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1 read distribution provide protection against recurrent infection.
2 ce VZV replication in skin during primary or recurrent infection.
3 ith Cryptosporidium infections, particularly recurrent infection.
4 verely defective in chemotaxis, resulting in recurrent infection.
5 a from this reservoir might be the source of recurrent infection.
6 airways, impairing respiration and promoting recurrent infection.
7 substantial risk for operative mortality or recurrent infection.
8 cell memory that confers protection against recurrent infection.
9 sia species appeared to result in chronic or recurrent infection.
10 that does not prevent invasive amebiasis or recurrent infection.
11 s who were seropositive before BMT underwent recurrent infection.
12 ls, but are frequent pathogens and can cause recurrent infection.
13 pressure and prevent or treat persistent and recurrent infection.
14 protective or nonprotective immunity against recurrent infection.
15 ently, which represents a lifelong source of recurrent infection.
16 ful patient outcomes including prevention of recurrent infection.
17 iated with an elevated risk of persistent or recurrent infection.
18 re less effective for treating refractory or recurrent infection.
19 led treatment at 17 weeks or later or from a recurrent infection.
20 load before transplantation will have rapid, recurrent infection.
21 therapy and a major cause of persistent and recurrent infections.
22 amages the airways and fails to protect from recurrent infections.
23 f the 12 mothers and 9 of the 21 infants had recurrent infections.
24 hould be performed, especially in those with recurrent infections.
25 en implicated in biofilms and in chronic and recurrent infections.
26 mes when antibody is present, such as during recurrent infections.
27 cy disorder characterized by neutropenia and recurrent infections.
28 proportion of patients suffer from multiple recurrent infections.
29 es, were found in a lymphopenic patient with recurrent infections.
30 cteria also serve as reservoirs important in recurrent infections.
31 least once, and 25% of these women will have recurrent infections.
32 nd mortality by plugging airways and causing recurrent infections.
33 women, and may be associated with chronic or recurrent infections.
34 rrow, ineffective neutrophil production, and recurrent infections.
35 rns of the organisms causing the initial and recurrent infections.
36 vers of cirrhosis patients with a history of recurrent infections.
37 m IgM to IgG and IgA, a defect that leads to recurrent infections.
38 ave significant morbidity and mortality from recurrent infections.
39 osed by environmental stresses, particularly recurrent infections.
40 ion of microbicidal oxidants leads to severe recurrent infections.
41 s indicate that ethanol addicts are prone to recurrent infections.
42 ssociated with thrombocytopenia, eczema, and recurrent infections.
43 ival of viruses that establish persistent or recurrent infections.
44 (9)/L, two had agranulocytosis, and four had recurrent infections.
45 clonal population and substrain shuffling in recurrent infections.
46 e to produce superoxide (O2-.), resulting in recurrent infections.
47 n part by pulmonary abnormalities, including recurrent infections.
48 on, are commonly associated with chronic and recurrent infections.
49 s, inadequate postnatal nutrient intake, and recurrent infections.
50 erized by impaired leukocyte recruitment and recurrent infections.
51 es the need for vaccines to prevent UTIs and recurrent infections.
52 in human disease, particularly in regards to recurrent infections.
53 ng the capacity to establish lifelong latent-recurrent infections.
54 mental contaminants, increasing the risk for recurrent infections.
55 plicated in resistance to antimicrobials and recurrent infections.
56 If untreated, patients succumb to recurrent infections.
57 ng patients had no evidence of persistent or recurrent infections.
58 may prevent the establishment of latency and recurrent infections.
59 ies diagnosed with hypogammaglobulinemia and recurrent infections.
60 tions in order to deal more efficiently with recurrent infections.
61 GR with primary infection and 3 asymptomatic recurrent infections.
62 ency, poor humoral response to antigens, and recurrent infections.
63 RA) and manifests chiefly as neutropenia and recurrent infections.
64 accines focused on preventing both acute and recurrent infections.
65 erized by impaired leukocyte recruitment and recurrent infections.
66 which reside in the urothelium and can seed recurrent infections.
67 only a minority of people experience severe recurrent infections.
68 f virus in HCMV-experienced hosts undergoing recurrent infection (0.61 units/day; 95% CI, 0.55-0.7 un
70 These bacteria may serve as a reservoir for recurrent infections, a common problem affecting million
72 ections at baseline were at highest risk for recurrent infection (adjusted odds ratio, 2.5 and 2.4, r
74 ociated with a reduced risk of persistent or recurrent infection after adjustment for other predictor
75 ferral of partners in reducing persistent or recurrent infection among patients with gonorrhea (3 per
76 Thus far, interventions designed to reduce recurrent infections among household members have had li
77 infection can impact the risk for developing recurrent infection and has implications for the develop
78 apoptosis (TWEAK; TNFSF12) in a kindred with recurrent infection and impaired antibody responses to p
79 rus-specific CD8 T cells home to the site of recurrent infection and participate in viral clearance.
81 hepatitis B virus infection were hampered by recurrent infection and subsequent allograft failure.
82 he male sexual partner of the patient with a recurrent infection and the patient, an isolate from the
83 m Rac2 knockout mice and from a patient with recurrent infections and a dominant-negative mutation in
84 ed primary immunodeficiency characterized by recurrent infections and a marked predisposition to deve
85 nt assimilation but is also characterized by recurrent infections and chronic inflammation, implying
90 ct in chronic granulomatous disease, causing recurrent infections and granulomatous complications.
91 on the efficacy of decolonization to prevent recurrent infections and highlights the adaptive potenti
93 phylaxis may not be effective for preventing recurrent infections and may result in antimicrobial res
94 transcription 3 (STAT3) mutations, including recurrent infections and mucocutaneous candidiasis, whic
95 rome characterized by hypogammaglobulinemia, recurrent infections and multiple autoimmune clinical fe
96 gene expressed by CD4(+) T cells suffer from recurrent infections and often develop autoimmune disord
97 gap hinders the ability to clinically manage recurrent infections and reconstruct transmission networ
98 mining those patients who are susceptible to recurrent infections and renal scarring following urinar
100 Secondary aims were analyses of the rate of recurrent infections and reoperations, and time trends i
101 from a consanguineous family suffering from recurrent infections and severe lupuslike autoimmunity.
102 In 3 families, we identified 4 children with recurrent infections and varying clinical manifestations
103 virologic response at 12 weeks, 10 (23%) had recurrent infection, and 3 (7%) died (2 from nonfunction
104 isk of HPV16 infection, high viral load, and recurrent infection, and was also more strongly associat
105 on, and less frequently, bowel inflammation, recurrent infections, and angiopathic thromboembolic dis
108 ncludes hyaline deposits in multiple organs, recurrent infections, and death within the first 2 years
110 rombocytopenia, small platelet size, eczema, recurrent infections, and increased risk of autoimmune d
111 racterized by microthrombocytopenia, eczema, recurrent infections, and susceptibility to autoimmunity
112 nd syndromes most frequently associated with recurrent infection are presented, along with discrimina
115 s a T-cell defect frequently presenting with recurrent infections, as well as associated immune dysre
118 f childhood-onset hypogammaglobulinemia with recurrent infections, autoimmune features, and adrenal i
119 hat autosomal recessive TPP2 mutations cause recurrent infections, autoimmunity, and neurodevelopment
120 tential of person-to-person transmission and recurrent infections but perhaps not for other genotypin
121 monocytes are most commonly associated with recurrent infection, but abnormalities of immunoglobulin
125 lower risk of mortality and similar odds of recurrent infections compared with nafcillin or oxacilli
126 We identified three unrelated children with recurrent infections, congenital leukopenia including ne
127 ial factors that contribute to the course of recurrent infection could inform strategies to reduce re
129 disorder of persistent lung inflammation and recurrent infection, defined by a common pathological en
130 pha (IkappaBalpha) are susceptible to severe recurrent infections, despite normal T and B cell number
132 in (Ig)M syndrome (X-HIGM), characterized by recurrent infections due to impaired immunoglobulin clas
133 lish SOCE cause severe immunodeficiency with recurrent infections due to impaired T cell function.
134 Staphylococcus aureus causes persistent, recurrent infections (e.g., osteomyelitis) by forming bi
135 (encoding GP130 p.N404Y) who presented with recurrent infections, eczema, bronchiectasis, high IgE,
136 of Wiskott-Aldrich syndrome (WAS), including recurrent infections, eczema, thrombocytopenia, defectiv
137 ry immunodeficiency that is characterized by recurrent infections, hematopoietic malignancies, eczema
139 ses of hypogammaglobulinemia predisposing to recurrent infections, higher incidence of autoimmunity,
140 s are increasingly utilized for treatment of recurrent infections (i.e., Clostridium difficile) in th
144 D46-Jagged1 crosstalk is responsible for the recurrent infections in subpopulations of these patients
145 al wisdom dictates that uropathogens causing recurrent infections in such individuals come from the f
146 ation.IMPORTANCE HSV-2 causes very localized recurrent infections in the skin and genital mucosa.
149 l mouse model of protective immunity against recurrent infection, in which S. aureus skin and soft ti
150 okinesis 8 (DOCK8) deficiency is typified by recurrent infections, increased serum IgE levels, eosino
154 le immunodeficiency (CVID), characterized by recurrent infections, is the most prevalent symptomatic
155 ic obstructive pulmonary disease patients to recurrent infections, leading to exacerbations and contr
156 imary ciliary dyskinesia, with upper-airways recurrent infections, left-right asymmetry perturbations
157 in M (IgM) syndrome (HIGM), characterized by recurrent infections, low serum IgG and IgA, normal or e
160 of viral infections in infancy, and in turn, recurrent infections may influence the development of im
165 ukocytes, consistent with the neuropathy and recurrent infection of the human patients possessing the
167 mophilus influenzae (NTHI) frequently causes recurrent infections of the respiratory tract in humans.
168 ons in the upper respiratory tract and skin, recurrent infections of the respiratory tract, and skin
169 isorder of ciliary beating, characterized by recurrent infections of the upper and lower airways, as
171 axis to sterilize the urine and thus prevent recurrent infection, or abolishing reflux via surgical i
172 also presented that in 45% of patients with recurrent infections, oral and vulvovaginal isolates wer
173 Chronically critically ill patients have recurrent infections, organ dysfunction, and at least ha
174 cent serum antibody levels with incidence of recurrent infection over 12 months follow-up in 235 chil
175 parasitemia did not differ between acute and recurrent infections (P = .48 in Thailand, P = .08 in In
176 eding diathesis, neutropenia, and history of recurrent infections prompted consideration of the diagn
178 ection was also associated with a history of recurrent infection (relative risk, 5.58 in the universi
180 The NEIL3 mutation was associated with fatal recurrent infections, severe autoimmunity, hypogammaglob
181 luded developmental delay, seizures, ataxia, recurrent infections, severe language deficit, and an un
185 d wound healing, which increases the risk of recurrent infections, tissue necrosis, and limb amputati
188 icans isolates from each of 18 patients with recurrent infections was assessed by sequential DNA fing
190 To determine whether PD-1 contributes to recurrent infection, we first established a model of rei
193 reus is responsible for numerous chronic and recurrent infections, which are frequently associated wi
194 nd lacks immunoglobulins and B cells and has recurrent infections, while the elder, affected brother
200 sphate oxidase activity and characterized by recurrent infections with a limited spectrum of bacteria
201 able immune deficiency, a syndrome marked by recurrent infections with encapsulated microorganisms, i
204 swelling often associated with fibrosis and recurrent infections with no available cures to date.
205 odeficiency (CID) is characterized by severe recurrent infections with normal numbers of T and B lymp
207 ous disease (CGD) is characterized by severe recurrent infections with Staphylococcus aureus, certain
208 patients have decreased quality of life and recurrent infections with treatments limited to palliati
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