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1 proximately 0.1 %) of the pigment content in red cones.
2 n--about 10% of total opsin--in dark-adapted red cones.
3  required for the maintenance or function of red cones.
4 hromophore did not affect the ability of the red cone and blue cone/green rod pigments to activate tr
5                                          The red cone and blue cone/green rod pigments were unstable
6 t important in the activation pathway of the red cone and blue cone/green rod pigments.
7 E65 was selectively expressed in human green/red cones but absent from blue cones and mediated ester
8 f photoresponsiveness of bleached salamander red cones but not of bleached salamander red rods.
9 ased opsin expression in individual rods and red cones, but decreased opsin expression in individual
10 tinal is reversible in darkness in amphibian red cones, but essentially irreversible in red rods.
11 s an approximately 2-fold desensitization in red cones, equivalent to that produced by a steady light
12                        Implicit times of the red cone ERGs were slightly faster for the Neuroline ski
13 normally bias their connectivity in favor of red cones fail to precisely recapture this synaptic part
14 lectrode recordings from the hypothalamus of red cone knockin mice (Opn1mwR).
15 us laevis genes, Prph2 (also called RDS) and red cone opsin (RCO) using a polymerase chain reaction-b
16 sion resulted in a decrease in rhodopsin and red cone opsin expression levels in Xenopus retinas.
17 y that Rx is co-expressed with rhodopsin and red cone opsin in maturing photoreceptors and demonstrat
18                                 When rod and red cone opsin probes were combined, the number of label
19 n of rAAV5-hCNGB3 with a long version of the red cone opsin promoter in younger animals led to a stab
20 nt therapy with different forms of the human red cone opsin promoter led to the restoration of cone f
21 monstrate that Rx binds to the rhodopsin and red cone opsin promoters in vivo.
22 r of photoreceptors expressing rod opsin and red cone opsin, and decreased the number of photorecepto
23 ess this deficit using mice expressing human red cone opsin, in which rod-, cone-, and melanopsin-dep
24 eceptor-specific genes such as rhodopsin and red cone opsin.
25        We also show that the degeneration of red cone photoreceptors in the mutants occurs independen
26 stability of the chromophore within the deep red cone pigment binding sites.
27                           Interestingly, the red cone pigment containing the retinal analogue remaine
28 f rhodopsin, the green cone pigment, and the red cone pigment in H2O (D2O) are found at 1656 (1623),
29 s question by expressing human or salamander red cone pigment in Xenopus rods, and human rod pigment
30                                     However, red cone pigment isomerizes spontaneously 10,000 times m
31 ow overcome this problem by expressing human red cone pigment transgenically in mouse rods in order t
32                             However, for the red cone pigment, the 9-methyl group of retinal appears
33 corresponding control cultures regarding the red cone pigment, which was expressed in all cases, and
34 nel catfish orthologues of rhodopsin and the red cone pigment-the full complement of retinal opsins i
35 Raman spectra of recombinant human green and red cone pigments have been obtained to examine the mole
36 f seven cone pigments indicate that the deep red cone pigments select 6- s- trans chromophore conform
37 igate theoretically the hypothesis that deep red cone pigments select a 6- s- trans conformation of t
38 flash cone, light-adapted 3.0 Hz flicker and red cone responses were analysed, as well as their respe
39 flash cone, light-adapted 3.0 Hz flicker and red cone responses were up to four times larger when rec
40 d opsin is the only known molecule unique to red cones, these data suggest that a novel gene is requi
41 e of transgenic cone pigment to native human red cones, we obtained a dark rate of approximately 10 f

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