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1 gitudinal study of offspring of parents with recurrent depression.
2 accompanied by an increased risk of new and recurrent depression.
3 ng stress, in particular in individuals with recurrent depression.
4 r schizophrenia, bipolar disorder and severe recurrent depression.
5 y in genes, is associated with resilience to recurrent depression.
6 ive impairment in older people with previous recurrent depression.
7 hich were observed only in participants with recurrent depression.
8 ortisol awakening response) in patients with recurrent depression.
9 instrumental activities of daily living, and recurrent depression.
10 not observed in children who later developed recurrent depression.
11 ly maintenance psychotherapy did not prevent recurrent depression.
12 ard-related learning as an endophenotype for recurrent depression.
13 fetal risk against the risk of persistent or recurrent depression.
14 quential treatment response among women with recurrent depression.
15 nly tool to prevent relapse in patients with recurrent depression.
16 to SLEs among adults with moderately severe, recurrent depression.
17 unced on the left of these structures and in recurrent depression.
18 th insomnia have a high risk of incident and recurrent depression.
19 e-control cohort study of Chinese women with recurrent depression.
20 otherapy alone for treatment of more severe, recurrent depressions.
21 ficant advantage was observed in more severe recurrent depressions.
22 nalysis of large, rare CNVs in 3106 cases of recurrent depression, 459 controls screened for lifetime
23 Subgroup A; chi(2)=5.34, p=.02), and highly recurrent depression (63.2% of Subgroup B vs 31.8% of Su
24 ntial in euthymic subjects with a history of recurrent depression and 2) the relationship between rec
25 d from a familial collection of early-onset, recurrent depression and were compared with screened con
26 onotherapy is effective for the treatment of recurrent depression, and the MRI-guided method of coil
27 hildren who later developed schizophrenia or recurrent depression as well as in healthy comparison su
28 nus age at onset), course (single episode or recurrent depression), baseline depression severity, tre
29 atment experiences of patients with chronic, recurrent depression being treated by primary care physi
30 fulness-based cognitive therapy (n = 50) for recurrent depression compared with treatment as usual (n
31 ectiveness of this approach in patients with recurrent depression (> or = 3 episodes of depression).
34 s associated with lower risk of incident and recurrent depression (hazard ratio, HR, 0.52, 95% CI 0.2
35 trols; however, no difference in the risk of recurrent depression (HR, 0.90 [95% CI, 0.62-1.28]; P =
36 no depression history) and 1696 at risk for recurrent depression (ie, depression history but no trea
37 e and psychotherapy were less likely to have recurrent depression if they received two years of maint
38 and adolescents is strongly associated with recurrent depression in adulthood; other mental disorder
39 raline is a safe and effective treatment for recurrent depression in patients with recent MI or unsta
41 s therefore do not support the proposal that recurrent depression is associated with long-standing de
43 showed genome-wide significant evidence of a recurrent depression locus in a previous sib-pair study.
44 e life may be part of the AD prodrome, while recurrent depression may be etiologically associated wit
46 atment for relapse prevention for those with recurrent depression, particularly those with more prono
47 ventral striatum in medication-free remitted recurrent depression patients (n = 36) versus healthy co
48 (no previous depression) and 1696 at risk of recurrent depression (previous depression, but not for t
49 2) an affected sibling pair linkage study of recurrent depression (probands from the Depression Netwo
50 ion severity, and melancholic, atypical, and recurrent depression subtypes were all independently ass
51 re collected from 1) a case-control study of recurrent depression (the Depression Case Control [DeCC]
53 ty-three persons aged 60 years or older with recurrent depression were studied: 34 had been depressed
54 istory of PMD should be alert to the risk of recurrent depression when discontinuing hormone therapy.
55 ound that deletion CNVs were associated with recurrent depression, whereas duplications were not.
56 ed, sham-controlled trial, 105 patients with recurrent depression who exhibited no responses to at le
57 isode of depression or more than 2 years for recurrent depression who were currently well enough to c