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1 via antidromic activation of corticostriatal recurrent inhibition.
2 ansformed into different spatial patterns of recurrent inhibition.
3 ent excitation and, to a lesser degree, more recurrent inhibition.
4 ts did not lead to any significant change in recurrent inhibition.
5  any descending facilitation of heteronymous recurrent inhibition.
6 mitter release from the granule cells during recurrent inhibition.
7  of network theta rhythm and the strength of recurrent inhibition (affecting capacity), as well as th
8 er half a century ago, their precise role in recurrent inhibition and ability to modulate motoneuron
9 ons, which mediate neuronal excitability and recurrent inhibition and thus contribute to the stabilit
10 rasegmental and intersegmental interactions, recurrent inhibition, and descending influences, produce
11  features--balanced afferent drive, dominant recurrent inhibition, and differential recruitment by af
12 interneurons responsible for feedforward and recurrent inhibition are anatomically segregated in laye
13      However, the regulation and function of recurrent inhibition are poorly understood in terms of t
14 his model of temporal lobe epilepsy, reduced recurrent inhibition contributes to layer II stellate ce
15                                         This recurrent inhibition declined during voluntary tonic con
16 escending from the brain reduce heteronymous recurrent inhibition during isolated quadriceps muscle c
17 ctivities differ in their ability to recruit recurrent inhibition, entrain field-potential oscillatio
18  to monitor the transmission of heteronymous recurrent inhibition from soleus to quadriceps motor neu
19 s concluded that the pathway of heteronymous recurrent inhibition from soleus to quadriceps motor neu
20                     Indirect measurements of recurrent inhibition have suggested only a weak modulato
21 oss of tyrosine hydroxylase and accompanying recurrent inhibition in a small number of their populati
22  on the rostral tongue) is known to initiate recurrent inhibition in cells in the nucleus of the soli
23                                              Recurrent inhibition in olfactory bulb mitral cells is m
24 responses and a suppression of GABA-mediated recurrent inhibition in the dentate gyrus of LCMV-infect
25 tory response dominates over the STN-GPe-GPe recurrent inhibition in the GPe, whereas the STN-GPe-GPi
26 our results suggest a general approach where recurrent inhibition is associated with stimulus 'recogn
27                             We conclude that recurrent inhibition is remarkably effective, in that a
28 GABA(B) agonist baclofen reduces mitral cell recurrent inhibition mediated by dendrodendritic synapse
29   We propose a model in which development of recurrent inhibition mediates development of temporal as
30 s, the interneuronal cell type that mediates recurrent inhibition of motor neurons.
31 ces and found that under control conditions, recurrent inhibition of principal neurons (mitral cells)
32 luded that the inhibition was a heteronymous recurrent inhibition of quadriceps motor neurons mediate
33  of cholinergic interneurons into widespread recurrent inhibition of these neurons via nicotinic exci
34 ries of action potentials in pyramidal cells recurrent inhibition rapidly shifts from their soma to t
35         Coloboma mice also exhibit increased recurrent inhibition, reduced theta rhythm by tail-pinch
36 s are consistent with the diverse effects on recurrent inhibition reported in subjects with upper mot
37 he brain, the recruitment of feedforward and recurrent inhibition shapes neural responses.
38 bsets of OFC neurons, potentially by driving recurrent inhibition though antidromic activation of cor
39 DP-PCN neurons, suggesting that they provide recurrent inhibition to DP-PCN.
40                                              Recurrent inhibition, wherein excitatory principal neuro
41                    It has been proposed that recurrent inhibition within the thalamic reticular nucle

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