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1 mitter release from the granule cells during recurrent inhibition.
2 via antidromic activation of corticostriatal recurrent inhibition.
3 ansformed into different spatial patterns of recurrent inhibition.
4 ent excitation and, to a lesser degree, more recurrent inhibition.
5 ts did not lead to any significant change in recurrent inhibition.
6  any descending facilitation of heteronymous recurrent inhibition.
7  due to local circuit constraints imposed by recurrent inhibition.
8 ion between distinct activity states through recurrent inhibition.
9 , such a denoising behavior is contingent on recurrent inhibition.
10  we also examined the temporal properties of recurrent inhibition.
11  of network theta rhythm and the strength of recurrent inhibition (affecting capacity), as well as th
12  is reduced in HD compared to WT mice, while recurrent inhibition also shows phenotype dependency.
13                Here, we provide evidence for recurrent inhibition: an experience-dependent plasticity
14 er half a century ago, their precise role in recurrent inhibition and ability to modulate motoneuron
15 st that segmental spinal mechanisms, such as recurrent inhibition and stretch reflex, probably play a
16 ons, which mediate neuronal excitability and recurrent inhibition and thus contribute to the stabilit
17 rasegmental and intersegmental interactions, recurrent inhibition, and descending influences, produce
18  features--balanced afferent drive, dominant recurrent inhibition, and differential recruitment by af
19 interneurons responsible for feedforward and recurrent inhibition are anatomically segregated in laye
20      However, the regulation and function of recurrent inhibition are poorly understood in terms of t
21                        Renshaw cells mediate recurrent inhibition between motoneurons within the spin
22 of a single module is set by the distance of recurrent inhibition between neurons.
23           A spiking network model shows that recurrent inhibition can control this activity-state swi
24 his model of temporal lobe epilepsy, reduced recurrent inhibition contributes to layer II stellate ce
25                                         This recurrent inhibition declined during voluntary tonic con
26 escending from the brain reduce heteronymous recurrent inhibition during isolated quadriceps muscle c
27 ctivities differ in their ability to recruit recurrent inhibition, entrain field-potential oscillatio
28  to monitor the transmission of heteronymous recurrent inhibition from soleus to quadriceps motor neu
29 s concluded that the pathway of heteronymous recurrent inhibition from soleus to quadriceps motor neu
30                     Indirect measurements of recurrent inhibition have suggested only a weak modulato
31 oss of tyrosine hydroxylase and accompanying recurrent inhibition in a small number of their populati
32 ng V2a neurons supports phasic firing, while recurrent inhibition in bifurcating V2a neurons helps pa
33  on the rostral tongue) is known to initiate recurrent inhibition in cells in the nucleus of the soli
34                  In contrast, at slow speeds recurrent inhibition in descending V2a neurons supports
35                                              Recurrent inhibition in olfactory bulb mitral cells is m
36  We present the first direct measurements of recurrent inhibition in primate upper limb motoneurons,
37 responses and a suppression of GABA-mediated recurrent inhibition in the dentate gyrus of LCMV-infect
38 tory response dominates over the STN-GPe-GPe recurrent inhibition in the GPe, whereas the STN-GPe-GPi
39  Startle disease is due to the disruption of recurrent inhibition in the spinal cord.
40 our results suggest a general approach where recurrent inhibition is associated with stimulus 'recogn
41                             We conclude that recurrent inhibition is remarkably effective, in that a
42 omputational model, supporting the idea that recurrent inhibition may function to reduce tremor.SIGNI
43 GABA(B) agonist baclofen reduces mitral cell recurrent inhibition mediated by dendrodendritic synapse
44   We propose a model in which development of recurrent inhibition mediates development of temporal as
45 s, the interneuronal cell type that mediates recurrent inhibition of motor neurons.
46 ces and found that under control conditions, recurrent inhibition of principal neurons (mitral cells)
47 luded that the inhibition was a heteronymous recurrent inhibition of quadriceps motor neurons mediate
48  of cholinergic interneurons into widespread recurrent inhibition of these neurons via nicotinic exci
49 ompetition between external visual input and recurrent inhibition, particularly within L2/3 and L4.
50 ries of action potentials in pyramidal cells recurrent inhibition rapidly shifts from their soma to t
51         Coloboma mice also exhibit increased recurrent inhibition, reduced theta rhythm by tail-pinch
52 s are consistent with the diverse effects on recurrent inhibition reported in subjects with upper mot
53 he brain, the recruitment of feedforward and recurrent inhibition shapes neural responses.
54 onvergent connectivity or a CPG network with recurrent inhibition that actively decorrelates common i
55 bsets of OFC neurons, potentially by driving recurrent inhibition though antidromic activation of cor
56 DP-PCN neurons, suggesting that they provide recurrent inhibition to DP-PCN.
57                                              Recurrent inhibition was strongest to motoneurons innerv
58                                              Recurrent inhibition, wherein excitatory principal neuro
59                    It has been proposed that recurrent inhibition within the thalamic reticular nucle
60 ng models in which excitation is balanced by recurrent inhibition, yet all excitatory synapses underg