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1 unlike EPO products, reduce the risk of pure red cell aplasia.
2 e transplant and may be associated with pure red cell aplasia.
3 enia gravis, hypogammaglobulinemia, and pure red cell aplasia.
4 isorders), the most common of which was pure red-cell aplasia.
5 disorder most commonly associated with pure red cell aplasia, (2) the presence of clonal cytogenetic
8 sociation between rituximab therapy and pure red cell aplasia, but the diagnostic and therapeutic uti
10 tor can correct anemia in patients with pure red-cell aplasia caused by antierythropoietin antibodies
11 d-Blackfan anemia (DBA) is a rare congenital red-cell aplasia characterized by anemia, bone-marrow er
12 graft-vs-host disease-like colitis, and pure red cell aplasia, different from the pattern observed in
14 B19 causes fifth disease, acute and chronic red cell aplasia, fetal hydrops, arthropathy, and other
15 obtained reports of epoetin-associated pure red-cell aplasia from the Food and Drug Administration a
18 ntified 47 adult patients with acquired pure red cell aplasia (median age, 64 years; range, 22 to 84
19 -eight episodes of HPV B19-induced transient red cell aplasia occurred with the following clinical ev
20 teins cause Diamond Blackfan Anemia (DBA), a red cell aplasia often associated with physical abnormal
21 nts with chronic kidney disease who had pure red-cell aplasia or hypoplasia due to antierythropoietin
22 Delayed donor red cell engraftment and pure red cell aplasia (PRCA) are well-recognized complication
24 months, respectively, before developing pure red cell aplasia (PRCA) confirmed by bone marrow biopsy.
27 lobulin (IVIG) therapy in patients with pure red cell aplasia (PRCA) related to human parvovirus B19
30 ce of this effect of rituximab therapy: pure red cell aplasia resulting from chronic parvovirus B19 i
31 n in transplant recipients can cause chronic red cell aplasia that generally responds to IVIG therapy
32 Diamond Blackfan anemia (DBA), an inherited red cell aplasia, the bone marrow is characterized by a
34 of administration, the country in which pure red-cell aplasia was identified, and the date on which p
35 the peak incidence of Eprex-associated pure red-cell aplasia was reached in 2001, interventions desi
36 ween 1988 and 1998, antibody-associated pure red-cell aplasia was reported in three patients who had
39 l 2004, 175 cases of epoetin-associated pure red-cell aplasia were reported for Eprex, 11 cases for N
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