ライフサイエンスコーパス: recurrent infection

1 ently, which represents a lifelong source of recurrent infection.

2 ful patient outcomes including prevention of recurrent infection.

3 iated with an elevated risk of persistent or recurrent infection.

4 re less effective for treating refractory or recurrent infection.

5 led treatment at 17 weeks or later or from a recurrent infection.

6 load before transplantation will have rapid, recurrent infection.

7 read distribution provide protection against recurrent infection.

8 ce VZV replication in skin during primary or recurrent infection.

9 ith Cryptosporidium infections, particularly recurrent infection.

10 us site colonisation as a possible source of recurrent infection.

11 verely defective in chemotaxis, resulting in recurrent infection.

12 to C. difficile and TcdB during primary and recurrent infection.

13 a from this reservoir might be the source of recurrent infection.

14 airways, impairing respiration and promoting recurrent infection.

15 substantial risk for operative mortality or recurrent infection.

16 cell memory that confers protection against recurrent infection.

17 sia species appeared to result in chronic or recurrent infection.

18 that does not prevent invasive amebiasis or recurrent infection.

19 s who were seropositive before BMT underwent recurrent infection.

20 ls, but are frequent pathogens and can cause recurrent infection.

21 pressure and prevent or treat persistent and recurrent infection.

22 ssociated with a significantly lower risk of recurrent infection.

23 superior to placebo in reducing the risk of recurrent infection.

24 es were 3-mo SAB-specific mortality rate and recurrent infection.

25 ormancy after antibiotic withdrawal leads to recurrent infection.

26 protective or nonprotective immunity against recurrent infection.

27 ion, specific hair shaft defects, atopy, and recurrent infections.

28 antibody bezlotoxumab is reported to reduce recurrent infections.

29 If untreated, patients succumb to recurrent infections.

30 may prevent the establishment of latency and recurrent infections.

31 ies diagnosed with hypogammaglobulinemia and recurrent infections.

32 tions in order to deal more efficiently with recurrent infections.

33 GR with primary infection and 3 asymptomatic recurrent infections.

34 ency, poor humoral response to antigens, and recurrent infections.

35 RA) and manifests chiefly as neutropenia and recurrent infections.

36 accines focused on preventing both acute and recurrent infections.

37 erized by impaired leukocyte recruitment and recurrent infections.

38 which reside in the urothelium and can seed recurrent infections.

39 only a minority of people experience severe recurrent infections.

40 amages the airways and fails to protect from recurrent infections.

41 f the 12 mothers and 9 of the 21 infants had recurrent infections.

42 hould be performed, especially in those with recurrent infections.

43 trains associated with greater likelihood of recurrent infections.

44 en implicated in biofilms and in chronic and recurrent infections.

45 mes when antibody is present, such as during recurrent infections.

46 cy disorder characterized by neutropenia and recurrent infections.

47 ntigens from viruses that produce chronic or recurrent infections.

48 proportion of patients suffer from multiple recurrent infections.

49 es, were found in a lymphopenic patient with recurrent infections.

50 cteria also serve as reservoirs important in recurrent infections.

51 least once, and 25% of these women will have recurrent infections.

52 nd mortality by plugging airways and causing recurrent infections.

53 women, and may be associated with chronic or recurrent infections.

54 eases consultation and associated with fewer recurrent infections.

55 rrow, ineffective neutrophil production, and recurrent infections.

56 rns of the organisms causing the initial and recurrent infections.

57 able treatment strategy for children without recurrent infections.

58 m IgM to IgG and IgA, a defect that leads to recurrent infections.

59 ave significant morbidity and mortality from recurrent infections.

60 osed by environmental stresses, particularly recurrent infections.

61 ion of microbicidal oxidants leads to severe recurrent infections.

62 s indicate that ethanol addicts are prone to recurrent infections.

63 ssociated with thrombocytopenia, eczema, and recurrent infections.

64 ival of viruses that establish persistent or recurrent infections.

65 ating the proportion of vivax relapses among recurrent infections.

66 (9)/L, two had agranulocytosis, and four had recurrent infections.

67 clonal population and substrain shuffling in recurrent infections.

68 burden on healthcare and frequently lead to recurrent infections.

69 e to produce superoxide (O2-.), resulting in recurrent infections.

70 reatment; 3 patients died, and 3 experienced recurrent infections.

71 s infections, some suffer from severe and/or recurrent infections.

72 c resistance, has been linked to chronic and recurrent infections.

73 cous secretions, bacterial colonization, and recurrent infections.

74 g parasite genotypes before treatment and in recurrent infections.

75 with microcephaly, primordial dwarfism, and recurrent infections.

76 endar year and followed up for 12 months for recurrent infections.

77 treatment, potentially leading to chronic or recurrent infections.

78 organ autoimmunity, lymphoproliferation, and recurrent infections.

79 ent immunosuppression with increased risk of recurrent infections.

80 lls may have an important role in chronic or recurrent infections.

81 therapy and a major cause of persistent and recurrent infections.

82 erized by impaired leukocyte recruitment and recurrent infections.

83 ng patients had no evidence of persistent or recurrent infections.

84 vers of cirrhosis patients with a history of recurrent infections.

85 n part by pulmonary abnormalities, including recurrent infections.

86 on, are commonly associated with chronic and recurrent infections.

87 s, inadequate postnatal nutrient intake, and recurrent infections.

88 es the need for vaccines to prevent UTIs and recurrent infections.

89 in human disease, particularly in regards to recurrent infections.

90 ng the capacity to establish lifelong latent-recurrent infections.

91 mental contaminants, increasing the risk for recurrent infections.

92 plicated in resistance to antimicrobials and recurrent infections.

93 f virus in HCMV-experienced hosts undergoing recurrent infection (0.61 units/day; 95% CI, 0.55-0.7 un

94 Of 76 patients with recurrent infections, 2 (11%), 2 (10%), and 10 (36%) in

95 ions were below the therapeutic level in all recurrent infections (24 of 40 patients), which were eff

96 s (4), gastrointestinal intolerance (4), and recurrent infections (3).

97 tment outcomes (4.7% treatment failure, 2.5% recurrent infection, 4.1% death, 6.8% loss to follow-up)

98 However, 44% of survivors developed recurrent infections; 43% of recurrences were late (285

99 These bacteria may serve as a reservoir for recurrent infections, a common problem affecting million

100 ozygous mutations in IL6R who presented with recurrent infections, abnormal acute-phase responses, el

101 ate, leading to severe immunodeficiency with recurrent infections (ADA-SCID).

102 ections at baseline were at highest risk for recurrent infection (adjusted odds ratio, 2.5 and 2.4, r

103 Recurrent infection after a negative posttreatment UBT a

104 ociated with a reduced risk of persistent or recurrent infection after adjustment for other predictor

105 ferral of partners in reducing persistent or recurrent infection among patients with gonorrhea (3 per

106 Thus far, interventions designed to reduce recurrent infections among household members have had li

107 infection can impact the risk for developing recurrent infection and has implications for the develop

108 apoptosis (TWEAK; TNFSF12) in a kindred with recurrent infection and impaired antibody responses to p

109 rus-specific CD8 T cells home to the site of recurrent infection and participate in viral clearance.

110 lux, but little is known about its impact on recurrent infection and renal scarring.

111 tant pathogen was a strong predictor of both recurrent infection and resistance in future infection e

112 hepatitis B virus infection were hampered by recurrent infection and subsequent allograft failure.

113 he male sexual partner of the patient with a recurrent infection and the patient, an isolate from the

114 m Rac2 knockout mice and from a patient with recurrent infections and a dominant-negative mutation in

115 ed primary immunodeficiency characterized by recurrent infections and a marked predisposition to deve

116 environments and specific microbes prevents recurrent infections and allergy development, likely due

117 a virulent Escherichia coli strain, whereas recurrent infections and asymptomatic bacteriuria common

118 stically overlapped diseases presenting with recurrent infections and autoimmunity.

119 nt assimilation but is also characterized by recurrent infections and chronic inflammation, implying

120 Staphylococcus aureus) biofilm, a source of recurrent infections and chronic inflammation.

121 In this report we describe a child with recurrent infections and clinical evidence of impaired p

122 Despite frequent drug exposures due to recurrent infections and comorbidities, the prevalence a

123 A patient is described who has recurrent infections and defective neutrophil cellular f

124 ory infections, but the host factors causing recurrent infections and exacerbations in some children

125 CGD patients suffer from recurrent infections and exuberant and persistent tissue

126 ct in chronic granulomatous disease, causing recurrent infections and granulomatous complications.

127 on the efficacy of decolonization to prevent recurrent infections and highlights the adaptive potenti

128 usceptibility of DOCK8-deficient patients to recurrent infections and malignant disease.

129 phylaxis may not be effective for preventing recurrent infections and may result in antimicrobial res

130 transcription 3 (STAT3) mutations, including recurrent infections and mucocutaneous candidiasis, whic

131 rome characterized by hypogammaglobulinemia, recurrent infections and multiple autoimmune clinical fe

132 gene expressed by CD4(+) T cells suffer from recurrent infections and often develop autoimmune disord

133 mizes the risk of local morbidity, including recurrent infections and pneumothorax, and avoids the ri

134 ncluding allograft rejection, drug toxicity, recurrent infections and postrenal obstruction.

135 ome of immune dysregulation characterized by recurrent infections and predisposition to humoral autoi

136 gap hinders the ability to clinically manage recurrent infections and reconstruct transmission networ

137 mining those patients who are susceptible to recurrent infections and renal scarring following urinar

138 iveness of interventions designed to prevent recurrent infections and renal scarring.

139 Secondary aims were analyses of the rate of recurrent infections and reoperations, and time trends i

140 Patients with CGD present with recurrent infections and severe inflammatory complicatio

141 from a consanguineous family suffering from recurrent infections and severe lupuslike autoimmunity.

142 ase, characterized by hypogammaglobulinemia, recurrent infections and various complications.

143 In 3 families, we identified 4 children with recurrent infections and varying clinical manifestations

144 virologic response at 12 weeks, 10 (23%) had recurrent infection, and 3 (7%) died (2 from nonfunction

145 relative lack of immediate hypersensitivity, recurrent infection, and an increased rate of lymphoma i

146 isk of HPV16 infection, high viral load, and recurrent infection, and was also more strongly associat

147 on, and less frequently, bowel inflammation, recurrent infections, and angiopathic thromboembolic dis

148 ned immunodeficiency characterized by atopy, recurrent infections, and cancer susceptibility.

149 ller (NK) cells which leads to autoimmunity, recurrent infections, and combined immune deficiency.

150 involving increased innate immune response, recurrent infections, and complex somatic features.

151 ncludes hyaline deposits in multiple organs, recurrent infections, and death within the first 2 years

152 clude temperature and electrolyte imbalance, recurrent infections, and failure to thrive.

153 tion, leading to impaired microbial killing, recurrent infections, and granulomatous inflammation.

154 rombocytopenia, small platelet size, eczema, recurrent infections, and increased risk of autoimmune d

155 racterized by microthrombocytopenia, eczema, recurrent infections, and susceptibility to autoimmunity

156 ed to protect patients from the morbidity of recurrent infections, and to extend the survival benefit

157 dhood with developmental delay, skin ulcers, recurrent infections, and, in some patients, autoimmune

158 nd syndromes most frequently associated with recurrent infection are presented, along with discrimina

159 It has been proposed that recurrent infections are a result of the reintroduction

160 ponsible for various diseases in humans, and recurrent infections are commonly observed.

161 proaches targeting high-risk populations for recurrent infections are needed.

162 uity of care for patients with refractory or recurrent infections, are ideally situated to provide gu

163 Recurrent infections arise sporadically from the periphe

164 s a T-cell defect frequently presenting with recurrent infections, as well as associated immune dysre

165 There was no history of infectious exposure, recurrent infection, aspiration, or choking.

166 ent duration and assessment of persistent or recurrent infection at the completion of therapy.

167 S gene, leading to thrombocytopenia, eczema, recurrent infections, autoimmune disease, and malignancy

168 f childhood-onset hypogammaglobulinemia with recurrent infections, autoimmune features, and adrenal i

169 oinsufficiency showed hypogammaglobulinemia, recurrent infections, autoimmunity, and allergy, but wit

170 DGS), other risk factors could predispose to recurrent infections, autoimmunity, and allergy.

171 ibody deficiencies (PADs) has been linked to recurrent infections, autoimmunity, and cancer, yet clin

172 hat autosomal recessive TPP2 mutations cause recurrent infections, autoimmunity, and neurodevelopment

173 umoral and cellular immunity, manifesting as recurrent infections, autoimmunity, hepatosplenomegaly,

174 tential of person-to-person transmission and recurrent infections but perhaps not for other genotypin

175 monocytes are most commonly associated with recurrent infection, but abnormalities of immunoglobulin

176 the host either more or less susceptible to recurrent infection, but the specific mechanisms that ti

177 Although recurrent infections can be observed in patients with de

178 y to further health deterioration, including recurrent infection, cardiovascular events, and acute re

179 AT patients suffer from recurrent infections caused by both cellular and humoral

180 tiology of PAP, polycystic lung disease, and recurrent infections caused by impaired CCL2-dependent m

181 chanism underlying the susceptibility to the recurrent infections commonly seen in HIES.

182 lower risk of mortality and similar odds of recurrent infections compared with nafcillin or oxacilli

183 We identified three unrelated children with recurrent infections, congenital leukopenia including ne

184 age in IEI has been historically ascribed to recurrent infections, contributions from potentially tar

185 ial factors that contribute to the course of recurrent infection could inform strategies to reduce re

186 Primary and recurrent infections cycle virus particles between neuro

187 disorder of persistent lung inflammation and recurrent infection, defined by a common pathological en

188 pha (IkappaBalpha) are susceptible to severe recurrent infections, despite normal T and B cell number

189 However, in patients with recurrent infections, different substrains of the establ

190 One patient developed a recurrent infection due to meropenem-vaborbactam non-sus

191 One patient developed a recurrent infection due to meropenem-vaborbactam-nonsusc

192 impaired specific Ab response, resulting in recurrent infections due to dysfunctional immune respons

193 in (Ig)M syndrome (X-HIGM), characterized by recurrent infections due to impaired immunoglobulin clas

194 lish SOCE cause severe immunodeficiency with recurrent infections due to impaired T cell function.

195 t month 6: high-level EBVd (>1500 IU/mL) and recurrent infection during the previous months (6 points

196 Staphylococcus aureus causes persistent, recurrent infections (e.g., osteomyelitis) by forming bi

197 (encoding GP130 p.N404Y) who presented with recurrent infections, eczema, bronchiectasis, high IgE,

198 of Wiskott-Aldrich syndrome (WAS), including recurrent infections, eczema, thrombocytopenia, defectiv

199 a with skewed memory phenotype, infant-onset recurrent infections, failure to thrive, and protracted

200 quitous important human pathogen that causes recurrent infections for the life of its host.

201 Recurrent infections have a diverse strain background.

202 ry immunodeficiency that is characterized by recurrent infections, hematopoietic malignancies, eczema

203 All patients presented with recurrent infections, hepatosplenomegaly, anaemia, and e

204 ses of hypogammaglobulinemia predisposing to recurrent infections, higher incidence of autoimmunity,

205 pioid use disorder reduces overdose risk and recurrent infections, hospitalisation remains common.

206 missense variants in NFATC1, presenting with recurrent infections, hypogammaglobulinemia, and decreas

207 s are increasingly utilized for treatment of recurrent infections (i.e., Clostridium difficile) in th

208 be central to the altered responsiveness to recurrent infection in these patients.

209 Among recurrent infections in 41 individuals, there were 42 re

210 t the presentation of IEIs might not include recurrent infections in 9% of cases, and that current di

211 fit in preclinical animal models and prevent recurrent infections in human clinical trials.

212 This review also summarizes studies on recurrent infections in individuals living in endemic re

213 D46-Jagged1 crosstalk is responsible for the recurrent infections in subpopulations of these patients

214 al wisdom dictates that uropathogens causing recurrent infections in such individuals come from the f

215 ation.IMPORTANCE HSV-2 causes very localized recurrent infections in the skin and genital mucosa.

216 provide a potential mechanism underlying the recurrent infections in WAS patients.

217 mechanism underlying phagocytic defects and recurrent infections in WAS patients.

218 l mouse model of protective immunity against recurrent infection, in which S. aureus skin and soft ti

219 P), progressive polycystic lung disease, and recurrent infections, including bacillus Calmette Guerin

220 f morbidity and mortality in early life, and recurrent infections increase the risk of developing chr

221 okinesis 8 (DOCK8) deficiency is typified by recurrent infections, increased serum IgE levels, eosino

222 We report a new syndrome with recurrent infections, inflammation, and hyperzincaemia a

223 Recurrent infection is infrequently transmitted (2%) and

224 The hyper-IgE syndrome with recurrent infections is a rare immunodeficiency characte

225 le immunodeficiency (CVID), characterized by recurrent infections, is the most prevalent symptomatic

226 Since patients with NS suffer from recurrent infections, it has been hypothesized that an u

227 ic obstructive pulmonary disease patients to recurrent infections, leading to exacerbations and contr

228 imary ciliary dyskinesia, with upper-airways recurrent infections, left-right asymmetry perturbations

229 in M (IgM) syndrome (HIGM), characterized by recurrent infections, low serum IgG and IgA, normal or e

230 ating differential effects of antibiotics on recurrent infection may be warranted.

231 The recurrent infection may have induced accumulation of AAT

232 Recurrent infections may arise from activation of dorman

233 of viral infections in infancy, and in turn, recurrent infections may influence the development of im

234 Patients show hypopigmentation, recurrent infections, mild coagulation defects and varyi

235 = 14, 93%), failure to thrive (n = 10, 67%), recurrent infections (n = 10, 67%), allergic symptoms (n

236 After treatment with AAQ + PQ, recurrent infection occurred in 0 of 167 patients within

237 Recurrent infections occurred in 3 of 7 patients who sur

238 Recurrent infection of the graft is universal in HCV pat

239 ukocytes, consistent with the neuropathy and recurrent infection of the human patients possessing the

240 Thus, recurrent infection of these developing B cell populatio

241 Considering the low pathogenicity and recurrent infections of parental hPIV2, BC-PIV can be us

242 mophilus influenzae (NTHI) frequently causes recurrent infections of the respiratory tract in humans.

243 ons in the upper respiratory tract and skin, recurrent infections of the respiratory tract, and skin

244 isorder of ciliary beating, characterized by recurrent infections of the upper and lower airways, as

245 Affected persons suffer from recurrent infections of upper and lower airways because

246 e [IQR], 8-10) therapy had a similar odds of recurrent infection or death within 30 days as those rec

247 axis to sterilize the urine and thus prevent recurrent infection, or abolishing reflux via surgical i

248 also presented that in 45% of patients with recurrent infections, oral and vulvovaginal isolates wer

249 Chronically critically ill patients have recurrent infections, organ dysfunction, and at least ha

250 cent serum antibody levels with incidence of recurrent infection over 12 months follow-up in 235 chil

251 parasitemia did not differ between acute and recurrent infections (P = .48 in Thailand, P = .08 in In

252 eding diathesis, neutropenia, and history of recurrent infections prompted consideration of the diagn

253 A 28-year-old man presented with recurrent infections, reduced class-switched memory B ce

254 ection was also associated with a history of recurrent infection (relative risk, 5.58 in the universi

255 pital environments, promoting resistance and recurrent infections, remains unknown.

256 ociated with eczema, elevated serum IgE, and recurrent infections resembling severe atopic dermatitis

257 may go undiagnosed in patients experiencing recurrent infections, resulting in increased morbidity a

258 592 was underrepresented in individuals with recurrent infection (RO, 0.59; P=.046).

259 1.3, 95% CI 0.9, 1.9) and persistent or recurrent infection (RR(adj).

260 sophageal reflux, constipation, dysmotility, recurrent infections, seizures, and structural brain ano

261 The NEIL3 mutation was associated with fatal recurrent infections, severe autoimmunity, hypogammaglob

262 luded developmental delay, seizures, ataxia, recurrent infections, severe language deficit, and an un

263 e antigen, a feature that may be critical in recurrent infections such as malaria.

264 hthyosis, many patients suffer from itching, recurrent infections, sweating impairment (hypohidrosis)

265 Recurrent infection that can progress to life-threatenin

266 lective compliance, otherwise giving rise to recurrent infections that are not only driven by pathoge

267 Urinary tract infections (UTIs) are common, recurrent infections that can be mild to life-threatenin

268 In each of the 18 patients with recurrent infections, the same strain was responsible fo

269 Recurrent infections, thermolability, and other complica

270 d wound healing, which increases the risk of recurrent infections, tissue necrosis, and limb amputati

271 cipants were examined for adverse events and recurrent infection twice per week.

272 A recurrent infection was defined as a MSSA blood culture

273 icans isolates from each of 18 patients with recurrent infections was assessed by sequential DNA fing

274 After one lung transplant patient with recurrent infections was noted to be severely hypogammag

275 A NAAT+/Ab+ status, signifying untreated/recurrent infection, was associated with increased cervi

276 To determine whether PD-1 contributes to recurrent infection, we first established a model of rei

277 Factors associated with recurrent infection were history of sternotomy (p = 0.00

278 val analysis, and risk factors for first and recurrent infections were identified by Cox regression m

279 lates from 74 trainees, 6 (8.1%) of whom had recurrent infection, were subjected to WGS.

280 rotective immunity and allowing prolonged or recurrent infection, which may be important for viral lu

281 reus is responsible for numerous chronic and recurrent infections, which are frequently associated wi

282 nd lacks immunoglobulins and B cells and has recurrent infections, while the elder, affected brother

283 should guide current treatment choices since recurrent infection will most likely be at least as resi

284 nce of a chronic state, and development of a recurrent infection with autoimmunity.

285 Lamivudine appears to limit recurrent infection with hepatitis B virus in a signific

286 Recurrent infection with hepatitis C virus (HCV) is almo

287 c fibrosis (CF), a disease in which there is recurrent infection with known pathogens.

288 as how it can lead to a chronic infection or recurrent infection with relapses and remissions.

289 sphate oxidase activity and characterized by recurrent infections with a limited spectrum of bacteria

290 able immune deficiency, a syndrome marked by recurrent infections with encapsulated microorganisms, i

291 Recurrent infections with high-risk human papillomavirus

292 Moreover, 78% of the pigs had recurrent infections with IAVs closely related to each o

293 swelling often associated with fibrosis and recurrent infections with no available cures to date.

294 odeficiency (CID) is characterized by severe recurrent infections with normal numbers of T and B lymp

295 inesis 8 (DOCK8) deficiency, who experienced recurrent infections with resistant HSV-1.

296 Recurrent infections with respiratory syncytial virus (R

297 ous disease (CGD) is characterized by severe recurrent infections with Staphylococcus aureus, certain

298 patients have decreased quality of life and recurrent infections with treatments limited to palliati

299 Recurrent infections with USA300 MRSA are common, yet in

300 Seventeen CRBSIs were recurrent; infection with gram-positive bacteria predict