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1 cause of transfusion-related mortality with red cell transfusion.
2 RDS risk and mortality including the role of red cell transfusion.
3 d significantly higher rates of bleeding and red-cell transfusion.
4 as the percentage of patients who received a red-cell transfusion.
5 o, 1.91; 95% confidence interval, 1.49-2.44) red cell transfusions.
6 er entry effectively decreased inappropriate red cell transfusions.
7 nd early death in patients receiving regular red cell transfusions.
8 ents, spleen response, and independence from red-cell transfusions.
9 tin (epoetin alfa) might reduce the need for red-cell transfusions.
10 ive vasopressor infusions (66.6% vs. 57.8%), red-cell transfusions (13.6% vs. 7.0%), and dobutamine (
11 e standard, autologous, leukoreduced, packed red cell transfusion after 1, 2, 3, 4, 5, or 6 weeks of
12 poetin alfa does not reduce the incidence of red-cell transfusion among critically ill patients, but
13 l failure have greatly reduced the number of red cell transfusions and hence the propensity to iron o
14 o identify patients who are at high risk for red cell transfusions and therefore most likely to benef
16 ter); 3 of them were previously dependent on red-cell transfusions and no longer needed transfusions.
17 of intravenous fluids, vasoactive drugs, and red-cell transfusions and reflected by significantly wor
19 estrictive threshold for hemoglobin level in red-cell transfusions, as compared with a liberal thresh
20 nces in the proportion of patients requiring red cell transfusions, changes in quality of life, or th
21 A-DR15, younger age, and shorter duration of red cell transfusion dependence as pretreatment variable
22 ent revision include stricter definitions of red cell transfusion dependency and independency and con
24 Jakob disease have been identified following red cell transfusions from donors who subsequently devel
25 ty and compliance with each of inotropes and red cell transfusions, glucocorticoids, and lung-protect
30 ique, reduces the requirement for allogeneic red cell transfusions in patients undergoing major hepat
31 iology of SCD is better understood; however, red cell transfusion is currently the most studied and a
35 was associated with decreased likelihood of red cell transfusion (odds ratio, 0.43; 95% confidence i
36 01) and platelet (P =.01) recovery and fewer red cell transfusions (P =.02) than 48 historical contro
39 =800 mL, ANH reduced not only the allogeneic red cell transfusion rate (18.2% vs. 42.4%, P = 0.045) b
41 reutzfeldt-Jakob disease and an asymptomatic red cell transfusion recipient, who did not die of varia
42 either the number of patients who received a red-cell transfusion (relative risk for the epoetin alfa
43 tients who received G-CSF + EPO had a higher red cell transfusion requirement (median, 161.0 mL/kg) t
44 rfarin resulted in faster reversal and lower red cell transfusion requirement with fewer adverse even
45 ary end point was the effect of EPO on total red cell transfusion requirements during induction thera
47 mpare outcomes of a restrictive to a liberal red cell transfusion strategy in 20% or more total body
48 The effect of a restrictive versus liberal red-cell transfusion strategy on clinical outcomes in pa
49 those in the G-CSF + EPO group received more red cell transfusions than did those given G-CSF alone (
50 eath after cardiac surgery) to a restrictive red-cell transfusion threshold (transfuse if hemoglobin
51 g from induction of anesthesia) or a liberal red-cell transfusion threshold (transfuse if hemoglobin
52 we randomly assigned patients who required a red-cell transfusion to receive blood that had been stor
54 ovided to the 1098 participants who received red-cell transfusion was 7 days in the shorter-term stor
55 for death, a restrictive strategy regarding red-cell transfusion was noninferior to a liberal strate
56 verity of renal and hepatic dysfunction, and red cell transfusions were all independent risk factors
58 in the critically ill, is often treated with red-cell transfusions, which are associated with poor cl
59 plasma hemoglobin increase immediately after red cell transfusion, with more significant increases ob
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