrudescent and new infections were distinguished by co
2 her wave of commitment is observed following recrudescent asexual parasitemia, and these gametocytes
3 tation at our center to identify factors for recrudescent coccidioidomycosis (despite antifungal prop
4 ntifungal prophylaxis effectively suppressed recrudescent coccidioidomycosis after solid organ transp
5 h quiescent infection, azoles suppressed any recrudescent coccidioidomycosis after transplantation.
6 less than 3 months (P<0.05); one subject had recrudescent disease after two years.
7             Some patients have persistent or recrudescent fever despite this therapy.
8 saki disease patients who have persistent or recrudescent fever, promising new therapies are under st
9 parasites and fever by day 7, and absence of recrudescent infection by parasites with the same molecu
10  findings are consistent with the origins of recrudescent infection by reactivation from many latentl
11               Among patients who experienced recrudescent infection despite antifungal prophylaxis, A
12 ampylobacteriosis as well as observations of recrudescent infection following treatment with C. jejun
13 ed large pool of cells and sites to rekindle recrudescent infection highlights the challenges in erad
14 cells are thought be the principal source of recrudescent infection, but this estimate is based on pe
15 erapy is associated with a high frequency of recrudescent infection, resulting in treatment failure.
16 response may resolve the clinical problem of recrudescent infection.
17 nts with recent SP intake are likely to have recrudescent infections and may need close follow-up if
18 ng mutations of new infections with those of recrudescent infections due to parasites that survived p
19                                              Recrudescent infections occurred in 4 of 79 patients in
20 ctericidal levels of antibiotics and lead to recrudescent infections resistant to therapy.
21 mpared with initial treatments, treatment of recrudescent infections was associated with a higher rat
22                                              Recrudescent infections were associated with the selecti
23 ome phylogenetics successfully discriminated recrudescent iNTS from reinfection, despite a high level
24 simplex virus 1 (HSV-1) most commonly causes recrudescent labial ulcers; however, it is also the lead
25 d those with secondary outcomes [eg, new non-recrudescent malaria infection] were censored on the las
26 lays a key role in artemisinin resistance in recrudescent malaria infections.
27 ion (TPI)' to define foetal infection from a recrudescent maternal infection acquired before pregnanc
28 related to the number of SNPs accumulated by recrudescent organisms, suggesting that there was little
29                                Patients with recrudescent P falciparum infections were more likely to
30                      The primary outcome was recrudescent P falciparum parasitaemia within 63 days.
31                                  Analysis of recrudescent parasites after ELQ or atovaquone monothera
32 asite polymorphisms were used to distinguish recrudescent parasites from new infections.
33                                              Recrudescent parasites had a higher prevalence of kelch1
34 group was uncommon, limiting the analysis of recrudescent parasites.
35 concentrations at baseline compared with non-recrudescent patients, but did not differ significantly
36                                              Recrudescent T-cell activity coincided with the presence
37  immunotherapeutic implications in combating recrudescent toxoplasmosis as well other chronic infecti
38                                    When only recrudescent treatment failures were considered, the ris
39 w often sepsis readmissions are for relapsed/recrudescent versus new infections.
40 d a large number of R/F viruses representing recrudescent viremia from multiple sources.
41 emory TFH and FDC-bound virions in promoting recrudescent viremia in the setting of ART cessation is
42                 In two of these animals, the recrudescent virus population contained only the vaccine
43 n IL-10R blocking Ab enhanced control of the recrudescent virus.
44 ebounding virus, resulting in restriction of recrudescent viruses and selection for preexisting and e

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