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1 serve as imaging biomarkers of breast cancer recurrence risk.
2 s that beta-blockers attenuate breast cancer recurrence risk.
3 alcium stone, increased fluid intake reduced recurrence risk.
4 ransplantation model for posttransplantation recurrence risk.
5 ute to resection completeness and reduce the recurrence risk.
6 ropriate for treating PSP patients with high recurrence risk.
7 of postpartum AD, duration of treatment, and recurrence risk.
8 clinical prediction rule that stratifies VTE recurrence risk.
9 ss an individual patient's tumor biology and recurrence risk.
10 of comorbidities were associated with higher recurrence risk.
11 -fetoprotein (AFP) have been associated with recurrence risk.
12 aracteristics and ERP, and estimated sibling recurrence risk.
13 rs in identifying patients with an increased recurrence risk.
14 n have emerged as the primary determinant of recurrence risk.
15 etastases in lymph nodes and better estimate recurrence risk.
16 is unlikely to cause substantially increased recurrence risk.
17 ion on mechanism of origin, inheritance, and recurrence risk.
18 mutation site may be important in assessing recurrence risk.
19 ogic, and treatment features known to affect recurrence risk.
20 involvement in breast cancer is a marker of recurrence risk.
21 ncluding rare individual pedigrees with high recurrence risk.
22 sus dizygotic (5%) twins as well as familial recurrence risk.
23 ening corrected QT interval and reducing TdP recurrence risk.
24 marginally associated with a decline in the recurrence risk.
25 6% not accepting a less than 5% reduction in recurrence risk.
26 m-effects model and used to calculate 5-year recurrence risk.
27 treatment, geography, Child-Pugh status, and recurrence risk.
28 f CHD with chromosome abnormalities and high recurrence risk.
29 rombotic burden can identify subjects at low recurrence risk.
30 ese predictors may be useful for stratifying recurrence risk.
31 ual SNPs were at most mildly associated with recurrence risk.
32 able couples to be counseled as to their low recurrence risk.
33 d parental mosaicism as central variables in recurrence risk.
34 h the procedure has not been shown to reduce recurrence risk.
35 dneys from living donors (LD) have increased recurrence risk.
36 f mosaic trisomy and UPD and (iii) potential recurrence risks.
37 nd 24.6% in validation set) without additive recurrence risks.
38 after ASD index cases were used to calculate recurrence risks.
39 8%) and that BCS has a slightly higher local recurrence risk (63%); most accurately identified the ma
41 xonidine treatment was associated with lower recurrence risk after adjustment for age, body mass inde
42 dated Vienna Prediction Model for estimating recurrence risk after an unprovoked venous thromboemboli
45 rnational bladder cancer nomogram predicting recurrence risk after radical cystectomy for bladder can
48 of non-RCC death exceeded that of abdominal recurrence risk already at 30 days after surgery, regard
50 having an affected child, and determined the recurrence risks among the siblings and offspring of aff
54 uantitative gene expression assays to assess recurrence risk and benefits from chemotherapy in patien
55 ing the underlying parameters that influence recurrence risk and could be useful for analyzing risk i
57 erage risk, they will not contribute much to recurrence risk and heritability unless they persist on
58 for preventing gastric cancer will depend on recurrence risk and individual and community factors.
59 genes showed a significant trend for reduced recurrence risk and longer recurrence-free survival as t
61 nderlying aetiology, theranostic strategies, recurrence risk and path to recovery are populated by a
64 linical cohort study, the authors determined recurrence risk and survival-analysis-based time to recu
67 h CD68 score and Ki-67 index correlated with recurrence risk, and Ki-67 index inversely correlated wi
69 of genomic predictors of ER pathway status, recurrence risk, and sensitivity to chemotherapeutics wa
70 ns of nipple viability, flap necrosis, local recurrence risk, and the technical challenge of this pro
71 agnosis, to counsel family members for their recurrence risk, and to classify these rare disorders mo
72 alysis of the symptom loadings, comorbidity, recurrence risks, and within-family correlations indicat
73 ans and mice, CHD has a low absolute sibling recurrence risk ( approximately 2.7%), suggesting a cons
74 idering Mendelian diseases in which familial recurrence risks are high, and mutant alleles are both n
75 Thus, although early mortality and stroke recurrence risks are higher among non-lacunar than lacun
77 the context of common traits, where familial recurrence risks are modest, and for the most part the r
78 mptom reduction for even a small increase in recurrence risk, are at substantially increased risk of
79 DCE MR imaging features were predictive of recurrence risk as determined by the surrogate assay, wi
81 tor-positive, HER2-negative disease and high recurrence risk, as defined by clinicopathologic charact
84 of non-RCC death exceeded that of abdominal recurrence risk at 6 months in patients age 80 years and
85 al study cannot specifically examine adenoma recurrence risk at intervals suggested for patients with
87 ive, face-based ABM reduced both measures of recurrence risk (Beck Depression Inventory and cortisol
89 .1, all P < 0.001) and accurately stratified recurrence risk beyond MC, ranging from 19% (CRS 0) to 6
91 , active dual-chamber pacing reduces syncope recurrence risk by 75% (95% confidence interval, 44-88).
92 istic, 0.82; 95% CI, 0.77-0.86) and superior recurrence risk classification compared with explant Mil
94 0.84]) each further reduced composite stone recurrence risk compared with placebo or control, althou
95 drug-treated patients, dual-site RA reduced recurrence risk compared with SP (HR = 0.638, p = 0.011)
96 ristic phenotype is important due to the low recurrence risk compared with the other (recessive) cere
103 according to whether or not the 5-year local recurrence risk exceeded 10% (<10%, 17,000 women; >10%,
106 lysis identified 3 statistically significant recurrence risk factors: advanced age, largest basal dia
108 first population-based study to examine the recurrence risk for autism spectrum disorders (ASDs), in
110 sm indicates that there may be a significant recurrence risk for DC/XLIS in families at risk, even wh
113 complications for their patients and predict recurrence risk for families of children with congenital
119 al half siblings (2.3%) was half the overall recurrence risk for maternal half siblings but was simil
122 The authors sought to determine the illness recurrence risk for women with bipolar disorder who disc
124 iting the range of genetic determination and recurrence risks for two-, three-, and four-locus purely
126 substantive implications for calculation of recurrence risk, genetic counseling, and potential treat
130 ant chemotherapy was associated with overall recurrence risk (hazard ratio [HR] = 0.56), while histor
131 -6 genotype was associated with an increased recurrence risk (hazard ratio [HR], 4.60; 95% CI, 1.24 t
133 le was associated with significantly reduced recurrence risk (HR = 0.25; 95% CI, 0.10 to 0.64) and im
134 PPARG genotype was associated with a reduced recurrence risk (HR, 0.41; 95% CI, 0.20 to 0.86) among u
136 ovides information in assessing the clinical recurrence risk in bladder cancer and that the specific
139 se of single affected parents shows that the recurrence risk in children is determined by genetic fac
140 multigene signature to improve prediction of recurrence risk in clear cell renal cell carcinoma.
141 clinically important question of predicting recurrence risk in colorectal cancer patients, we demons
142 tative distributions in nature, estimates of recurrence risk in families have never previously consid
145 e of genetics in ASDs, while the significant recurrence risk in maternal half-siblings may support th
147 of these findings, we hypothesized a higher recurrence risk in older than in younger patients when t
148 ance (MR) imaging features and breast cancer recurrence risk in patients with estrogen receptor-posit
149 ted in a prospective study for assessment of recurrence risk in patients with stage II colon cancer a
155 a genomic signature developed both to assess recurrence risk in stage II patients and to assist in tr
161 ; however, an optimistic under-estimation of recurrence risk is a common problem associated with thes
166 t, identification of patients at low risk of recurrence risk is very difficult (that is, such people
167 bar intracerebral haemorrhage (with its high recurrence risk) is now well recognised, a number of man
169 Age and gender-adjusted OR(s) and sibling recurrence risk (lambda(s)), with different thresholds d
171 nature predicted particularly well for early recurrence risk (<2 years), especially when combined wit
172 solute gains from radiotherapy; 5-year local recurrence risks (mainly at these sites) 6% versus 23% (
173 to the conserved breast), with 5-year local recurrence risks (mainly in the conserved breast, as mos
174 Degrees of gonosomal mosaicism mean that recurrence risks may well be <50% in the index case when
176 staining did not confer an increased risk of recurrence (risk of recurrence, 0.86, P = 0.78), whereas
183 ermine the true rate of familial ALS and the recurrence risk of ALS in family members, and to identif
184 identifiable provocative risk factors have a recurrence risk of approximately 25% at 4 years with the
188 cs, with 98% both incorrectly estimating the recurrence risk of deafness and misunderstanding the con
195 ed low and high RS groups had average 5-year recurrence risks of 13% (95% CI, 10% to 16%) and 21% (95
196 findings have important consequences for the recurrence risks of disorders caused by de novo mutation
199 us at enrollment was associated with a lower recurrence risk (OR = 0.67, 95% CI 0.45, 0.99, for the m
201 liability, approximate heritability, sibling recurrence risk, overall genetic variance using a logari
205 and might ultimately lead to improvements in recurrence risk prediction, treatment, and prognosis.
207 RETREAT was able to stratify 5-year post-LT recurrence risk ranging from less than 3% with a score o
208 associated with an increased risk of disease recurrence (risk rate, 3.12; P =.02) and decreased risk
209 therapy significantly reduced risk of stroke recurrence (risk ratio, 0.69; 95% confidence interval, 0
214 da(R)/lambda(jR), where lambda(R) is Risch's recurrence risk ratio and lambda(jR) is the contribution
220 netic contribution of >10% to the AS sibling recurrence risk ratio) within this area contributing to
221 ex diseases with large values of the sibling recurrence risk ratio, sequencing unselected affected in
222 es with small values for the overall sibling recurrence risk ratio, such as Alzheimer's disease and m
223 We introduce the idea of a restricted sib recurrence-risk ratio (lambda*S) estimated by restrictio
225 asure of familial aggregation is the sibling recurrence-risk ratio, which is defined as the ratio of
230 t interbirth interval was observed, with the recurrence risk reaching 14.4% for an interbirth interva
231 because data suggest increased locoregional recurrence risks (relative to luminal subtypes) with bre
233 can benefit families with information about recurrence risk, resolve concerns about etiology, provid
234 HRT did not seem to affect breast cancer recurrence risk (RR = 0.64, 95% confidence interval [CI]
235 The prevalence of TS/CT and OCD and relative recurrence risk (RRR) for TS/CT or OCD among individuals
237 omy and open nephroureterectomy, investigate recurrence risks specific to laparoscopic nephroureterec
238 enetic etiology is indicated by an increased recurrence risk, sporadic occurrence suggests that CHD g
239 ts that mutations in parental blood increase recurrence risk substantially more than parental mutatio
240 sms had significantly worse DFS and a higher recurrence risk than patients with fewer combined risk g
242 ved little (<10%) difference in 5-year local recurrence risk there was little difference in 15-year b
244 olute treatment effect across a continuum of recurrence risk to individualize endocrine therapy decis
245 tionship R for the true model and KRI is the recurrence risk to relatives for a multiplicative model
246 nteraction ratio, CR=KR/KRI, where KR is the recurrence risk to relatives with relationship R for the
247 sex bias in AS, and there are differences in recurrence risk to the offspring of affected mothers and
248 Reproductive fitness of adults with TOF and recurrence risks to offspring are of increasing interest
249 al with secondary data on long-term melanoma recurrence risks to project the cost-effectiveness of ad
251 y regimens should take into account baseline recurrence risk, toxicities, likelihood of benefit, and
260 on, a validated model of posttransplantation recurrence risk was produced with a concordance statisti
264 versus continued mood stabilizer treatment, recurrence risk was twofold greater, median time to firs
266 al (>10%) differences, however, 5-year local recurrence risks were 7% active versus 26% control (abso
267 nd combined polymorphisms, the above similar recurrence risks were particularly higher among patients
268 nosis, and model predictions of age-specific recurrence risks were tested against outcome data from S
269 does not define a subgroup that is at higher recurrence risk when compared with patients with RT-PCR-
271 d fosters genetic counseling with respect to recurrence risks while assuring reproductive choices.
272 urgery, trauma, pregnancy) have a low annual recurrence risk, while patients without identifiable pro
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